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1 who exhibited airway wall thickening without airway narrowing.
2 ; in allergic mice, gVPLA2 caused persistent airway narrowing.
3 erapeutic target in CF and other diseases of airway narrowing.
4 nges in enhanced pause (Penh) as an index of airway narrowing.
5 MCh challenge are associated with conducting airway narrowing.
6 ilies also suggests a propensity for dynamic airway narrowing.
7 n of integrin alpha9beta1 increased in vitro airway narrowing and airway smooth muscle contraction in
8                              They both cause airway narrowing and are increasing in incidence through
9 s been linked to complex behaviour in sudden airway narrowing and avalanche-like reopening.
10 ctively structural abnormalities involved in airway narrowing and bronchial reactivity, particularly
11 ubjects, asthmatics have (1) more collateral airway narrowing and closure and lower segmental complia
12 truct, to our knowledge, a new model wherein airway narrowing and closure dynamics are modulated by c
13 s via the regulation of airway inflammation, airway narrowing and remodelling.
14 n obtaining objective evidence of reversible airways narrowing and inflammation.
15 cked fully gVPLA2-induced cell migration and airway narrowing as marked by reduction of migrating leu
16 airway dilatation) at low airway volumes and airway narrowing at high airway volumes (P < 0.05).
17 , co-activation produced volume compression (airway narrowing) at large airway volumes (P < 0.05), bu
18                                   To confirm airway narrowing by MCh in this setting and to determine
19 of nonspecific agonists and that the induced airway narrowing can be reversed by the inhalation of a
20 duals develop this condition, the associated airway narrowing can materially interfere with performan
21 steinyl leukotrienes are potent mediators of airway narrowing derived from the lipoxygenation of arac
22                           The maintenance of airway narrowing despite MCh clearance in humans is attr
23                    With placebo, significant airway narrowing developed at all times (mean [+/-SE] de
24 yngeal walls play an important role in upper airway narrowing during sleep in normal subjects.
25 eta6 integrin are protected from exaggerated airway narrowing in a model of allergic asthma.
26 s naturally to the suggestion that excessive airway narrowing in asthma may be associated with the de
27 cluding cough, and are a potent stimulus for airway narrowing in asthmatic patients, but the mechanis
28  trapping, with a contribution from proximal airway narrowing in asthmatic patients.
29  gVPLA2 mutant with reduced activity, caused airway narrowing in immune-sensitized mice.
30 on of goblet cells may contribute to chronic airway narrowing in moderate asthma.
31         Asthma is characterized by increased airway narrowing in response to nonspecific stimuli.
32 id peroxidation is associated with pulmonary airway narrowing in the general population.
33                                              Airway narrowing in the RP region was associated with a
34 sthma, the mechanisms by which they regulate airway narrowing in vivo remain to be elucidated.
35                                              Airway narrowing is maintained for a prolonged period af
36 cells may contribute at least in part to the airway narrowing observed in patients with asthma.
37 l area, particularly during inspiration; (2) airway narrowing occurred during inspiration without evi
38 symptoms and the demonstration of reversible airways narrowing on lung function testing, which is dif
39 nd (2) after challenge, increased collateral airway narrowing or closure without a change in complian
40 ation is not correlated with the severity of airway narrowing or the severity of bronchial hyperrespo
41                                              Airway narrowing showed no predilection for particular a
42 ed bronchoconstriction (EIB) describes acute airway narrowing that occurs as a result of exercise.
43 ion has been postulated to contribute to the airway narrowing that occurs following exercise or hyper
44 ary disease and asthma by 1) contributing to airway narrowing through hyperplasia and hypertrophy and
45  hyperventilation (ISH) is believed to cause airway narrowing through noninflammatory mechanisms.
46 ogically and by differential cell count, and airway narrowing was measured by calibrated micrometry.
47  these changes include sputum production and airway narrowing, which lead to asthma exacerbations.
48  phase reaction (EPR), that leads to maximal airway narrowing within 15-30 min, followed by a recover
49       Because loss of T(H)17 cells inhibited airway narrowing without any obvious effects on airway i

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