ライフサイエンスコーパス: air pollutant

1 ation of tropospheric ozone (O3), a powerful air pollutant.

2 nged between 0.6% and 4.5%, depending on the air pollutant.

3 of atrial fibrillation after exposure to an air pollutant.

4 emit black carbon (BC), a climate forcer and air pollutant.

5 improving soybean responses to this damaging air pollutant.

6 rgen can be enhanced by prior exposure to an air pollutant.

7 may modify susceptibility to the effects of air pollutants.

8 calculate an average exposure parameter for air pollutants.

9 n increased population exposure to unhealthy air pollutants.

10 mortality and long-term exposure to several air pollutants.

11 is a key oxidant in the degradation of most air pollutants.

12 egression to account for colinearity between air pollutants.

13 inesses, or dump sites, is a large source of air pollutants.

14 predict long-term average concentrations of air pollutants.

15 ion properties commonly observed in ambient air pollutants.

16 that encompasses well-studied and ubiquitous air pollutants.

17 concentrations of fungal spores, pollen, and air pollutants.

18 conventional fossil fuels for six regulated air pollutants.

19 hes, with a particular focus on the criteria air pollutants.

20 ward large multiwell pads, release hazardous air pollutants.

21 better characterize health effects of indoor air pollutants.

22 dren associated with modeled traffic-related air pollutants.

23 in ambient inhalable particulate matter from air pollutants.

24 These associations were independent of air pollutants.

25 ffered by levels of oxidant-stress-producing air pollutants.

26 -km resolution were estimated for 11 ambient air pollutants.

27 lic concerns regarding exposure to hazardous air pollutants.

28 lwood usage, and climate and health-relevant air pollutants.

29 ure to hazardous concentrations of smoke and air pollutants.

30 ay potentiate the adverse effects of oxidant air pollutants.

31 ons and concerns about exposure to hazardous air pollutants.

32 he observed associations were independent of air pollutants.

33 ng CH4 emissions while controlling for other air pollutants.

34 considered effects from exposure to ambient air pollutants.

35 d drug to modulate responsiveness to oxidant air pollutants.

36 tion and may be useful for analysis of other air pollutants.

37 The estimates slopes for air pollutants (28 days cumulated) were higher in partic

38 Very little is currently known about air pollutants' adverse effects on neurodegenerative dis

39 Inhalation of air pollutants affects heart rate, heart rate variabilit

40 ed associations of birth outcomes with toxic air pollutants (air toxics) in traffic exhaust.

41 ses, providing a possible mechanism by which air pollutants alter antimicrobial immunity.

42 Benzene is a common air pollutant and confirmed carcinogen, especially in re

43 Nitrogen dioxide (NO2) is an environmental air pollutant and endogenously generated oxidant that co

44 Ozone is a highly toxic air pollutant and global health concern.

45 There is a pressing need to verify air pollutant and greenhouse gas emissions from anthropo

46 NO is a precursor to tropospheric ozone, an air pollutant and greenhouse gas.

47 ation between indoor exposure to NO (2) , an air pollutant and known asthmogen, and severe asthma amo

48 tions of ground-level ozone (O3 ) - a common air pollutant and phytotoxin - currently being experienc

49 Ozone is a common urban environmental air pollutant and significantly contributes to hospitali

50 We aimed to investigate whether ambient air pollutant and traffic exposures in early gestation c

51 Associations between air pollutants and ACOS were evaluated using Cox regress

52 We investigated criteria air pollutants and air toxics during the period before c

53 d associations between perinatal exposure to air pollutants and autism spectrum disorder (ASD) in chi

54 Previous studies on air pollutants and birth outcomes have reported inconsis

55 studies have evaluated associations between air pollutants and cardiac arrhythmia.

