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5 nRNA) expression in rats that were intact or adrenalectomized (ADX) and replaced with corticosterone
6 le was administered daily to gonadectomized, adrenalectomized (ADX) male (experiment 1) and female (e
9 nduced c-fos expression was characterized in adrenalectomized (ADX) or adrenalectomized and corticost
10 ases in duration of inhibitory action in the adrenalectomized (adx) rat after intravenous injection,
11 e effects of two adrenal steroid agonists on adrenalectomized (ADX) rats' performance on the Y-maze.
12 s characterized in adrenalectomized (ADX) or adrenalectomized and corticosterone replaced (ADX/B) mal
13 Rats implanted with telemetry probes were adrenalectomized and given low-corticosterone pellets or
15 is of thrombotic microangiopathy, SHRSP were adrenalectomized and infused with vehicle, Ang II, or AL
16 ne expression in male C57BL/6 mice that were adrenalectomized and replaced with defined levels of cor
17 stimulation, and carbachol were compared in adrenalectomized and sham-operated halothane-anesthetize
18 hypothalamo-pituitary-adrenal axis, as both adrenalectomized and sham-operated mice exhibited simila
19 n IL-1beta protein 2 h after IS, but only in adrenalectomized (and basal corticosterone replaced) sub
20 eceded the onset of anorexia, and dehydrated adrenalectomized animals (which also develop anorexia) h
22 type I adrenal steroid receptor agonist) to adrenalectomized animals did not have a significant effe
23 hydration because PVH CRH mRNA in dehydrated adrenalectomized animals is unchanged from euhydrated ad
24 62 (a specific type II receptor agonist), to adrenalectomized animals produced changes in leukocyte d
27 , in the presence of constant B (B-replaced, adrenalectomized animals), cold stress led to increased
28 This effect of stress was not observed in adrenalectomized animals, and was reproduced in nonstres
31 stered intraperitoneally to sham-operated or adrenalectomized/castrated (ADX/CX) male rats dose-depen
33 ed decrements in PPG mRNA were attenuated in adrenalectomized-corticosterone-replaced rats, suggestin
35 hetic nervous system, we compared unilateral adrenalectomized, desynchronized rats that had undergone
36 atory hormones in overnight-fasted conscious adrenalectomized dogs that were given somatostatin and i
37 the formation of partner preferences, while adrenalectomized females form preferences more quickly t
40 onitoring explants taken from rats that were adrenalectomized, intact, or treated with corticosterone
41 l corticosteroid receptor gene expression in adrenalectomized male C57BL/6 mice with fixed glucocorti
45 ne dependent, as evidenced by its absence in adrenalectomized mice, in which plasma corticosterone le
49 AFC in beta(2)AR overexpressing lungs from adrenalectomized or propranolol-treated rats revealed cl
50 activation, whereas corticosterone-depleted (adrenalectomized) paired odor-shock pups showed odor-pre
51 in branched-chain amino acid catabolism, in adrenalectomized rat skeletal muscle and liver, the two
53 terminal IMCD was significantly increased in adrenalectomized rats and reduced in dexamethasone-treat
55 RNA levels were not altered significantly in adrenalectomized rats before and after cortisone treatme
56 ve challenge increased LC discharge rates of adrenalectomized rats by a magnitude greater than that p
58 ed at 4 hr after peripheral LPS injection in adrenalectomized rats compared with sham-operated rats.
59 dent of circulating glucocorticoids, because adrenalectomized rats displayed an identical pattern.
60 nt glucoprivic feeding were also examined in adrenalectomized rats in which the source of endogenous
61 immediate precursor of 3alpha,5alpha-THP, to adrenalectomized rats not only restored the ethanol-indu
62 icosteroid-regulated proteins in livers from adrenalectomized rats over 11 time points after drug dos
63 ized or given sham surgery, with half of the adrenalectomized rats receiving corticosterone replaceme
65 aint stress were examined in both intact and adrenalectomized rats receiving glucocorticoid replaceme
67 onic CRF secretion in the LC is increased in adrenalectomized rats suggest that activity of certain C
68 F antagonist decreased LC discharge rates of adrenalectomized rats to rates comparable with those obs
69 hrough a different receptor, three groups of adrenalectomized rats were prepared: (1) vehicle, (2) ad
74 nse curve was shifted in a complex manner in adrenalectomized rats, suggesting that a proportion of C
75 al allodynia after CCI also was prevented in adrenalectomized rats, whereas the GR agonist dexamethas
84 oto rat (WKY, 69.6+/-6.5 mm Hg; P<0.0001) or adrenalectomized SHR (73.5+/-2.1 mm Hg; P=0.0009) at the
85 e increases in brain IL-1beta protein in the adrenalectomized subjects 2 hr after stress, whether bas
86 one in an amount that led to blood levels in adrenalectomized subjects that match those produced by I
87 e hypothalamus and in other brain regions in adrenalectomized subjects, and no longer present 24 h la
92 increase corticosterone production, because adrenalectomized wild-type mice had similar disease cour
93 pression, male wild-type and ob/ob mice were adrenalectomized (with saline supplementation) or sham a
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