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1 atment with 50 nM Compound 49b, a novel beta-adrenergic receptor agonist.
2 onist, or isoprenaline, a non-selective beta-adrenergic receptor agonist.
3 ation of UCP1 after stimulation with a beta3-adrenergic receptor agonist.
4 free [Ca2+] oscillations evoked by an alpha1 adrenergic receptor agonist.
5 nucleus when cells were treated with alpha1-adrenergic receptor agonist.
6 both amphetamine and guanfacine, an alpha2A adrenergic receptor agonist.
7 This is reversed by isoproterenol, a beta-adrenergic receptor agonist.
8 ase activity induced by UK14304, an alpha(2)-adrenergic receptor agonist.
9 ed neonatal cardiac myocytes exposed to beta-adrenergic receptor agonists.
10 sensitizes the channel to activation by beta-adrenergic receptor agonists.
11 ased sensitivity to the lipolytic effects of adrenergic receptor agonists.
12 threefold greater than that elicited by beta-adrenergic receptor agonists.
13 the anti-allodynic effect elicited by alpha2-adrenergic receptor agonists.
15 ells with phenylephrine, a specific alpha(1)-adrenergic receptor agonist, activated p70 S6 kinase but
16 tal secretion by secretin (through cAMP) and adrenergic receptor agonist activation (through Ca(2+)/P
17 myocardium to exogenous and endogenous beta-adrenergic receptor agonists after burn injury may be at
18 nsulin receptor substrate-1, as the alpha(1)-adrenergic receptor agonist also inhibited the platelet-
19 s study tests whether clonidine, an alpha(2)-adrenergic receptor agonist, alters excitatory glutamate
20 teristics in response to treatment with beta-adrenergic receptor agonists and activators of adenylate
21 l currently available therapies such as beta-adrenergic receptor agonists and antagonists, phosphodie
24 ume less oxygen after treatment with a beta3-adrenergic-receptor agonist and that they are sensitive
25 easured in response to isoproterenol, a beta-adrenergic receptor agonist, and carbachol, a cholinergi
27 chanism of Rett syndrome and introduce beta2-adrenergic receptor agonists as potential therapeutic ag
30 AR) in wound scarring, the ability of beta 2 adrenergic receptor agonist (beta2ARag) to alter HDF dif
31 ides were synthesized and evaluated as beta3-adrenergic receptor agonists (beta3-AR) for the treatmen
32 otected myocytes from death induced by beta1-adrenergic receptor agonists by preventing cytosolic and
33 evealed that formoterol, a long-acting beta2 adrenergic receptor agonist, caused significant improvem
34 rality were treated with the selective beta3 adrenergic receptor agonist CL 316, 243 and underwent me
36 and given intraperitoneally either the beta3-adrenergic receptor agonist CL-316,243, 1 mg/kg (n = 8),
39 lementation of a high fat diet with a beta 3-adrenergic receptor agonist (CL316,243) prevented obesit
42 Here, we show that treatment with the beta2-adrenergic receptor agonist clenbuterol increases surviv
44 dministration of the centrally acting alpha2-adrenergic receptor agonist, clonidine), and hypoxia and
45 eptor antagonist, prazosin, and the alpha(2)-adrenergic receptor agonist, clonidine, reduced the numb
46 her transdermal administration of the alpha2 adrenergic-receptor agonist, clonidine, can be used as a
48 induced by growth promoters, such as alpha1-adrenergic receptor agonists (e.g. phenylephrine (PE)).
50 activation in MDCK-D1 cells triggered by the adrenergic receptor agonist epinephrine and its mechanis
52 of this study was to determine whether alpha-adrenergic receptor agonists have a role in alveolar flu
53 ating airway obstructive diseases, and beta2 adrenergic receptor agonists have been the most commonly
54 hma exacerbation is the short-acting beta(2)-adrenergic receptor agonist; however, there is variation
55 an inhibit the Cl- current activated by beta-adrenergic receptor agonists in guinea-pig ventricular m
56 ced responsiveness to alpha(1)- and alpha(2)-adrenergic receptor agonists in skeletal muscle vasculat
59 with phenylephrine (PE), a specific alpha(1) adrenergic receptor agonist, increased protein synthesis
60 her insulin or phenylephrine (PE), an alpha1-adrenergic receptor agonist, induced Ser-9 phosphorylati
61 Rho and inhibited the characteristic alpha1-adrenergic receptor agonist-induced myofibrillar organiz
64 d electrophysiological responses to the beta-adrenergic receptor agonist isoprenaline (Iso) in CA1 py
65 owing intra-LS injections of either the beta-adrenergic receptor agonist isoproterenol (10 mug or 30
69 reperfused with the addition of the beta(2)-adrenergic receptor agonist isoproterenol (iso), and rol
70 es, we show that norepinephrine and the beta-adrenergic receptor agonist isoproterenol also inhibited
71 were conducted using forskolin and the beta-adrenergic receptor agonist isoproterenol as agonists.
