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1 en enrichment of room air can overcome this 'aggression'.
2 re-escalation phase but not during escalated aggression.
3 it from a broader conceptualization of human aggression.
4  social behavior and the neural circuitry of aggression.
5 g NMDARs in the PLmPFC in alcohol-heightened aggression.
6  are essential for male but not female mouse aggression.
7 gnaling is necessary and sufficient to limit aggression.
8 apply to forms of aggression beyond reactive aggression.
9 eventing the development of chronic physical aggression.
10 wired hypothalamus-mediated male territorial aggression.
11 factor facilitate, while serotonin inhibits, aggression.
12  frequent sleep disturbances and episodes of aggression.
13 henotype that frequently includes persistent aggression.
14 erstanding the nature and evolution of human aggression.
15 uring the task) and parent-reported reactive aggression.
16 es in the neural regulation of dominance and aggression.
17 about implicit attitudes toward violence and aggression.
18 y serve as an important biomarker of disease aggression.
19 ve an elevated risk for maladaptive reactive aggression.
20 mus (VMHvl)-a region required for male mouse aggression.
21 and BLA reactivity was positively related to aggression.
22 al properties through reduction in impulsive aggression.
23 gorical expression of pathological impulsive aggression.
24 ating implicit attitudes toward violence and aggression.
25 have critical influence on behavior, such as aggression.
26 e behaviour but had no impact on mobility or aggression.
27 uli displaying an expression associated with aggression.
28 ition (e.g., freeloading, displacements) and aggression.
29 atively correlated with rates of territorial aggression.
30 ts an effective pathway for suppressing male aggression.
31 n observed in humans-by signs of distress or aggression.
32 re environmental anxiety, training, and fear/aggression.
33  and Self-control in Humans (CLASH) model of aggression.
34  environments to rigid routine behaviors and aggression.
35 tions from the range cause impulsiveness and aggression.
36 male prairie voles displaying affiliation or aggression.
37 any tumors and their correlation with cancer aggression.
38 vl(Esr1+) cells are indispensable for female aggression.
39 at tempers rather than fuels poverty-induced aggression.
40 l pathways that bias Drosophila males toward aggression.
41 p or aggression song has opposite effects on aggression.
42  of NE, is known to promote both arousal and aggression.
43 gins and underemphasizes economic origins of aggression.
44 orrectly notes the low frequency of reactive aggression.
45 circuits to impede processing and to promote aggression.
46 ect behaviours such as olfactory learning or aggression.
47 (VMH) that are critical for male territorial aggression.
48 esting an evolutionary origin for compulsive aggression.
49 to dysregulate threat reactivity and promote aggression.
50  in the fly's auditory organ elicit abnormal aggression.
51 o developmental challenges and environmental aggressions.
52  of the sexual coercion hypothesis [7]: male aggression (1) is greatest against cycling females, (2)
53 nding in promoting social order and reducing aggression [4, 5].
54  humans that also engages in male intergroup aggression [7].
55  staff intervention in response to escalated aggression, a continuum between support and control.
56  humans have a high propensity for proactive aggression, a trait shared with chimpanzees but not bono
57 e1 and -e2 mutant males displayed heightened aggression accompanied by convergent expression changes
58           In explaining variation in violent aggression across populations, the age structures of tho
59 sive behaviors, leading to paradoxical tumor aggression after therapy.
60 group discrimination and sometimes defensive aggression against threatening (members of) out-groups.
61 e which aims to extinguish or reduce patient aggression/agitation irrespective of its cause, and impr
62 und a more positive implicit attitude toward aggression among individuals with lesions to the dlPFC a
63 eptides and their interactions in modulating aggression and affiliation.
64 terone's influence on males often centers on aggression and antisocial behavior, contemporary theoris
65  at scan-time was negatively related to both aggression and basolateral amygdala (BLA) reactivity to
66                                    Impulsive aggression and borderline personality disorder, but not
67 ckdown of Chrna7 in the DG enhanced baseline aggression and eliminated the serenic effects of both ni
68  and can induce antisocial effects including aggression and envy.
69  involves circuits that link innate feeding, aggression and fear.
70 project axons to brain regions implicated in aggression and fear.
71  were examined in the context of measures of aggression and impulsivity in physically healthy subject
72 role of serotonin 1B receptors (5-HT1BRs) in aggression and impulsivity, but this has never been eval
73 m harassment and punishment, by showing that aggression and matings are temporally decoupled.
