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1 mbic centers, and the hypothalamic-pituitary-adrenal axis.
2 ess and regulates the hypothalamic-pituitary-adrenal axis.
3 of CFS, including the hypothalamic-pituitary-adrenal axis.
4 sal regulation of the hypothalamic-pituitary-adrenal axis.
5 ted activation of the hypothalamic-pituitary-adrenal axis.
6 dysregulation of the hypothalamic-pituitary-adrenal axis.
7 pal regulation of the hypothalamic-pituitary-adrenal axis.
8 ing activation of the hypothalamic pituitary adrenal axis.
9 ns in function of the hypothalamic-pituitary-adrenal axis.
10 an equivalent of the hypothalamus-pituitary-adrenal axis.
11 ey to controlling the hypothalamic-pituitary-adrenal axis.
12 ant regulation of the hypothalamic-pituitary adrenal axis.
13 via activation of the hypothalamic-pituitary-adrenal axis.
14 eak activation of the hypothalamic-pituitary-adrenal axis.
15 the regulation of the hypothalamic-pituitary-adrenal axis.
16 etely mediated by the hypothalamic-pituitary-adrenal axis.
17 istent changes in the hypothalamic-pituitary-adrenal axis.
18 e sympathoadrenal and hypothalamic-pituitary adrenal axis.
19 and in particular the hypothalamo-pituitary adrenal axis.
20 context of the limbic-hypothalamo-pituitary adrenal axis.
21 bitory control of the hypothalamic-pituitary-adrenal axis.
22 in the function of the hypothalamo-pituitary-adrenal axis.
23 ons is mediated by the hypothalamo-pituitary-adrenal axis.
24 ich is mediated by the hypothalamo-pituitary-adrenal axis.
25 oid production by the hypothalamic-pituitary-adrenal axis.
26 he hypothalamo-pituitary-adrenal or sympatho-adrenal axis.
27 he hypothalamo-pituitary-adrenal or sympatho-adrenal axis.
28 rons that control the hypothalamic-pituitary-adrenal axis.
29 d to disruption of the hypothalamo-pituitary-adrenal axis.
30 halamic regulator of the endocrine pituitary-adrenal axis.
31 via activation of the hypothalamic pituitary adrenal axis.
32 and activation of the hypothalamic-pituitary-adrenal axis.
33 modulation of the activity of the pituitary-adrenal axis.
34 nterregulatory systems such as the pituitary-adrenal axis.
35 ility to activate the hypothalamic-pituitary-adrenal axis.
36 the amygdala and the hypothalamic-pituitary-adrenal axis.
37 ced regulation of the hypothalamic-pituitary-adrenal axis.
38 normally functioning hypothalamic-pituitary-adrenal axis.
39 by hormones from the hypothalamic-pituitary-adrenal axis.
40 ons, by affecting the hypothalamic-pituitary-adrenal axis.
41 ajor regulator of the hypothalamic-pituitary-adrenal axis.
42 dysregulation of the hypothalamic-pituitary-adrenal axis.
43 also regulated by the hypothalamic-pituitary-adrenal axis.
44 gnaling and an intact hypothalamic-pituitary-adrenal axis.
45 une activation of the hypothalamic-pituitary-adrenal axis.
46 ative feedback on the hypothalamic-pituitary-adrenal axis.
47 disinhibition of the hypothalamus-pituitary-adrenal axis.
48 ced activation of the hypothalamic-pituitary-adrenal axis.
49 edback control of the hypothalamic-pituitary-adrenal axis.
50 in the corticotropic (hypothalamo-pituitary-adrenal) axis.
51 ude (1) a hyperactive hypothalamic-pituitary-adrenal axis; (2) structural and functional abnormalitie
52 3) the stress system (hypothalamic-pituitary-adrenal axis), (4) the (gastrointestinal) corticotropin-
53 ase and activates the hypothalamic-pituitary-adrenal axis, a reflex central to the stress response an
55 her than an activated hypothalamic-pituitary-adrenal axis, ACTH-independent regulators have been repo
56 d by activation of the hypothalamo-pituitary-adrenal-axis acting in concert with endogenous EAAs from
57 ight loss may decrease hypothalamo-pituitary-adrenal axis activation and reduce glucocorticoid metabo
58 in, representative of hypothalamic-pituitary-adrenal axis activation, and the tyrosine hydroxylase ge
59 nstead of consecutive hypothalamus-pituitary-adrenal axis activation, we report that acute SCI in mic
64 ess the role of MR in hypothalamic-pituitary-adrenal axis activity and anxiety-related behaviors, we
66 nt role in regulating hypothalamic-pituitary-adrenal axis activity and mediating physiological respon
67 iposity, and abnormal hypothalamic-pituitary-adrenal axis activity could be predisposing factors.
