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1 eather events that have been associated with adverse health outcomes.
2 nce problems, adverse drug events, and other adverse health outcomes.
3  and identifies a state of vulnerability for adverse health outcomes.
4 s an early warning system of risk for future adverse health outcomes.
5 ociated with cancer susceptibility and other adverse health outcomes.
6 ism; impaired methylation is associated with adverse health outcomes.
7 e of a healthy vaginal ecosystem, can reduce adverse health outcomes.
8 s a strong and independent predictor of such adverse health outcomes.
9  poverty), inability to obtain services, and adverse health outcomes.
10 nowledge of the links between overweight and adverse health outcomes.
11 ociated with functional measures or risk for adverse health outcomes.
12 -based multiorgan phenotypes associated with adverse health outcomes.
13 oxide (NO2) air pollution is associated with adverse health outcomes.
14 tencies; it is unclear how exposure leads to adverse health outcomes.
15 dwide but also are critical risk factors for adverse health outcomes.
16 ions that can lower SBPV and their impact on adverse health outcomes.
17          Insomnia is associated with several adverse health outcomes.
18 standards can potentially be used to predict adverse health outcomes.
19 irths are at substantially increased risk of adverse health outcomes.
20 itamin D deficiency has been associated with adverse health outcomes.
21  Working long hours has been associated with adverse health outcomes.
22 nnot be synthesized by humans, are linked to adverse health outcomes.
23  2.5 mum (PM2.5) in aerodynamic diameter and adverse health outcomes.
24 e action that may be relevant to a number of adverse health outcomes.
25 of biological systems and with it associated adverse health outcomes.
26 the aging population and are associated with adverse health outcomes.
27 s that climate change can be associated with adverse health outcomes.
28 aged social environments are associated with adverse health outcomes.
29 ium intake and high blood pressure and other adverse health outcomes.
30 CA) increases stress sensitivity and risk of adverse health outcomes.
31 affeine has been linked to both positive and adverse health outcomes.
32 ductive infections, preterm birth, and other adverse health outcomes.
33 ations was associated with a higher risk for adverse health outcomes.
34 ch use to patients may not outweigh costs or adverse health outcomes.
35 hat show the association between frailty and adverse health outcomes.
36 re known to be at risk for treatment-related adverse health outcomes.
37 senic (iAs) causes cancer and possibly other adverse health outcomes.
38 will reduce PM emissions but not necessarily adverse health outcomes.
39 he developing fetus and child from potential adverse health outcomes.
40  decrease the risk of hypertension and other adverse health outcomes.
41 ssing may place a child at a bigger risk for adverse health outcomes.
42 ve of BV status and correlates strongly with adverse health outcomes.
43 re, and is associated with increased risk of adverse health outcomes.
44 at lower concentrations) and a wide range of adverse health outcomes.
45 m of the vitamin in blood is associated with adverse health outcomes.
46  a high lifetime risk of a broad spectrum of adverse health outcomes.
47 gical studies on the association of BPA with adverse health outcomes.
48 nitive function are associated with multiple adverse health outcomes across the life course.
49 r interaction for predictors of positive and adverse health outcomes across the lifespan.
50 etween exposures and health to help mitigate adverse health outcomes across the lifespan.
51        Purpose To provide new information on adverse health outcomes (AHOs) in testicular cancer surv
52 s associated with immunologic impairment and adverse health outcomes among adults infected with human
53 ined associations between PFOA exposures and adverse health outcomes among residents in highly expose
54 phasizes the relationship between stigma and adverse health outcomes and health access measures.
55 nded follow-up evaluation increases risk for adverse health outcomes and medical or legal issues.
56  is a major public health problem leading to adverse health outcomes and neurodevelopmental abnormali
57  refer to older people at particular risk of adverse health outcomes and to organise care.
58 otential associations among TDCIPP exposure, adverse health outcomes, and DNA methylation status with
59 pollution has been implicated in a number of adverse health outcomes, and the effect of particulate m
60                                         Many adverse health outcomes are associated with obstructive
61 ween subclinical vitamin B-12 deficiency and adverse health outcomes as well as appropriate cutoffs f
62     Appreciating the quality and quantity of adverse health outcomes associated with CSA has helped c
63 enetic mechanisms in the pathogenesis of the adverse health outcomes associated with this important i
64  interesting mechanistic explanation of some adverse health outcomes associated with toxic cyanobacte
65 h a history of DMDD were more likely to have adverse health outcomes, be impoverished, have reported
66             Their likelihood of experiencing adverse health outcomes because of these behaviors may b
67        Edentulism is associated with various adverse health outcomes but treatment options in low- an
68 c beverages is hypothesized to contribute to adverse health outcomes, but the beverages and populatio
69            Sedentary behaviors are linked to adverse health outcomes, but the total amount of time sp
70 leep problems have been linked to a range of adverse health outcomes, but there is limited knowledge
71 how associations between sleep duration with adverse health outcomes, but these have not systematical
72 millions of Americans suffer from a range of adverse health outcomes due to noise exposure, including
73 d exposure to finer particles can thus cause adverse health outcomes during hazy days.
