ライフサイエンスコーパス: after birth

1 were performed on variable dates (10-29 days after birth).

2 ygous mice than in wild-type mice at 21 days after birth.

3 cular-stage lung morphogenesis and died soon after birth.

4 ubset of infants (n = 307) in the first 14 d after birth.

5 nsible for DMD and other monogenic disorders after birth.

6 depletion of BRAG2 during the first 2 weeks after birth.

7 that are usually diagnosed at or immediately after birth.

8 ressed in fetal brains but rapidly decreases after birth.

9 uilibrium following a dynamic priming period after birth.

10 eurodevelopment that occurs within 2-3 weeks after birth.

11 to 20.92 mm at midgestation to 32.88 mm just after birth.

12 ert T1D suggests that the DVPs likely emerge after birth.

13 expressed at very low levels in most tissues after birth.

14 y's birth, and at 3, 6, 9, 12, and 18 months after birth.

15 n potential during development and very soon after birth.

16 fetal development and rapidly down-regulated after birth.

17 death during the third trimester or shortly after birth.

18 rs, at 12 to 18 hours, and at 36 to 42 hours after birth.

19 onsequently, prostasin null mice die shortly after birth.

20 in (HbA) normally occurs within a few months after birth.

21 ving treatment as compared with comfort care after birth.

22 ive effect of a peripheral OT treatment just after birth.

23 ption or why RTT features appear 6-18 months after birth.

24 hyltransferase DNMT3A during the first weeks after birth.

25 tes were genotyped and underwent MRI shortly after birth.

26 tent hypoxemia or bradycardia for many weeks after birth.

27 ry stimuli shape the organization of the map after birth.

28 nd kidney hemorrhage and die within 24 hours after birth.

29 tionally expressed anxiety-related behaviour after birth.

30 iferative and regenerative potential shortly after birth.

31 myocyte centrosome integrity is lost shortly after birth.

32 visits at 1, 3, 5, 9, 12, 18, and 24 months after birth.

33 at differentiates en masse before or shortly after birth.

34 sing air-displacement plethysmography </=3 d after birth.

35 lation of albumin and AFP genes in the liver after birth.

36 imarily by growing faster during the 6 weeks after birth.

37 ic transmission during ENS development, even after birth.

38 gut is rapidly colonized by microbes shortly after birth.

39 ng both subunits developed severe dermatitis after birth.

40 retardation phenotype and die within a month after birth.

41 ith animals followed up for a 4-month period after birth.

42 of vitamin A or placebo in the first 3 days after birth.

43 n cardiomyocytes become post-mitotic shortly after birth.

44 etting social behaviors during a period just after birth.

45 cells are likely depleted shortly before or after birth.

46 schooling, and maternal smoking was obtained after birth.

47 that show sharply reduced expression shortly after birth.

48 unction of pulmonary surfactant prior to and after birth.

49 dosing, as well as at 1, 3, 6, and 12 months after birth.

50 ntially lifesaving intervention administered after birth.

51 d maternal and infant iron status at 1 month after birth.

52 ctors (Pbx) led to lethal PH in mice shortly after birth.

53 to 34 degrees C for 72 hours within 6 hours after birth.

54 rs, at 12 to 18 hours, and at 36 to 42 hours after birth.

55 ic surge of testosterone occurring up to 5 h after birth.

56 RC- and RW/RW-mutant mice died within 9 days after birth.

57 empt and greasy hair coat and hair loss soon after birth.

58 nbred strains to hyperoxia (95% O2) for 72 h after birth.

59 y using routine histology sections collected after birth.

60 paralysis and ataxia and die around 6 weeks after birth.

61 e infant sex and breastfeeding within 1 hour after birth.

62 resent results from the trial cohort 2 years after birth.

63 ryonic development and abruptly decline soon after birth.

64 endochondral bone formation and die shortly after birth.

65 s grew rapidly and were lethal within 3-4 mo after birth.

66 their infants were evaluated until 24 weeks after birth.

67 rteen infants were enrolled; 1 died 3 months after birth.

68 to death from dehydration within a few days after birth.

69 high contribution to resident cMPhi declined after birth.

70 tion of the entire exocrine pancreas shortly after birth.

71 al skeletogenesis and lose MSC activity soon after birth.

72 OV with conventional ventilation immediately after birth.

73 ing detected only in neonatal thymi by day 3 after birth.

74 alities and increases seizure susceptibility after birth.

75 continued to mature until approximately 3 wk after birth.

76 ain protein product, and its levels decrease after birth.

77 ects remain unrepaired for prolonged periods after birth.