69 his suggests that low hypothalamic-pituitary-adrenal axis activity is a correlate of severe and persi
71 hyperresponsivity in hypothalamic-pituitary-adrenal axis activity secondary to hypersecretion of cor
72 elated energy intake, hypothalamic-pituitary-adrenal axis activity, and the glucoregulatory response
73 anxiety behavior and hypothalamic-pituitary-adrenal axis activity, likely through modulation of hipp
79 key regulator of the hypothalamic-pituitary-adrenal axis, also has proinflammatory effects, apparent
80 mary regulator of the hypothalamic-pituitary-adrenal axis and a key element in the response to stress
81 sed activation of the hypothalamic-pituitary-adrenal axis and a poor antibody response to influenza v
82 ghrelin regulates the hypothalamic-pituitary-adrenal axis and affects anxiety and mood disorders, suc
83 after ablation of the hypothalamic-pituitary-adrenal axis and after high-dose glucocorticoid administ
84 yperactivation of the hypothalamic-pituitary-adrenal axis and altered accumulation of important growt
85 ghrelin regulates the hypothalamic-pituitary-adrenal axis and associated stress-induced behaviors, in
86 hat SEA activates the hypothalamic-pituitary-adrenal axis and augments gustatory neophobic behaviors.
87 findings suggest that hypothalamic-pituitary-adrenal axis and autonomic nervous system hyperreactivit
88 ; in that i.c.v. CRH activates the pituitary-adrenal axis and autonomic nervous system, these respons
89 N Sirt1 activates the hypothalamic-pituitary-adrenal axis and basal GC levels by enhancing the produc
90 patible with enhanced hypothalamic-pituitary-adrenal axis and decreased central sympathetic system ac
91 used to activate the hypothalamic-pituitary-adrenal axis and elevate corticosterone (CORT) levels in
92 ethanol activates the hypothalamic-pituitary-adrenal axis and elevates 3alpha,5alpha-THP in plasma, c
93 dysregulation of the hypothalamic-pituitary-adrenal axis and hippocampus and implicate corticotropin
94 Dysregulation of the hypothalamic pituitary adrenal axis and hyperactivity of the subgenual cortex a
99 ne (CRH), through the hypothalamic pituitary adrenal axis and other brain stress systems, is involved
100 onnection between the hypothalamic-pituitary-adrenal axis and RCT that can be triggered by a stress-i
101 alivary biomarkers of hypothalamus-pituitary-adrenal axis and sympathetic nervous system (SNS) activi
102 c nervous system, the hypothalamic-pituitary-adrenal axis and the cardiovascular, metabolic, and immu
104 the activation of the hypothalamus-pituitary-adrenal axis and the potentiation of acute-phase protein
105 ve was to examine the hypothalamic-pituitary-adrenal axis and the subjective and physiologic response
106 responsiveness of the hypothalamic-pituitary-adrenal axis and the sympathetic nervous system may repr
107 ressors stimulate the hypothalamic-pituitary-adrenal axis and the sympathetic nervous system, resulti
109 stimuli activate the hypothalamic-pituitary-adrenal axis and through which cortisol feedback modulat
110 ohorts, activated the hypothalamic-pituitary-adrenal axis, and caused reactivation of latent HSV type
111 ction, activity of the hypothalamo-pituitary-adrenal axis, and sympathovagal balance in 11 young men
112 , we have studied the hypothalamic-pituitary-adrenal axis, and the activities of the 11beta-hydroxyst
113 region modulating the hypothalamic-pituitary-adrenal axis-and somatosensory, viscerosensory, and inte
115 be independent of the hypothalamo-pituitary-adrenal axis, as both adrenalectomized and sham-operated
116 omote arousal via the hypothalamic-pituitary-adrenal axis, but rather probably acts via brainstem crh
117 urther activity in the hypothalamo-pituitary-adrenal axis, but the chronic actions (across days) of t
118 ons by regulating the hypothalamic-pituitary-adrenal axis, but the mechanisms of inhibition of hypoth
119 eroid release via the hypothalamic-pituitary-adrenal axis) by alcohol intoxication and withdrawal and
121 t the activity of the hypothalamic-pituitary-adrenal axis can be influenced by classical (Pavlovian)
122 opment of the mouse's hypothalamic-pituitary-adrenal axis can be modified by neonatal rearing conditi
123 ress responses of the hypothalamic-pituitary-adrenal axis can produce adverse effects on the brain.
