ライフサイエンスコーパス: airflow

1 scle, thus airway diameter and resistance to airflow.

2 , and that drug deposition is independent of airflow.

3 d have a high sampling efficiency and a high airflow.

4 icularly trees, have the potential to affect airflow, air quality, and production of aeroallergens.

5 acute respiratory distress syndrome in whom airflow, airway pressure, and esophageal pressure were r

6 seed dispersal by redirecting and bellowing airflow and by increasing the likelihood of seed uplift.

7 control of song production, both can affect airflow and frequency.

8 cally presents with a diurnal variability in airflow and is a characteristic that is usually not seen

9 gh-level bending of the micropillar under an airflow and its optical read-out enables mm s(-1) scale-

10 hanges in airway dynamics impairs expiratory airflow and leads to progressive air trapping.

11 s that can be used to measure nasal patency, airflow and resistance, mainly peak nasal inspiratory fl

12 rons to detect increases in the wing-induced airflow and that JO neurons are involved in a response t

13 was delivered with respect to initiation of airflow as variation in this parameter would influence l

14 Frontal airflow, as experienced during forward flight, causes an

15 e of oxygen dependence at 36 weeks rose, and airflows at 8 years of age were worse in 2005 than in ea

16 ocal fold oscillation is passively driven by airflow between the lungs and upper respiratory spaces,

17 A high airflow blower enables rapid cooling.

18 tive expiration and reduced early expiratory airflow but only during wake.

19 ir surrounding the pileus creates convective airflows capable of carrying spores at speeds of centime

20 It has long been desired to match airflow conditions during formulation evaluation to thos

21 than polyester filters under similar RH and airflow conditions.

22 e measured in surgically opened nose without airflow constraints is similar to the shape of the sorpt

23 y administration of azithromycin can improve airflow decline-free survival after allogeneic HSCT.

24 The primary efficacy end point was airflow decline-free survival at 2 years after randomiza

25 nistration of azithromycin resulted in worse airflow decline-free survival than did placebo; these fi

26 ithromycin vs 50 placebo) had experienced an airflow decline; 138 patients (30%) died (78 azithromyci

27 hma by age 7 (14%) already had a significant airflow deficit as neonates (forced expiratory flow at 5

28 trial protocol as a significant clinical or airflow deterioration), and the secondary outcome was as

29 volutionary scenarios: either unidirectional airflow evolved independently in archosaurs and monitor

30 mouth, highlighting the importance of nasal airflow for generating respiratory oscillations.

31 ent (15%-25% relative humidity and 2-5 L/min airflow) for 90 minutes.

32 Observations made in westerly airflow (from parts of London where traffic is a smaller

33 ut not allergic sensitization, also hampered airflow growth.

34 tive expiration and reduced early expiratory airflow (i.e. increased upper airway resistance) only du

35 ajor risk factor for developing this chronic airflow impairment, but the early progression of disease

36 s can be tested by investigating patterns of airflow in members of the outgroup to birds and crocodil

37 ons project a westward shift of anticyclonic airflow in summer, but uncertainty is larger for spring

38 Here we show unidirectional airflow in the green iguana, a lizard with a strikingly

39 e demonstrate region-specific unidirectional airflow in the lungs of the savannah monitor lizard (Var

40 The expiratory time constants of regional airflows in the segmented airway tree were quantified as

41 By fast manipulation of the airflows into the chamber, a step-change in RH over a ti

42 a oscillations, and are abolished when nasal airflow is bypassed by tracheotomy.

43 If unidirectional airflow is plesiomorphic for Diapsida, this respiratory

44 the avian respiratory system, unidirectional airflow, is that it is an adaptation for efficiency of g

45 patients with fixed as compared to variable airflow limitation (69.76 vs 43.84 pg/ml, P < 0.05) and

46 Most patients with airflow limitation (70.6%) had no previous spirometry te

47 osure (PE) to dust and fumes (P = 0.006) and airflow limitation (AFL) (P = 0.033).

