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1  the locus coeruleus and decreased pituitary adrenocorticotropic and vasopressin content.
2 logs (alpha-melanocyte-stimulating hormone = adrenocorticotropic hormone > gamma-melanocyte-stimulati
3                                              Adrenocorticotropic hormone (250 mug IV) stimulation tes
4 e unresponsive to medical therapy, including adrenocorticotropic hormone (ACTH) administration.
5 e expected to stimulate pituitary release of adrenocorticotropic hormone (ACTH) and affect input to a
6 n was not influenced significantly by plasma adrenocorticotropic hormone (ACTH) and CORT levels.
7          In males, gonadectomy increases the adrenocorticotropic hormone (ACTH) and corticosterone (C
8                                       Plasma adrenocorticotropic hormone (ACTH) and corticosterone (C
9 aken to determine levels of the HPA hormones adrenocorticotropic hormone (ACTH) and corticosterone ac
10                                       Plasma adrenocorticotropic hormone (ACTH) and corticosterone le
11        Acute ether stress provokes increased adrenocorticotropic hormone (ACTH) and corticosterone se
12 ation, rats were sacrificed, blood levels of adrenocorticotropic hormone (ACTH) and corticosterone we
13 rmalized blood pressure and decreased plasma adrenocorticotropic hormone (ACTH) and corticosterone.
14                        At 1.5 and 2.5 years, adrenocorticotropic hormone (ACTH) and cortisol concentr
15                                       Plasma adrenocorticotropic hormone (ACTH) and cortisol levels w
16 ions in plasma levels of leptin and those of adrenocorticotropic hormone (ACTH) and cortisol that cou
17 terial plasma was then assayed for levels of adrenocorticotropic hormone (ACTH) and cortisol.
18 release as well as the response to pituitary adrenocorticotropic hormone (ACTH) and hypothalamic lute
19 ng into ES-like stem cells by treatment with adrenocorticotropic hormone (ACTH) and leukaemia inhibit
20 which the pituitary gland releases excessive adrenocorticotropic hormone (ACTH) as a result of an ade
21 tress hormones corticosterone (CORT) and the adrenocorticotropic hormone (ACTH) before and after slee
22 P) or in vivo treatment of rat adrenals with adrenocorticotropic hormone (ACTH) decreased the express
23  2 had growth hormone (GH) deficiency, 1 had adrenocorticotropic hormone (ACTH) deficiency, and 1 had
24 risk factor being cortisol deficiency due to adrenocorticotropic hormone (ACTH) deficiency.
25 ruct (FLAG-pro-GX sPLA2), we determined that adrenocorticotropic hormone (ACTH) enhanced FLAG-pro-GX
26 es the actin cytoskeleton, shifts endogenous adrenocorticotropic hormone (ACTH) from mature granules
27 99 attenuates the synthesis and secretion of adrenocorticotropic hormone (ACTH) from the anterior pit
28 y-adrenal (HPA) axis activation by exogenous adrenocorticotropic hormone (ACTH) in blubber of norther
29 ion of renin, arginine vasopressin (AVP) and adrenocorticotropic hormone (ACTH) in late-gestation fet
30  vivo injections of the melanocortin peptide adrenocorticotropic hormone (ACTH) increase the amplitud
31 e activation of the pregnant female by daily adrenocorticotropic hormone (ACTH) injection across a 2-
32                                              Adrenocorticotropic hormone (ACTH) injections prevented
33  a suppressed cortisol response to exogenous adrenocorticotropic hormone (ACTH) irrespective of the p
34   Whereas the constitutive-like secretion of adrenocorticotropic hormone (ACTH) is increased by expre
35 icant increase in plasma DHEA, cortisol, and adrenocorticotropic hormone (ACTH) levels (all p = .02).