74  analysis of chronological sequences between aggression and matings ruled out other coercive mechanis
75 aring in the control of Drosophila intermale aggression and open perspectives to decipher how hearing
76 o nerves is associated with increased cancer aggression and poor patient outcome.
77 simple relationship between testosterone and aggression and provide causal evidence for a more comple
78 ria terminalis, which suppresses territorial aggression and reduces contextual fear.
79 n variable of interest for predicting cancer aggression and response to therapy.
80                                              Aggression and responsiveness to noxious stimuli are ada
81 al role of the VMHvl in both male and female aggression and reveal the existence of two previously un
82 re less understood and often associated with aggression and social unrest.
83 pport of CLASH, and other possible causes of aggression and violence (e.g., wealth, income inequality
84                                  Maladaptive aggression and violence are associated with dysfunction
85  evolutionarily explains the biogeography of aggression and violence as strategic adaptation.
86                                  Maladaptive aggression and violence can lead to interpersonal confli
87  differences within and between countries in aggression and violence in terms of differences in clima
88 e various exceptions, a general rule is that aggression and violence increase as one moves closer to
89 erent predictions about the distributions of aggression and violence within and between societies.
90  explanations for these large differences in aggression and violence within countries and around the
91 rol are important determinants of inhibiting aggression and violence.
92 aping individual and societal differences in aggression and violence.
93 ltimately explain the poleward declension of aggression and violence.
94 mpletely through the causal chain underlying aggression and violence.
95  differences within and between countries in aggression and violence.
96 exhibited consistent individual variation in aggression and web weight only.
97 volution of traits that favour between-group aggression and within-group cooperation (e.g. parochial
98 critical for regulating retaliation/reactive aggression and, when dysfunctional, contributes to react
99 ypes characterized by dominance, competitive aggression, and active coping strategies appear to be mo
100 ronger and better coordinated than out-group aggression, and defender groups survived roughly 70% of
101 her implicate alpha7 nAChRs in regulation of aggression, and demonstrate that hippocampal alpha7 nACh
102 c subnucleus previously known as a locus for aggression, and discovered that social-fear and aggressi
103 hich involve negative emotions such as fear, aggression, and envy and lead to avoidance behavior.
104 s support previous neurobiological models of aggression, and extend them by demonstrating that fast a
105 as diverse as body temperature, respiration, aggression, and mood.
106 ion recapitulates learning impairment, hyper-aggression, and motor defects, all of which are insensit
107 nctionally link mucinous proteins with tumor aggression, and offer a new view of the cancer glycocaly
108  with a special focus on feeding, courtship, aggression, and postmating behaviors.
109  Mental health nurses are exposed to patient aggression, and required to manage and de-escalate aggre
110 ry serves as the foundation for the CLimate, Aggression, and Self-control in Humans (CLASH) model of
111             We propose a new model, CLimate, Aggression, and Self-control in Humans (CLASH), that hel
112 mmented on the theoretical model of CLimate, Aggression, and Self-control in Humans (CLASH).
113 ry processing, locomotor control, courtship, aggression, and sleep.
114 aring of a limited water resource, decreased aggression, and strongly defined dominance ranks (an ind
115 unctions, such as circadian rhythms, reward, aggression, anxiety, and fear.
116  14 techniques used in response to escalated aggression applied on a continuum between support and co
117              We show that although levels of aggression are driven, in part, by between-group selecti
118 olved in the development of chronic physical aggression are generally the product of an intergenerati
119 tus), in which both exploratory tendency and aggression are heritable.
120 es of white matter connectivity in impulsive aggression are warranted.
121 f the central amygdala and the hypothalamic 'aggression area', suggest that the ACo can initiate defe
122  be a promising therapeutic intervention for aggression arising in certain forms of neuropsychiatric
123 the concept of de-escalation of violence and aggression as described within the healthcare literature
124  each neuromodulatory molecule on Drosophila aggression, as well as effects of receptor proteins wher
125 mptoms of ADHD, sleep disruption, hypotonia, aggression, ASD, and ID.
126 amus and then the lateral septum to modulate aggression associated with mate guarding.
127 nstruction-carried over to affect focal male aggression at a later time and with a new opponent.
128 tanding of links between nurses' exposure to aggression, attitudes to, and actual involvement in, coe
129 ediating antidepressant responses and normal aggression behavior.
130 alue by reducing the potential for escalated aggression, benefiting both senders and receivers by fac
131 that shrub landcover significantly moderates aggression between nests, suggesting nests are more rela
132 t that the CLASH model may apply to forms of aggression beyond reactive aggression.
