ライフサイエンスコーパス: airway pressure

1 /- 7% (P < 0.0001) going from 5 to 45 cm H2O airway pressure.

2 ts who are intolerant to continuous positive airway pressure.

3 roduction of less invasive forms of positive airway pressure.

4 tomography measurements when increasing mean airway pressure.

5 reduced extravascular lung water and plateau airway pressure.

6 y small tidal volumes cycling at a high mean airway pressure.

7 te ventilation when challenged with negative airway pressure.

8 line of therapy is nasal continuous positive airway pressure.

9 ol subjects in the fractional volumes at any airway pressure.

10 oss-sectional area was decreased by lowering airway pressure.

11 in the duration of nasal continuous positive airway pressure.

12 spontaneous breathing or continuous positive airway pressure.

13 ay pressure, but can be controlled under low airway pressure.

14 less than 34 weeks under continuous positive airway pressure.

15 essure support on top of expiratory positive airway pressure.

16 ng CT scans were obtained at 5 and 45 cm H2O airway pressure.

17 irst hours of life under continuous positive airway pressure.

18 xia or respiratory acidosis and high plateau airway pressures.

19 be augmented or sustained with generous mean airway pressures.

20 oration in gas exchange and the increases in airway pressures.

21 litude (50, 60, 70, 80, and 90 cm H2O), mean airway pressure (20, 30, and 40 cm H2O), test lung compl

22 kg predicted body weight, p < .001), in peak airway pressure (31-25 cm H2O, p < .001), and in the per

23 or the fraction of inspired oxygen 0.25/mean airway pressure 4 definition (i.e., increase in minimum

24 ng the fraction of inspired oxygen 0.25/mean airway pressure 4 thresholds to identify pediatric venti

25 derwent lung recruitment continuous positive airway pressure 40 cm H2O for 40 secs to normalize volum

26 e; the fraction of inspired oxygen 0.30/mean airway pressure 7 definition yielded ventilator-associat

27 s, a group that has poor continuous positive airway pressure adherence and difficulty in achieving we

28 Biphasic positive airway pressure/airway pressure release ventilation more

29 , and damage compared with biphasic positive airway pressure/airway pressure release ventilation more

30 release ventilation of 0%, biphasic positive airway pressure/airway pressure release ventilation more

31 Compared with biphasic positive airway pressure/airway pressure release ventilation of 0

32 Biphasic positive airway pressure/airway pressure release ventilation was

33 distress syndrome in pigs, biphasic positive airway pressure/airway pressure release ventilation with

34 elease ventilation, 0%; 2) biphasic positive airway pressure/airway pressure release ventilation, > 0

35 e ventilation, > 0-30%; 3) biphasic positive airway pressure/airway pressure release ventilation, > 3

36 tilation, > 30-60%, and 4) biphasic positive airway pressure/airway pressure release ventilation, > 6

37 per group, 6 hr each): 1) biphasic positive airway pressure/airway pressure release ventilation, 0%;

38 Driving pressures calculated from airway pressures alone (plateau airway pressure--positiv

39 s with a 10-sec pause between increments for airway pressure and alveolar confirmation to stabilize.

40 The old modes of continuous positive airway pressure and bilevel positive airway pressure hav

41 During combined (T9+L1) stimulation, airway pressure and expiratory flow rate increased to 13

42 itor them properly, especially understanding airway pressure and flow graphics.

43 Observations of airway pressure and flow tracings before and after bronc

44 Airway pressure and flow were continuously recorded at t

45 between respiratory impedance (obtained from airway pressure and flow) and model-predicted impedance

46 Studies show that continuous positive airway pressure and non-invasive positive pressure venti

47 he current management of continuous positive airway pressure and noninvasive positive pressure ventil

48 al treatment, the use of continuous positive airway pressure and noninvasive positive pressure ventil

49 r be chosen from a table of recommended mean airway pressure and oxygen concentration combinations, o

50 h improvement in functional properties (peak airway pressure and oxygenation) and histologic appearan

51 dent by near normal pulmonary function (peak airway pressure and oxygenation), histological appearanc

52 S including preinduction continuous positive airway pressure and postextubation NRS for high-risk ind

53 of acute lung injury, despite its lower mean airway pressure and reduced risk for hemodynamic comprom

54 scle pressure can be estimated from the peak airway pressure and the percentage of assistance (gain).

