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1 en enrichment of room air can overcome this 'aggression'.
2 re-escalation phase but not during escalated aggression.
3 it from a broader conceptualization of human aggression.
4 social behavior and the neural circuitry of aggression.
5 g NMDARs in the PLmPFC in alcohol-heightened aggression.
6 are essential for male but not female mouse aggression.
7 gnaling is necessary and sufficient to limit aggression.
8 apply to forms of aggression beyond reactive aggression.
9 eventing the development of chronic physical aggression.
10 wired hypothalamus-mediated male territorial aggression.
11 factor facilitate, while serotonin inhibits, aggression.
12 frequent sleep disturbances and episodes of aggression.
13 henotype that frequently includes persistent aggression.
14 erstanding the nature and evolution of human aggression.
15 uring the task) and parent-reported reactive aggression.
16 es in the neural regulation of dominance and aggression.
17 about implicit attitudes toward violence and aggression.
18 y serve as an important biomarker of disease aggression.
19 ve an elevated risk for maladaptive reactive aggression.
20 mus (VMHvl)-a region required for male mouse aggression.
21 and BLA reactivity was positively related to aggression.
22 al properties through reduction in impulsive aggression.
23 gorical expression of pathological impulsive aggression.
24 ating implicit attitudes toward violence and aggression.
25 have critical influence on behavior, such as aggression.
26 e behaviour but had no impact on mobility or aggression.
27 uli displaying an expression associated with aggression.
28 ition (e.g., freeloading, displacements) and aggression.
29 atively correlated with rates of territorial aggression.
30 ts an effective pathway for suppressing male aggression.
31 n observed in humans-by signs of distress or aggression.
32 re environmental anxiety, training, and fear/aggression.
33 and Self-control in Humans (CLASH) model of aggression.
34 environments to rigid routine behaviors and aggression.
35 tions from the range cause impulsiveness and aggression.
36 male prairie voles displaying affiliation or aggression.
37 any tumors and their correlation with cancer aggression.
38 vl(Esr1+) cells are indispensable for female aggression.
39 at tempers rather than fuels poverty-induced aggression.
40 l pathways that bias Drosophila males toward aggression.
41 p or aggression song has opposite effects on aggression.
42 of NE, is known to promote both arousal and aggression.
43 gins and underemphasizes economic origins of aggression.
44 orrectly notes the low frequency of reactive aggression.
45 circuits to impede processing and to promote aggression.
46 ect behaviours such as olfactory learning or aggression.
47 (VMH) that are critical for male territorial aggression.
48 esting an evolutionary origin for compulsive aggression.
49 to dysregulate threat reactivity and promote aggression.
50 in the fly's auditory organ elicit abnormal aggression.
51 o developmental challenges and environmental aggressions.
52 of the sexual coercion hypothesis [7]: male aggression (1) is greatest against cycling females, (2)
56 humans have a high propensity for proactive aggression, a trait shared with chimpanzees but not bono
57 e1 and -e2 mutant males displayed heightened aggression accompanied by convergent expression changes
60 group discrimination and sometimes defensive aggression against threatening (members of) out-groups.
61 e which aims to extinguish or reduce patient aggression/agitation irrespective of its cause, and impr
62 und a more positive implicit attitude toward aggression among individuals with lesions to the dlPFC a
64 terone's influence on males often centers on aggression and antisocial behavior, contemporary theoris
65 at scan-time was negatively related to both aggression and basolateral amygdala (BLA) reactivity to
67 ckdown of Chrna7 in the DG enhanced baseline aggression and eliminated the serenic effects of both ni
71 were examined in the context of measures of aggression and impulsivity in physically healthy subject
72 role of serotonin 1B receptors (5-HT1BRs) in aggression and impulsivity, but this has never been eval
74 analysis of chronological sequences between aggression and matings ruled out other coercive mechanis
75 aring in the control of Drosophila intermale aggression and open perspectives to decipher how hearing
77 simple relationship between testosterone and aggression and provide causal evidence for a more comple
81 al role of the VMHvl in both male and female aggression and reveal the existence of two previously un
83 pport of CLASH, and other possible causes of aggression and violence (e.g., wealth, income inequality
87 differences within and between countries in aggression and violence in terms of differences in clima
88 e various exceptions, a general rule is that aggression and violence increase as one moves closer to
89 erent predictions about the distributions of aggression and violence within and between societies.
