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1 y benign tumors of the ovary and uterus, and adrenal adenoma.
2 f hypertension, especially when caused by an adrenal adenoma.
3 teral renal-adrenal fusion with a concurrent adrenal adenoma.
4 ns resulted in unilateral cortisol-producing adrenal adenomas.
5 sence and amount of histologic lipid in many adrenal adenomas accounts for their low attenuation on u
6 e adrenal gland that consisted of contiguous adrenal adenoma and metastasis, which represented a coll
7 ens from 10 patients with cortisol-producing adrenal adenomas and evaluated recurrent mutations in ca
8 sity analysis allows differentiation between adrenal adenomas and nonadenomas, reflecting an improved
9 rcentage of lipid-rich cortical cells in the adrenal adenomas and the unenhanced CT attenuation numbe
10 , KCNJ5, as a cause of aldosterone-producing adrenal adenomas (APAs) and one inherited KCNJ5 mutation
11 docrine tumors such as aldosterone-producing adrenal adenomas (APAs), a cause of severe hypertension,
13 the 10-HU threshold method for diagnosis of adrenal adenomas at enhanced CT, with specificity mainta
15 uced sensitivity for the characterization of adrenal adenomas compared with results from prior studie
16 eview of 2 years of clinical CT records, 223 adrenal adenomas in 193 patients (115 with contrast mate
17 The higher estimates encourage search for adrenal adenomas in patients with elevated ratios of pla
19 ear-old woman had hyperaldosteronemia and an adrenal adenoma that showed no evidence of lipid on in-p
22 esent a case of a patient with a preexisting adrenal adenoma who only presented with clinical signs o
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