124 gents can disrupt the hypothalamic-pituitary-adrenal axis, cause thyroid abnormalities, and result in
125 responses to standard hypothalamic-pituitary-adrenal axis challenge tests in adult female survivors o
126 d hypoactivity of the hypothalamic-pituitary-adrenal axis constitute other findings, but the question
127 ovascular system, and hypothalamic pituitary adrenal axis, contribute both to pathogenesis of disease
128 s hormone system, the hypothalamic-pituitary-adrenal axis, contributes to variability in stress-relat
129 in key relays of the hypothalamic-pituitary-adrenal axis could contribute to the brain's region-spec
130 ed suppression of the hypothalamic-pituitary-adrenal axis coupled with increased glucocorticoid trans
131 basal activity of the hypothalamic-pituitary-adrenal axis, CRF mRNA expression in the central nucleus
133 al that impairment of hypothalamic-pituitary-adrenal axis during depression can lead to olfactory def
134 ce the function of the hypothalamo-pituitary-adrenal axis during hypoxemic challenges to homeostasis
135 for activation of the hypothalamic-pituitary-adrenal axis during immunological challenge in the absen
137 ; it is possible that hypothalamic-pituitary-adrenal axis dysregulation is an important mechanism.
138 h the hypothesis that hypothalamic-pituitary-adrenal axis dysregulation may be a risk factor for poor
139 with both biological (hypothalamic-pituitary-adrenal axis dysregulation) and psychosocial processes (
140 thetic activation and hypothalamic-pituitary-adrenal axis dysregulation, have been linked to arterial
142 end-effectors of the hypothalamic-pituitary-adrenal axis, endogenous glucocorticoids also play an im
144 ised, and GR-mediated hypothalamic-pituitary-adrenal axis feedback is desensitized (as in the case of
145 ressant drugs improve hypothalamic-pituitary-adrenal axis feedback regulation and reduce plasma gluco
146 ytokines activate the hypothalamic-pituitary-adrenal axis for glucocorticoid release, and these hormo
147 nts was impaired, but hypothalamic-pituitary-adrenal axis function did not differ from that of contro
148 suggest that altered hypothalamic-pituitary-adrenal axis function due to preterm birth may be a sign
150 oendocrine markers of hypothalamic-pituitary-adrenal axis function were examined in a sample of 122 c
151 lammation, attenuated hypothalamus-pituitary-adrenal axis function, cognitive impairment, and large a
152 noamine transmission, hypothalamus-pituitary-adrenal axis function, immune function, neurogenesis, mi
155 (GR) sensitivity and hypothalamic-pituitary-adrenal axis functioning and has been associated with ma
156 neuroregulator of the hypothalamus-pituitary-adrenal axis, has broad central and peripheral distribut
157 l (as a marker of the hypothalamic-pituitary-adrenal axis), heart rate variability (as a marker of th
158 nown to influence the hypothalamic-pituitary-adrenal axis, here we test whether PFC and amygdala resp
159 the regulation of the hypothalamus-pituitary-adrenal axis hormones and that it augments CRS-induced o
160 al subiculum inhibits hypothalamic-pituitary-adrenal axis (HPA) activity following psychological, but
161 in mice activates the hypothalamic-pituitary-adrenal axis (HPA) and induces long-term behavioral chan
162 tor (LIF), affects the hypothalamo-pituitary-adrenal axis (HPA) by stimulating in vitro and in vivo p
163 ethanol activates the hypothalamic pituitary adrenal axis (HPA) causing release of glucocorticoids.
