ライフサイエンスコーパス: afferent fiber

1 chanism (e.g., inhibition of exocytosis from afferent fibers).

2 tonin gene-related peptide (CGRP) (marker of afferent fibers).

3 ted predominately by activation of group III afferent fibers).

4 s provides the entire input to each cochlear afferent fiber.

5 from their ribbon synapses onto the auditory afferent fiber.

6 ibutes by observing the activity of a single afferent fiber.

7 inhibits exocytosis from pulpal peptidergic afferent fibers.

8 nsed by vagal, spinal, and intrinsic primary afferent fibers.

9 rrents elicited by electrical stimulation of afferent fibers.

10 ly located near central terminals of primary afferent fibers.

11 rization-induced suppression of excitation") afferent fibers.

12 /7-immunopositive terminals are from primary afferent fibers.

13 90 min of the study to block parasympathetic afferent fibers.

14 iors, suggesting that ET-1 activates primary afferent fibers.

15 ity of the branches were produced by type II afferent fibers.

16 d after electrical stimulation of peripheral afferent fibers.

17 eatic secretion via intestinal vagal mucosal afferent fibers.

18 of capsaicin-sensitive, nociceptive primary afferent fibers.

19 e)- and small (nociceptive)-diameter primary afferent fibers.

20 til about birth, a time after the arrival of afferent fibers.

21 for the effects of L-ARG on the RA of crista afferent fibers.

22 l fusiform cells, granule cells, and primary afferent fibers.

23 geneous population of small diameter primary afferent fibers.

24 single unit activity of 54% of hepatic vagal afferent fibers.

25 presentations of fat USs may depend on vagal afferent fibers.

26 ts ability to trigger phase-locked spikes in afferent fibers.

27 nsitive, substance P (SP)-containing primary afferent fibers.

28 lves CCK-A receptors and capsaicin-sensitive afferent fibers.

29 g in the terminals of small-diameter primary afferent fibers.

30 ic changes within the cells of origin of the afferent fibers.

31 s) did not differ between the two groups of afferent fibers.

32 nent of the response to burst stimulation of afferent fibers.

33 nals are mainly endings of thinly myelinated afferent fibers.

34 ase staining, and the pattern of cholinergic afferent fibers.

35 round vesicles, formed synapses with radial afferent fibers.

36 ne of the three proteins were present in all afferent fibers.

37 ors associated with the membranes of primary afferent fibers.

38 tral terminal projections of abdominal vagal afferent fibers.

39 CR immunoreactivity of cell populations and afferent fibers.

40 t pruritogens activate the same or different afferent fibers.

41 ically through the recruitment of myelinated afferent fibers.

42 luconeogenesis to the brain depends on vagal afferent fibers.

43 dial to the solitary tract contained labeled afferent fibers.

44 li is not a common property of gastric vagal afferent fibers.

45 tivates P2X2 and P2X3 receptors on gustatory afferent fibers.

46 r neurons, receiving direct input from vagal afferent fibers.

47 lectrical stimulation of ipsilateral primary afferent fibers.

48 channel expressed on a subset of peripheral afferent fibers.

49 the site of termination of the chemosensory afferent fibers.

50 yers of termination of the primary gustatory afferent fibers.

51 orsal horn-the target zone of many cutaneous afferent fibers.

52 al cord dorsal horn by A delta and C primary afferent fibers.

53 ts, and (4) excitation of intestinal sensory afferent fibers.

54 formation arriving from Adelta and C primary afferent fibers.

55 0-kD neurofilament-immunopositive myelinated afferent fibers, a subpopulation of which did not expres

56 l capsaicin-treated animals (NNCAP, group IV afferent fibers ablated).

57 y measure dendritic and somatic responses to afferent fiber activation in two different populations o

58 Auditory afferent fiber activity is driven by high-fidelity infor

59 with a piezoelectric device while recording afferent fiber activity.