72 ministration of either the nonselective beta-adrenergic receptor agonist isoproterenol or the beta3-a
73 Both angiotensin II (Ang II) and the beta-adrenergic receptor agonist isoproterenol selectively in
74 ATP and UTP were not additive with the beta-adrenergic receptor agonist isoproterenol, but were syne
75 either alone or in the presence of the beta-adrenergic receptor agonist isoproterenol, failed to ind
76 ng periodic pacing in the presence of a beta-adrenergic receptor agonist isoproterenol, was significa
77 27 complex in response to the selective beta adrenergic receptor agonist isoproterenol, was subsequen
78 d enhanced inotropic sensitivity to the beta-adrenergic receptor agonist isoproterenol, with impairme
82 se-response curve of the effect of the beta2-adrenergic-receptor agonist isoproterenol was constructe
84 re incubated with forskolin or with the beta-adrenergic receptor agonist, isoproterenol, to stimulate
86 wing administration of isoproterenol, a beta-adrenergic receptor agonist known to induce cardiac inju
87 or in combination with a long-acting beta2 -adrenergic receptor agonist (LABA) on GCM in the bronchi
88 ytic effects we observed suggest that alpha2 adrenergic-receptor agonists may offer a pharmacologic m
90 sistent with this idea, the selective alpha1-adrenergic receptor agonist methoxamine was still able t
92 rbol 12-myristate 13-acetate or the alpha(1)-adrenergic receptor agonist norepinephrine via a mechani
94 vity by forskolin, cAMP analogs, or the beta-adrenergic receptor agonists norepinephrine and isoprote
96 o information exists regarding the effect of adrenergic receptor agonists on ductal secretion in a mo
97 he effect of salmeterol, a long-acting beta2-adrenergic receptor agonist, on ovalbumin-induced plasma
98 e induction of these responses by the alpha1-adrenergic receptor agonist phenylephrine, whereas its i
99 luated the effects of the alpha-1 and beta-1 adrenergic receptor agonists (phenylephrine and dobutami
100 ly) or saline, the response to the a) alpha1-adrenergic receptor agonist, phenylephrine; b) endotheli
102 ent stimulation of beating rate by the beta2 adrenergic receptor agonist procaterol was significantly
103 f the two genotypes with a selective beta(3)-adrenergic receptor agonist produced similar increases i
104 me indicates that chronic exposure to alpha2-adrenergic receptor agonists produces strong dependence.
105 se receptors with BRL37344, a specific beta3-adrenergic receptor agonist, promoted migration that was
106 ments revealed that treatment with a beta(3)-adrenergic receptor agonist resulted in ligand-induced c
108 short-acting and long-acting inhaled beta(2)-adrenergic receptor agonists (SABA and LABA, respectivel
112 c TGF-beta type II receptor restored beta(2)-adrenergic receptor agonist-stimulated alveolar epitheli
113 iator of acute lung injury, inhibits beta(2)-adrenergic receptor agonist-stimulated vectorial fluid a
115 uced in cultured myocardial cells by alpha1- adrenergic receptor agonists, such as phenylephrine (PE)
116 catecholamine agonists tested, only beta(1)-adrenergic receptor agonists supported encystation, wher
117 e after treatment with isoproterenol, a beta-adrenergic receptor agonist that causes turnover of palm
118 exmedetomidine (DEX; Precedex) is an alpha-2 adrenergic receptor agonist that produces anxiolysis and
120 es were treated with phenylephrine, an alpha-adrenergic receptor agonist, to drive HDACS out of the n
122 (1.0 microm), and attenuated by the alpha(2)-adrenergic receptor agonist, UK 14,304 (1.0 microm), whi
123 ifically stimulated by the selective alpha2A-adrenergic receptor agonist UK14304 and by lysophosphati
124 turbed by intravenous injection of an alpha1-adrenergic receptor agonist, unilateral pressure changes
125 turbed by intravenous injection of an alpha1-adrenergic receptor agonist, unilateral pressure changes
127 pithelial bilayer, when stimulated with beta adrenergic receptor agonists, vasointestinal peptide, or
130 d some cells with Compound 49b, a novel beta-adrenergic receptor agonist we have reported previously
131 e development of orally bioavailable beta(3) adrenergic receptor agonists, we have identified a serie
132 Significance statement: Clonidine, an alpha2-adrenergic receptor agonist, which decreases the level o
133 ere treated with CL-316243, a specific beta3-adrenergic receptor agonist, which sensitizes insulin ac
134 hat orally administered DL-isoproterenol, an adrenergic receptor agonist whose skeleton includes 1,2-
135 in Ts65Dn mice, formoterol or similar beta2 adrenergic receptor agonists with ability to cross the b
136 hase of LTP by pairing isoproterenol, a beta-adrenergic receptor agonist, with a weak train, subthres
137 the effects of increasing doses of the beta2-adrenergic receptor agonist zinterol on Ca2+ transient a
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