48 measures as z-score, and a classification of airflow limitation (AL) based on this parameter has rece

49 xpression was also associated with increased airflow limitation (FEV1/forced vital capacity and resid

50 associated with a higher risk of subsequent airflow limitation (odds ratio [95% confidence interval]

51 ated with increased risk of incident stage 2 airflow limitation (ratio of FEV1 to forced expiratory v

52 Enrollment criteria included irreversible airflow limitation and AECOPD requiring corticosteroids,

53 We sought to prospectively assess airflow limitation and airway inflammation in children 4

54 P < 0.001), which increased with severity of airflow limitation and are suggestive of hypertensive or

55 ion does not mediate the association between airflow limitation and atherosclerosis.

56 Instead, airflow limitation and endothelial dysfunction seem to b

57 ith asthma had more respiratory symptoms and airflow limitation and higher levels of inflammatory and

58 nificantly down-regulated in smokers without airflow limitation and in patients with COPD compared wi

59 tein level, was decreased in smokers without airflow limitation and in patients with COPD, and correl

60 lable, and inexpensive global measurement of airflow limitation and lung function.

61 ributable to variation in the definitions of airflow limitation and the treatment of people with asth

62 and CMH, how symptoms during life related to airflow limitation at 60-64 years, and how CMH duration

63 Airflow limitation at day 0 was reversible after broncho

64 ermediate-onset wheezers showed irreversible airflow limitation by 18 years.

65 y disease (COPD) is characterized by chronic airflow limitation caused by a combination of airways di

66 Airflow limitation compatible with chronic obstructive p

67 tinguishable, but many patients with chronic airflow limitation demonstrate features of both conditio

68 flammation-predominant asthma and persistent airflow limitation despite high-intensity anti-inflammat

69 ations with decline in FEV1 and incidence of airflow limitation for adults who were free from COPD at

70 els in sputum are associated with persistent airflow limitation in asthma patients with airway eosino

71 The aim of the ALICE (Airflow Limitation in Cardiac Diseases in Europe) study

72 Emphysema is a key contributor to airflow limitation in chronic obstructive pulmonary dise

73 C because of CS exposure might contribute to airflow limitation in COPD.

74 ) study was to investigate the prevalence of airflow limitation in patients with ischemic heart disea

75 etic factors and is strongly associated with airflow limitation in smaller airways.

76 Airway remodeling burden is not limited to airflow limitation in the assessment of COPD severity an

77 re less likely to have undergone testing for airflow limitation in the community at the time of initi

78 ne in adulthood, and development of moderate airflow limitation in the general adult population.

79 iation between atopy and post-bronchodilator airflow limitation in the general population aged 40 yea

80 iation between atopy and post-bronchodilator airflow limitation in the general population appears to

81 ls from 4,724 subjects with mild-to-moderate airflow limitation in the Lung Health Study.

82 Although airflow limitation is associated with additional morbidi

83 Treatments for airflow limitation might improve survival and both respi

84 h emphysema were matched for the severity of airflow limitation of those with bronchiolitis.

85 disease (COPD) is defined by the presence of airflow limitation on spirometry, yet subjects with COPD

86 g people with known pulmonary disease and/or airflow limitation on spirometry.

87 The no airflow limitation or air-trapping criteria (None) pheno

88 nt asthma (defined as wheeze and presence of airflow limitation or airway hyper-reactivity, or both).

89 ficant for spirometric phenotypes related to airflow limitation or COPD.

90 The airflow limitation phenotype (A Limit) had an FEV1/FVC z

91 e pulmonary component is characterized by an airflow limitation that is not fully reversible.

92 y, 6-AMCH, DHC and 4-OPA would not result in airflow limitation to the airways.

93 Airflow limitation was associated with greater respirato

94 Airflow limitation was defined as post-bronchodilator FE

95 Airflow limitation was defined as post-bronchodilator sp

96 Airflow limitation was observed in 30.5% of patients wit

97 assessment of (1) symptoms, (2) severity of airflow limitation, (3) history of exacerbations, and (4

98 with ischemic heart disease: 11.3% had mild airflow limitation, 15.8% moderate airflow limitation, 3

99 ator spirometry, to identify the presence of airflow limitation, 18,475 subjects (99%) were assigned

100 had mild airflow limitation, 15.8% moderate airflow limitation, 3.3% severe airflow limitation, and

101 .8% moderate airflow limitation, 3.3% severe airflow limitation, and 0.1% very severe airflow limitat

102 mpassing 8 never-smokers, 10 smokers without airflow limitation, and 12 smokers with COPD.

