ライフサイエンスコーパス: aerodigestive tract

1 promyelocytic leukemia (ie, not of the upper aerodigestive tract).

2 plasms that arise in the mucosa of the upper aerodigestive tract.

3 has developed a second primary tumor in the aerodigestive tract.

4 HN) is the most common neoplasm in the upper aerodigestive tract.

5 CYP1B1 in cell lines derived from the human aerodigestive tract.

6 he epithelium but also the mesenchyme of the aerodigestive tract.

7 of tobacco smoke-related malignancies of the aerodigestive tract.

8 neoplasm arising in the mucosa of the upper aerodigestive tract.

9 ma originally described to involve the upper aerodigestive tract.

10 ctivity in certain malignancies of the upper aerodigestive tract.

11 known to cause cancers of the lung and upper aerodigestive tract.

12 arly for carcinomas that derive not from the aerodigestive tract.

13 opharyngeal cancers and cancers of the upper aerodigestive tract.

14 over time throughout the mucosa of the upper aerodigestive tract.

15 ans developed a squamous cancer of the upper aerodigestive tract.

16 e presence of cancer cells shed in the upper aerodigestive tract.

17 ent of squamous cell carcinomas of the upper aerodigestive tract.

18 ons and a high risk to develop cancer of the aerodigestive tract.

19 iated with a relative's cancers in the upper aerodigestive tract (2.14) and lung (1.74).

20 We eliminated tap water exposure to the aerodigestive tract among high-risk patients, and the in

21 eart disease, COPD, and cancers of the upper aerodigestive tract and lung.

22 ed by ATRA via retinoid receptors in certain aerodigestive tract cancer cells, and its induction by A

23 onstrated activity in the chemoprevention of aerodigestive tract cancer.

24 Twenty-four patients with advanced aerodigestive tract cancers were enrolled; 79% had non-s

25 ssociated with decreased risk of a number of aerodigestive tract cancers, including esophageal cancer

26 has been shown to be a risk factor for upper aerodigestive tract cancers.

27 sed interventions focus on the prevention of aerodigestive tract colonization (avoidance of unnecessa

28 arises from the mucosal lining of the upper aerodigestive tract, demonstrates squamous differentiati

29 ng adenotonsillitis (3.29, 2.41-4.50); lower aerodigestive tract diseases, including asthma (2.69, 2.

30 Unique anesthetic complications and aerodigestive tract disorders have been identified.

31 his region that is expressed in normal upper aerodigestive tract epithelium.

32 netic basis for "field cancerization" of the aerodigestive tract has also remained obscure.

33 studies suggest that many SPTs in the upper aerodigestive tract have a common clonal origin, challen

34 Aerodigestive tract inflammatory injuries, such as decli

35 be managed conservatively; however, owing to aerodigestive tract injury and other associated injuries

36 the action of RA on the mucosa of the upper aerodigestive tract is mediated via downregulation of st

37 The upper aerodigestive tract is predisposed to the formation of m

38 the breast, endometrium, lung, kidney, upper aerodigestive tract, liver, and esophagus but not for pr

39 management of regional metastases from upper aerodigestive tract malignancies has shifted from the tr

40 Individuals with one aerodigestive tract malignancy have a high incidence of

41 clearly related to malignancies of the upper aerodigestive tract, may also increase risk of cancer at

42 Field cancerization predisposes the upper aerodigestive tract mucosa to the formation of multiple

43 The upper aerodigestive tract mucosal lining is often disrupted du

44 ter methylation in cells exfoliated from the aerodigestive tract of current and former smokers.

45 ty for gene promoter hypermethylation in the aerodigestive tract of smokers.

46 UGT1A10, extrahepatic enzymes present in the aerodigestive tract, preferentially formed both BPD-7R-G

47 sly expressed with highest expression in the aerodigestive tract, prostate, adrenal gland, and saliva

48 val, 1.10 to 1.91), and cancers of the upper aerodigestive tract (relative risk, 2.02; 95 percent con

49 (Ecad-lo) and high vimentin within the upper aerodigestive tract SCCs.

50 romosome arm 3p is a frequent event in upper aerodigestive tract squamous cell carcinoma (UADT SCC).

51 e associations were observed for lung, upper aerodigestive tract, stomach, and prostate cancer mortal

52 st peak observed, whereas in microsomes from aerodigestive tract tissues, peak 2 (both BPD-7R-Gluc an

53 iated with abnormal proximal exposure of the aerodigestive tract to refluxate; these patients respond

54 ulting from field-wide exposure of the upper aerodigestive tract to tobacco smoking.

55 CE-inhibitor-induced angioedema of the upper aerodigestive tract to treatment with 30 mg of subcutane

56 inoids are reported to reduce second primary aerodigestive tract tumors in patients with prior lung o

57 identified as an oncogene involved in upper aerodigestive tract (UADT) carcinomas.

58 human HNSCC tumors, but not in normal upper aerodigestive tract (UADT) epithelia.