56 supports a relationship between exposure to air pollutants and cardiovascular disease, but populatio

57 Inhalation exposure to indoor air pollutants and cigarette smoke increases the risk of

58 ease energy security and reduce emissions of air pollutants and CO2 from coal use, China is attemptin

59 tive associations between a diverse group of air pollutants and cognitive functioning in children and

60 nitric oxide production--may be affected by air pollutants and contribute to the pathogenesis of ast

61 cive to forest fires, which generate harmful air pollutants and damage agriculture, are likely to bec

62 fects models to estimate the main effects of air pollutants and effect modification by DNA methylatio

63 Regulation of air pollutants and efforts that focus on limiting childh

64 ical studies addressing maternal exposure to air pollutants and fetal growth during gestation as asse

65 is a key oxidant involved in the removal of air pollutants and greenhouse gases from the atmosphere.

66 pes of oxidized lipids formed in response to air pollutants and how this occurs and their relevance t

67 ich temperature increases with altitude-trap air pollutants and lead to higher pollutant concentratio

68 Wildfire smoke contains numerous hazardous air pollutants and many studies have documented populati

69 Interactions between exposure to ambient air pollutants and respiratory pathogens have been shown

70 kin aging, but no direct link between indoor air pollutants and skin aging manifestations has ever be

71 relationship between early-life exposure to air pollutants and subsequent asthma diagnosis.

72 e commuters experience increased exposure to air pollutants and that traffic characteristics, land us

73 relations between ambient concentrations of air pollutants and the production rates and concentratio

74 no studies evaluating the impact of ambient air pollutants and the propensity to develop type 2 diab

75 eview, we focus on the major constituents of air pollutants and their impacts on chronic respiratory

76 Emissions of air pollutants and their precursors determine regional a

77 Exposure to ambient air pollutants and traffic during pregnancy is associate

78 t moist meteorological conditions in winter, air pollutants and water vapour accumulate in a shallow

79 is paper, we assess the relationship between air pollutants and weather conditions with outpatient vi

80 ips between variants in CAT and MPO, ambient air pollutants, and acute respiratory illness.

81 diameter <2.5 mum [PM2.5] or <10 mum [PM10]) air pollutants, and heart failure hospitalisations or he

82 y inflammatory responses, including those to air pollutants, and is an asthma candidate gene.

83 ess to neurologists and exposure to selected air pollutants, and restricting to never movers and urba

84 is growing evidence that multiple genes and air pollutants are associated with asthma.

85 Ambient air pollutants are important determinants of bronchitis

86 global data on the health effects of indoor air pollutants are lacking.

87 ly, immune responses elicited by exposure to air pollutants are mediated by specific TLR- and NLR-dep

88 ologic studies show that increased levels of air pollutants are positively associated with cardiovasc

89 hat neither noise levels nor traffic-related air pollutants are responsible for this relationship.

90 resented here suggest that emissions of many air pollutants are significantly underestimated in curre

91 Traditional depictions of MB for criteria air pollutants are such that each additional ton of emis

92 Pulmonary responses to ozone, a common air pollutant, are augmented in obese individuals.

93 d carcinogen), 3-nitrobenzanthrone (an urban air pollutant), aristolochic acid (a component of Chines

94 rotocol, we assessed residential exposure to air pollutants as annual average concentrations of parti

95 oring site that is instrumented for criteria air pollutants, associated gases, and particle compositi

96 We evaluated the lag structure of air pollutant associations with lung function and potent

97 rotection Agency-modeled levels of hazardous air pollutants at the time and place of birth and ASD in

98 Radon is a pervasive indoor air pollutant believed to cause significant incidence of

99 surement platform to characterize a suite of air pollutants (black carbon (BC), particle-bound polycy

100 Residential exposures to traffic-related air pollutants (black carbon, particulate matter <2.5 mu

101 itting conditions in the truck fleet affects air pollutants by 20% to 65%; although these estimates a

102 y effects of exposure to biomass and traffic air pollutants by promoting awareness and supporting ind

103 ease, and post-translational modification by air pollutants can enhance the allergenic potential of p

104 Exposure to air pollutants can influence cardiac autonomic tone and

105 ehicle greenhouse gas (GHG) and conventional air pollutant (CAP) emissions to speed, weight, age, and

106 It is not known whether exposure to air pollutants causes systemic oxidative stress in child

107 A range of air pollutant classes was also added to the EVA sampler

108 eway closure on traffic and several criteria air pollutants (CO, NO2, O3, PM10, PM2.5).

