ライフサイエンスコーパス: afterdischarge

1 t broadening of action potentials during the afterdischarge.

2 rane to ensure adequate secretion during the afterdischarge.

3 reproduction by secreting hormone during an afterdischarge.

4 oductive behaviors by generating a prolonged afterdischarge.

5 as Type II neurons typically had a sustained afterdischarge.

6 the cation channel and, thereby, trigger the afterdischarge.

7 aneous action potentials referred to as the "afterdischarge.

8 ly stimulated or to other electrodes showing afterdischarges.

9 restored the ability of stimulation to evoke afterdischarges.

10 unstimulated clusters inhibited the onset of afterdischarges.

11 slices, accompanied by the appearance of CA3 afterdischarges.

12 or the commissural system was used to elicit afterdischarges.

13 ala stimulation (baseline), (2) during focal afterdischarge (AD) and (3) during generalized AD.

14 as to test the hypothesis that an electrical afterdischarge (AD) causes prolonged elevation in cytoso

15 etized rat during the initiation of a single afterdischarge (AD) evoked by perforant path stimulation

16 GalKO showed increased duration of afterdischarge (AD) evoked from the dentate gyrus by per

17 Kindling trials and afterdischarge (AD) were controlled to ascertain their r

18 ated kindling rate, defined as the number of afterdischarges (ADs) required to achieve the first stag

19 on the rapid kindling induced by a series of afterdischarges (ADs) triggered in hippocampus over a 3-

20 outs of synchronized network activity called afterdischarges (ADs).

21 Irrespective of the evoking conditions, afterdischarges always terminated first in the CA1 regio

22 tion, play important roles in generating the afterdischarge and triggering egg-laying behaviors.

23 ministration was also associated with longer afterdischarges and abnormal wind-up to transcutaneous e

24 tion velocity, (2) increase in IED secondary afterdischarges and their reverberation between CA3a and

25 y period, intact clusters were stimulated to afterdischarge, and neurons were isolated after the clus

26 ed spike failure, single spike transmission, afterdischarge, and spontaneous spiking.

27 In juvenile animals, such afterdischarges are inhibited by a high density of Ca2+

28 The likelihood of an afterdischarge at an individual site after stimulation w

29 Afterdischarge can be triggered in vitro by a variety of

30 Adult bag cell neurons exhibit an afterdischarge, consisting of prolonged depolarization a

31 A neuron prone to afterdischarge could function normally unless it was swi

32 Normal, long-lasting afterdischarges could be triggered by stimulation of an

33 which focuses on the neuronal mechanisms of afterdischarges, demonstrates that a single pair of inte

34 Afterdischarge duration (ADD) was assessed via electroen

35 Two indices of seizure severity, afterdischarge duration (Mean +/- S.E.M., sec.) (stimula

36 red-pulse inhibition, decreased epileptiform afterdischarge durations during 8 hours of subsequent st

37 After a complete lesion was made, afterdischarges evoked on one half of a slice were not d

38 inch) stimulation and increased incidence of afterdischarge firing 2-3 days after injection.

39 We did this by studying cortical afterdischarges following electrical stimulation of the

40 nstrate that: (1) the threshold to elicit an afterdischarge from the BL was lower than that of either

41 A interneuron (Cr-Aint)] is responsible for afterdischarge generation in the network.

42 nature of the depolarization generating the afterdischarge, however, has remained unclear.

43 gs revealed increased spontaneous discharge, afterdischarge, hyperresponsiveness to innocuous and nox

44 as given below the threshold that elicits an afterdischarge (i.e., a neuronal discharge that occurs a

45 An increase in this current also follows an afterdischarge in mature animals, contributing to a subs

46 to adults, juvenile neurons did not exhibit afterdischarge in response to pleural-abdominal connecti

47 the anlage of the mechanism responsible for afterdischarge in the adult.

48 e to Conus textile venom (CtVm), triggers an afterdischarge in the bag cell neurones of Aplysia.