164 dulate activity of the hypothalamo-pituitary-adrenal axis (HPA) following stress, but the regulatory
171 rgic, neurons induced hypothalamic-pituitary-adrenal axis hyperactivity and reduced fear- and anxiety
172 glial activation, and hypothalamic-pituitary-adrenal axis hyperactivity in stress vulnerability.
173 of activation of the hypothalamus-pituitary-adrenal axis, hypoglycemia, serum amyloid A production,
174 one and stimulate the hypothalamic-pituitary-adrenal axis in circumstances such as pain, hypoxia or h
175 insufficiency of the hypothalamic-pituitary-adrenal axis in critical illness, which is diagnosed by
176 tion of CXCL5, suggesting a key role for the adrenal axis in driving CXCL5 expression and pulmonary n
177 responsiveness of the hypothalamic-pituitary-adrenal axis in individual patients generally remained w
178 for activation of the hypothalamic-pituitary-adrenal axis in response to stress, and has been a targe
180 acutely activate the hypothalamic pituitary adrenal axis in unstressed CD-1 mice or have the abortiv
181 abnormalities in the hypothalamic-pituitary-adrenal axis, including signaling by corticotropin-relea
182 nic stress alters the hypothalamic-pituitary-adrenal axis, increases gut motility, and increases the
183 In the case of the hypothalamic-pituitary-adrenal axis, induction of c-fos and/or NGFI-B mRNAs in
184 , end products of the hypothalamic-pituitary-adrenal axis, influence functions of virtually all organ
185 the anti-inflammatory hypothalamo-pituitary-adrenal axis, inhibition of both the production and hypo
186 hat neurogenesis, via hypothalamic-pituitary-adrenal axis interactions, is directly involved in preci
189 ugh activation of the hypothalamic-pituitary-adrenal axis is associated with a large variety of stres
190 factor (CRF) and the hypothalamic-pituitary-adrenal axis is controlled by the master circadian pacem
192 rating that the fetal hypothalamic-pituitary-adrenal axis is fully functional when the genitalia diff
194 hyperactivity of the hypothalamic-pituitary-adrenal axis is one of the most consistent biological fi
196 dysregulation of the hypothalamic-pituitary-adrenal axis, leading to changes in glucocorticoid relea
197 catecholaminergic and hypothalamic-pituitary-adrenal axis leads to splenic atrophy and contraction of
198 normalisation of the hypothalamic-pituitary-adrenal axis, maintenance of pituitary function, and avo
199 of evidence that the hypothalamic-pituitary-adrenal axis may influence the behavioral expression of
200 nsmitter systems, the hypothalamic-pituitary-adrenal axis, metabolic hormonal pathways, inflammatory
201 ack regulation of the hypothalamic-pituitary-adrenal axis occurs through a dual-receptor system of mi
203 idual elements of the hypothalamic pituitary adrenal axis on the pathogenesis of hypoglycemia-associa
204 k between CRF and the hypothalamic-pituitary-adrenal axis, on the one hand, and stress (including str
205 nal cascade along the hypothalamic-pituitary-adrenal axis orchestrates bodily responses to stress.
206 e of an immune system-hypothalamic-pituitary-adrenal axis pathway for regulating endogenous responses
208 sion to study whether hypothalamic-pituitary-adrenal axis perturbation could be sufficient to provoke
209 the activation of the hypothalamic pituitary adrenal axis plays an important role in stimulating IL-6
210 its activation of the hypothalamic-pituitary-adrenal axis, plays a key role in engaging the transitio
212 the stress-responsive hypothalamo-pituitary-adrenal axis raises the possibility that cocaine-related
213 ys, suggesting normal hypothalamic-pituitary-adrenal axis reactivity to painful stressors in CBP pati
216 central component of hypothalamic-pituitary-adrenal axis regulation that prepares the organism for s
217 ay overlap with GR in hypothalamic-pituitary-adrenal axis regulation, but they dissociate significant
221 with an insufficient hypothalamic-pituitary-adrenal axis response and the optimum treatment for this
222 lack of difference in hypothalamic pituitary adrenal axis response between the cocaine-dependent and
223 information mediating hypothalamic-pituitary-adrenal axis response habituation to repeated loud noise
224 Evaluation of the hypothalamic-pituitary-adrenal axis response in these animals revealed an incre
225 novelty have a larger hypothalamic-pituitary-adrenal axis response than do nonfearful individuals.