103 by enhanced airway inflammation, reversible airflow limitation, and asthma-related symptoms.

104 d in lungs of never-smokers, smokers without airflow limitation, and patients with COPD; and in C57BL

105 but without current or previous evidence of airflow limitation, bronchial reversibility, or airway h

106 With regard to the causes of airflow limitation, CT can be used to quantify the two m

107 COPD is characterized by persistent airflow limitation, neutrophilia and oxidative stress fr

108 ntiating asthma from other causes of chronic airflow limitation, such as chronic obstructive pulmonar

109 ic obstructive pulmonary disease with severe airflow limitation, symptoms of chronic bronchitis, and

110 commended for patients with COPD with severe airflow limitation, symptoms of chronic bronchitis, and

111 ording to simple clinical measures (level of airflow limitation, symptoms, and frequency of previous

112 a with low cumulated smoking exposure and no airflow limitation, those with COPD, those with asthma-C

113 ditional common characteristic is reversible airflow limitation.

114 ith COPD and explore their relationship with airflow limitation.

115 ed with the AA genotype demonstrated greater airflow limitation.

116 ere airflow limitation, and 0.1% very severe airflow limitation.

117 rrelated with the type 2 immune response and airflow limitation.

118 y disease (COPD) is characterized by chronic airflow limitation.

119 post-bronchodilator measures for those with airflow limitation.

120 deration in the diagnosis of asthma based on airflow limitation.

121 lly characterized by incompletely reversible airflow limitation.

122 y variable respiratory symptoms and variable airflow limitation.

123 decline is a rare feature of biomass-induced airflow limitation.

124 be associated with severe exacerbations and airflow limitation.

125 diseases, both of which are characterized by airflow limitation.

126 d to ensure proper evaluation of severity of airflow limitation.

127 isk increased significantly with severity of airflow limitation.

128 status and smoking history, and severity of airflow limitation.

129 roke and the risk increases with severity of airflow limitation.

130 on was stratified by country and severity of airflow limitation.

131 ults in largely irreversible and progressive airflow limitation.

132 Here we use 3D airflow modelling to demonstrate that local dune topogra

133 Airflow models at length scales relevant to landform siz

134 do discriminate well, they do so with lower airflow, more sniffs, and lower frequency of sniffing th

135 ment periods for total symptom scores, nasal airflow, nasal secretion weight, and nasal congestion sc

136 d safety, nasal and nonnasal symptoms, nasal airflow, nasal secretions, basophil activation, and plas

137 COPD (198 [4%] of 4439) and smokers without airflow obstruction (255 [5%] of 5547).

138 To examine sex differences in the risk of airflow obstruction (a COPD hallmark) in relation to smo

139 acute exacerbation (AE) in patients without airflow obstruction (Asthma AE group).

140 ed physical activity (multisensory armband), airflow obstruction (FEV1), health status (St. George's

141 Moderate/severe airflow obstruction (FEV1/forced vital capacity <0.70 an

142 f lung for carbon monoxide (Dlco%) than with airflow obstruction (forced expiratory volume in 1 secon

143 .16, 1.03-1.32), and incompletely reversible airflow obstruction (RR 1.28, 1.04-1.57) than did those

144 ntial shared genetic architecture underlying airflow obstruction across individuals, irrespective of

145 acute respiratory events in smokers without airflow obstruction affect lung function decline is unkn

146 that could be related to symptom control and airflow obstruction after standardized treatment.

147 but not total IgE, is associated with fixed airflow obstruction and a number of radiological abnorma

148 Our findings suggest that early airflow obstruction and air trapping in infants with cys

149 t PiMZ heterozygotes have significantly more airflow obstruction and COPD than PiMM individuals and c

150 control subjects and influenced by worsening airflow obstruction and corticosteroid use.

151 al dysfunction, which results in progressive airflow obstruction and debility.

152 apoA-I and large HDLNMR particles can reduce airflow obstruction and disease severity in asthma.

