ライフサイエンスコーパス: agnosia

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1 the routing of information in the forebrain (agnosia).

2 visual stream and suffered from visual form agnosia.

3 d can produce the typical symptoms of finger agnosia.

4 ent in the published data cohort (eg, visual agnosia [5.6%, 3.9-7.2], aphasia [23.0%, 20.0-26.0], and

5 derate Alzheimer's disease, including visual agnosia (55.1%, 95% CI 45.7-64.6), aphasia (57.9%, 48.6-

6 the need for patients to exhibit associative agnosia and/or prosopagnosia: many authors have used the

7 vides a framework for understanding auditory agnosias and makes specific predictions to direct future

8 mechanisms and brain substrates of auditory agnosias and related disorders of auditory object proces

9 entrotemporal-fusiform network (resulting in agnosia) and the left hemisphere language network (resul

10 nce of amusia as a distinct form of auditory agnosia, but does not support the hypothesis that bilate

11 We tested a patient (HJA) with visual agnosia due to bilateral lesions of the ventral occipito

12 vioral switching or a possible interoceptive agnosia following hippocampal damage.

13 However, in humans category specific visual agnosia follows inferior temporal cortex but not LPFC da

14 owledge for objects in a patient with visual agnosia (H.J.A.).

15 D.F., a patient with severe visual form agnosia, has been the subject of extensive research duri

16 to confirm directly that D.F.'s visual form agnosia is associated with extensive damage to the ventr

17 ance on tasks commonly used to assess finger agnosia is modulated by changes in hand posture.

18 Loss of shape recognition in visual-form agnosia occurs without equivalent losses in the use of v

19 this imbalance can be symptoms like apraxia, agnosia or sundowning.

20 Aphasia, agnosia, seizures and visual field defects were observed

21 range from sensory or motor deficits (visual agnosia, sensory hypersensitivity, atonic episodes, rest

22 , which resulted in a persisting visual-form agnosia that has been extensively characterized at the b

23 yday life; others interpret the criterion of agnosia to require pervasive recognition impairments aff

24 mporal ablations in monkeys produce a visual agnosia which causes severe 'psychic blindness' in the f

25 suoperceptual skills and apperceptive visual agnosia with predominant posterior atrophy corresponding

26 deficits in object perception in visual-form agnosia without the exploitation of both structural and

27 g their fingers, a condition known as finger agnosia, yet are relatively unimpaired in sensation and

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