133 ychopathy) scores, and with higher levels of aggression (but not rule-breaking).
134 gher levels of externalizing symptoms (e.g., aggression) but not internalizing symptoms (e.g., depres
135  has highlighted monoaminergic influences on aggression, but a mechanistic account of how monoamines
136                                              Aggression by dominant individuals within groups was the
137     Bullying data support the CLASH model of aggression by suggesting that climate may moderate the f
138                  Monge referred to 'climatic aggression', by which he meant the negative consequences
139 d cellular mechanisms controlling Drosophila aggression can shed light on neural principles governing
140                           This modulation of aggression cannot be accounted for by linear integration
141 such as pair bonding, social recognition and aggression causally increases humans' willingness to eng
142 ing evidence on the neural basis of explicit aggression centered on the ventromedial prefrontal corte
143 t, humans have a low propensity for reactive aggression compared with chimpanzees, and in this respec
144                                              Aggression directed by male baboons at females during th
145 evolved in bonobos as the risk of intergroup aggression dissipated and the benefits of bonding betwee
146 rdination of collective action for out-group aggression, doubling the aggressor's success rate (Exp.
147 ation promoted wing extension and suppressed aggression during photostimulation, whereas aggression r
148 nd have a greater prevalence of violence and aggression (e.g., higher homicide rates).
149 P < .001); 5 of 7 syndrome scales, including aggression (effect size, 0.36; P < .001), sleep (effect
150 underlie synaptic plasticity associated with aggression escalation.
151 structure is a key predictor of coalitionary aggression even in the absence of meaningful resource st
152  individuals will seek out opportunities for aggression, even in the absence of threat-provoking cues
153  latitudinal gradients of heat, poverty, and aggression finds that heat-induced aggression is mediate
154 om ANAs, we tested additional mice for their aggression following preferential antagonism of GluN2D-c
155 , animals often experience elevated rates of aggression from conspecifics, and they may also be threa
156 aimed at subordinating out-groups (out-group aggression) from those aimed at defending the in-group a
157 lected data on infants' social interactions (aggression, grooming, and play) and self-scratching (a p
158   The debate on the developmental origins of aggression has historically opposed genetic and environm
159              The involvement of serotonin in aggression has traditionally been attributed to impaired
160    Psychosis (hazard ratio=2.007), agitation/aggression (hazard ratio=2.946), and any one clinically
161 ood to select an alternative reinforcer over aggression, heightened relapse vulnerability and progres
162          The bimodal classification of human aggression helps solve two important puzzles.
163 c circuits are known to encode initiation of aggression; however, little is known about the neural me
164 ed social interaction, compulsive behaviors, aggression, hyperactivity, anxiety, depression, and soma
165 tures underpinning implicit attitudes toward aggression in a unique sample of combat veterans with tr
166 ypothesis that lithium use reduces impulsive aggression in addition to stabilizing mood.
167 mantine, or intra-PLmPFC memantine increased aggression in AHAs, but only in the absence of alcohol.
168 ohol produced a pathological-like pattern of aggression in AHAs; these mice shifted their bites to mo
169 , are used to treat psychosis, agitation and aggression in Alzheimer's disease.
170 racted with prior alcohol intake to escalate aggression in ANAs.
171                                              Aggression in borderline personality disorder (BPD) is t
172 ikely to be effective in treating pathologic aggression in both female and male subjects.
173 be sensitive to "addiction-like" features of aggression in CD-1 mice.
174                                              Aggression in Drosophila is context-dependent and can th
175 ptor-expressing neurons (OARNs) that control aggression in Drosophila.
176      Intra-ILmPFC memantine had no effect on aggression in either AHAs or ANAs.
177 e disproportionately responsible for violent aggression in every society, and increases in violence t
178          Until now, data comparing anger and aggression in female and male patients with BPD have bee
179 mic injection of a 5-HT1a agonist stimulated aggression in female hamsters and inhibited aggression i
180           Experience-dependent escalation of aggression in female hamsters depends on activation of m
181 n the hypothalamus to regulate dominance and aggression in females and males.
182 in males, whereas injection of AVP inhibited aggression in females and stimulated aggression in males
183 stemically administered fluoxetine increased aggression in females and substantially reduced aggressi
184 irst-line response to potential violence and aggression in healthcare settings.
