ライフサイエンスコーパス: agalactiae

1 id detection of Streptococcus agalactiae (S. agalactiae).

2 genomic region including the pur genes in S. agalactiae.

3 activity and other cellular functions of S. agalactiae.

4 genes specific to the streptococci and to S. agalactiae.

5 n used to distinguish GAS from Streptococcus agalactiae.

6 3 clinical blood cultures with Streptococcus agalactiae.

7 conserved in Gap homologs from Streptococcus agalactiae.

8 pected of being S. pseudoporcinus and not S. agalactiae.

9 ts of Streptococcus mutans and Streptococcus agalactiae.

10 ure alone for the detection of Streptococcus agalactiae.

11 The most frequent species was Streptococcus agalactiae (34%), and 52% of all cases were hematogenous

12 ltered the expression of several genes in S. agalactiae 874391 that encode key virulence factors, inc

13 We found that covR-deficient serotype III S. agalactiae 874391 was significantly attenuated for colon

14 Streptococcus bovis group (5), Streptococcus agalactiae (9), the Streptococcus anginosus group (1), S

15 th in human urine observed in ABU-causing S. agalactiae (ABSA) that was not seen among uropathogenic

16 affect the persistence or progression of S. agalactiae ABU.

17 th-enhanced PCR nominally detected 10 CFU S. agalactiae after 4 h of carrot broth incubation with com

18 enzymes, from S.pneumoniae and Streptococcus agalactiae, allowed for insights into this enzyme's mole

19 corresponding homologues from Streptococcus agalactiae also interacted with each other and formed a

20 secondary immunization with conjugate and S. agalactiae, although not S. pneumoniae, results in a boo

21 , vancomycin, and ceftriaxone, Streptococcus agalactiae, ampicillin, and cefotaxime, Escherichia coli

22 detection of Candida albicans, Streptococcus agalactiae and Chlamydia trachomatis with a single bioch

23 Detection of Streptococcus agalactiae and detection of bacteremia at <1 CFU/ml were

24 perform a similar function in Streptococcus agalactiae and Enterococcus faecalis In conclusion, the

25 presence of a supragenome for Streptococcus agalactiae and Haemophilus influenzae, it appears that t

26 h a putative peptidoglycan hydrolase from S. agalactiae and S. pneumoniae, indicative of a role in mu

27 th, as well as in Streptococcus pyogenes, S. agalactiae and S. suis.

28 Gap1 homologues from Streptococcus agalactiae and Staphylococcus aureus also interacted wit

29 Furthermore, the Gap1 homologues from S. agalactiae and Streptococcus sanguinis rescued the Fap1

30 homologous to the CAMP factor genes from S. agalactiae and Streptococcus uberis.

31 nce between the crystal structures of the S. agalactiae and the S. pneumoniae hyaluronate lyases.

32 y comparing the crystal structures of the S. agalactiae and the Streptococcus pneumoniae enzymes, and

33 s), group B streptococci (GBS; Streptococcus agalactiae), and Streptococcus pneumoniae were designed

34 m, Neisseria meningitidis, and Streptococcus agalactiae, and 3% of the residues in its deduced amino

35 calis, Streptococcus pyogenes, Streptococcus agalactiae, and viridans streptococci.

36 iv) dissemination of antibiotic-resistant S. agalactiae appears to include both clonal spread of resi

37 Group B Streptococcus (GBS) or Streptococcus agalactiae are beta-hemolytic gram-positive bacteria tha

38 Group B streptococci (GBS; Streptococcus agalactiae) are a major cause of invasive infections in

39 Group B streptococci (GBS) (Streptococcus agalactiae) are a major cause of sepsis and meningitis i

40 Group B streptococci (GBS; Streptococcus agalactiae) are beta-hemolytic, Gram-positive bacteria t

41 he group B streptococcus (GBS) Streptococcus agalactiae, are an important cause of systemic disease,

42 s which appear to synthesize glutathione (S. agalactiae ATCC 12927, S. pyogenes ATCC 8668, and Entero

43 Group B Streptococcus agalactiae bacteria (group B streptococci [GBS]) are the

44 f the laboratories that tested Streptococcus agalactiae by disk diffusion, 17% reported nonsusceptibl

45 Streptococcus agalactiae can cause urinary tract infection (UTI).

46 ctor containing a promoterless Streptococcus agalactiae cat gene was constructed.

47 we show that crude extracts of Streptococcus agalactiae catalyze the gamma-GCS and GS reactions and c

48 Streptococcus agalactiae causes both symptomatic cystitis and asymptom

49 Streptococcus agalactiae causes severe invasive disease in humans and

50 ve and highly sensitive quantification of S. agalactiae cells in a concentration range of 10(1)-10(7)

51 uC homologue from serotype III Streptococcus agalactiae complements DeltaneuC.

52 aromyces cerevisiae and one of Streptococcus agalactiae constructed using the KEGG database.