49 gh-frequency stimulation of the VTA provoked afterdischarge in the central amygdala and enhanced kind

50 clude that a subiculum--CA1 circuit supports afterdischarges in both regions and synchronizes their a

51 region between subiculum and CA1 eliminated afterdischarges in subicular and CA1 events, but did not

52 ulse excitation or inhibition or duration of afterdischarges in the intact hippocampal preparation.

53 In vivo stimulation of a paroxysmal afterdischarge increased both pan-BDNF and NGF mRNA leve

54 A single afterdischarge increased exon I-IV-containing mRNA level

55 nisomycin modestly attenuated the paroxysmal afterdischarge-induced increase of both transcripts.

56 rons, which initiate reproduction through an afterdischarge involving multiple Ca(2+)-dependent proce

57 The afterdischarge is followed by a prolonged (approximately

58 Moreover, the refractory period of the afterdischarge itself may also be initiated by Ca2+ entr

59 A vigorous, long-lasting sound-evoked afterdischarge (LSA) is seen in a subpopulation of both

60 Afterdischarge occurred at g(Na)/g(L) just below the thr

61 astic" slices were characterized by multiple afterdischarges, occurring at a significantly higher rep

62 The afterdischarge of Aplysia bag cell neurons has served as

63 stimulation of the CeM elicited seizures and afterdischarges of shorter duration than those evoked by

64 was to investigate the effects of electrical afterdischarge on protein kinase C (PKC) activity from b

65 lectrical stimulation (ES), subthreshold for afterdischarge, on delayed item RM in epilepsy patients

66 h electrical stimulation and CtVm to trigger afterdischarges or elevate [Ca2+]i is severely attenuate

67 P levels or direct electrical stimulation of afterdischarge rapidly enhanced formation of these clust

68 Afterdischarges represent a prominent characteristic of

69 The mechanism of afterdischarge termination in the various hippocampal re

70 itic layers associated with a low amplitude "afterdischarge termination oscillation" (ATO) at 40 to 8

71 tion of action potentials during a prolonged afterdischarge that triggers a series of reproductive be

72 evelopment allows mature neurons to generate afterdischarges that are required for reproduction.

73 ecordings revealed increased tetanus-induced afterdischarges that could be kindled in the absence of

74 During the first few seconds of this afterdischarge, the action potentials of the bag cell ne

75 were divided: one cluster was stimulated to afterdischarge, the other was a control.

76 After an approximately 30 min afterdischarge, these neurons enter an approximately 18

77 he left piriform cortex every 5 min for 6 h (afterdischarge threshold, 60 Hz, 1 ms, 1 s).

78 The rats were kindled once daily using afterdischarge-threshold electrical stimulation until th

79 Rats were kindled to stage 5 by afterdischarge-threshold electrostimulation of the left

80 d chronic antiepileptic action by increasing afterdischarge thresholds in perforant path (but not olf

81 antly higher spontaneous activity and longer afterdischarges to noxious mechanical stimuli than wide

82 lock, probably by preventing newly generated afterdischarges to travel backwards to the original focu

83 Afterdischarge triggered a rapid and persistent increase

84 the intensely discharging neurons during the afterdischarge triggers propagating waves of depolarizat

85 Vocalization afterdischarges (VADs) are a validated model of the affe

86 zations during shock (VDSs) and vocalization afterdischarges (VADs) was challenged by the intrathecal

87 ations during shock (VDSs), and vocalization afterdischarges (VADs) when administered into the spinal

88 s during shock), and forebrain (vocalization afterdischarges, VADs) levels of the neuraxis were elici

89 ng shock, VDSs), and forebrain (vocalization afterdischarges, VADs) levels were elicited by noxious t

90 Termination of the afterdischarge was heralded by a large DC shift initiate

91 rrupted motor, sensory or language function, afterdischarges were more likely to occur at other sites

92 human infantile spasms: electrodecrement or afterdischarges were observed.