185 ChR signaling have also been shown to reduce aggression in human 15q13.3DS.
186 ng-standing debate about the significance of aggression in human nature is misconceived, because both
187 cture of the central neuromodulatory role of aggression in human subjects.
188 tween plasma markers of oxidative stress and aggression in human subjects.
189 kers of oxidative stress would be related to aggression in human subjects.
190 he neural basis of implicit attitudes toward aggression in humans using a modified version of the Imp
191 favored increased in-group prosociality over aggression in late human evolution.
192 d inflammation is associated with behavioral aggression in lower mammals and humans, we hypothesized
193 ression in females and substantially reduced aggression in males, there may be substantial gender dif
194  aggression in female hamsters and inhibited aggression in males, whereas injection of AVP inhibited
195 hibited aggression in females and stimulated aggression in males.
196 iques are recommended to manage violence and aggression in mental health settings yet restrictive pra
197           In contrast, GTS-21, which reduces aggression in mice, reduced DG granule cell activity dur
198 haviour: explorative genotypes elicited most aggression in opponents.
199 ss variable than participation in intergroup aggression in other primate species; (ii) males often pa
200 Pharmacological treatments for agitation and aggression in patients with Alzheimer's disease have sho
201        Recent work argued that the levels of aggression in social spider colonies are explained by gr
202     By contrast, these neurons cannot elicit aggression in socially housed males that intrude in anot
203 the neurocognitive processes associated with aggression in such individuals, but we know remarkably l
204 ity to anger expressions on women's reactive aggression in the Social Threat Aggression Paradigm (STA
205 erlying neurobiology of maladaptive reactive aggression in youths with DBD who have relatively low le
206 ding the in-group against possible out-group aggression (in-group defense).
207 ation, in addition to efficacy for agitation/aggression, included reductions in the frequency of irri
208 s that mediate a specific influence of OA on aggression, independent of any effect on arousal.
209  when dysfunctional, contributes to reactive aggression, independent of level of callous-unemotional
210                                    Escalated aggression is a behavioral sign of numerous psychiatric
211                                              Aggression is a prevalent behavior in the animal kingdom
212 as an instance of interspecific combat; such aggression is a social feature of modern ants.
213                                              Aggression is a universal social behavior important for
214                                    Increased aggression is common after traumatic brain injuries and
215                            In such contexts, aggression is commonly used to obtain desired resources.
216 s an essential means of resolving conflicts, aggression is expressed by both sexes but often at a hig
217           Instead, our analyses suggest that aggression is favoured primarily as a selfish strategy t
218                     The biology of proactive aggression is less well known and merits increased atten
219 ituations (Routine Activity Theory) in which aggression is likely to unfold.
220 erty, and aggression finds that heat-induced aggression is mediated by poverty and that heat tempers
221 hat D1-like receptor regulation of selective aggression is mediated through downstream activation of
222 rence between these two species is that male aggression is more frequent and intense in male-dominate
223 social interactions such as male territorial aggression is poorly understood.
224 reased between dMBD-R2-deficient males while aggression is reduced.
225        The neurobiology underlying escalated aggression is unknown and is particularly understudied i
226  studied using anxiety (light/dark test) and aggression (mirror test) paradigms.
227 factor that triggers aggression (The General Aggression Model), or the notion that warm temperature a
228  whereas peer punishment increased out-group aggression more than in-group defense without affecting
229 rkers correlated with a composite measure of aggression; more specifically, 8-hydroxy-2'-deoxyguanosi
230 ression, and discovered that social-fear and aggression neurons are intermixed but largely distinct.
231 and its expression correlates with increased aggression of human gliomas.
232 rocesses within each stage, including venom, aggression, olfactory recognition as well as growth and
233 se intergenerational mechanisms and physical aggression onset in infancy, it appears clear that preve
234 n can bias the output of P1 neurons to favor aggression over inter-male courtship.
235 n's reactive aggression in the Social Threat Aggression Paradigm (STAP).
236 es have shown that the frequency of physical aggression peaks in early childhood and then decreases u
237                                       In the aggression phase, men with BPD exhibited higher activity
238                           Two major types of aggression, proactive and reactive, are associated with
239  more aggression than controls and receiving aggression reduces juvenile hormone (JH) titers.
240 d that dogs were more likely to develop fear/aggression related issues early on, whilst issues relate
241 oss of antidepressant efficacy and increased aggression-related behavior.
242     In experiment 3, cluster analysis of the aggression-related measures identified a subset of "addi
243 l neural basis for this type of pathological aggression remains elusive.
