ライフサイエンスコーパス: adult offspring

1 hippocampal Dio3 and Igf2 expressions in the adult offspring.

2 ssociated with a more atherogenic profile in adult offspring.

3 e contributes to the development of NAFLD in adult offspring.

4 rain and subsequent cognitive impairments in adult offspring.

5 d maternal undernutrition to the size of the adult offspring.

6 changes in gene expression in the brains of adult offspring.

7 opiate exposure blunts the fever response of adult offspring.

8 ened cardiovascular response to restraint in adult offspring.

9 ) and latent inhibition (LI) deficits in the adult offspring.

10 erability of ischemic injury in the heart of adult offspring.

11 ning and by a shortage of follow-up data for adult offspring.

12 mesenteric artery endothelial dysfunction in adult offspring.

13 -term effects on cardiovascular responses in adult offspring.

14 rmanently enhances memory performance of the adult offspring.

15 increased risk of nicotine dependence among adult offspring.

16 me of birth and risk of schizophrenia in the adult offspring.

17 their spouses (parents' generation) or their adult offspring.

18 10%) by prenatal exposure to cocaine only in adult offspring.

19 cular dysfunction in rat weanlings and young adult offspring.

20 birth weight and causes hyperglycemia in the adult offspring.

21 nd its relation to testicular dysfunction in adult offspring.

22 l thickness were measured in male and female adult offspring.

23 ne environment alters vascular reactivity in adult offspring.

24 uses hypertension and cardiac hypertrophy in adult offspring.

25 ring pregnancy leads to metabolic changes in adult offspring.

26 iation has rarely been investigated in young adult offspring.

27 ing diabetes, obesity and premature death in adult offspring.

28 fetal programming of vascular reactivity in adult offspring.

29 ring pregnancy promotes physical activity in adult offspring.

30 ight, and obesity and premature mortality in adult offspring.

31 rations in the fine motor performance of the adult offspring.

32 nd the efficiency of immune responses in the adult offspring.

33 otypes but to a greater extent in the Ts65Dn adult offspring.

34 e rats and in the brain of their neonate and adult offspring.

35 f selected BER-related genes in the brain of adult offspring.

36 d with cardiometabolic risk factors in young adult offspring.

37 se persisting abnormalities (programming) in adult offspring.

38 ain responses to the fear conditioned cue in adult offspring.

39 increases the risk for schizophrenia in the adult offspring.

40 ophysiological consequences in the hearts of adult offspring.

41 ents of hepatic and muscle Akt expression in adult offspring.

42 o PKCepsilon gene repression in the heart of adult offspring.

43 genotyped these two SNPs in 1679 CLHNS young adult offspring.

44 (2.5, 5, and 10 mg/kg i.p.) was assessed in adult offspring (70-90 days of age) using a rate-frequen

45 The study examined 197 adult offspring (95 male and 102 female) of alcoholic bi

46 tional weight gain (GWG) are associated with adult offspring adiposity.

47 Young adult offspring (age, 21 years [N = 433]) provided self-

48 3 to 84 years in 1987 through 1988 and their adult offspring aged 21 to 84 years in 2005 through 2008

49 middle-aged women and their female and male adult offspring (aged 18-23 y) and to examine the associ

50 is "interneuronopathy" persists in the young adult offspring and contributes to enduring changes in (

51 iatal DAT availability that were apparent in adult offspring and were associated with behavioral char

52 latively leading to long-term improvement in adult offspring; and (ii) maternal behavior can attenuat

53 f MgSO4 in clinical practice, its effects on adult offspring are not well known nor have sex-specific

54 ding were observed in the dorsal striatum of adult offspring as a consequence of germline THC exposur

55 ing of Holocaust survivors, thus identifying adult offspring as a possible high-risk group within whi

56 Parental prebirth, parental concurrent, and adult offspring assessments occurred in 1971-1983, 1998-

57 and p38 MAPK activity in left ventricular of adult offspring at resting state.