153 iated with a significantly increased risk of airflow obstruction and emphysema but the risk of chroni

154 medium-dose inhaled corticosteroids reduces airflow obstruction and improves asthma control in patie

155 begins in infancy or childhood with variable airflow obstruction and intermittent wheezing, cough, an

156 response (BDR) reflects the reversibility of airflow obstruction and is recommended as an adjunctive

157 early-onset nonsevere asthma and reversible airflow obstruction and normal airway structure.

158 ung function are at increased risk for fixed airflow obstruction and possibly COPD in early adulthood

159 constrict airway smooth muscle, but elicits airflow obstruction and pulmonary inflammation in patien

160 mmunological biomarkers are related to fixed airflow obstruction and radiological abnormalities in mo

161 Fixed airflow obstruction and radiological abnormalities were

162 Understanding the genetic basis of airflow obstruction and smoking behaviour is key to dete

163 ve effects of etoricoxib on allergen-induced airflow obstruction and sputum eosinophils, basal lung f

164 In adults, differences in airflow obstruction and symptoms between SA and NONSA pe

165 Preliminary studies have shown that both airflow obstruction and systemic inflammation may contri

166 nd to lesser extent of Twist, was related to airflow obstruction and to expression of a canonical EMT

167 m the association between skin wrinkling and airflow obstruction and to identify genetic polymorphism

168 particular showed a direct correlation with airflow obstruction and treatment requirement in patient

169 We found no association between airflow obstruction and use of solid fuels for cooking o

170 pulmonary disease, who had at least moderate airflow obstruction and were taking part in PR, were ran

171 Air trapping and airflow obstruction are being increasingly identified in

172 Airflow obstruction assessed from post-bronchodilator sp

173 eling and contributes to the mucus plugs and airflow obstruction associated with severe asthma phenot

174 osing COPD in patients with mild to moderate airflow obstruction at baseline.

175 is similar to cluster T2 in terms of chronic airflow obstruction but is composed of nonsmokers.

176 us by goblet cells, which leads to worsening airflow obstruction by luminal obstruction of small airw

177 d-onset persistent asthma is associated with airflow obstruction by mid-adult life, but this does not

178 for INtrinsic and EXtrinsic skin Aging) and airflow obstruction by spirometry, using the ratio of fo

179 ontrol, recurrent exacerbations, and chronic airflow obstruction despite adequate and, in many cases,

180 Patients with moderate chronic airflow obstruction experienced a reduction in exacerbat

181 Asthmatics with persistent airflow obstruction had greater airway smooth muscle (As

182 A proportion of 26.3% of smokers without airflow obstruction had ND-E/I greater than the 90th per

183 tantial proportion of subjects without overt airflow obstruction have significant respiratory morbidi

184 Cluster analysis of adults with symptomatic airflow obstruction identifies 5 disease phenotypes, inc

185 f the gut-lung axis can be targeted to treat airflow obstruction in asthma.

186 all conducting airways are the major site of airflow obstruction in chronic obstructive pulmonary dis

187 fferences in gene expression were related to airflow obstruction in epithelial cells (C3, ALOX5AP, CC

188 be a marker of neutrophilic inflammation and airflow obstruction in patients with asthma, who have a

189 ated in relation to both symptom control and airflow obstruction in severe asthmatics.

190 function, greater risk of the development of airflow obstruction in smokers, a predisposition to lowe

191 usceptible (n = 64) to emphysema with severe airflow obstruction in the Pittsburgh Specialized Center

192 ched among genes associated with more severe airflow obstruction in these COPD cohorts (P < 0.001), s

193 ammation and remodeling, although persistent airflow obstruction in these patients was associated wit

194 Cohort studies suggest that airflow obstruction is established early in life, manife

195 In asthma, airflow obstruction is thought to result primarily from

196 EBC LXA4 levels correlate with the degree of airflow obstruction measured by using FEV1 (r = 0.28, P

197 ndent association between skin wrinkling and airflow obstruction of the lung.

198 th all-cause mortality among persons without airflow obstruction or COPD in a general population samp

199 iratory tract disease can manifest itself as airflow obstruction or viral pneumonia, which can be fat

200 th muscle function and may contribute to the airflow obstruction phenotype observed in human CF.

201 h severe uncontrolled asthma and the chronic airflow obstruction phenotype.