53 sular polysaccharide (PPS14) and type III S. agalactiae containing a PPS14 core capsule identical to

54 S. agalactiae CovR promotes bladder infection and inflammat

55 I sequences were compared to those of the S. agalactiae cpsIa locus, and the primary difference betwe

56 dis, Streptococcus pneumoniae, Streptococcus agalactiae, cytomegalovirus, enterovirus, herpes simplex

57 The S. agalactiae detection rate by early-aliquot carrot broth-

58 ered from the bottles with S. pneumoniae, S. agalactiae, E. coli, N. meningitidis, or H. influenzae i

59 One of these new structures resembles the S.agalactiae enzyme conformation, and provides evidence of

60 aecalis, Enterococcus faecium, Streptococcus agalactiae, Escherichia coli, Klebsiella pneumoniae, and

61 oniae, Streptococcus pyogenes, Streptococcus agalactiae, Escherichia coli, Klebsiella pneumoniae, Hae

62 The putative gene for S. agalactiae gamma-GCS was identified and cloned, and the

63 e loop region in GSH binding, chimeras of S. agalactiae gamma-GCS-GS were made containing gamma-GCS d

64 Streptococcus agalactiae (GBS) is a major cause of serious newborn bac

65 macrophages induced by group B Streptococcus agalactiae (GBS) is likely an important virulence mechan

66 Streptococcus agalactiae (GBS) is the leading cause worldwide of neona

67 The role of the S. agalactiae global virulence regulator, CovR, in UTI path

68 i), and recombinant SCPB, from Streptococcus agalactiae (group B streptococci), were compared in the

69 shown that the human pathogen Streptococcus agalactiae (Group B Streptococci, GBS) encodes a single

70 The Gram-positive bacteria Streptococcus agalactiae (group B streptococci, GBS) is an important h

71 S. agalactiae (Group B streptococci, GBS), E. faecalis, S.

72 Human isolates of serotype III Streptococcus agalactiae (group B streptococcus [GBS]) can be divided

73 Streptococcus agalactiae (group B Streptococcus [GBS]) causes serious

74 Streptococcus agalactiae (group B streptococcus [GBS]) colonizes the r

75 Streptococcus agalactiae (group B Streptococcus [GBS]) has not been de

76 Streptococcus agalactiae (group B streptococcus [GBS]) is a Gram-posit

77 Streptococcus agalactiae (group B streptococcus [GBS]) is a leading ca

78 Neonatal infection with Streptococcus agalactiae (group B Streptococcus [GBS]) is a leading ca

79 Streptococcus agalactiae (group B Streptococcus [GBS]) is a leading ca

80 rnal vaginal colonization with Streptococcus agalactiae (Group B Streptococcus [GBS]) is a precursor

81 Streptococcus agalactiae (group B Streptococcus [GBS]) is an important

82 Streptococcus agalactiae (group B Streptococcus [GBS]) remains a leadi

83 Escherichia coli, E. faecalis, Streptococcus agalactiae (group B streptococcus [GBS]), or Streptococc

84 Streptococcus agalactiae (group B Streptococcus or GBS) is a common ca

85 Streptococcus agalactiae (group B Streptococcus or GBS) is a common co

86 Streptococcus agalactiae (Group B Streptococcus) is a commensal of the

87 Streptococcus agalactiae (Group B Streptococcus, GBS) causes life-thre

88 Streptococcus agalactiae (group B streptococcus, GBS) causes neonatal

89 Streptococcus agalactiae (group B streptococcus, GBS) expresses either

90 Streptococcus agalactiae (Group B Streptococcus, GBS) is a commensal o

91 Streptococcus agalactiae (group B Streptococcus, GBS) is a leading cau

92 Streptococcus agalactiae (group B Streptococcus, GBS) is the predomina

93 Streptococcus agalactiae (group B Streptococcus; GBS) is a significant

94 Streptococcus agalactiae (group B Streptococcus; GBS) produces a CPS t

95 by the Gram-positive bacteria Streptococcus agalactiae (Group B Streptococcus; GBS) type III (GBSIII

96 In Streptococcus agalactiae, GSH synthesis is catalyzed by a single enzym

97 he burden of disease caused by Streptococcus agalactiae has increased significantly among older adult

98 Skizzle (SkzL), secreted by Streptococcus agalactiae, has moderate sequence identity to streptokin

99 Streptococcus agalactiae hyaluronate lyase degrades primarily hyaluron

100 Streptococcus agalactiae hyaluronate lyase is a virulence factor that

101 exasaccharide hyaluronan complex with the S. agalactiae hyaluronate lyase was determined at 2.2 A res

102 ides a discriminatory subtype analysis of S. agalactiae; (ii) most human invasive and bovine S. agala

103 itis and peptic ulcer disease, Streptococcus agalactiae, implicated in neonatal meningitis, and sever

104 s a rapid detection and quantification of S. agalactiae in environmental samples but also opens up ne

105 (ABU); however, growth characteristics of S. agalactiae in human urine have not previously been repor

106 atment of women colonized with Streptococcus agalactiae include vancomycin prophylaxis for those with

107 Group B streptococcus (GBS; Streptococcus agalactiae) induces apoptosis of macrophages, and this m

108 Neonatal Streptococcus agalactiae infections cause significant morbidity and mo

109 ction as rare complications of Streptococcus agalactiae infective endocarditis.