60 This ATP excites primary sensory afferent fibers and also stimulates neighboring taste bu

61 dose inducing functional ablation of sensory afferent fibers and by devazepide, a CCK-A receptor anta

62 synapses, including those formed by sensory afferent fibers and by intrinsic interneurons.

63 f mGlu1alpha receptors on peripheral primary afferent fibers and determine the behavioral effects of

64 lized spine-like contacts between individual afferent fibers and hair cells that are surrounded by la

65 en functional properties of mechanoreceptive afferent fibers and intrinsic discharge properties of la

66 BS may activate 5-HT3 receptors on extrinsic afferent fibers and motor neurons, giving rise to viscer

67 DA receptors have been demonstrated on vagal afferent fibers and on second-order neurons in the media

68 eceptors have been found on both the sensory afferent fibers and on the GCs.

69 gthening of the polysynaptic pathway between afferent fibers and pattern-initiating neurons of the bu

70 e peripheral auditory pathway with misrouted afferent fibers and reduced synaptic contacts with hair

71 receive primary inputs from vibrissa sensory afferent fibers and send monosynaptic connections to fac

72 ological data revealed that numbers of vagal afferent fibers and synapses in the NTS were significant

73 Plastic changes in vagal afferent fibers and synapses were investigated at the mo

74 The distribution of gravity-sensing, otolith afferent fibers and terminals was studied in the vestibu

75 tudy to identify the type(s) of VR1-positive afferent fibers and terminals.

76 ators of sensory information between primary afferent fibers and the spinal cord, activate Erk in dor

77 likely attributable to damage to myelinated afferent fibers and their abnormal sprouting in lamina I

78 Afferent fibers and their terminal distributions within

79 on at superficial lamina IA among horizontal afferent fibers and unique axonal targeting pattern sugg

80 ses suggested normal function of hair cells, afferent fibers, and efferent feedback.

81 cells, on peripheral nerves, on fine sensory afferent fibers, and on brain endothelial cells, respect

82 udy found that both VIP and PACAP containing afferent fibers are abundant in the BSTLd (dorsolateral

83 Subpopulations of these afferent fibers are activated by capsaicin.

84 Our results suggest that large afferent fibers are critical for the timing of automatic

85 and glutamatergic responses in CVNs, C-type afferent fibers are critical to the afferent stimulation

86 pite the normal presence of Bdnf transcript, afferent fibers are disoriented near the organ of Corti.

87 ggesting that both hair cells and vestibular afferent fibers are normally recruited by GVS.

88 s, then the LOC terminals synapsing with IHC afferent fibers are not particularly rich in these pepti

89 tric fish Eigenmannia, P- and T-type primary afferent fibers are specialized for encoding the amplitu

90 Additionally, evoked EPSCs in afferent fibers are unaffected by glutamate transporter

91 In addition, efferent axons that use the afferent fibers as a scaffold during pathfinding also sh

92 ional terminals, presumed to be from primary afferent fibers, at the center of glomerular arrangement

93 of CCKA receptors in the terminals of vagal afferent fibers because these receptors have been implic

94 In control mice, the labeled afferent fibers belong to fine, beaded axons diffusely d

95 a severe reduction in axonal arborization in afferent fibers, but no deficit in neurogenesis.

96 terminals are mainly endings of unmyelinated afferent fibers; C2 terminals are mainly endings of thin

97 rimary sensory circuits from locus coeruleus afferent fibers can produce a spectrum of modulatory act

98 eding, (2) capsaicin-sensitive hepatic vagal afferent fibers carry the signal that stimulates feeding

99 istent with the notion that group IV phrenic afferent fibers comprise the afferent arm of a fatigue-i

100 anistic insight into the subtypes of primary afferent fibers damaged by chemotherapy.

101 tment groups before histological analysis of afferent fiber density at the injection sites.

102 The activity of auditory afferent fibers depends strongly on the frequency of sti

103 by application of capsaicin, which destroys afferent fibers, directly to the cervical vagi.