226 g and terminating the hypothalamic-pituitary-adrenal axis response to both acute and repeated stress.
227 r impairment of their hypothalamic-pituitary-adrenal axis response to stress than that of the CRFR1-m
228 n cocaine craving and hypothalamic-pituitary-adrenal axis responses are each associated with specific
229 knockdown had reduced hypothalamic-pituitary-adrenal axis responses to both acute and chronic stress
230 robust behavioral and hypothalamic-pituitary-adrenal axis responses to DR infusion of NBI 35965 and C
231 raving and associated hypothalamic-pituitary-adrenal axis responses to evaluate cocaine relapse prope
232 group showed altered hypothalamus-pituitary-adrenal axis responses to stress, evidenced by lower ACT
236 of CGRP activates the hypothalamo-pituitary-adrenal axis resulting in increased corticosterone secre
237 stimuli activate the hypothalamic-pituitary-adrenal axis resulting in increased steroidogenic activi
239 se examination of the hypothalamic-pituitary-adrenal axis showed that MC3-R(-/-) mice exhibit elevate
241 gnificantly increased hypothalamic-pituitary-adrenal axis stress response and impaired sensorimotor g
242 mitter system and the hypothalamic-pituitary-adrenal axis stress-response system in the diathesis for
243 edback control of the hypothalamus-pituitary-adrenal axis suggests that PP5 may be an important modul
245 functions altered the hypothalamic-pituitary-adrenal axis, sympathetic adrenal-medullary activation a
246 opin-releasing factor/hypothalamic-pituitary-adrenal axis system, as assessed by 24-hour urinary cort
247 gon secretion, and the hypothalamo-pituitary-adrenal axis that is commonly impaired in diabetes.
248 opioid system and the hypothalamic-pituitary-adrenal axis that may contribute to the development of n
250 al differences in the hypothalamic-pituitary-adrenal axis that underlie propensities for aggression,
251 C), the amygdala, and hypothalamic-pituitary-adrenal axis, the precise genetic and experiential contr
252 abetes influences the hypothalamic-pituitary-adrenal axis, the role of this neuroendocrine system in
253 the regulation of the hypothalamic-pituitary-adrenal axis, thereby affecting an individual's ability
254 ing activation of the hypothalamic-pituitary-adrenal axis through direct projections to paraventricul
255 immune system is the hypothalamic-pituitary-adrenal axis, through the hormones of the neuroendocrine
256 a might sensitise the hypothalamic-pituitary-adrenal axis to development of persistent central fatigu
257 itical element of the hypothalamic-pituitary-adrenal axis to provide effective glucocorticoid-depende
260 , which activates the hypothalamic-pituitary-adrenal axis under stress, also has proinflammatory peri
261 luence of CRF and the hypothalamic-pituitary-adrenal axis upon the circadian pacemaker is less well e
262 er, activation of the hypothalamic-pituitary-adrenal axis using restraint stress did not activate the
263 ory regulation of the hypothalamic-pituitary-adrenal axis via hypothalamic glutamatergic neurons and
264 asting changes in the hypothalamic-pituitary-adrenal axis, we found that social recognition memory wa
265 tokine activating the hypothalamic-pituitary-adrenal axis, were markedly increased (495 +/- 131 vs. 2
266 e system includes the hypothalamic-pituitary-adrenal axis, which centrally drives glucocorticoid prod
267 e and the tone of the hypothalamic-pituitary-adrenal axis, which might negatively affect the cardiova
268 pid activation of the hypothalamic-pituitary-adrenal axis, which typically resolves within 60-90 min
269 ing activation of the hypothalamic-pituitary-adrenal axis with atherosclerosis are not well-understoo
270 The activation of the hypothalamic-pituitary-adrenal axis with cocaine appears to depend on feed-forw
272 d inactivation of the hypothalamic-pituitary-adrenal axis, without affecting energy expenditure or gl
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