ライフサイエンスコーパス: aged mouse

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1 progeny in the ischemic brain of the middle-aged mouse.

2 ischemic striatum and corpus callosum in the aged mouse.

3 progeny in the ischemic brain of the middle-aged mouse.

4 levels decreased significantly in NMJs from aged mouse.

5 xpression significantly decreases in healthy-aged mouse Achilles tendons.

6 ations in the lymphoid progenitor content of aged mouse bone marrow (BM) have been described, irradia

7 the chronic presence of type I interferon in aged mouse brain impedes cognitive ability by altering m

8 dicate an almost 50% aneuploidy frequency in aged mouse brain.

9 8-specific E3 ligase, Ube3a, is decreased in aged mouse brain.

10 re extensive expression of NT-3 in adult and aged mouse brains with cortical expression apparent at 4

11 L1 is disrupted in human OA cartilage and in aged mouse cartilage.

12 is a decline in eIF2alpha phosphorylation in aged mouse cerebral cortex that is associated with highe

13 In moderately aged mouse epidermis, we find that abnormal acidificatio

14 imary-cultured NPCs from the young adult and aged mouse forebrain.

15 RNA inductions after fornix transections in aged mouse hippocampus.

16 level by transplanting individual young and aged mouse HSCs labeled with barcoded viral vector, foll

17 etion and glucose tolerance are preserved in aged mouse islets by the heightened metabolic sensitivit

18 In this study, aged mouse kidneys displayed a reduced epithelial prolif

19 that the ratio of ASK1/Trx-ASK1 increases in aged mouse livers and that this correlates with the incr

20 We find that nuclear extracts from normal aged mouse livers have decreased p42(C/EBPalpha) levels

21 an LPS challenge is delayed significantly in aged mouse livers.

22 effects of low-dose lithium treatment in an aged mouse model expressing a parkin mutation within dop

23 Using an aged mouse model of RSV pathogenesis, we found that aged

24 Here, we used an aged mouse model to investigate the protective efficacy

25 ed conditional knockout [cKO]) and naturally aged mouse models.

26 n of active zones in developing, mature, and aged mouse NMJs by immunohistochemical detection of the

27 or-dependent survival of neurons and, in the aged mouse, paradoxical upregulation of myelin and ephri

28 esions were 30-100-fold more frequent in the aged mouse paravertebral SCG than in the prevertebral ce

29 f both the mitochondria-injured and in vitro-aged mouse platelets.

30 positively affects memory mechanisms in the aged mouse, possibly by acting on the septohippocampal c

31 oprotein is expressed in neonatal but not in aged mouse skin.

32 libraries from apparently healthy young and aged mouse ventricular cardiac muscle cells.

33 Treatment of the ischemic middle-aged mouse with Sildenafil increased nestin expressing n

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