1 MITO-
8 aimed to verify this hypothesis commonly used in clinical pra
2 ip between endoplasmic reticulum (ER) stress and cGVHD,
and aimed to create effective treatment of cGVHD.
3 ized that bile acids regulate colonic HbetaD expression
and aimed to test this by investigating the effects of deoxycholi
4 Here we introduce a package that
is aimed to simplify this process.
5 This
review aimed to describe core constipation symptoms and their freque
6 This
review aimed to provide a critical overview of the current knowledge
7 This
study aimed to determine the osteoclastogenic role of H4R in rheuma
8 This
study aimed to determine the T-cell response to Phl p 12 in profili
9 This
study aimed to elucidate potential clinical and biological function
10 This
study aimed to evaluate clinical response and survival in patients
11 This
study aimed to examine the effectiveness and safety of TMVR in a co
12 This study
was aimed to determine both total and bioaccessible of trace elem
13 We aimed to assess colposcopy referral and CIN2+ detection rates
14 We aimed to assess mortality by occupation in the UK, difference
15 We aimed to assess the efficacy and safety of ceftazidime-avibac
16 We aimed to assess the efficacy and safety of pacritinib versus
17 We aimed to assess the electrophysiological properties of CI cha
18 We aimed to assess the scale of this problem and whether it is a
19 We aimed to assess whether a patient-specific molecular signatur
20 We aimed to assess whether provision of mindfulness courses to u
21 We aimed to clarify the underlying molecular mechanisms of suspe
22 We aimed to compare in-hospital outcomes of patients with CKD or
23 We aimed to comprehensively describe the treatment cascade for R
24 We aimed to define an association between mtDNA and fibroblast r
25 We aimed to determine apixaban pharmacokinetics at steady state
26 We aimed to determine if associations between maternal body mass
27 We aimed to determine whether changes in the serum levels of the
28 We aimed to determine whether higher mortality rates with high m
29 We aimed to develop a methodical approach that would yield a bro
30 We aimed to elucidate the origin and hydrological pathways of BT
31 We aimed to estimate the difference in opioid relapse-free survi
32 We aimed to evaluate the efficacy of the parenteral (intravenous
33 We aimed to identify genetic and pathological markers that have
34 We aimed to identify the effectiveness of prolonged versus short
35 We aimed to identify the role of the enzyme acid sphingomyelinas
36 We aimed to investigate urinary NP and BPA levels in relation to
37 We aimed to profile the microbiota at different stages of Barret
38 We aimed to quantify changes in projected ranges and threat leve
39 We aimed to quantify the independent and joint associations of t
40 We aimed to systematically evaluate serum levels of tryptophan a
41 We aimed to trace the geographic origin of HIV-1 infection for m
42 We aimed to use molecular genotyping to assess antimalarial drug
43 Additionally,
we aimed to identify microbial patterns associated with the onse
44 Here,
we aimed to gain insights into the genetic architecture of bioma
45 Herein,
we aimed to assess the potential role of protein-modifying varia
46 opamine neurons while exhibiting milder cell loss in PD,
we aimed to define the electrophysiological properties of DMV ne
47 In this study,
we aimed to define the mechanisms by which noninflammatory SF mo
48 In this study,
we aimed to explore the roles of secondary surgical cytoreductio
49 In this study,
we aimed to reveal the mechanisms underlying the behavioral acti
50 The present
work aimed to examine how the doxycycline-inducible lentiviral-med