78 NTBC) at a dose of 1 mg/kg per day initially after birth.

79 the abdomen and pelvis was performed 6 days after birth.

80 results into a solidified compact myocardium after birth.

81 oncentrations were measured for up to 7 days after birth.

82 city for regeneration, which is lost shortly after birth.

83 ne may be functionally required in the brain after birth.

84 nce between NIRS SctO2 and aEEG for 78 hours after birth.

85 ging scans were acquired in neonates shortly after birth.

86 mite (HDM) allergens from days 3, 15, or 60 after birth.

87 mic encephalopathy enrolled at 6 to 24 hours after birth.

88 knocked out Celsr3 in the hippocampus 1 week after birth.

89 on parental disease activity both before and after birth.

90 ly, expression of GAS6 initiated only 3-4 wk after birth.

91 idity and mortality from infectious diseases after birth.

92 cular cell progenitors, but is downregulated after birth.

93 ated rate, often within the first few months after birth.

94 lls were generated during embryogenesis, not after birth.

95 d in profound deficits in maternal care soon after birth.

96 KO) mice and found that most died within 3 d after birth.

97 in 99.5% of infants within the first 30 days after birth.

98 uces target RNA expression for up to 4 weeks after birth.

99 infant and maternal outcomes up to 24 months after birth.

100 mples from infants were collected 4 and 9 mo after birth.

101 and bone area (BA) were measured within 2 wk after birth.

102 nocytes that colonize the tissue immediately after birth.

103 a functioning pulse oximeter by two minutes after birth, (2) Delayed intubation, (3) Normothermia on

104 (5/5), sensorineural deafness diagnosed soon after birth (5/5), congenital cataracts (4/5), and hypot

105 TDP-43 protein starting as early as 3 months after birth, a pathology that was exacerbated with age.

106 nerative capacity for a short period of time after birth, after which the majority of cardiomyocytes

107 st children with these disorders die shortly after birth, although limited data suggest some children

108 receptor, VPAC2R, reached detectable levels after birth and after the onset of circadian synchrony.

109 ot admitted to the neonatal nursery) shortly after birth and at age 6 months.

110 prospectively from pregnancy through 2 years after birth and completed several multimethod assessment

111 crobiome is established within a few minutes after birth and consists of stable multi-species communi

112 Neonatal nasal AECs were collected soon after birth and cultured to tertiary passage.

113 samples were collected beginning immediately after birth and for 1 year; the children were followed u

114 whole-body p31(comet) knockout mice die soon after birth and have reduced hepatic glycogen.

115 yses indicate that Individual 1 died shortly after birth and Individual 2 was a late-term fetus, maki

116 rapid weight gain during the first 6 months after birth and overweight status at age 1 year.

117 er of MCs in this region gradually increased after birth and peaked at the time of eyelid opening in

118 in human heart samples that KLF15 is induced after birth and reduced in heart failure, a myocardial e

119 hat acquisition of RSV infection occurs only after birth and shift attention to the prenatal effects

120 ealed a degenerative myopathy that developed after birth and specifically affected smooth muscle in t

121 l signal for cardiomyocyte cell-cycle arrest after birth and suggest interventions to augment the reg

122 NBDPS received a diagnosis more than 3 days after birth and therefore might have benefited from rout

123 DREAM downregulation was observed early after birth and was associated with endogenous neuroprot

124 natural lactoferrin concentrate, immediately after birth and, ideally, continue on breast milk throug

125 ll, 40.4% of deaths occurred within 12 hours after birth, and 17.3% occurred after 28 days.

126 Differences appear 2 weeks after birth, and depletion and recolonization in adultho

127 to interact with others measured within days after birth, and differences in infants' early social en

128 sion during different periods (before birth, after birth, and during child ages 1-5, 6-10, and 11-16

129 pression at different periods (before birth, after birth, and during child ages 1-5, 6-10, and 11-16

130 ytes lose the ability to proliferate shortly after birth, and further increase in cardiac mass is ach

131 ord blood of newborns and rates of clearance after birth, and how these correlated with drug concentr

132 embryonic tissues but is strongly repressed after birth, and significant transcription persists only

133 unique (neomorphic) features already shortly after birth, and these features persist throughout postn

134 nerally been considered to be completed soon after birth, and to be reactivated in later life only un

135 he postnatal mastery of suckling at 4 months after birth; and (2) thereafter, from preparation to the

136 blood culture positive sepsis more than 72 h after birth; and death before discharge from hospital.

137 The first 2 weeks after birth are particularly dynamic for differential ge

138 The days immediately after birth are the most risky for human survival, yet n

139 al mortality (death within the first 28 days after birth) as the main outcome measure.