109 ve a higher information gain than other gene-air pollutant combinations.

110 virtually complete spatially resolved global air pollutant concentration estimates.

111 attenuation factor," the ratio of the indoor air pollutant concentration to the pollutant concentrati

112 thickness and long-term exposure to ambient air pollutant concentrations (PM2.5, NOX, and black carb

113 t model (CMAQ) simulations to estimate daily air pollutant concentrations across Georgia.

114 ing fine scale spatial variations in average air pollutant concentrations across urban areas.

115 ictors in estimating on-road traffic-related air pollutant concentrations and GAMs perform better for

116 ities of air quality (including both average air pollutant concentrations and high pollution episodes

117 ed provides daily spatial field estimates of air pollutant concentrations and uncertainties that are

118 mum in diameter (PM2.5) and traffic-related air pollutant concentrations are associated with cardiov

119 eased birth weight associated with increased air pollutant concentrations during pregnancy.

120 d with interquartile range increases in mean air pollutant concentrations in the first, second, and t

121 Contributions from vaping to air pollutant concentrations in the home did not exceed

122 Air pollutant concentrations near major highways are usu

123 s is needed to achieve targets for household air pollutant concentrations or fuel savings.

124 captures the response of spatially resolved air pollutant concentrations to changes in electricity-g

125 associations of SES measures with predicted air pollutant concentrations, demonstrating the importan

126 e investigated short-term changes in ambient air pollutant concentrations, including speciated partic

127 To assess determinants of variability in air pollutant concentrations, we develop land use regres

128 his risk because a wide range of deleterious air pollutants contribute to the pathogenesis of asthma

129 ndings suggest that prenatal exposure to PAH air pollutants contributes to slower processing speed, a

130 humans, inhalational exposure to particulate air pollutants decreases heart rate variability, causes

131 Acute controlled exposure to air pollutants did not increase the short-term risk of a

132 l was to investigate the association between air pollutants, DNA methylation, and respiratory outcome

133 hort-term elevations in the level of ambient air pollutants does not increase the incidence of UGIB s

134 date found that exposure to certain ambient air pollutants during pregnancy is negatively associated

135 en exposed to high levels of traffic-related air pollutants during pregnancy.

136 omarker of oxidative stress, and exposure to air pollutants during various time windows of pregnancy.

137 We investigated maternal exposures to air pollutants during weeks 2-8 of pregnancy and their a

138 the advantages and challenges of quantifying air pollutant effects on ES in a decision making context

139 Adding a traffic-related air pollutant (elemental carbon) to models explained 20-

140 Air pollutant emission levels in China remain substantia

141 Difficulty in obtaining air pollutant emission permits and the cost of air pollu

142 rado O&G activities, production volumes, and air pollutant emission rates from two Colorado basins to

143 Our analysis of air pollutant emissions and controls can assist biorefin

144 development programs, often lack historical air pollutant emissions data, which can pose challenges

145 missions, fossil energy demand, and criteria air pollutant emissions for the GTW-biodiesel process, i

146 lity of federal air regulations and quantify air pollutant emissions for two feasibility-level bioref

147 We show that 50-60% of China's air pollutant emissions in 2007 were associated with goo

148 impacts of declining transportation-related air pollutant emissions on disparities in exposure have

149 The effects of international trade on air pollutant emissions, air quality and health have bee

150 that climate change, exclusive of changes in air pollutant emissions, can significantly impact ozone

151 mass combustion have become major sources of air pollutant emissions, with substantial spatial and te

152 ated external costs due to energy-associated air pollutant emissions.

153 Diesel vehicles are a major source of air pollutant emissions.

154 assume uniformly an aggressive reduction, of air pollutant emissions.