36 Subjective stress, cardiovascular responses, adrenocorticotropic hormone (ACTH) levels, and cortisol
37                Melanocortin therapy by using adrenocorticotropic hormone (ACTH) or non-steroidogenic
38 -L-Pro-L-Val, and L-Lys-L-Pro-D-Val) and two adrenocorticotropic hormone (ACTH) peptides (1-17 and 1-
39 steroidogenesis in response to physiological adrenocorticotropic hormone (ACTH) perturbations, rangin
40 utant animals was attributed to insufficient adrenocorticotropic hormone (ACTH) production during the
41                               We report that adrenocorticotropic hormone (ACTH) protects against oste
42 ocortin-2 receptor (MC2R), also known as the adrenocorticotropic hormone (ACTH) receptor, plays an im
43 (V(m)) and couple angiotensin II (AngII) and adrenocorticotropic hormone (ACTH) receptors to membrane
44 1) receptor (K(i) = 3.5, 2.9 nM) and inhibit adrenocorticotropic hormone (ACTH) release from rat pitu
45 AAR suppresses basal and secretagogue-evoked adrenocorticotropic hormone (ACTH) release in a manner i
46 ardiorespiratory stimulation, adrenaline and adrenocorticotropic hormone (ACTH) release, and arousal.
47 mal, Crhr2-/- mice show early termination of adrenocorticotropic hormone (Acth) release, suggesting t
48 ity in the form of peer rearing to influence adrenocorticotropic hormone (ACTH) response to stress an
49 ude reduced; whereas, the peak of the plasma adrenocorticotropic hormone (ACTH) rhythm is also reduce
50 ocortical (HPA) axis responses and increased adrenocorticotropic hormone (ACTH) secretagogue biosynth
51 ch activates adenylyl cyclase and stimulates adrenocorticotropic hormone (ACTH) secretion from cortic
52 sol secretion by primary adrenal lesions and adrenocorticotropic hormone (ACTH) secretion from cortic
53 ated proopiomelanocortin gene expression and adrenocorticotropic hormone (ACTH) secretion, inhibiting
54 elanocortin (POMC) gene expression, and thus adrenocorticotropic hormone (ACTH) secretion, which then
55 distinguish Cushing disease from the ectopic adrenocorticotropic hormone (ACTH) syndrome is accurate
56                                              Adrenocorticotropic hormone (ACTH) treatment of HPX rats
57 his, leptomycin B-induced nuclear import and adrenocorticotropic hormone (ACTH) treatment result in t
58 ed for analysis of plasma corticosterone and adrenocorticotropic hormone (ACTH) under basal condition
59             Serum cortisol (CORT) and plasma adrenocorticotropic hormone (ACTH) were assessed in resp
60 ovanillic acid (HVA, a dopamine metabolite), adrenocorticotropic hormone (ACTH), and cortisol were as
61 alpha), interleukins (IL) IL-1beta and IL-6, adrenocorticotropic hormone (ACTH), and cortisol.
62 elicited by sodium deficiency, stress-evoked adrenocorticotropic hormone (ACTH), and reproduction.
63 uch as prolactin (PRL), growth hormone (GH), adrenocorticotropic hormone (ACTH), and thyroid stimulat
64                       The pituitary hormones adrenocorticotropic hormone (ACTH), beta-endorphin, and
65 c CRH release, and measured plasma levels of adrenocorticotropic hormone (ACTH), cortisol, and catech
66                                              Adrenocorticotropic hormone (ACTH), cortisol, and interl
67                                              Adrenocorticotropic hormone (ACTH), cortisol, glucagon,
68 ordinately regulated with steroidogenesis by adrenocorticotropic hormone (ACTH), human chorionic gona
69 ization stress, assessed by measuring plasma adrenocorticotropic hormone (ACTH), in both strains, alt
70                       Standard treatments of adrenocorticotropic hormone (ACTH), oral corticosteroids
71 elease of alpha-melanotropin (alpha-MSH) and adrenocorticotropic hormone (ACTH), which induce cyclic
72                              The presence of adrenocorticotropic hormone (ACTH)-immunoreactive cells
73      In adrenal cells, MRAP is essential for adrenocorticotropic hormone (ACTH)-induced activation of
74 blood pressure, and it specifically mediates adrenocorticotropic hormone (ACTH)-induced hypertension
75 ith wild-type (WT) mice under both basal and adrenocorticotropic hormone (ACTH)-induced stress condit
76                                              Adrenocorticotropic hormone (ACTH)-secreting tumors acco
77 e Ca(2+) channels that function pivotally in adrenocorticotropic hormone (ACTH)-stimulated cortisol s