58 Adult offspring (both Ts65Dn and 2N) of choline-suppleme

59 caused low birth-weight and changes in young adult offspring brain, mimicking those in human neuropsy

60 induce insulin resistance and reduce IST in adult offspring, but such alterations are not attributab

61 ovascular and metabolic dysfunction in their adult offspring, but the intrauterine mechanisms involve

62 cient mice indeed reduced atherosclerosis in adult offspring by up to 56%, but the protective effect

63 of parental age with a suite of outcomes in adult offspring, comparing the results from an array of

64 Subjects were adult offspring derived from a 2 x 2 experiment in which

65 Subsequent genotyping of CLHNS young adult offspring detected an uncommon variant [minor alle

66 dy examined associations between parents and adult offspring diagnoses of the MS and its risk factors

67 Mother-adult offspring dietary resemblance in this Australian c

68 Adult offspring dyslipidemia is associated with maternal

69 In young adult offspring exposed to ethanol in utero, this effect

70 Furthermore, young adult offspring exposed to the 3 day binge-type ethanol

71 Analyses in the AA adolescent/young adult (offspring from COGA families) subsample indicated

72 Kidneys of the adult offspring had few structural or functional abnorma

73 Remarkably, Pl-OGT adult offspring had reduced body weights and elevated hy

74 mpus, with effects on memory that persist in adult offspring; higher choline intake is associated wit

75 At the time of testing of adult offspring, hormone status had returned to control

76 In hearts of both fetuses and adult offspring, hypoxia-induced methylation of SP1 site

77 , offspring viability) and higher numbers of adult offspring (i.e., productivity) than MOC females, s

78 bility to ischemia and reperfusion injury in adult offspring in rats.

79 of maternal cholestasis on the metabolism of adult offspring in the mouse.

80 Our previous brain imaging findings in adult offspring in these high-risk families also reveale

81 d with reduced Na(+),K(+)-ATPase activity in adult offspring kidney.

82 L-C levels explained 13% of the variation in adult offspring LDL-C levels beyond common genetic varia

83 Adult offspring LDL-C levels were associated with matern

84 Adult offspring LDL-C levels were examined as both a con

85 concurrent LDL-C levels in association with adult offspring LDL-C levels.

86 ity to ischaemia-reperfusion (I/R) injury in adult offspring male and female rats.

87 d sites in H9c2 cells, hearts of fetuses and adult offspring, methylation of both SP1 sites reduced S

88 vioral and cognitive characterization of the adult offspring (n = 12 per group), unbiased capture arr

89 sured in Holocaust survivors (n = 32), their adult offspring (n = 22), and demographically comparable

90 Adult offspring (n = 38) were imaged by positron emissio

91 allelic expression of Dio3 in the fetal and adult offspring of alcohol-consuming and control dams, w

92 ces cardiovascular and metabolic function in adult offspring of C57BL/6J mice in comparison with anim

93 Specifically, the adult offspring of choline-supplemented Ts65Dn dams perf

94 Participants were 114 adult offspring of depressed and nondepressed parents, f

95 major depression, assessed prospectively, in adult offspring of depressed probands who reported that

96 sistent changes were determined in F1 and F2 adult offspring of F0 mothers exposed to two relevant hu

97 aptive" responses in a rodent model in which adult offspring of fat-fed dams develop characteristics

98 l mGluR1-induced long-term depression in the adult offspring of high-LG compared with low-LG mothers,

99 imethylation of Grm1 in hippocampus from the adult offspring of high-LG compared with low-LG mothers.

100 nd other psychiatric diagnoses in a group of adult offspring of Holocaust survivors (N=100) and a dem

101 rinary cortisol excretion was measured in 35 adult offspring of Holocaust survivors and 15 healthy co

102 The data show that although adult offspring of Holocaust survivors did not experienc

103 stress disorder (PTSD) have been observed in adult offspring of Holocaust survivors in both glucocort

104 on cortisol negative feedback inhibition in adult offspring of Holocaust survivors with PTSD (N=13)

105 tamin C prevented these effects in fetal and adult offspring of hypoxic pregnancy.