244 s depression, childhood abuse, and impulsive aggression, report inconsistent results.
245 d that an ecological account of violence and aggression requires consideration of societal and cultur
246  aggression during photostimulation, whereas aggression resumed and wing extension was inhibited foll
247  insight into the neural circuits modulating aggression reward processing.
248 weakened by their state of stress (rendering aggression risky and/or unnecessary).
249  current smokers showed significantly higher aggression scores compared with both nonsmokers and ex-s
250                    In stage 2, NPI Agitation/Aggression scores were reduced from 5.8 to 3.8 with dext
251           We then tested them for relapse to aggression seeking after forced abstinence or punishment
252 uppression of aggression seeking, relapse to aggression seeking, progressive ratio responding, and pu
253 em for choice-based voluntary suppression of aggression seeking, relapse to aggression seeking, progr
254 on of the VMHvl accelerated moment-to-moment aggression-seeking and intensified future attack.
255 ound that VMHvl neurons became active during aggression-seeking and that their activity tracked chang
256            Inactivation of the VMHvl reduced aggression-seeking behavior, whereas optogenetic stimula
257     To explore this directly, we utilized an aggression-seeking task in which male mice self-initiate
258 s that promote self-initiated or 'voluntary' aggression-seeking when no threat is present.
259 th a known role in attack, was essential for aggression-seeking.
260 R availability and from increased behavioral aggression seen in active smokers.
261 nding, and punishment-induced suppression of aggression self-administration.
262 tinence or punishment-induced suppression of aggression self-administration.
263 , the occurrence of two major types of human aggression solves the execution paradox, concerned with
264 rther, we show that exposure to courtship or aggression song has opposite effects on aggression.
265 ing rates, however, were not associated with aggression, suggesting that reduced dispersal rates were
266                                    Following aggression testing, messenger RNA expression and protein
267        Female hamsters were given five daily aggression tests with or without prior treatment with th
268 ccurately high fighting ability receive more aggression than controls and receiving aggression reduce
269 anxiety-like behaviour and altered levels of aggression that are modulated by the neurotransmitter se
270 ullying was defined as repeated, intentional aggression that is targeted at a person who cannot easil
271 verage temperature as a factor that triggers aggression (The General Aggression Model), or the notion
272 en by traits reflecting good manners and non-aggression (the politeness aspect of Big Five agreeablen
273 asures of their exposure to various types of aggression, their attitudes towards seclusion and restra
274 en mental health nurses' exposure to patient aggression, their emotions, their attitudes towards coer
275  functioning and support the use of physical aggression to solve problems.
276  Fluoro-Gold, and tested for affiliation and aggression toward a female partner or novel female subje
277 on in animals [4, 5], in which repeated male aggression toward a female provides the aggressor with d
278                   Here, we test whether male aggression toward females functions as sexual coercion i
279  female mouse model of RSDS by inducing male aggression toward females through chemogenetic activatio
280 y toward closer genetic relatives and direct aggression toward less closely related individuals.
281 intained through the expression of selective aggression toward novel conspecifics.
282  who self-harm may have an increased risk of aggression toward others, but this association has been
283 terpersonal rejection and directing physical aggression toward others.
284 ral judgements regarding the function of the aggression; trial-and-error; ingrained local custom (esp
285 eking task in which male mice self-initiated aggression trials to gain brief and repeated access to a
286 idence of a link between these and increased aggression/use of restrictive practices.
287                                    Since the aggression used by executioners is proactive, the execut
288 sence of wing extension triggered persistent aggression via an internal state that could endure for m
289 ) Why is there so much variation in reactive aggression/violence between people living in the same en
290                                        Trait aggression was found to positively modulate connectivity
291                                              Aggression was significantly higher in dyads using only
292         Exposure to mild and severe physical aggression was unrelated to nurses' emotions.
293 rcuit mechanism of LS-mediated modulation of aggression, we examined the influence of LS input on the
294 ntrol study, markers of oxidative stress and aggression were assessed in human subjects with historie
295                      However, T's effects on aggression were strongly influenced by variation in trai
296 ncreased, while ad lib spiders reduced their aggression when their adult condition increased.
297   Inactivation of these cells reduced female aggression whereas their activation elicited attack.
298                                       Verbal aggression which appears targeted, demeaning or humiliat
299 inical studies of the treatment of escalated aggression, with the added benefit that emerging therape
300 d OA neuronal genome exhibited a decrease in aggression without altering male-male courtship.

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