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1   At admission, 25% (19/76) of children were afebrile.
2 e initially afebrile and 6 patients remained afebrile.
3 nd 16 (17%) and six (12%), if the patient is afebrile.
4 566 (34%) were febrile, and 1,125 (66%) were afebrile.
5 arum and 36 had P. vivax infections) and 162 afebrile adults (of whom 20 had P. falciparum and 20 had
6 hea, although 21% of patients were initially afebrile and 6 patients remained afebrile.
7 ymmetric, descending flaccid paralysis in an afebrile and alert patient without sensory findings.
8                                  He remained afebrile and asymptomatic.
9                            Adults were often afebrile and complained of shortness of breath, chills,
10                   Patients were grouped into afebrile and febrile groups, based on initial pulmonary
11 he start of therapy, 220 (30%) patients were afebrile and had normal white blood cell counts.
12 her had neutropenic fever, and the third was afebrile and non-neutropenic at the time of presentation
13             On clinical examination, she was afebrile and tachypneic.
14 ve therapy (80% v 53%); and (3) treatment of afebrile and uncomplicated febrile neutropenia (30% v 60
15 st likely to reduce CSF use for treatment of afebrile and uncomplicated febrile neutropenia.
16                                       He was afebrile, and the physical examination was notable for m
17                              The patient was afebrile at admission.
18 s and Treg cells using flow cytometry in 168 afebrile children (of whom 15 had P. falciparum and 36 h
19 e illnesses other than KD to those of normal afebrile children and to KD patients.
20 om both virus-negative afebrile controls and afebrile children with the same viruses present in the f
21 ve febrile children from both virus-negative afebrile controls and afebrile children with the same vi
22 ion or with acute bacterial infection and 22 afebrile controls.
23 NP specimens of febrile patients compared to afebrile controls.
24 P) specimens of febrile patients compared to afebrile controls.
25 y considered only admissions for febrile and afebrile convulsions.
26 ts with PKC also have a history of infantile afebrile convulsions.
27 for emergency treatment of acute febrile and afebrile (epileptic) seizures in children.
28                               Almost half of afebrile episodes (47.6% [204/429]) were RDT positive.
29      Fifteen affected members presented with afebrile focal or generalized tonic-clonic seizures duri
30                    Subjects were tested when afebrile for (i) psychophysical loudness adaptation to c
31  100 mg per kilogram per day until they were afebrile for 48 hours and 3 to 5 mg per kilogram per day
32          However, post-infectious refractory afebrile form of seizures in previously healthy children
33 exclude febrile HCWs from working, but allow afebrile HCWs with respiratory symptoms to have contact
34                                              Afebrile healthy infants served as controls.
35 nd 190 without bacterial infections), and 19 afebrile healthy infants.
36    Clinical bias was greatest in febrile vs. afebrile intensive care unit patients.
37       Geometric mean iron absorptions in the afebrile malaria and hookworm groups were 12.9% and 32.2
38 zation ranged between 79.1% and 88.0% in the afebrile malaria and hookworm groups with no significant
39                                 In contrast, afebrile malaria causes inflammation, increases hepcidin
40                                 Treatment of afebrile malaria reduced inflammation (P < 0.001) and se
41 tion pre- and posttreatment in children with afebrile malaria, hookworm, and Schistosoma haematobium
42 v 4.6%); and (3) secondary prophylaxis after afebrile neutropenia following chemotherapy for germ cel
43                                              Afebrile neutropenia is rarely followed by CSF initiatio
44 of either established febrile neutropenia or afebrile neutropenia remains uncertain.
45 d: development of fever and/or infections in afebrile neutropenic outpatients and recovery without co
46              Secondary outcomes included: in afebrile neutropenic outpatients, infection-related mort
47 ividuals presented with febrile and multiple afebrile, often focal seizure types, multifocal epilepti
48 ulocyte colony-stimulating factor (G-CSF) in afebrile outpatients with severe chemotherapy-induced ne
49 TTMV was equally present in both febrile and afebrile patient specimens.
50  found in similar percentages of febrile and afebrile patient specimens.
51                                              Afebrile patients had lower rates of antibiotic administ
52 es F documented in the emergency department; afebrile patients lacked both.
53 gency department patients with septic shock, afebrile patients received lower rates of emergency depa
54  Routine therapeutic application of G-CSF in afebrile patients with severe neutropenia can reduce the
55 ge number of specimens from both febrile and afebrile patients, they were more prevalent in the plasm
56  time of high bacteremia that alternate with afebrile periods of relative well being during low bacte
57      Nearly half of HCWs with influenza were afebrile prior to their diagnosis.
58                                           In afebrile rats, alpha-MSH infusion caused a modest transi
59                          In both febrile and afebrile rats, CCK-8 induced dose-dependent skin vasodil
60 n combination, affected body temperatures in afebrile rats.
61 emergency department IV fluids volume, being afebrile remained a significant predictor of in-hospital
62                     Like the distribution of afebrile seizure duration in children, the distribution
63 followed by treatment-refractory febrile and afebrile seizures and psychomotor decline.
64                  Eight individuals developed afebrile seizures between ages 5 and 13 years.
65                                              Afebrile seizures consisted of generalized tonic-clonic,
66                                Patients with afebrile status epilepticus had a variety of imaging abn
67 illnesses are larger than those in normative afebrile subjects but smaller than dimensions in patient
68 nces in clinical characteristics (history of afebrile v febrile neutropenia), drug characteristics (G
69   They are the principal etiologic agents of afebrile viral upper-respiratory-tract infections (the c
70 ation required the patient with asthma to be afebrile with normal chest x-ray and white blood cell co
71 ive clusters, 59.7% were febrile, 20.2% were afebrile with other symptoms, and 20.2% were asymptomati
72 clinical and radiologic improvement and were afebrile within 24 hrs after drainage.

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