106 Adult offspring of immune-challenged mothers displayed h

107 Adult offspring of LP0.5-exposed mothers exhibited gluco

108 We found that 2 weeks of ISO treatment in adult offspring of LPS-treated mothers led to augmented

109 To determine the effects in adult offspring of maternal exposure to stress and alcoh

110 uR1 mRNA and protein in hippocampus from the adult offspring of mothers showing an increased frequenc

111 AR-dependent long-term potentiation (LTP) in adult offspring of mothers that varied in the frequency

112 mere length in young (4 mo) and aged (15 mo) adult offspring of normoxic or hypoxic pregnancy with or

113 Studying adult offspring of pregnant mice subjected to PRS, we ex

114 itivity (euglycemic clamp) were performed in adult offspring of probands.

115 To examine this, adult offspring of rat dams either fed a control diet (C

116 Perfused livers of 48-h- starved adult offspring of rat dams fed 8% protein diets during

117 In the adult offspring of rats exposed to dexamethasone in late

118 We recently reported vascular dysfunction in adult offspring of rats fed a fat-rich (animal lard) die

119 ed whether abnormal vascular function in the adult offspring of rats fed a high saturated fat diet in

120 Adult offspring of rats that were given sufficient choli

121 Adult offspring of RES-treated mothers showed increased

122 ave examined vascular and renal structure in adult offspring of Sprague-Dawley rats fed a control die

123 or spirituality with major depression in the adult offspring of the original sample using a 10-year p

124 BOSS (2005-2008) is a study of aging in the adult offspring of the population-based Epidemiology of

125 Adult offspring of time-pregnant dams that were given a

126 her studies on the health and pregnancies of adult offspring of transplant patients are warranted.

127 We studied 206 adult offspring of women with gestational diabetes melli

128 Participants were 1086 mother-adult offspring pairs (59% female) from the New England

129 At 4 months, tissues from 1 male adult offspring per litter per group were either perfusi

130 ero and suckling periods in establishing the adult offspring phenotype in response to an environmenta

131 ellets on a progressive-ratio (PR) schedule, adult offspring prenatally exposed to cocaine were obser

132 ion has a profound effect on the behavior of adult offspring, probably via an effect of the maternal

133 long-lasting deleterious consequences in the adult offspring solely mediated by its ability to disrup

134 d heart susceptibility to ischemic injury in adult offspring, suggesting an in utero programming of P

135 damage responding to isoproterenol (ISO) in adult offspring that underwent maternal inflammation (mo

136 iation into distinct effector populations in adult offspring that were infected with influenza A viru

137 As adults, offspring that received low perinatal n-6/n-3 ra

138 y, and during 1 h of restraint stress in the adult offspring using indwelling arterial catheters impl

139 y for metabolic dysregulation and obesity in adult offspring via epigenetic modifications.

140 Adult offspring were assessed for sensory inhibition.

141 s, striatum and midbrain of prepubescent and adult offspring were determined by measuring: (1) the co

142 Adult offspring were evaluated for effects of placental

143 After foster rearing, adult offspring were exposed to the stressor and trained

144 percent egg-to-adult survival and number of adult offspring were higher for PMC than MMC females, sh

145 As adults, offspring were fed either a low-fat diet (LFD) o

146 success (measured as the number of returning adult offspring) when spawned in captivity compared with

147 contributes toward the NAFLD progression in adult offspring, which is mediated through impaired hepa

148 DNA damage levels in subcortical regions of adult offspring, which may increase sensitivity to oxida

149 Adult offspring with at least one Holocaust survivor par

150 ith hyperglycemia at age 39 years and in two adult offspring with impaired insulin secretion.

151 family functioning when there is a child or adult offspring with intellectual disability.

152 Females homozygous for tuh-1h always produce adult offspring with more bristles than females homozygo

153 des or both, was significantly higher in the adult offspring with parental CAD.

154 to enhance the probability that they produce adult offspring with rarer phenotypes with survival bene