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1 ohol and tobacco consumption and exposure to aflatoxin B1.
2 sitive foci) in the liver of rats exposed to aflatoxin B1.
3 highly related to hepatitis B infection and aflatoxin B1.
4 HCCs from areas of high dietary exposure to aflatoxin B1.
5 n with the hepatitis viruses and exposure to aflatoxin B1.
6 iolet (UV) light to fungal metabolites, like Aflatoxin B1.
7 cryostat liver sections of rats treated with aflatoxin B1.
8 an important step in the carcinogenicity of aflatoxin B1.
9 robisfuran is key to the mutagenic nature of aflatoxin B1.
11 were positive for the following mycotoxins: aflatoxin B1 (50 mug/kg), alternariol monomethyl ether (
12 diol epoxide, N-hydroxy-2-aminofluorene, and aflatoxin B1 8,9-epoxide in (1) naked intact genomic DNA
13 diol epoxide, benzo(g)chrysene diol epoxide, aflatoxin B1 8,9-epoxide, and N-acetoxy-2-acetylaminoflu
16 nsist of a unique abasic site or ring-opened aflatoxin B1 adduct but rather is consistent with the pr
18 considered for human use to protect against aflatoxin B1 (AFB)-induced hepatocarcinogenesis based on
19 , 8, 9-dihydro-8-(S-glutathionyl)-9-hydroxyl-aflatoxin B1 (AFB-GSH), was found to bind with a stoichi
21 ection of the mycotoxins ochratoxin A (OTA), aflatoxin B1 (AFB1) and deoxynivalenol (DON) which are s
22 as these toxins, such as ochratoxin A (OTA), aflatoxin B1 (AFB1) and deoxynivalenol (DON), are subjec
23 ution with only the specific toxin, which is aflatoxin B1 (AfB1) and mixture of AfB1 with other non-s
24 p reduced the number of revertants caused by aflatoxin B1 (AFB1) and proliferation of cells M12.C3.F6
29 HCCs occur in geographical regions with high aflatoxin B1 (AFB1) exposure, concomitant with hepatitis
30 There is a prompt need for determination of aflatoxin B1 (AFB1) in food products to avoid distributi
34 , exposure to the genotoxic hepatocarcinogen aflatoxin B1 (AFB1) is a significant factor in the genes
37 r for determination and quantification of an aflatoxin B1 (AFB1) level using a reduced graphene oxide
39 is initiated through metabolic activation of aflatoxin B1 (AFB1) to its epoxide form that reacts with
40 rochemical aptasensor for trace detection of aflatoxin B1 (AFB1) was developed by using an aptamer as
42 isk of liver cancer upon exposure to dietary aflatoxin B1 (AFB1), a carcinogenic product of the mold
45 5% of NNK-induced, 59% of VC-induced, 58% of aflatoxin B1 (AFB1)-induced, 14% of N-ethyl-N-nitrosoure
46 ue has been developed for the preparation of aflatoxin B1 (AFB1)-tagged liposomes encapsulating a vis
49 certain mycotoxins, such as deoxynivalenol, aflatoxin B1, aflatoxin B2, aflatoxin G1, aflatoxin G2,
52 tify 14 fungus secondary metabolites, namely aflatoxin B1, aflatoxin G1, aspergillic acid, aspyrone,
53 ylaminofluorene, benzidine, 2-naphthylamine, aflatoxin B1, aflatoxin G1, sterigmatocystin, N-nitrosod
54 , sodium azide, mitomycin C, benzo[a]pyrene, aflatoxin B1 and 2-aminofluorene, were compared with the
56 plied to cultured cells treated with H2O2 or aflatoxin B1 and then to cryostat liver sections of rats
59 e between 0.05mugL(-1) (for aflatoxin G1 and aflatoxin B1) and 15mugL(-1) (for deoxynivalenol and fum
62 hratoxin A and aflatoxin M1 (a metabolite of aflatoxin B1), as well as other aflatoxins, under compet
66 etection method for the determination of the aflatoxin B1, B2, G1 and G2 in peanuts, rice and chilli
68 ed), were investigated for their contents of aflatoxins (B1, B2, G1 and G2), patulin, and ergosterol.
70 arker - ergosterol and important mycotoxins (aflatoxins B1, B2, G1 and G2, and ochratoxin A) were als
73 s measured in two populations: (a) placental aflatoxin B1 DNA (AFB1-DNA) adducts in a group of Taiwan
75 has been applied to label-free detection of aflatoxin B1 in a competitive immunoassay format, with t
78 inogenic mycotoxin and secondary metabolite, aflatoxin B1 in the filamentous fungus and an important
81 hesis of the potent environmental carcinogen aflatoxin B1 involves ca. 15 steps beyond the first poly
83 ihydrobisfuran ring system characteristic of aflatoxin B1 is essential to the covalent reaction of it
84 nigmatic step in the complex biosynthesis of aflatoxin B1 is the oxidative rearrangement of versicolo
85 biosynthesis of the environmental carcinogen aflatoxin B1, is one of the multidomain iterative polyke
87 ]anthracene, benzo[a]pyrene-7,8-dihydrodiol, aflatoxin B1, naphthalene, and styrene, with high turnov
89 zone containing immobilized antibodies; then aflatoxin B1-tagged, dye-containing liposomes are allowe
91 observed between Cr and total Aflatoxin and Aflatoxin B1; whereas Ochratoxin A was related to Cu and
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