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1 lowers tHcy levels potentially ameliorating age-related hearing loss.
2 that they play a role in the progression of age-related hearing loss.
3 Eps8 and is a candidate gene for progressive age-related hearing loss.
4 r leads to neurodegeneration and exacerbates age-related hearing loss.
5 association between 17 tagSNPs for MSRB3 and age-related hearing loss.
6 orms of deafness, as well as progressive and age-related hearing loss.
7 essing of the central auditory system during age-related hearing loss.
8 2 and Skp1 as potential genetic modifiers in age-related hearing loss.
9 a well-known correlation between smoking and age-related hearing loss.
10 as mutations in its gene cause deafness and age-related hearing loss.
11 jury and disease, and a major determinant of age-related hearing loss.
12 psychiatric treatments for older adults with age-related hearing loss.
13 ure and in analyses excluding those with non-age-related hearing loss.
14 uity has been identified as one component of age-related hearing loss.
15 t environmental exposures may play a role in age-related hearing loss.
16 d characterization of a novel mouse model of age-related hearing loss.
17 ying progressive sensorineural hearing loss (age-related hearing loss 5, ahl5) and audiogenic seizure
19 on to many inbred mouse strains, accelerates age-related hearing loss (AHL) and can worsen auditory p
24 iety of species and slows the progression of age-related hearing loss (AHL), a common age-related dis
25 of mice vary widely in onset and severity of age-related hearing loss (AHL), an important considerati
31 e whether deficiencies in Cu/Zn SOD increase age-related hearing loss and cochlear pathology, we coll
33 ing an opportunity to study both progressive age-related hearing loss and possible interventional the
35 nd noise-induced hearing loss because of its age-related hearing loss and susceptibility to acoustic
36 vironmental, and sex-related determinants of age-related hearing loss and to identify potential inter
38 ges and suffered a base to apex gradient and age-related hearing loss, and that mutations in cdh23 we
39 d outer hair cell loss characteristic of the age-related hearing loss (ARHL) in the background strain
42 ric hearing deficits are a common symptom of age-related hearing loss (ARHL), as are specific histopa
45 conceptual framework for understanding human age-related hearing loss (ARHL, or presbycusis) holds th
48 protective effects of caloric restriction on age-related hearing loss by promoting the mitochondrial
51 earing thresholds in these mice demonstrated age-related hearing loss in all homozygous-null, but not
53 se SIRT3 was essential for the prevention of age-related hearing loss in mice fed a calorically restr
54 ts an age-noise interaction that exacerbates age-related hearing loss in previously noise-damaged ear
56 del suggests that the primary factor in true age-related hearing loss is an energy-starved cochlear a
63 he objective was to test the hypothesis that age-related hearing loss may be associated with poor vit
64 animal model by comparing noise-induced and age-related hearing loss (NIHL; AHL) in groups of CBA/Ca
65 m response (ABR) which confirmed that severe age-related hearing loss occurred in 8-month-old mice, w
67 ion cells, were consistent with the observed age-related hearing loss of these mice beginning with th
69 rol subjects and patients with noise- and/or age-related hearing loss (p < 0.05 at all dilutions test
70 ly subjects challenge the common belief that age-related hearing loss (presbyacusis) is based primari
76 er hair cells of the C57BL/6J mouse model of age-related hearing loss, which reveal that cholinergic
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