104 or prolonged stimuli, like tetanic bursts of afferent fiber discharge at high frequencies.

105 tivity, group IV, but not group III, phrenic afferent fibers discharged more (p<0.05) during rhythmic

106 We found that high-frequency stimulation of afferent fibers elicits synaptic currents mediated by al

107 cell, voltage-clamp recordings from IHCs and afferent fiber endings in excised postnatal rat cochleae

108 ents rather than in the terminals of primary afferent fibers, even though labeling overlapped with th

109 Furthermore, LES rat afferent fiber-evoked APs showed a pronounced loss of te

110 The central terminals of primary afferent fibers experience depolarization upon activatio

111 To test whether primary gustatory afferent fibers express Ca(2+)-permeable AMPA/kainate re

112 th duodenal input transmitted by sympathetic afferent fibers expressing TRPV1; spinal neuronal respon

113 tion when activated by a small population of afferent fibers firing asynchronously at physiologically

114 of motoneurons, interneurons, and myelinated afferent fibers for antagonistic ankle muscles.

115 and internodal distance were quantified for afferent fibers from eight sites within the vestibular n

116 rainstem structure that receives the primary afferent fibers from electroreceptors in the skin.

117 ing the presynaptic terminals of cholinergic afferent fibers from projection neurons (Pn).

118 clei receive axosomatic endings from primary afferent fibers from the cochlea and have projections th

119 Sympathetic afferent fibers from the heart enter the upper thoracic

120 , layer Ia, which is the unique recipient of afferent fibers from the olfactory bulb.

121 s, which has resulted from the re-routing of afferent fibers from the third spinal nerve into the hyp

122 All afferent fibers had characteristic frequencies of 16-32

123 Hepatic vagal afferent fibers have been implicated in the feeding resp

124 mination region for unmyelinated (C) primary afferent fibers; however, how the input it receives from

125 Electrophysiological recordings from primary afferent fibers in control and hyperalgesic mice with tu

126 s autoreceptors in terminals of Adelta and C afferent fibers in laminae I-III, presynaptic IGRs may p

127 , cytoarchitecture, and projection fields of afferent fibers in tandem revealed input target selectiv

128 rotid sinus and anodal block of baroreceptor afferent fibers in the carotid sinus nerve to examine th

129 ion of contraction-sensitive skeletal muscle afferent fibers in the cat; and (ii) synaptic input from

130 her naturalistic activation of barosensitive afferent fibers in the glossopharygneal and vagus nerves

131 itu hybridization to determine if neurons or afferent fibers in the hindbrain possess both TRH type 1

132 nsistent with our previous report, medullary afferent fibers in the solitary tract and spinal trigemi

133 are coexpressed on the terminals of primary afferent fibers in the spinal cord where they may mediat

134 low-frequency stimulation (5 Hz) of primary afferent fibers in the tractus solitarius resulted in a

135 The central projections of primary afferent fibers in the utricular nerve, which convey lin

136 egister with the major terminal fields of CT afferent fibers, in the central third of the rostral 1.0

137 c conditions, noxious stimulation of primary afferent fibers induces release of CatS from microglia,

138 e monosynaptic input from trigeminal primary afferent fibers innervating extracranial orofacial struc

139 cts of opioid receptor-selective agonists on afferent fibers innervating the colon.

140 Discrete tracing of afferent fibers innervating the individual semicircular

141 extrinsic NOS-IR nerve fibers represented an afferent fiber input that was separate from the substanc

142 synaptic EPSP, and EPSP in cells receiving C-afferent fiber input.