78 DNA whose expression is rapidly inhibited by adrenocorticotropic hormone (ACTH).
79 bility to form corticosterone in response to adrenocorticotropic hormone (ACTH).
80 lating concentrations of glucocorticoids and adrenocorticotropic hormone (ACTH).
81 e (GA) for assessment of pCRH, cortisol, and adrenocorticotropic hormone (ACTH).
82 C2 nulls contained highly elevated pituitary adrenocorticotropic hormone (ACTH); the neurointermediat
83 with resultant increased levels of pituitary adrenocorticotropic hormone and adrenal hormones.
84 the animals demonstrate a normal increase in adrenocorticotropic hormone and corticosterone after res
85                                        Basal adrenocorticotropic hormone and corticosterone levels ar
86 st MK-801 to infected mice normalized plasma adrenocorticotropic hormone and corticosterone levels, i
87 rain (GRov) display normal basal circulating adrenocorticotropic hormone and corticosterone levels.
88 lood samples were subsequently collected for adrenocorticotropic hormone and corticosterone measureme
89 lipopolysaccharide, rats showed a pattern of adrenocorticotropic hormone and corticosterone that was
90 sted as an increase in circulating levels of adrenocorticotropic hormone and corticosterone, beginnin
91  attenuated basal and postprandial levels of adrenocorticotropic hormone and cortisol and curbed the
92 can in order to measure the subjects' plasma adrenocorticotropic hormone and cortisol levels.
93 rafting, there is a massive increase in both adrenocorticotropic hormone and cortisol secretion.
94  There was an initial rise in both levels of adrenocorticotropic hormone and cortisol to supranormal
95                Ultradian pulsatility of both adrenocorticotropic hormone and cortisol was maintained
96 tration, but modest increases in circulating adrenocorticotropic hormone and cortisol were observed.
97    In single anesthetic dose studies, plasma adrenocorticotropic hormone and cytokine concentrations
98 s with greater increases in plasma levels of adrenocorticotropic hormone and modest reductions in glu
99                         Hormonal treatments (adrenocorticotropic hormone and prednisolone) have been
100 nd that its expression in vivo is induced by adrenocorticotropic hormone and suppressed by glucocorti
101 eased slightly from baseline to week 52, but adrenocorticotropic hormone and the cortisol:adrenocorti
102     We also studied the content of pituitary adrenocorticotropic hormone and vasopressin, representat
103                            We also show that adrenocorticotropic hormone appears to be involved in a
104     alpha-Melanocyte-stimulating hormone and adrenocorticotropic hormone are well documented to regul
105 of agouti-signaling protein) and imitated by adrenocorticotropic hormone but not elicited by the trip
106 normal or greater than normal morning plasma adrenocorticotropic hormone concentration, and a pituita
107                                              Adrenocorticotropic hormone controls the function and ex
108 hyroid-stimulating hormone (TSH) deficiency, adrenocorticotropic hormone deficiency, and primary hypo
109 uclear translocation, resulting in CVID with adrenocorticotropic hormone deficiency, growth hormone d
110 rabenazine for chorea in Huntington disease, adrenocorticotropic hormone for infantile spasms, and en