155 12 per cent (411,100 deaths) were related to air pollutants emitted in a region of the world other th

156 Mercury is a toxic air pollutant, emitted from the combustion of coal.

157 Our results suggest that exposure to air pollutants, especially traffic-related pollutants, m

158 sociations with concentrations of 11 ambient air pollutants estimated by combining Community Multisca

159 investigated associations between 11 ambient air pollutants, estimated by combining Community Multisc

160 rely on complete and accurate spatiotemporal air pollutant estimates.

161 ally related to observations linking ambient air pollutant exposure and adverse neurological/neurodev

162 A possible link between particulate air pollutant exposure and antimycobacterial immunity ha

163 were geocoded and used to assign cumulative air pollutant exposure estimates based on data derived f

164 Recent studies link air pollutant exposure to adverse neurodevelopmental out

165 The contribution of childhood air pollutant exposure to cardiovascular health has not

166 ession (LUR) models have been used to assess air pollutant exposure, but limited evidence exists on w

167 halation, which is the model traffic-related air pollutant exposure, is associated with vascular dysf

168 males-to the deleterious effects of prenatal air pollutant exposure, which may be due to a synergism

169 birth, we assessed long-term traffic-related air pollutant exposures (represented by nitrogen dioxide

170 fects models to examine associations between air pollutant exposures and SBP, taking into account mea

171 for estimating associations between CVDs and air pollutant exposures by providing clues about the und

172 ssess the interaction of maternal asthma and air pollutant exposures in relation to PTB risk.

173 Acute and long-term air pollutant exposures merit further study in relation

174 findings outline the lung-brain axis, where air pollutant exposures result in circulating, cytokine-

175 Air pollutant exposures were analyzed as both quintiles

176 ated the association of long- and short-term air pollutant exposures with flow-mediated dilation (FMD

177 r (10), before adjusting for risk factors or air pollutant exposures.

178 ric ozone is considered the most detrimental air pollutant for vegetation at the global scale, with n

179 asthma and data on inversions, weather, and air pollutants for Salt Lake County, Utah, during the wi

180 organic pollutants and the beneficial use of air pollutants for the removal of water pollutants since

181 c aromatic hydrocarbons (PAHs) are hazardous air pollutants formed during incomplete combustion.

182 d in a full-scale biological filter treating air pollutants from a pig facility.

183 atic hydrocarbons (PAH) are widespread urban air pollutants from fossil fuel burning and other combus

184 lly explicit life cycle inventories (LCI) of air pollutants from gasoline, ethanol derived from corn

185 d on a chemical mass balance model, criteria air pollutants from government monitoring data, and land

186 r when meteorological inversions concentrate air pollutants from oil and gas activities, but when sol

187 energy, are exposed to high levels of indoor air pollutants from the inefficient burning of biomass f

188 ement results and indicated the transport of air pollutants from wildfires burning in southern Califo

189 ry increases surface-level ozone, a criteria air pollutant, greenhouse gas and source of atmospheric

190 mental coexposures including traffic-related air pollutants, greenness, and neighborhood walkability.

191 and production sites the major stressors are air pollutants, ground and surface water contamination,

192 timate annual concentrations of 40 hazardous air pollutants (HAPs) across the continental United Stat

193 nogen in outdoor air among the 187 hazardous air pollutants (HAPs) identified by the U.S. Environment

194 e transformation and speciation of hazardous air pollutants (HAPs), including SO(2)/SO(3), NO(x), HCl

195 on (HC) species, including several hazardous air pollutants (HAPs).

196 Near-road exposure to air pollutants has been associated with decreased lung f

197 actions between GSTP1 and different types of air pollutants have a higher information gain than other

198 Air pollutants have been associated with childhood asthm

199 pulmonary effects of the individual criteria air pollutants have been well investigated, but little i

200 There are few large population-based multi-air pollutant health assessments.

201 The case study illustrates how air pollutant health impacts can be cost-effectively min

202 Indoor air pollutants (IAPs) cause multiple health impacts.

203 teractions with climate change variables and air pollutants, impacts of increased climate variability

204 and NO2 (collectively called NOx) are major air pollutants in automotive emissions.