90 explanations for these large differences in aggression and violence within countries and around the
97 volution of traits that favour between-group aggression and within-group cooperation (e.g. parochial
98 critical for regulating retaliation/reactive aggression and, when dysfunctional, contributes to react
99 ypes characterized by dominance, competitive aggression, and active coping strategies appear to be mo
100 ronger and better coordinated than out-group aggression, and defender groups survived roughly 70% of
101 her implicate alpha7 nAChRs in regulation of aggression, and demonstrate that hippocampal alpha7 nACh
102 c subnucleus previously known as a locus for aggression, and discovered that social-fear and aggressi
103 hich involve negative emotions such as fear, aggression, and envy and lead to avoidance behavior.
104 s support previous neurobiological models of aggression, and extend them by demonstrating that fast a
106 ion recapitulates learning impairment, hyper-aggression, and motor defects, all of which are insensit
107 nctionally link mucinous proteins with tumor aggression, and offer a new view of the cancer glycocaly
109 Mental health nurses are exposed to patient aggression, and required to manage and de-escalate aggre
110 ry serves as the foundation for the CLimate, Aggression, and Self-control in Humans (CLASH) model of
114 aring of a limited water resource, decreased aggression, and strongly defined dominance ranks (an ind
116 14 techniques used in response to escalated aggression applied on a continuum between support and co
118 olved in the development of chronic physical aggression are generally the product of an intergenerati
121 f the central amygdala and the hypothalamic 'aggression area', suggest that the ACo can initiate defe
122 be a promising therapeutic intervention for aggression arising in certain forms of neuropsychiatric
123 the concept of de-escalation of violence and aggression as described within the healthcare literature
124 each neuromodulatory molecule on Drosophila aggression, as well as effects of receptor proteins wher
127 nstruction-carried over to affect focal male aggression at a later time and with a new opponent.
128 tanding of links between nurses' exposure to aggression, attitudes to, and actual involvement in, coe
130 alue by reducing the potential for escalated aggression, benefiting both senders and receivers by fac
131 that shrub landcover significantly moderates aggression between nests, suggesting nests are more rela
134 gher levels of externalizing symptoms (e.g., aggression) but not internalizing symptoms (e.g., depres
135 has highlighted monoaminergic influences on aggression, but a mechanistic account of how monoamines
137 Bullying data support the CLASH model of aggression by suggesting that climate may moderate the f
139 d cellular mechanisms controlling Drosophila aggression can shed light on neural principles governing
141 such as pair bonding, social recognition and aggression causally increases humans' willingness to eng
142 ing evidence on the neural basis of explicit aggression centered on the ventromedial prefrontal corte
143 t, humans have a low propensity for reactive aggression compared with chimpanzees, and in this respec
145 evolved in bonobos as the risk of intergroup aggression dissipated and the benefits of bonding betwee
146 rdination of collective action for out-group aggression, doubling the aggressor's success rate (Exp.
147 ation promoted wing extension and suppressed aggression during photostimulation, whereas aggression r
149 P < .001); 5 of 7 syndrome scales, including aggression (effect size, 0.36; P < .001), sleep (effect
151 structure is a key predictor of coalitionary aggression even in the absence of meaningful resource st
152 individuals will seek out opportunities for aggression, even in the absence of threat-provoking cues
153 latitudinal gradients of heat, poverty, and aggression finds that heat-induced aggression is mediate
154 om ANAs, we tested additional mice for their aggression following preferential antagonism of GluN2D-c
155 , animals often experience elevated rates of aggression from conspecifics, and they may also be threa
156 aimed at subordinating out-groups (out-group aggression) from those aimed at defending the in-group a
157 lected data on infants' social interactions (aggression, grooming, and play) and self-scratching (a p
158 The debate on the developmental origins of aggression has historically opposed genetic and environm
160 Psychosis (hazard ratio=2.007), agitation/aggression (hazard ratio=2.946), and any one clinically
161 ood to select an alternative reinforcer over aggression, heightened relapse vulnerability and progres
163 c circuits are known to encode initiation of aggression; however, little is known about the neural me
164 ed social interaction, compulsive behaviors, aggression, hyperactivity, anxiety, depression, and soma
165 tures underpinning implicit attitudes toward aggression in a unique sample of combat veterans with tr
167 mantine, or intra-PLmPFC memantine increased aggression in AHAs, but only in the absence of alcohol.