111      The sensitivity of the adrenal gland to adrenocorticotropic hormone increased markedly at around
112                The decrease was prevented by adrenocorticotropic hormone injections.
113 or necrosis factor (TNF)-alpha (p < .01) and adrenocorticotropic hormone levels (p < .03) were signif
114            Stress reactivity was measured by adrenocorticotropic hormone levels and by cortisol conce
115 ing this observation is the fact that normal adrenocorticotropic hormone levels are substantially red
116 significantly suppressed plasma cortisol and adrenocorticotropic hormone levels compared with anti-CD
117  Because of the impaired cortisol secretion, adrenocorticotropic hormone levels rise due to impairmen
118                                              Adrenocorticotropic hormone levels then returned toward
119                           Serum cortisol and adrenocorticotropic hormone levels were drawn twice dail
120                                              Adrenocorticotropic hormone levels were lower at the tim
121 cortisol measurements--whether pre- or post- adrenocorticotropic hormone or as a calculated delta--we
122         The partition and structure of three adrenocorticotropic hormone peptides ACTH(1-10), ACTH(1-
123 t of the neuroendocrine circuits involved in adrenocorticotropic hormone production, may be more prom
124                    No patient had a cortisol:adrenocorticotropic hormone ratio <3.0 (nmol/pmol).
125 adrenocorticotropic hormone and the cortisol:adrenocorticotropic hormone ratio did not change.
126                        Adrenal levels of the adrenocorticotropic hormone receptor (melanocortin type
127 egion is 98% identical to part of the bovine adrenocorticotropic hormone receptor.
128 hat secretagogin's loss of function occludes adrenocorticotropic hormone release from the pituitary a
129                                              Adrenocorticotropic hormone replacement elevated plasma
130                                          The adrenocorticotropic hormone response to acute stress was
131 rmone stimulation test, we found an impaired adrenocorticotropic hormone response, particularly in no
132 s-related behaviors and showed an attenuated adrenocorticotropic hormone response.
133  voxel-wise analyses, the authors found that adrenocorticotropic hormone responses were covaried with
134 he CRF-BP ligand inhibitor did not stimulate adrenocorticotropic hormone secretion or elevate heart r
135                                 The elevated adrenocorticotropic hormone stimulates both the accumula
136                                              Adrenocorticotropic hormone stimulation of cortisol was
137 ary-adrenal function, commonly utilizing the adrenocorticotropic hormone stimulation test to diagnose
138                           Notably, using the adrenocorticotropic hormone stimulation test, we found a
139 ered by intensive care therapies or frequent adrenocorticotropic hormone stimulation testing with one
140 ) or undergo either a total of four or seven adrenocorticotropic hormone stimulation tests over 96 hr
141 d mechanical ventilation, completion of more adrenocorticotropic hormone stimulation tests, or dexame
142                                   Failure of adrenocorticotropic hormone to augment plasma cortisol a
143                 Despite a subsequent fall of adrenocorticotropic hormone to basal levels, cortisol re
144   Finally, in contrast to the adrenal gland, adrenocorticotropic hormone treatment did not increase t
145 e its levels are elevated by acute stress or adrenocorticotropic hormone treatment.
146                                 Cortisol and adrenocorticotropic hormone were measured every 10 and 6
147                    Plasma corticosterone and adrenocorticotropic hormone were measured from blood col
148 ontained neuropeptide-processing enzymes and adrenocorticotropic hormone, a product of proopiomelanoc
149 de for various bioactive peptides, including adrenocorticotropic hormone, alpha-, beta-, and gamma-me
150 tural ligands are the melanocortin peptides, adrenocorticotropic hormone, and alpha-, beta-, and gamm
151             Plasma levels of norepinephrine, adrenocorticotropic hormone, and cortisol were assessed.
152                         Vitamin E, cortisol, adrenocorticotropic hormone, and gonadal hormones were a
153 s, alpha-melanocyte-stimulating hormone, and adrenocorticotropic hormone, are important mediators of
154 anocortin, from which other peptides such as adrenocorticotropic hormone, beta-lipotropic hormone, an
155 ary proopiomelanocortin processing to mature adrenocorticotropic hormone, blood corticosterone levels
156 ma corticosterone secretion and basal plasma adrenocorticotropic hormone, but surprisingly, have a pr