202 Their effects in patients with milder airflow obstruction remain unclear.

203 ations of asthma are thought to be caused by airflow obstruction resulting from airway inflammation,

204 r matrix (ECM) processes specifically in the airflow obstruction study.

205 se of smoking, women showed a higher risk of airflow obstruction than men.

206 syndrome (BOS) is a condition of progressive airflow obstruction that affects a majority of lung tran

207 COPD is characterised by poorly reversible airflow obstruction usually due to cigarette smoking.

208 ver diagnosed asthma and post-bronchodilator airflow obstruction was 44.8%, 19.3% and 7.5%, respectiv

209 Airflow obstruction was assessed by FEV1% predicted.

210 Increasing PRM(FSA) in subjects without airflow obstruction was associated with increased FVC (P

211 bution relative to PRM(FSA) in those without airflow obstruction was limited by its low burden.

212 Lack of airflow obstruction was the principal (89%) reason patie

213 lity and the predictors of age, dyspnea, and airflow obstruction were available.

214 eparate models for subjects without and with airflow obstruction were generated using baseline clinic

215 s were obese female patients with reversible airflow obstruction who exhibited airway wall thickening

216 te-onset older male subjects with persistent airflow obstruction who exhibited significant air trappi

217 In both sexes, the association of airflow obstruction with cigarettes per day, smoking dur

218 To assess the association of airflow obstruction with self-reported use of solid fuel

219 The association of airflow obstruction with smoking characteristics was ass

220 The association of airflow obstruction with smoking status was stronger in

221 re was also no evidence of an association of airflow obstruction with use of solid fuels (ORmen=1.00,

222 The association of airflow obstruction with use of solid fuels for cooking

223 idence supporting the association of COPD or airflow obstruction with use of solid fuels is conflicti

224 methods with GWASs of pulmonary function and airflow obstruction would identify a broader repertoire

225 Perform meta-analyses of GWAS for airflow obstruction, a key pathophysiologic characterist

226 In subjects without airflow obstruction, acute respiratory events were not a

227 In never- and ever-smokers without airflow obstruction, aging is associated with increased

228 etric abnormalities, and was correlated with airflow obstruction, air trapping, and diffusing capacit

229 common chronic lung disease characterized by airflow obstruction, airway hyperresponsiveness (AHR), a

230 Asthma is characterized by variable airflow obstruction, airway inflammation, airway hyper-r

231 vel of CerS2 was associated with significant airflow obstruction, airway inflammation, and increased

232 irways hyperresponsiveness (AHR), reversible airflow obstruction, airway remodeling, and episodic exa

233 and 80% had late-onset asthma, 50% had fixed airflow obstruction, and 66% showed a Th2-high phenotype

234 The presence of air trapping, airflow obstruction, and airway size reduction in newbor

235 yper-responsiveness, incompletely reversible airflow obstruction, and asthma-related school and work

236 al-appearing lung regions in smokers without airflow obstruction, and it is associated with respirato

237 smooth muscle bronchoconstriction leading to airflow obstruction, and mucous hypersecretion are clini

238 ously that lower respiratory tract symptoms, airflow obstruction, and neutrophilic airway inflammatio

239 d in lungs of never smokers, smokers without airflow obstruction, and patients with COPD by reverse t

240 ovel candidate gene in emphysema with severe airflow obstruction, and rs61754411 is a previously unre

241 ilic inflammation, the attributes of chronic airflow obstruction, and the notion of corticosteroid in

242 before age 18 years were more likely to have airflow obstruction, but a sex difference in this associ

243 ve or multiplicative effects on the risk for airflow obstruction, but this has not been demonstrated

244 ight into the specific mechanisms underlying airflow obstruction, COPD, and tobacco addiction, and sh

245 to -13.2]; p=0.006), higher Body Mass Index, Airflow Obstruction, Dyspnea, and Exercise Capacity (BOD

246 total score (SGRQ), and the body mass index, airflow obstruction, dyspnea, and exercise capacity (BOD

247 to -8.556; P < 0.001), and body mass index, airflow obstruction, dyspnea, and exercise capacity inde

248 ore, -0.6 points; and BODE (body mass index, airflow obstruction, dyspnea, and exercise capacity) ind