110 act Neisseria meningitidis nor Streptococcus agalactiae inhibited the OVA-specific IgG response.

111 Group B streptococcus (GBS) or Streptococcus agalactiae is a beta-hemolytic, Gram-positive bacterium

112 Streptococcus agalactiae is a primary cause of neonatal morbidity and

113 Streptococcus agalactiae is the leading cause of bacterial sepsis and

114 Streptococcus agalactiae isolates (n = 1,056) were highly susceptible

115 We conclude that (i) human and bovine S. agalactiae isolates represent distinct populations; (ii)

116 tiae; (ii) most human invasive and bovine S. agalactiae isolates represent distinct subtypes, suggest

117 characteristics of 52 human and 83 bovine S. agalactiae isolates.

118 The Gram-positive pathogen Streptococcus agalactiae, known as group B Streptococcus (GBS), is the

119 Inhibition remained S. agalactiae-like (i.e., very weak).

120 pyogenes (</=0.12 microg/mL), Streptococcus agalactiae (</=0.12 microg/mL), Streptococcus anginosus

121 treptococcus iniae (CpsY), and Streptococcus agalactiae (MtaR) that regulate methionine transport, am

122 dder uroepithelial cell models of UTI and S. agalactiae mutants in covR and related factors, includin

123 Streptococcus agalactiae, or group B Streptococcus (GBS), is an import

124 reservoir available for inclusion in the S. agalactiae pan-genome is vast and that unique genes will

125 The results suggest a role for SkzL in S. agalactiae pathogenesis through fibrinolytic enhancement

126 that an unannotated homodimeric TetR from S. agalactiae (PDB 3KKC) is the bona fide zinc efflux regul

127 Mice infected with covR-deficient S. agalactiae produced less proinflammatory cytokines than

128 ent H10, similar to an unknown Streptococcus agalactiae protein, was present in 31% of middle ear iso

129 ll promote comparative genomic studies of S. agalactiae recovered from diverse sources.

130 Homologous GtfA and GtfB from Streptococcus agalactiae rescued the glycosylation defect in the gtf1g

131 sor for the rapid detection of Streptococcus agalactiae (S. agalactiae).

132 ptococcal species, including S. pyogenes, S. agalactiae, S. dysgalactiae, S. equi, S. mutans, S. pneu

133 m SALSA, bound to Streptococcus pyogenes, S. agalactiae, S. gordonii, and Escherichia coli.

134 ococci, including Streptococcus pyogenes, S. agalactiae, S. pneumoniae, and S. equi.

135 the crystal structures of the Streptococcus agalactiae SAG2603 V/R sortase SrtC1 in two space groups

136 mutant of SCP from group B Streptococcus (S. agalactiae, SCPB) revealed SCPB is composed of five dist

137 The reporting of accurate Streptococcus agalactiae screening results in a short time frame is of

138 se available in databases showed that the S. agalactiae species can be described by a pan-genome cons

139 Srr2, an SRRP from S. agalactiae strain COH1, has been implicated in bacterial

140 e shotgun draft sequence for a Streptococcus agalactiae strain representing multilocus sequence type

141 Many group B Streptococcus agalactiae strains and other pathogenic streptococci exp

142 We conclude that some S. agalactiae strains can grow in human urine, and this rel

143 quenced serotype V strain 2603 V/R and 19 S. agalactiae strains from several serotypes using whole-ge

144 revealed the genetic heterogeneity among S. agalactiae strains, even of the same serotype, and provi

145 Genome comparisons among S. agalactiae, Streptococcus pneumoniae, Streptococcus pyog

146 ccus aureus in 8 patients, and Streptococcus agalactiae, Streptococcus pyogenes, and Streptococcus sa

147 t populations; (ii) human host-associated S. agalactiae subtypes may occasionally be transmitted to b

148 ,160,267 bp genome sequence of Streptococcus agalactiae, the leading cause of bacterial sepsis, pneum

149 r disease-causing serotypes of Streptococcus agalactiae, the main cause of neonatal infection in huma

150 For S. agalactiae, there was a single false-positive and false-

151 By contrast, in Streptococcus agalactiae, there were a number of cases in which both s

152 rain of group B Streptococcus (Streptococcus agalactiae) type III (GBS-III) that expresses desialylat

153 n additional 26 (12.8%) were positive for S. agalactiae upon subculture.

154 SA) that was not seen among uropathogenic S. agalactiae (UPSA) strains isolated from patients with ac

155 The pathogenesis of S. agalactiae UTI is complex, multifactorial, and influence

156 ission of antibiotic-resistant Streptococcus agalactiae, we compared phenotypic and genotypic charact

157 hich only 2 (Escherichia coli, Streptococcus agalactiae) were cultivated.

158 region of a GspB homologue of Streptococcus agalactiae, which is acidic rather than basic, showed no

159 mproved with inocula of 100 and 1,000 CFU S. agalactiae, with the majority of these aliquots demonstr