157                                       Plasma adrenocorticotropic hormone, corticosterone, and cytokin
158                 Blood plasma was assayed for adrenocorticotropic hormone, cortisol, and testosterone.
159 is hormones-corticotropin-releasing hormone, adrenocorticotropic hormone, dehydroepiandrosterone sulf
160 protein partitioned from endogenous hormone (adrenocorticotropic hormone, growth hormone) into separa
161 ant increase in cortisol, despite suppressed adrenocorticotropic hormone, indicating primary (adrenal
162 rease in the serum levels of corticosterone, adrenocorticotropic hormone, interleukin-6, and tumor ne
163 one (GHRH), pituitary proopiomelanocortin to adrenocorticotropic hormone, islet proinsulin to insulin
164 mportant regulators are MC1 receptor its and adrenocorticotropic hormone, melanocyte stimulating horm
165 opiomelanocortin (POMC) is the precursor for adrenocorticotropic hormone, melanocyte-stimulating horm
166 e, and p27-null mice showed evidence of TSH, adrenocorticotropic hormone, prolactin, and luteinizing
167 y with scores on state-anxiety and levels of adrenocorticotropic hormone, serum cortisol, and saliva
168 ctor binding protein-3, prolactin, cortisol, adrenocorticotropic hormone, thyroid-stimulating hormone
169  a major change in adrenal responsiveness to adrenocorticotropic hormone, we used a reverse translati
170 he plasma concentration of corticosterone or adrenocorticotropic hormone, whereas their F/? counterpa
171 he primary receptor mediating the release of adrenocorticotropic hormone, which regulates endogenous
172 nse in these animals revealed an increase of adrenocorticotropic hormone, which was only partly refle
173 ere is an increase in adrenal sensitivity to adrenocorticotropic hormone, which we confirmed in our a
174 luding Rituximab, mycophenolate mofetil, and adrenocorticotropic hormone, with an emphasis on results
175 D4-P-selectin fusion proteins accumulated in adrenocorticotropic hormone-containing granules in D16/1
176 gmin I are diverted away from the endogenous adrenocorticotropic hormone-containing secretory granule
177 s, cortisol remains elevated and coordinated adrenocorticotropic hormone-cortisol pulsatility is main
178 ylated and silenced, although prolactin- and adrenocorticotropic hormone-producing tumors, which were
179 pression in GH3 lacto-somatotroph but not in adrenocorticotropic hormone-secreting AtT20 pituitary tu
180 /CYP11B2 selectivity resulted in blunting of adrenocorticotropic hormone-stimulated cortisol secretio
181 elial growth factor (VEGF) mRNA stability in adrenocorticotropic hormone-stimulated primary adrenocor
182     A minority of subjects developed blunted adrenocorticotropic hormone-stimulated release of cortis
183 es but lacks its potent inhibitory affect on adrenocorticotropic hormone-stimulated steroid synthesis
184  results in increased pituitary secretion of adrenocorticotropic hormone.
185 revents its proteolytic conversion to mature adrenocorticotropic hormone.
186 of cortisol in the face of "basal" levels of adrenocorticotropic hormone.
187 ss to increase anterior pituitary release of adrenocorticotropic hormone.
188 ed CORT following systemic administration of adrenocorticotropic hormone.
189 es corticotropin-releasing hormone (CRH) and adrenocorticotropic hormone.
190 s were analyzed for cytokines, cortisol, and adrenocorticotropic hormone.
191 of interleukin-6, cholesterol, cortisol, and adrenocorticotropic hormone.
192                           Short cosyntropin (adrenocorticotropic hormone; ACTH) stimulation tests.
193            We performed a 1-microg (low-dose adrenocorticotropic hormone; LD-ACTH) and 249 microg (hi
194 pe mice and in Y1 adrenal cells treated with adrenocorticotropic hormone; the latter effect is inhibi

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