140 Moderate cooling after birth asphyxia is associated with substantial redu

141 nhance the neuroprotective effect of cooling after birth asphyxia.

142 LRRK2 expression levels rise after birth at a time when synapses are forming and are

143 ly blind child who gains sight several years after birth be able to acquire this skill?

144 ow that mice deficient in GAS2L3 die shortly after birth because of heart failure.

145 covered that genes that acquire elevated mCH after birth become preferentially misregulated in mouse

146 ic regions, BCG vaccine is administered soon after birth, before in utero and peripartum HIV infectio

147 specific IgE measured before conception than after birth [BHR: aRRR = 2.96 (95% CI: 1.92, 4.57) and 1

148 anently withdraw from the cell cycle shortly after birth but can re-enter the cell cycle and prolifer

149 of peripheral T cells within the first 2 wk after birth but failed to populate the peripheral T cell

150 sory integration develops in cats 1-4 months after birth but only after acquiring extensive visual-au

151 ally, QIPAVA is present in utero and shortly after birth, but is reduced in older (>50 years) adults

152 ual cortex is adult-like within a few months after birth, but is subsequently refined through develop

153 d undergoes significant proliferative stages after birth, but little is known about how the developme

154 93A mice have a higher rate of proliferation after birth, but stop proliferation at the age of 2 mont

155 of CYP2C11 is irreversibly imprinted shortly after birth by a hormone other than the customary testos

156 Data on potential confounders was collected after birth by questionnaire.

157 ism and malnutrition, which start a few days after birth by the onset of nursing.

158 Two weeks after birth, by which time most neurites in the neurogen

159 rse environmental events in utero or shortly after birth can lead to abnormal brain development and i

160 Correcting fetal cardiac diagnosis after birth can lead to significant changes in neonatal

161 Following CRP and NT-proBNP levels after birth can potentially monitor severity and progres

162 nting normal BM into newborn MPS I mice soon after birth can prevent skeletal dysplasia.

163 After birth cardiomyocytes undergo terminal differentiat

164 athy, hypothermia initiated at 6 to 24 hours after birth compared with noncooling resulted in a 76% p

165 e following: Apgar score of 5 or less 10 min after birth, continued need for resuscitation 10 min aft

166 After birth, daughters underwent exams that included two

167 Only after birth did B cells acquire CXCL13 responsiveness, a

168 RIM1 and RIM2 from rods starting a few weeks after birth did not change Cav1.4 protein expression at

169 TOR(EKO) mutants are viable, but die shortly after birth due to deficits primarily during the early e

170 R3 function, appear cyanotic and die shortly after birth due to failure of lung inflation.

171 complete loss of LCs in the epidermis early after birth due to impaired proliferation and increased

172 of onset is related to physiological changes after birth (e.g., hormonal, immunological, circadian),

173 caffeine initiation: within the first 2 days after birth (early) and on or after the third day follow

174 hether cigarette smoke exposure in utero and after birth (environmental tobacco smoke [ETS]) modifies

175 t's clinical condition in the first 24 hours after birth (EOS calculator period [July 1, 2014, throug

176 at are briefly exposed to ascr#3 immediately after birth exhibit increased ascr#3-specific avoidance

177 FGF2 administered on the day after birth facilitated extinction learning and retentio

178 fetal liver and become PPARalpha responsive after birth following an epigenetic switch triggered by

179 ded in the prenatal period and first 3 years after birth for the most disadvantaged children and fami

180 , we collected freshly voided urine at day 1 after birth from neonates born at 31-36 weeks of gestati

181 Right after birth, hair follicle (HF) morphogenesis was transi

182 function of PGC-1 coactivators in the heart after birth has been incompletely understood.

183 intervention strategies in the first 2 years after birth has been limited.

184 ce develop dramatic cardiac dilation shortly after birth; heart size increases almost twofold.

185 ility to the illusion is present immediately after birth; however, eliciting reliable responses from

186 he uterus, and conditional deletion of Foxa2 after birth impedes uterine gland development.

187 MRI was performed at </=2 wk after birth in 333 neonates born at >/=34 wk of gestatio

188 obesity affect baby's condition immediately after birth in general.

189 n the lipids of colostrum and milk with time after birth in giant pandas.

190 rhythms is reached late during development, after birth in humans and by the end of the second postn

191 Shortly after birth in mice, specialized stromal cell subsets ar

192 ng development but only up until a few weeks after birth in mice.

193 (P) 11 and found that cell death peaks just after birth in most regions.

194 hepatocellular carcinoma developed by 1 year after birth in nearly 60% of the mice.