168 ohol produced a pathological-like pattern of aggression in AHAs; these mice shifted their bites to mo
177 e disproportionately responsible for violent aggression in every society, and increases in violence t
179 mic injection of a 5-HT1a agonist stimulated aggression in female hamsters and inhibited aggression i
182 in males, whereas injection of AVP inhibited aggression in females and stimulated aggression in males
183 stemically administered fluoxetine increased aggression in females and substantially reduced aggressi
186 ng-standing debate about the significance of aggression in human nature is misconceived, because both
190 he neural basis of implicit attitudes toward aggression in humans using a modified version of the Imp
192 d inflammation is associated with behavioral aggression in lower mammals and humans, we hypothesized
193 ression in females and substantially reduced aggression in males, there may be substantial gender dif
194 aggression in female hamsters and inhibited aggression in males, whereas injection of AVP inhibited
196 iques are recommended to manage violence and aggression in mental health settings yet restrictive pra
199 ss variable than participation in intergroup aggression in other primate species; (ii) males often pa
200 Pharmacological treatments for agitation and aggression in patients with Alzheimer's disease have sho
202 By contrast, these neurons cannot elicit aggression in socially housed males that intrude in anot
203 the neurocognitive processes associated with aggression in such individuals, but we know remarkably l
204 ity to anger expressions on women's reactive aggression in the Social Threat Aggression Paradigm (STA
205 erlying neurobiology of maladaptive reactive aggression in youths with DBD who have relatively low le
207 ation, in addition to efficacy for agitation/aggression, included reductions in the frequency of irri
209 when dysfunctional, contributes to reactive aggression, independent of level of callous-unemotional
216 s an essential means of resolving conflicts, aggression is expressed by both sexes but often at a hig
220 erty, and aggression finds that heat-induced aggression is mediated by poverty and that heat tempers
221 hat D1-like receptor regulation of selective aggression is mediated through downstream activation of
222 rence between these two species is that male aggression is more frequent and intense in male-dominate
227 factor that triggers aggression (The General Aggression Model), or the notion that warm temperature a
228 whereas peer punishment increased out-group aggression more than in-group defense without affecting
229 rkers correlated with a composite measure of aggression; more specifically, 8-hydroxy-2'-deoxyguanosi
230 ression, and discovered that social-fear and aggression neurons are intermixed but largely distinct.
232 rocesses within each stage, including venom, aggression, olfactory recognition as well as growth and
233 se intergenerational mechanisms and physical aggression onset in infancy, it appears clear that preve
236 es have shown that the frequency of physical aggression peaks in early childhood and then decreases u
240 d that dogs were more likely to develop fear/aggression related issues early on, whilst issues relate
242 In experiment 3, cluster analysis of the aggression-related measures identified a subset of "addi
245 d that an ecological account of violence and aggression requires consideration of societal and cultur
246 aggression during photostimulation, whereas aggression resumed and wing extension was inhibited foll
249 current smokers showed significantly higher aggression scores compared with both nonsmokers and ex-s
252 uppression of aggression seeking, relapse to aggression seeking, progressive ratio responding, and pu
253 em for choice-based voluntary suppression of aggression seeking, relapse to aggression seeking, progr
255 ound that VMHvl neurons became active during aggression-seeking and that their activity tracked chang
257 To explore this directly, we utilized an aggression-seeking task in which male mice self-initiate
263 , the occurrence of two major types of human aggression solves the execution paradox, concerned with
264 rther, we show that exposure to courtship or aggression song has opposite effects on aggression.
265 ing rates, however, were not associated with aggression, suggesting that reduced dispersal rates were
268 ccurately high fighting ability receive more aggression than controls and receiving aggression reduce
269 anxiety-like behaviour and altered levels of aggression that are modulated by the neurotransmitter se
270 ullying was defined as repeated, intentional aggression that is targeted at a person who cannot easil
271 verage temperature as a factor that triggers aggression (The General Aggression Model), or the notion
272 en by traits reflecting good manners and non-aggression (the politeness aspect of Big Five agreeablen
273 asures of their exposure to various types of aggression, their attitudes towards seclusion and restra
274 en mental health nurses' exposure to patient aggression, their emotions, their attitudes towards coer
276 Fluoro-Gold, and tested for affiliation and aggression toward a female partner or novel female subje
277 on in animals [4, 5], in which repeated male aggression toward a female provides the aggressor with d
279 female mouse model of RSDS by inducing male aggression toward females through chemogenetic activatio
280 y toward closer genetic relatives and direct aggression toward less closely related individuals.
282 who self-harm may have an increased risk of aggression toward others, but this association has been
284 ral judgements regarding the function of the aggression; trial-and-error; ingrained local custom (esp
285 eking task in which male mice self-initiated aggression trials to gain brief and repeated access to a
288 sence of wing extension triggered persistent aggression via an internal state that could endure for m
289 ) Why is there so much variation in reactive aggression/violence between people living in the same en
293 rcuit mechanism of LS-mediated modulation of aggression, we examined the influence of LS input on the
294 ntrol study, markers of oxidative stress and aggression were assessed in human subjects with historie
299 inical studies of the treatment of escalated aggression, with the added benefit that emerging therape
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