55 ssure ventilation (i.e., continuous positive airway pressure and/or intubation).

56 athing and is associated with decreased peak airway pressures and improved oxygenation/ventilation wh

57 ntilator volumes lower in patients with high airway pressures and poor compliance (8.4-10.6 mL/kg int

58 f high frequency jet ventilation in reducing airway pressures and, perhaps, barotraumas are cited.

59 spiratory distress syndrome in whom airflow, airway pressure, and esophageal pressure were recorded d

60 ut, lowers respiratory compliance, increases airway pressure, and impairs cardiac function.

61 has a small deadspace, is unaffected by mean airway pressure, and is therefore suitable for clinical

62 ntilation treatment with continuous positive airway pressure, and other potential ocular and systemic

63 combinations of recruitment maneuvers, mean airway pressure, and oxygen concentration.

64 the managing clinician, the ventilator flow, airway pressure, and volume/time waveforms were continuo

65 pwise recruitment maneuver without sustained airway pressure appeared to associate with less biologic

66 eration within 48 hrs of continuous positive airway pressure applied via the endobronchial blocker.

67 ten pigs with acute lung injury at multiple airway pressures, as well as a theoretical model relatin

68 y/expiratory ratio was fixed at 1:1 and mean airway pressure at 10 cm H2O.

69 g HFO, identified approaches to setting mean airway pressure based on lung mechanics, and identified

70 d obese patients had higher peak and plateau airway pressures before enrollment because of higher set

71 ve ventilation delivered as bilevel positive airway pressure (BiPAP) is often used to avoid reintubat

72 ation pressures (i.e., reduced mean and peak airway pressures) but control sheep did not.

73 hepatic vein cannot be controlled under high airway pressure, but can be controlled under low airway

74 icus, as evidenced by a reduction in plateau airway pressure, but the magnitude of this effect is rel

75 fraction of inspired oxygen by 0.25 or mean airway pressure by 4), rates ranged from 2.9 to 3.2 per

76 0.20, 0.25, and 0.30) and daily minimum mean airway pressure (by 4, 5, 6, and 7 cm H2O).

77 cm H2O) resulted in an increase in change in airway pressure, change in pleural pressure, change in p

78 tients with OSAS without continuous positive airway pressure (CPAP) (n = 13); (2) patients with OSAS

79 cmH(2)O l(-1) s; optimal continuous positive airway pressure (CPAP) = 11.3 +/- 0.7 cmH(2)O) and with

80 Continuous positive airway pressure (CPAP) and mandibular advancement device

81 and the clinical use of continuous positive airway pressure (CPAP) and positive end-expiratory press

82 Although continuous positive airway pressure (CPAP) can mitigate these risks, effecti

83 Meta-analysis found that continuous positive airway pressure (CPAP) compared with sham was significan

84 ms Questionnaire (SASQ), continuous positive airway pressure (CPAP) compliance, and physician decisio

85 and economic benefits of continuous positive airway pressure (CPAP) for moderate to severe obstructiv

86 lar alternative to nasal continuous positive airway pressure (CPAP) for noninvasive respiratory suppo

87 ether OSA treatment with continuous positive airway pressure (CPAP) has metabolic benefits.

88 rapy delivered by bubble continuous positive airway pressure (CPAP) improved outcomes compared with s

89 Continuous positive airway pressure (CPAP) in asthma patients with concomita

90 Continuous positive airway pressure (CPAP) is considered the treatment of ch

91 Continuous positive airway pressure (CPAP) is the first-line treatment for p

92 Continuous positive airway pressure (CPAP) is the treatment of choice in pat

93 Although continuous positive airway pressure (CPAP) is used frequently for preterm in

94 'drops' from therapeutic continuous positive airway pressure (CPAP) levels.