195 CD34(+) Gp38(+) cells develop after birth in the intestinal submucosa and expand aroun

196 projection neurons, progressively increases after birth in the somatosensory cortex.

197 o 27 weeks 6 days) to early (within 24 hours after birth) inhaled budesonide or placebo until they no

198 n, and layer formation dominate, development after birth involves more subtle processes, such as acti

199 32 degrees C in utero and in the first year after birth is associated with a 0.1% reduction in adult

200 stigate whether maternal depression 6 months after birth is associated with psychological difficultie

201 in a single high dose during the first month after birth is readily acquired but not retained efficie

202 hese symptoms evolve during the first months after birth is unknown.

203 25-hydroxyvitamin D [25(OH)D] concentration after birth is unknown.

204 zation by microbes, which begins immediately after birth, is influenced by gestational age and the mo

205 1a B-cell subset (present as early as 4 days after birth), late-onset lymphoid cancer development, an

206 knockout of Vangl2 in the hippocampus 1 week after birth led to a large increase in synaptic density,

207 ASO treatment starting after birth led to a significant decrease in Abeta patho

208 gut bacterial microbiome is rapidly acquired after birth, less is known about the viral microbiome (o

209 From weeks 2-4 after birth, levels of steatosis and apoptosis decreased

210 e nearly all of their proliferative capacity after birth, limiting the heart's ability to regenerate.

211 However, at weeks 4-8 after birth, livers became inflamed, with hepatocellular

212 Two weeks after birth, livers of ALR-L-KO mice contained low level

213 CI, -0.11 to 0.52; P = .20); and at 1 month after birth: maternal hemoglobin concentration: 12.89 g/

214 Hypothermia initiated at 6 to 24 hours after birth may have benefit but there is uncertainty in

215 providing evidence that manipulation of PAX6 after birth may lead to corrective tissue remodeling.

216 Measurements were performed after birth (median age: 13 d) and at 6-12 wk of age.

217 d), and post-neonatal (between 28 d and 1 y after birth) mortality.

218 to 9.0; P = .01) as well as anxiety shortly after birth (mothers: 13% vs 48%; OR = 6.5; 95% CI, 3.3

219 fants had higher rates of depression shortly after birth (mothers: 6% vs 40%; odds ratio [OR] = 9.9;

220 However, within the first 2 weeks after birth, mTOR-deficient islets became dysmorphic, be

221 Shortly after birth, myocyte cell division ceases.

222 itiated very early treatment within 12 weeks after birth (n = 14), or early treatment between week 12

223 in their skeletal muscles, but within weeks after birth nebulin expression rapidly falls to barely d

224 directly from a bipotent progenitor, whereas after birth new AT1 cells derive from rare, self-renewin

225 Infant outcome after birth of symptomatic vs asymptomatic disease was c

226 assessment before conception as compared to after birth of the child.

227 y and severe arthrogryposis who died shortly after birth, one 2-month-old baby, and two placentas fro

228 After birth, oocytes arrest at the prolonged dictyate st

229 istal bowel and is usually diagnosed shortly after birth or during childhood.

230 rth, continued need for resuscitation 10 min after birth, or acidosis within 1 h of birth.

231 in which cells undergo G2/M at a fixed time after birth, or by adding a critical size increment betw

232 Cp110(-/-) mice die shortly after birth owing to organogenesis defects as in ciliopa

233 Length of hospital stay after birth (P = .002) and maternal colonization (P = .0

234 FAP expression rose during the first 2 weeks after birth, paralleled by a steady increase in GLAST im

235 After birth, patients were randomly assigned to receive

236 s (Miz1DeltaPOZ) causes a neuropathy at 90 d after birth [postnatal day (P) 90], with a subsequent sp

237 Microbial colonization after birth profoundly affects development of the host.

238 of TSB in the early neonatal period (5 days after birth) promotes activation of the myelin basic pro

239 processes during endochondral bone formation after birth, recent studies have demonstrated the direct

240 Hypothermia initiated at less than 6 hours after birth reduces death or disability for infants with

241 that hypothermia initiated at 6 to 24 hours after birth reduces the risk of death or disability at 1

242 aneous head movements as early as 1-2 months after birth, relative to HR infants who showed more rapi

243 an cardiomyocytes out of the cell cycle soon after birth remain largely unknown.

244 The transition to pulmonary respiration after birth requires rapid alterations in the structure

245 h decrease sharply in wild-type mouse hearts after birth, resulting in cardiomyocytes with dysfunctio

246 hy with an onset within a few days to months after birth, resulting in persistent watery diarrhea.

247 spital discharge, infection more than 3 days after birth, severe retinopathy of prematurity, severe i

248 mice are born alive but most die within 3 d after birth showing no overt defects in anatomy.