205 ssessing the relative importance of specific air pollutants in different regions of the world, showin

206 f future work to further clarify the role of air pollutants in epidemiologic studies of extreme tempe

207 mias during and after controlled exposure to air pollutants in healthy volunteers and patients with c

208 Prenatal exposure to multiple air pollutants in the first trimester was associated wit

209 thacholine (MCh) and particulate matter (PM) air pollutants, in the absence of underlying inflammatio

210 nsights for understanding evolution steps of air pollutants including volatile organic carbons (VOCs)

211 sures to community noise and traffic-related air pollutants, including black carbon, particulate matt

212 Higher short-term exposure to air pollutants, including PM2.5, black carbon, and parti

213 The transient concentration of the other air pollutants, including PM2.5, PM10, and O3, did not s

214 Although exposure to ambient air pollutants increases cardiovascular disease risk in

215 Exposure to ambient air pollutants increases risk for cardiovascular health

216 ty income, or prenatal exposure to hazardous air pollutants, indicating that spatial variation is not

217 In experimental settings, ultrafine air pollutants instilled directly into the cardiac vascu

218 Increased levels of urban air pollutants interacting with parental stress have bee

219 The air pollutant is highly variable over time and space, wh

220 Exposure to traffic-related air pollutants is an important public health issue.

221 re to carbon monoxide (CO) and other ambient air pollutants is associated with adverse pregnancy outc

222 ed the hypothesis that perinatal exposure to air pollutants is associated with ASD, focusing on pollu

223 iologic literature suggests that exposure to air pollutants is associated with fetal development.

224 c studies that increased exposure to ambient air pollutants is associated with olfactory dysfunction.

225 Findings suggest that particle air pollutant levels in Canadian metros are substantiall

226 ssociations between the risk of CRAO and the air pollutant levels in the days preceding each event.

227 repeated biomarker measurements and ambient air pollutant levels.

228 two putative biological mechanisms by which air pollutants may adversely affect the brain.

229 st that prenatal exposure to traffic-related air pollutants may be associated with adverse birth outc

230 adverse health effects of ambient levels of air pollutants may be insidious and potentially underest

231 Air pollutants may impact anatomy and/or physiological f

232 Ambient air pollutants may increase preterm birth (PTB) risk, bu

233 Perinatal exposure to air pollutants may increase risk for ASD.

234 g in utero, environmental factors, including air pollutants, may permanently organize these systems t

235 ine concentrations of continuously monitored air pollutants measured in 2008 near a departure runway

236 Land use regression (LUR) models rely on air pollutant measurements for their development, and ar

237 The association persisted after air pollutant measures were added to the model, although

238 Regional air pollutant measures were based on the Environmental P

239 Prenatal exposure to ambient air pollutants might cause adverse birth outcomes; howev

240 human health impacts of exposure to complex air pollutant mixtures and the key features that drive t

241 e will summarize current knowledge about how air pollutants modify TLR- and NLR-dependent signaling a

242 We identified air pollutant monitoring stations located near these pat

243 del concentrations of mainly traffic-related air pollutants (nitrogen oxides (NOx), particulate matte

244 r concentrations of a transportation-related air pollutant, nitrogen dioxide (NO2), in the United Sta

245 We aimed to examine the effects of ambient air pollutants, NOS2 promoter haplotypes, and NOS2 promo

246 papers to estimate national mean effects of air pollutants on daily deaths in time-series analyses.