143 eurons, but also to regulate local growth of afferent fibers into target tissues and, in some cells,

144 nd behavior coincided with the projection of afferent fibers into the central vestibular nuclei, indi

145 on of NMDA responses to burst stimulation of afferent fibers is described, which required GABAA block

146 hat the modulatory function of noradrenergic afferent fibers is exerted through paracrine interaction

147 nerve growth factor (NGF)-responsive primary afferent fibers is thought to contribute to their hypera

148 During paired recordings of hair cells and afferent fibers, L-type voltage-gated Ca(2+) currents sh

149 Shock stimulation of afferent fibers (lateral olfactory tract) and associatio

150 Based on their anatomic localization, these afferent fibers may function to transmit important senso

151 at receptor-Ca2+ channel coupling in primary afferent fibers may have little functional significance

152 response to colorectal distension, and colon afferent fiber mechanosensitivity were assessed in contr

153 induce long-lasting depression of A primary afferent fibers-mediated monosynaptic excitatory postsyn

154 preparation, cats were anesthetized and the afferent fibers mediating the pressor reflex entered the

155 Examination of over 400 isolated afferent fibers, more than 200 from each species, indica

156 Sublamination requires the activity of the afferent fibers, NMDA receptors, and nitric oxide syntha

157 Synaptic currents in afferent fibers occurred with high failure rates of appr

158 erent fibers within the dorsal root ganglia, afferent fibers of the dorsal root, dorsal root entry zo

159 The distribution and synaptology of the afferent fibers of the glossopharyngeal nerve (IXN) in t

160 to identify putative synaptic contacts from afferent fibers of the two nerves onto individual projec

161 Gustatory afferent fibers of the vagus nerve that innervate taste

162 to determine the effect of activating vagal afferent fibers on the discharge of median preoptic (MnP

163 We conclude that group III and IV afferent fibers originating from the skeletal muscle of

164 dpuppy, PACAP is found primarily in visceral afferent fibers, originating from cells in either the do

165 Trigeminal, spinal and vagal afferent fibers overlap in C1-C2 segments.

166 ransmission from sympathetic and peptidergic afferent fibers participates in the regulation of pulpal

167 hat selective CST injury induces maladaptive afferent fiber plasticity remote from the lesion.

168 mply that, in conjunction with local primary afferent fiber plasticity, injury-induced sprouting of D

169 tio-temporal patterning of cutaneous sensory afferent fiber projections to the dorsal, but not the ve

170 Afferent fibers regrew through the transection site with

171 no effect; in that situation, the RA of the afferent fibers remained and the preparation still respo

172 r combined; in that situation, the RA of the afferent fibers remained.

173 ive colonic neurons, consistent with reduced afferent fiber responses to UTP and ADP in Na(v)1.9(-/-)

174 In addition, whereas stretch-evoked afferent fiber responses were enhanced by chemical infla

175 ith a significant and selective reduction in afferent fiber sensitivity to circumferential stretch of

176 Of interest, 10% of the trigeminal primary afferent fibers showed glycine localization.

177 Using single TS afferent fiber stimulation in slices from control and CI

178 synaptic current elicited in mPFC neurons by afferent fiber stimulation shows linear correlation with

179 secreting the enzyme, as a result of primary afferent fiber stimulation.

180 sed the synaptic transmission in response to afferent fibers stimulation.

181 activity generally associated with 8th nerve afferent fibers, strongly suggest that GABA serves an im

182 odel, suggest sustained release at hair cell afferent fiber synapse is dictated by Ca(2+)-dependent v

183 ling statistical properties of the hair cell-afferent fiber synapse suggested that the ability of one

184 Cardiac vagal afferent fibers synapse in the nucleus tractus solitariu

185 ontaining glomus cells (GCs) and the sensory afferent fibers synapsing onto the GCs.

186 mpal GABA cells with extrinsic and intrinsic afferent fiber systems.

187 opy results, imply that unmyelinated primary afferent fibers terminating in the superficial dorsal ho

188 Along this row the number of afferent fibers terminating on IHCs increases toward the

189 inea pig intrinsic cardiac ganglia are vagal afferent fibers that also contain NOS.