249 low skeletal TnI (ssTnI) isoform and shortly after birth ssTnI is completely and irreversibly replace

250 ry, with stage I typically performed shortly after birth, stage II at 4 to 6 months of age, and stage

251 arding HOXA5 functions in the nervous system after birth, such as a potential role in the establishme

252 lls complete their differentiation 2-3 weeks after birth, suggesting a link to postnatal maturation.

253 ough the kidney cannot generate new nephrons after birth, suggesting a low level of regenerative comp

254 rapid decrease in circulating reticulocytes after birth suggests that they may have a limited role i

255 alcn in excitatory preBotC neurons died soon after birth; surviving mice developed apneas in adulthoo

256 between 10 (adolescent) and 15 (adult) weeks after birth, symptom initiation and progression upon rem

257 is a good understanding of the early events after birth that lead to the rapid increase in serum bil

258 During the first 3 days after birth, the bacterial community was relatively simp

259 During the first 2 weeks after birth, the bacterial load in the lungs increased,

260 We report that within the first weeks after birth, the embryo-derived population of resident C

261 After birth, the gastric epithelium is maintained by the

262 After birth, the growth trajectory and metabolic paramet

263 al step in mammalian development immediately after birth, the mechanism of which remains unresolved.

264 sults show that except for the first 2 weeks after birth, the thalamic reticular nucleus of the mouse

265 s demonstrate that except for a short period after birth, the TRN of the mouse lacks intrinsic GABAer

266 e for parents of VPT infants in the 12 weeks after birth: the mean (95% CI) change in depression scor

267 ases in the use of less invasive ventilation after birth, there was no significant decline in oxygen

268 unting usually begins in utero and continues after birth; therefore, its reduction must involve actio

269 ll as clinical growth measures obtained from after birth through midchildhood.

270 index percentile trajectories from 3 months after birth to 6 years of age.

271 s are programmed to develop in the intestine after birth to constitute a specialized microenvironment

272 s system, as it requires the increase in mCH after birth to guide differential MeCP2 binding among mC

273 providing interventions during pregnancy and after birth to support breastfeeding.

274 outcomes of individuals who spent from soon after birth to up to 43 months in severe deprivation in

275 ere sampled every 10 seconds within 24 hours after birth until at least 36 weeks' postmenstrual age.

276 hanges in environmental exposure occur right after birth, upon weaning, and during pubertal maturatio

277 ibiotic use occurred between 24 and 72 hours after birth; use decreased from 0.5% to 0.4% (adjusted d

278 tricular-subventricular zone (V-SVZ) shortly after birth was also largely unaffected, except for a re

279 ose erythropoietin treatment within 42 hours after birth was associated with a reduced risk of brain

280 gan at 3 mo and progressed to blindness 8 mo after birth was reversed by intraocular injection of MTS

281 ) is the only organ developing predominantly after birth, we utilized it as a powerful system to stud

282 emic episodes during the first 2 to 3 months after birth were associated with adverse 18-month outcom

283 d by auditory brainstem response within 48 h after birth, were measured and compared between infants

284 The first few months after birth, when a child begins to interact with the en

285 Thus, there is a switch after birth, when AT2 cells function as stem cells that

286 nd pleural cavities constitutively, starting after birth, where they require IRF4 and signals likely

287 ent in the mouse brain drastically decreases after birth, whereas Ddo mRNA levels concomitantly incre

288 d in unusually high numbers as early as 10 d after birth, whereas differentiating spermatogonia were

289 the hematologic and neurologic complications after birth, whereas TCblR/CD320 gene defects appear to

290 ck-out mice gradually develop hypothyroidism after birth, which leads to reduced body weight and shor

291 gs from two unrelated families who died soon after birth with a profound neurodevelopmental disorder

292 nt in all wild-type hair cells but declining after birth with emergence of the normal-polarity curren

293 Atrial-specific Nkx2-5 mutants died shortly after birth with hyperplastic working myocytes and condu

294 oth Lmnb1- and Lmnb2-deficient mice die soon after birth with neuronal layering abnormalities in the

295 Pur-alpha-deficient mice die after birth with pleiotropic neuronal defects.

296 s or inborn errors of metabolism may present after birth with psychosis.

297 h a DGC-like morphology at approximately 7 d after birth, with a primary dendrite pointing to the mol

298 The mice die shortly after birth, with cystic kidneys and proteinaceous debri

299 pecific IgEs, measured before conception vs. after birth, with offspring asthma and hayfever.

300 it is not clear how reduction of apoE levels after birth would affect amyloid deposition.