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1 ndings regarding Pl(A) genotype and platelet aggregability.
2 vels were associated with increased platelet aggregability.
3 t with Pl(A) genotype in modulating platelet aggregability.
4 asminogen activator inhibitor-1 and platelet aggregability.
5 ulating red cell mass and increases platelet aggregability.
6 5 micrograms/ml of collagen showed increased aggregability (68.6% in hypercholesterolemic patients vs
7 tic cholesterol content and reduced platelet aggregability and occurred despite severe and persistent
9 nogen, factor VII, factor VIII, and platelet aggregability) and fibrinolytic factors [tissue plasmino
11 llele was associated with increased platelet aggregability, as indicated by lower epinephrine thresho
12 d, in part, to the morning surge in platelet aggregability, but its mechanisms are poorly understood.
13 dian effect on hemostasis, although platelet aggregability had two peaks: at approximately noon and a
15 ive stress, inflammation, increased platelet aggregability, metabolic dysregulation), may be implicat
16 s hypothesis is based on reports of enhanced aggregability of erythropoietin-driven platelets and an
17 lso act on the vessel wall by modulating the aggregability of platelets and the proliferation of lymp
18 e ability of clopidogrel to inhibit platelet aggregability; one suggested that CCBs reduce the effica
23 (A) genotype, fibrinogen level, and platelet aggregability were ascertained in 1340 subjects enrolled
24 c, however, in that the increase in platelet aggregability with higher fibrinogen was present for the
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