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1 nd a promoter selective to noradrenergic and adrenergic neurons.
2 of the central nervous system (CNS) rich in adrenergic neurons.
3 (SNA) after the depletion of bulbospinal C1 adrenergic neurones.
6 a (RVLM) region that contains bulbospinal C1 adrenergic neurons and is involved in blood pressure con
7 s expressed selectively in noradrenergic and adrenergic neurons and neuroendocrine cells in the nervo
9 ship between relay visceral sensory and (nor)adrenergic neurons, and suggest that it may be a common
10 cardioprotection: NHE inhibition in cardiac adrenergic neurons as a means to prevent ischemic arrhyt
11 that the fate and innervation pattern of the adrenergic neurons, as well as the development of the ce
12 In a separate group, pretreatment with the adrenergic neuron blocker, guanethidine (20 mg/kg), also
14 ological properties of the rostral medullary adrenergic neurons (C(1)) that express neuropeptide Y (N
15 In summary, the loss of 84% of bulbospinal adrenergic neurones does not alter the ability of RVLM t
18 , and respiration that may be mediated by C1 adrenergic neurons in the rostral ventrolateral medulla
19 ected 3,4-dihydroxyphenylglycolaldehyde onto adrenergic neurons in the rostral ventrolateral medulla.
20 either region were equally likely to contact adrenergic neurons in the RVL (21% for NTS, 25% for CVL)
21 with immunocytochemical identification of C1 adrenergic neurons in the RVL to compare the morphology
23 ed by tyrosine hydroxylase (TH) antibody, or adrenergic neurons labeled by phenylethanolamine-N-methy
24 stral ventrolateral medulla (including to C1 adrenergic neurons), locus coeruleus, A5 cell group, par
26 DNPI/VGLUT2 mRNA was detected in most of the adrenergic neurons of the C1, C2, and C3 groups (75-80%
30 luding the rostral ventrolateral medulla (C1 adrenergic neurons), rostral ventromedial medulla, cauda
31 ne acetyltransferase positive) and intrinsic adrenergic neurons (tyrosine hydroxylase positive) follo
34 a mode of endogenous AR cross-talk in native adrenergic neurons whereby canonical betaAR-mediated sig
35 t neuronal firing rate change in a subset of adrenergic neurons with an EC(50)=2.7 microM, a dose wel
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