ライフサイエンスコーパス: aganglionic

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1 efficiently both wild-type and RET-deficient aganglionic bowel in organ culture.

2 , nine identified genes had higher levels in aganglionic bowel than in WT animals suggesting that int

3 two-fold higher expression in wild type than aganglionic bowel.

4 ent of enteric neurons and accumulate in the aganglionic colon of ls/ls mice.

5 features typical of Hirschsprung's disease (aganglionic colon).

6 s of the extrinsic nerve fibers found in the aganglionic colon.

7 f donor ENCCs to invade a recipient piece of aganglionic colon.

8 Intracellular recordings from aganglionic gastrointestinal muscles showed normal slow

9 ere also capable of colonising wild-type and aganglionic gut in organ culture and had the potential t

10 mouse bowel into wild-type or RET-deficient aganglionic gut in organ culture, results in extensive r

11 's disease) based on the colonisation of the aganglionic gut with progenitors derived from normogangl

12 ural tube adjacent to somites 3-6 to produce aganglionic gut.

13 us system precursor cells and thus create an aganglionic hindgut model in vivo.

14 In aganglionic hindguts, TNC expression is strong throughou

15 The terminal colon is aganglionic in mice lacking endothelin-3 or its receptor

16 laminin alpha 1 was examined in the totally aganglionic intestine of E15 and newborn c-ret -/- mice,

17 variation in penetrance and expressivity of aganglionic megacolon analogous to the variation observe

18 n receptor type B (EDNRB) produce congenital aganglionic megacolon and pigment abnormalities in mice

19 in spotting lethal (sl) rats, which exhibit aganglionic megacolon associated with white coat color.

20 ndgut of Hirschsprung's disease in humans or aganglionic megacolon in animals.

21 Congenital aganglionic megacolon, commonly known as Hirschsprung di

22 Hirschsprung disease (HSCR), or congenital aganglionic megacolon, is the most common cause of conge

23 Hirschsprung disease (HSCR), or congenital aganglionic megacolon, is the most frequent cause of con

24 ion to compare gene expression in E14 and P0 aganglionic or wild type mouse intestine.

25 pe and RET-deficient gut and showed that the aganglionic phenotype observed in vivo is consistently r

26 gut by advancing on extrinsic fibers and, in aganglionic preparations, they form a small number of ne

27 fail to colonize the terminal hindgut, this aganglionic region becomes non-functional and results in

28 nsplanting wild-type NCSCs directly into the aganglionic region of the Ednrb(sl/sl) gut, where they e

29 ncomplete penetrance, variation in length of aganglionic segment, and sex bias observed in human HSCR

30 Slow waves developed normally in aganglionic segments of small bowel placed into organ cu

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