95 ients who cannot tolerate continous positive airway pressure (CPAP) machines or intraoral devices.

96 Adherence to short-term continuous positive airway pressure (CPAP) may predict long-term use.

97 determine the effect of continuous positive airway pressure (CPAP) of patients with OSA on renal hem

98 ence about the effect of continuous positive airway pressure (CPAP) on glycemic control in patients w

99 n premature infants with continuous positive airway pressure (CPAP) preserves surfactant and keeps th

100 Continuous positive airway pressure (CPAP) reduces blood pressure, but adher

101 g volume on the level of continuous positive airway pressure (CPAP) required to prevent flow limitati

102 Continuous positive airway pressure (CPAP) therapy is the most common treatm

103 to examine the effect of continuous positive airway pressure (CPAP) therapy on atrial fibrillation (A

104 The effects of continuous positive airway pressure (CPAP) therapy on endothelial function a

105 ontaneous breathing with continuous positive airway pressure (CPAP) therapy on the relative distribut

106 rm studies indicate that continuous positive airway pressure (CPAP) therapy reduces blood pressure in

107 ents had been prescribed continuous positive airway pressure (CPAP) therapy to manage OSA and were id

108 Prolonged continuous positive airway pressure (CPAP) therapy with supplemental oxygen

109 ood pressure response to continuous positive airway pressure (CPAP) treatment is highly variable and

110 ence about the effect of continuous positive airway pressure (CPAP) treatment on blood pressure in pa

111 n women, and the role of continuous positive airway pressure (CPAP) treatment on this association.

112 apeutic decision-making, continuous positive airway pressure (CPAP) treatment or a healthy habit asse

113 valuation in response to continuous positive airway pressure (CPAP) treatment.

114 ve ventilation (NIV) and continuous positive airway pressure (CPAP) use in patients with OHS, informa

115 pressure associated with continuous positive airway pressure (CPAP) use, with smaller or uncontrolled

116 of early treatment with continuous positive airway pressure (CPAP) versus early surfactant treatment

117 breathing (BB) and nasal continuous positive airway pressure (CPAP) was applied to reduce negative pr

118 similar to that of nasal continuous positive airway pressure (CPAP) when used as postextubation suppo

119 We delivered continuous positive airway pressure (CPAP) with the helmet under a variety o

120 We aimed to determine if continuous positive airway pressure (CPAP), a form of non-invasive ventilati

121 o receive treatment with continuous positive airway pressure (CPAP), a weight-loss intervention, or C

122 t for symptomatic OSA is continuous positive airway pressure (CPAP), but its value in patients withou

123 rough the application of continuous positive airway pressure (CPAP), which remains a primary therapeu

124 tilation (IPPV) or nasal continuous positive airway pressure (CPAP)--at the time of the first use of

125 the standard therapy of continuous positive airway pressure (CPAP).

126 reated and adherent with continuous positive airway pressure (CPAP).

127 r their SDB, using nasal continuous positive airway pressure (CPAP).

128 Noninvasive ventilation (continuous positive airway pressure [CPAP] or noninvasive intermittent posit

129 Under low airway pressure, CVP did not increase or often decreased

130 Under high airway pressure, CVP was persistently higher than pneumo

131 and regression analyses were performed among airway pressure, CVP, and pneumoperitoneum pressure.

132 Plateau airway pressure decreased by approximately 2 cm H2O (25.

133 Increasing the airway pressures decreased but did not abolish the exten

134 tment to total lung capacity, using stepwise airway pressure decrements.

135 We aimed to thoroughly determine airway pressure distribution, how this is influenced by

136 ume loop [in Joules]) and stress relaxation (airway pressure drop during an end-inspiratory pause [in

137 Pmusc/Eadi index was also calculated from airway pressure drop during end-expiratory occlusions.

138 ic simulations determined flow, velocity and airway pressure drops.

