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1 who exhibited airway wall thickening without airway narrowing.
2 ; in allergic mice, gVPLA2 caused persistent airway narrowing.
3 erapeutic target in CF and other diseases of airway narrowing.
4 nges in enhanced pause (Penh) as an index of airway narrowing.
5 MCh challenge are associated with conducting airway narrowing.
6 ilies also suggests a propensity for dynamic airway narrowing.
7 n of integrin alpha9beta1 increased in vitro airway narrowing and airway smooth muscle contraction in
10 ctively structural abnormalities involved in airway narrowing and bronchial reactivity, particularly
11 ubjects, asthmatics have (1) more collateral airway narrowing and closure and lower segmental complia
12 truct, to our knowledge, a new model wherein airway narrowing and closure dynamics are modulated by c
15 cked fully gVPLA2-induced cell migration and airway narrowing as marked by reduction of migrating leu
17 , co-activation produced volume compression (airway narrowing) at large airway volumes (P < 0.05), bu
19 of nonspecific agonists and that the induced airway narrowing can be reversed by the inhalation of a
20 duals develop this condition, the associated airway narrowing can materially interfere with performan
21 steinyl leukotrienes are potent mediators of airway narrowing derived from the lipoxygenation of arac
26 s naturally to the suggestion that excessive airway narrowing in asthma may be associated with the de
27 cluding cough, and are a potent stimulus for airway narrowing in asthmatic patients, but the mechanis
37 l area, particularly during inspiration; (2) airway narrowing occurred during inspiration without evi
38 symptoms and the demonstration of reversible airways narrowing on lung function testing, which is dif
39 nd (2) after challenge, increased collateral airway narrowing or closure without a change in complian
40 ation is not correlated with the severity of airway narrowing or the severity of bronchial hyperrespo
42 ed bronchoconstriction (EIB) describes acute airway narrowing that occurs as a result of exercise.
43 ion has been postulated to contribute to the airway narrowing that occurs following exercise or hyper
44 ary disease and asthma by 1) contributing to airway narrowing through hyperplasia and hypertrophy and
46 ogically and by differential cell count, and airway narrowing was measured by calibrated micrometry.
47 these changes include sputum production and airway narrowing, which lead to asthma exacerbations.
48 phase reaction (EPR), that leads to maximal airway narrowing within 15-30 min, followed by a recover
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