74 vorable multiorgan phenotype associated with adverse health outcomes, especially in elderly American
75 ated to significant short-term and long-term adverse health outcomes for both mothers and offspring.
76 ese factors may help prevent obesity-related adverse health outcomes for future generations.
77 mmon obstetric complications associated with adverse health outcomes for the mother and child.
78 ics and policy for understanding and abating adverse health outcomes from ambient particles.
79 ds to determine a) whether the occurrence of adverse health outcomes has changed, and b) the extent t
80 iations observed between passive smoking and adverse health outcomes have generated controversy.
81 nation are associated with increased risk of adverse health outcomes; however, it is unknown whether
82 ve long-lasting epigenetic marks, leading to adverse health outcomes in adulthood.
83 ential pesticide exposure has been linked to adverse health outcomes in adults and children.
84 ough body fat patterning has been related to adverse health outcomes in adults, its importance in chi
85  age and strongly associated with a range of adverse health outcomes in both women and men.
86  studies in Europe and California that found adverse health outcomes in children associated with mode
87  smoking is a major risk factor for multiple adverse health outcomes in children, but the underlying
88 ween major depression, early life stress and adverse health outcomes in diseases associated with infl
89 droxyvitamin D (25(OH)D) are associated with adverse health outcomes in diverse populations.
90 ure has been associated with a wide range of adverse health outcomes in limited epidemiologic studies
91  (SHS) has been associated with a variety of adverse health outcomes in nonsmokers, including emphyse
92 especially when sustained, leads to numerous adverse health outcomes in offspring.
93 al growth restriction and the programming of adverse health outcomes in the adult.
94 s consistently document a high prevalence of adverse health outcomes in this population, including HI
95 rial vaginosis (BV) may increase the risk of adverse health outcomes in women.
96 n people worldwide, resulting in substantial adverse health outcomes, including cancer.
97 as been found to be associated with numerous adverse health outcomes, including cancers.
98  air pollution and increased risk of various adverse health outcomes, including cardiopulmonary morta
99 ions between alcohol intake and a variety of adverse health outcomes, including coronary heart diseas
100 dently associated with an increased risk for adverse health outcomes, including impaired pulmonary fu
101  disorder (PTSD), and poses a major risk for adverse health outcomes, including neurodegeneration and
102   Chronic exposure to wood smoke can lead to adverse health outcomes, including respiratory infection
103 n disruptions are implicated in a variety of adverse health outcomes, including substance use disorde
104 hether accelerated epigenetic aging leads to adverse health outcomes later in life, these data point
105 ess than 2.5 microm; PM2.5) constituents and adverse health outcomes rather than focusing solely on t
106 nflammation, DSB, and emphysema and explains adverse health outcomes seen in smokers while implicatin
107  with complicated grief have greater risk of adverse health outcomes, should be diagnosed and assesse
108 nal association between monoHAA exposure and adverse health outcomes such as oxidative stress, inflam
109 ism by which tobacco exposure predisposes to adverse health outcomes, such as cancers, osteoporosis,
110 ith PM2.5 may appear more closely related to adverse health outcomes than other constituents even if
111 logy to estimate numbers of deaths and other adverse health outcomes that would have been avoided dur
112 e linked elevations in tropospheric ozone to adverse health outcomes, the effect of long-term exposur
113  bipolar disorder is associated with various adverse health outcomes, the relationship with violent c
114 ugh fast food consumption has been linked to adverse health outcomes, the relative contribution of fa
115 red effects cannot be directly translated to adverse health outcomes, this study demonstrates the app
116 ivors of childhood cancer, the prevalence of adverse health outcomes was high, and a systematic risk-
117 g clinical criteria, the crude prevalence of adverse health outcomes was highest for pulmonary (abnor
118 nges in rates and geographic distribution of adverse health outcomes were detected, and, in each inst
119 hat of oxidative stress, for associations of adverse health outcomes with PM2.5 mass and supports con
120 norganic arsenic is associated with numerous adverse health outcomes, with susceptibility differing b

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