190 use sprouting of nociceptive pelvic visceral afferent fibers that correlates with increased hypertens

191 rried by action potentials of S-type primary afferent fibers that project to the inner cellular layer

192 selectively taken up by unmyelinated primary afferent fibers that terminate in the outer part of lami

193 The vagus nerve contains afferent fibers that transmit sensory information from t

194 ion was afferent in origin, we traced apical afferent fibers that were retrogradely labeled by extrac

195 The major class of cochlear afferent fibers, the type-I or radial-fiber (RF) populat

196 ve ivy cells and SM-expressing O-LM cells to afferent fiber theta burst stimulation.

197 142,801 decreased responses of pelvic nerve afferent fibers to colorectal distention.

198 that relays sensory information from primary afferent fibers to higher order cells of ELLS.

199 vated 5-HT(3) and 5-HT(2) receptors on vagal afferent fibers to mediate luminal factor-stimulated pan

200 ent duodenal factors stimulate vagal mucosal afferent fibers to mediate pancreatic enzyme secretion v

201 tion to the central nervous system via vagal afferent fibers to mediate pancreatic secretion.

202 endently attenuate responses of pelvic nerve afferent fibers to noxious colonic distension was unaffe

203 eculate that they may be branches of type II afferent fibers to outer hair cells and that a smaller p

204 tterns of peptidergic small-diameter primary afferent fibers to the cat sacrocaudal spinal cord, a re

205 e information is transmitted by baroreceptor afferent fibers to the central network by glutamate bind

206 tter from glomus cells activates the sensory afferent fibers to transmit information to the nucleus o

207 gustatory receptor cells and primary sensory afferent fibers transmit the output signal from taste bu

208 at climbing fibers and some other cerebellar afferent fibers transported the virus retrogradely from

209 inal common pathway through which convergent afferent fibers using a variety of neurotransmitter agen

210 topic array of hair cells and transmitted to afferent fibers via fast, repetitive synaptic transmissi

211 t group III (n=7) and group IV (n=8) phrenic afferent fibers was recorded during rhythmic diaphragmat

212 elated peptide (CGRP)-immunoreactive primary afferent fibers was selectively altered at specific spin

213 from presynaptic hair cells and postsynaptic afferent fibers, we established that the rate of evoked

214 understand the effects of capsaicin on adult afferent fibers, we examined DRG neurons retrogradely la

215 ordings from adult frog hair cells and their afferent fibers, we show here that during sine-wave stim

216 ctrophysiological recordings of pelvic nerve afferent fibers, we studied drug effects on responses to

217 Group IV afferent fibers were ablated in neonatal Sprague-Dawley

218 ponses and neuronal activity in unmyelinated afferent fibers were assessed in monkey after topical ap

219 Responses of rat jejunal afferent fibers were examined by electrophysiology.

220 CAPs of the large and the small myelinated afferent fibers were observed in response to SCS at all

221 In contrast, myelinated afferent fibers were observed to be sprouting into lamin

222 tide (CGRP)-immunoreactive (CGRP-IR) primary afferent fibers were observed within the superficial lam

223 Labeled afferent fibers were of two types: type I fibers contact

224 Mechanosensitive pelvic nerve afferent fibers were recorded from the decentralized S1

225 nal evidence suggests that CCK acts on vagal afferent fibers, which may explain how CCK doses that pr

226 hanisms to maximize the dynamic range of its afferent fibers, which operate at the physiological limi

227 e activated by different distinct subsets of afferent fibers with minimal overlap.

228 Rats denervated of primary afferent fibers with neonatal capsaicin injection and an

229 to correlate the influence of sensory vagal afferent fibers with the functional organization of mGlu

230 ceived direct ST inputs from only one or two afferent fibers, with one-half also receiving smaller am

231 r) was constitutively expressed on cells and afferent fibers within the dorsal root ganglia, afferent

232 Here, we describe that stimulation of GLP-1 afferent fibers within the PVN is sufficient to suppress