247 The effects of ECV and air pollutants on repeated wheeze (>/= 2 episodes) were

248 The effect of indoor air pollutants on respiratory morbidity among patients w

249 High concentrations of air pollutants on roadways, relative to ambient concentr

250 relationship between suicide and exposure to air pollutants on the day of the suicide and during the

251 ntial confounders, including traffic-related air pollutants or noise, elevations in noise and black c

252 idence suggests that longer-term exposure to air pollutants over years confers higher risks of cardio

253 of air humidity, darkness, and pulses of the air pollutant ozone (O3), involves the SLOW ANION CHANNE

254 to estimate average annual exposure to five air pollutants: ozone, nitrogen dioxide (NO(2)), sulfur

255 estimate on-road concentrations of four key air pollutants, particle-bound polycyclic aromatic hydro

256 at were robust to adjustment by other common air pollutants (particles <2.5 mum in diameter, ozone, n

257 to humans after a single exposure to a toxic air pollutant, particularly when in the presence of unde

258 ngs suggest that early exposures to criteria air pollutants, particularly from transport emissions, a

259 s were used to estimate associations between air pollutants, percent DNA methylation, and respiratory

260 Formaldehyde is an air pollutant present in both indoor and outdoor atmosph

261 exposure to outdoor carbon monoxide (CO), an air pollutant primarily generated by traffic, is inconsi

262 From a mechanistic perspective, air pollutants probably cause oxidative injury to the ai

263 Acrolein (Acr), a hazardous air pollutant, reacts readily with deoxyguanosine (dG) i

264 ts of air pollution and accurately assessing air pollutant-related asthma outcomes are needed to bett

265 A large portion of air pollutants result from residential combustion of sol

266 entified as an important source of ultrafine air pollutants resulting in elaborated treatment systems

267 alibration method was proposed for low-level air pollutant sampling using this high capacity membrane

268 tact inhaled inflammatory insults, including air pollutants, smoke, and microorganisms.

269 pigenetic regulation of responses to ambient air pollutants, specifically respirable PM, and their as

270 d by a substantial increase of certain other air pollutants, specifically the number of ultrafine par

271 We recently reported that air pollutants such as cigarette smoke and cadmium negat

272 otosynthesis, loss of water, and entrance of air pollutants such as ozone.

273 deling, and episodic exacerbations caused by air pollutants, such as particulate matter (PM; PM <2.5

274 ur heavy fuel oil are an important source of air pollutants, such as sulfur dioxide and particulate m

275 Indoor air pollutants [sulfur dioxide (SO2), nitrogen dioxide (

276 production of ground-level ozone (O(3)), an air pollutant that damages human health, plants, and mat

277 door CO2 with concentrations of other indoor air pollutants that are also influenced by rates of outd

278 oad transport remains an important source of air pollutants that are linked with acute and chronic he

279 to personal exposures to complex mixtures of air pollutants that change rapidly in space and time bec

280 ictors to resolve the spatial variability of air pollutants together with complementary national esti

281 This interaction controls thermal and air pollutant transport and distribution.

282 cific associations between breast cancer and air pollutants using Cox regression models, adjusting fo

283 We assessed the women's exposure to air pollutants using the monitor located nearest to each

284 nt method), and confounding by other gaseous air pollutants were also investigated.

285 The associations between CIMT and air pollutants were assessed using linear regression ana

286 Several ambient air pollutants were associated with preterm birth; assoc

287 Complete monitoring data for the ambient air pollutants were collected from Taiwan Environmental

288 Ambient air pollutants were dominated by fine and ultrafine part

289 residence-level exposures to traffic-related air pollutants were estimated using a land use regressio

290 ent for coexposure to noise, traffic-related air pollutants were not associated with the incidence of

291 In 2012 air pollutants were responsible of seven million human d

292 Concentrations of UFP and other air pollutants were simultaneously monitored in and out

293 Nitrogen dioxide is a common air pollutant with growing evidence of health impacts in

294 Numerous studies have linked criteria air pollutants with adverse birth outcomes, but there is

295 .5 mum in diameter (PM2.5) and other ambient air pollutants with adverse birth outcomes; yet, to our

296 ons not only of greenhouse gases but also of air pollutants with high radiative forcing (RF), particu

297 PM2.5 mortality costs per tonne of inorganic air pollutants with the 36 km x 36 km spatial resolution

298 All the main air pollutants, with the exception of ozone, were signif

299 All the main air pollutants, with the exception of ozone, were signif

300 Tropospheric ozone is a serious air-pollutant, with large impacts on plant function.