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1 ricted to the juxtaglomerular portion of the afferent arteriole.
2 (JG) cells, located in the pole of the renal afferent arterioles.
3 d granulated cells located in the glomerular afferent arterioles.
4 d not induce significant vasoconstriction of afferent arterioles.
5 st pronounced in the most distal part of the afferent arterioles.
6 in preglomerular microvessels, predominantly afferent arterioles.
7 arcuate arteries and to the beginning of the afferent arterioles.
8 reased the diameters of the interlobular and afferent arterioles.
9 creases in diameters of the interlobular and afferent arterioles.
10 ing (juxtaglomerular) cells in the mammalian afferent arterioles.
11 n by transducing TGF-mediated signals to the afferent arteriole, a function that is independent of ni
14 mpairs acetylcholine-induced vasodilation of afferent arterioles (Aff) in AngII-induced hypertension
15 O2-) mediates enhanced contractions of renal afferent arterioles (Aff) of angiotensin II (Ang II)-inf
16 n constitutive accumulation of HIF-2alpha in afferent arterioles and glomerular cells and HIF-1alpha
17 e the recruitment of immunoreactive renin in afferent arterioles and in the juxtaglomerular apparatus
18 e smooth muscle cells, freshly isolated from afferent arterioles and interlobular arteries averaging
19 ed glomerular filtration rate, hyalinosis of afferent arterioles, and striped cortical tubulo-interst
20 effect on diameters of the interlobular and afferent arterioles at concentrations up to 1 microM.
21 st, 5,6-EET constricted the interlobular and afferent arterioles by 16 +/- 3% (N = 6) and 21 +/- 3% (
22 reased the diameters of the interlobular and afferent arterioles by 18 +/- 2% (N = 10) and 20 +/- 3%
23 preferentially vasoconstricts the glomerular afferent arterioles by depleting endothelial nitric oxid
24 wild-type littermate controls, an attenuated afferent arteriole constrictor response to endothelin-1
25 dministration of 10 muM S1P, the diameter of afferent arterioles decreased to 35%+/-5% of the control
27 +]i responses to 40 mM K+ were suppressed in afferent arterioles from diabetic rats (delta = 63+/-5 n
30 (3 to 300 microM) had virtually no effect on afferent arterioles from sham rats; however, this K(ATP)
32 e of the BSC2 protein at the juxtaglomerular afferent arteriole, in a juxtaglomerular structure proba
33 I-ANG II binding studies on freshly isolated afferent arterioles indicated that ANG II receptor densi
34 at the constriction response to adenosine in afferent arterioles is mediated by A1AR coupled to a PTX
36 In contrast with LacZ-positive cells in the afferent arterioles, LacZ-positive cells in the glomerul
38 -type numbers of renin-producing cells along afferent arterioles of the glomeruli rather than by up-r
39 damide (1 microM) vasodilates juxtamedullary afferent arterioles perfused in vitro; the vasodilation
43 alpha1-resistant/alpha2-resistant mice, and afferent arteriole responsiveness again was confirmed by
45 he functional connection between tubules and afferent arterioles (so-called tubuloglomerular feedback
46 Adenosine induces vasoconstriction of renal afferent arterioles through activation of A1 adenosine r
47 ubule perfusate confirmed the ability of the afferent arteriole to contract in the presence of ouabai
50 highly expressed near the glomerulus, in the afferent arteriole, where it may also dilate renal arter
51 s the glomerular filtration rate by dilating afferent arterioles while constricting efferent arteriol
52 ype 1B (AT(1B)) subtypes in freshly isolated afferent arterioles, while there was very little AT2 rec
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