249 OPD assessment test scores, Body-mass index, airflow Obstruction, Dyspnea, and Exercise index, or Glo

250 morbidity, more severe BDR and BHR, greatest airflow obstruction, high smoking prevalence, higher sym

251 tive associations with emphysema with severe airflow obstruction, including a suggestive association

252 isorder marked by inflammation and recurrent airflow obstruction, is associated with elevated levels

253 ute worsening of asthma symptoms, reversible airflow obstruction, or bronchial hyperresponsiveness af

254 exacerbations include previous exacerbation, airflow obstruction, poor overall health, home oxygen us

255 linear regression, for participants without airflow obstruction, PRM(fSAD) but not PRM(emph) was ass

256 n men and women is similarly associated with airflow obstruction, respiratory symptoms, more emphysem

257 sex), NO2 levels were associated highly with airflow obstruction, such that each 10-ppb increase in N

258 tics of chronic persistent asthma, including airflow obstruction, use of corticosteroid medications,

259 sorders encompassing different phenotypes of airflow obstruction, which might differ in their respons

260 alyses were conducted to identify effects on airflow obstruction, YKL-40 levels, and asthma severity.

261 with increased cough, sputum production, and airflow obstruction.

262 links between asthma and subsequent chronic airflow obstruction.

263 co smoking are risk factors for irreversible airflow obstruction.

264 a have additive or multiplicative effects on airflow obstruction.

265 io are considered the standard assessment of airflow obstruction.

266 ol, and apoB are associated with more severe airflow obstruction.

267 NO2 levels can be associated with increased airflow obstruction.

268 richment analysis to a meta-analyzed GWAS of airflow obstruction.

269 y, cell adhesion, epigenetic regulation, and airflow obstruction.

270 the presence of comorbidities compared with airflow obstruction.

271 the lungs that leads to progressive chronic airflow obstruction.

272 erapy in 5,887 smokers with mild to moderate airflow obstruction.

273 der characterized by incompletely reversible airflow obstruction.

274 PRM(FSA) and age in subjects with or without airflow obstruction.

275 asthma with a history of smoking and chronic airflow obstruction.

276 ts with mild to moderate post-bronchodilator airflow obstruction.

277 iation contributing to emphysema with severe airflow obstruction.

278 stratified by asthma symptom control and by airflow obstruction.

279 ncreased in both groups with the severity of airflow obstruction.

280 acity (FVC) ratio is used as a criterion for airflow obstruction; however, the test characteristics o

281 BVC profiling via dynamic (i.e., continuous airflow) or static headspace sampling using solid-phase

282 and the extent and strength of anticyclonic airflow over eastern North America varies with season.

283 The unidirectional airflow patterns in the lungs of birds have long been co

284 ocodilians suggest that these structures and airflow patterns may be homologous.

285 including: misalignments, propeller-induced airflows, power loss, intermodal crosstalk, and system b

286 lse responses convolved with the respiratory airflow predict the classical respiration-locked firing

287 was (1) to determine what characteristics of airflow predicted HFIS intensity, and (b) to determine i

288 whether animals make use of odorant sorption-airflow relationships as part of an active odor-sampling

289 ew structure's utility, we configure it into airflow sensors, in which the micropillars and microsphe

290 ape of the sorption profile imposed by nasal airflow, strongly indicating a tuning process.

291 B4) phase under topographic confinement with airflow that can induce a shear force and temperature gr

292 data (n = 135); level 3 (L3), which included airflow, thoracoabdominal bands, body position, electroc

293 e conducted to determine fan curves relating airflow to duct static pressure, sound levels, and exhau

294 frequency sound intensity was an estimate of airflow turbulence as reflected by the Reynold's number

295 Lower airflow values are present by spirometry for prepubertal

296 Expiratory airflow was measured at 8 years of age, and values were

297 mol; P=.01), whereas differences in baseline airflow were not significant for forced expiratory volum

298 adband, passive, low-cost approach to detect airflow with full fidelity over a frequency bandwidth th

299 dimensional spider silk captures fluctuating airflow with maximum physical efficiency (Vsilk/Vair app

300 ddenly rupture and are replaced with a rapid airflow with the characteristic slurping sound.