139 tubation risk factors included lower maximum airway pressure during airway occlusion (aPiMax) preextu

140 vation of the Pmusc/Eadi index from Eadi and airway pressure during an expiratory occlusion enables a

141 Flow, airway pressure, esophageal pressures, and peak electric

142 Data from an esophageal catheter and airway pressure/flow sensor were used to measure POB(I).

143 patients were placed on continuous positive airway pressure for 1-2 mins to measure their spontaneou

144 ve treatment of OSA with continuous positive airway pressure for 3 months significantly reduced sever

145 ressure of 30 cm H2O: 1) continuous positive airway pressure for 30 seconds (CPAP-30); 2) stepwise ai

146 alternative treatment to continuous positive airway pressure for patients with obstructive sleep apne

147 t compensation for more negative inspiratory airway pressures generated during heavy exercise occurs

148 olic blood pressure occurred in the positive airway pressure group than in the usual care group (-3.5

149 patients who adhered to continuous positive airway pressure >/=4 hours daily.

150 , changes in compliance, and changes in mean airway pressure had little effect.

151 asal cannula therapy and continuous positive airway pressure had similar efficacy (RR, 1.11; 95% CI,

152 ntilation treatment with continuous positive airway pressure have an increased risk of second eye inv

153 ositive airway pressure and bilevel positive airway pressure have been actively introduced in clinica

154 s ventilatory consequences include increased airway pressures, hypercarbia, and decreased pulmonary c

155 r whether treatment with continuous positive airway pressure improves daytime function in these patie

156 ructive sleep apnea with continuous positive airway pressure improves not only patient-reported outco

157 the main alternative to continuous positive airway pressure, improves endothelial function in patien

158 ume was decreased by lowering end-expiratory airway pressure in a stepwise manner.

159 improved after starting continuous positive airway pressure in asthmatics with moderate to severe ob

160 al recruitment, but decreased at the maximum airway pressure in nine patients, indicative of a reduct

161 ntilation treatment with continuous positive airway pressure in patients with severe OSAS increased t

162 tilation was superior to continuous positive airway pressure in preventing extubation failure (RR, 0.

163 ta as to the efficacy of continuous positive airway pressure in severe OSA have come from randomized,

164 similar to that of CPAP-30; and 3) stepwise airway pressure increase (5 cm H2O/step, 5 s at each ste

165 essure for 30 seconds (CPAP-30); 2) stepwise airway pressure increase (5 cm H2O/step, 8.5 s at each s

166 ve intra-abdominal pressure 5 mm Hg, plateau airway pressure increased linearly by ~ 50% of the appli

167 Continuous positive airway pressure is a useful second-line treatment for ch

168 ) may increase observed DeltaP, whereas mean airway pressure is decreased with air leaks.

169 Continuous positive airway pressure is the treatment of choice, with adheren

170 proved allograft lung function based on peak airway pressure, less infiltrates/consolidation on micro

171 +10, +15) in a randomized order, with a mean airway pressure level determined by adding 5, 10, or 15

172 id not improve oxygenation whatever the mean airway pressure level.

173 ies that use lower end-inspiratory (plateau) airway pressures, lower tidal volumes (VT), and higher p

174 ter; both plans included continuous positive airway pressure, mandibular advancement splints, or cons

175 Changes in peak airway pressure may not always reflect the changes in dy

176 < .05 vs. control]) and the increase in peak airway pressure (mbar; control group at BL, 20 +/- 1, an

177 bjects were treated with continuous positive airway pressure (mean duration of 26 weeks), after which

178 cycles with supporting a continuous positive airway pressure model.

179 auto-titrating CPAP (n = 122) or no positive airway pressure (n = 122).

180 ort noninferior to nasal continuous positive airway pressure (nCPAP) or bilevel nCPAP (BiPAP) as a pr

181 is optimal: noninvasive continuous positive airway pressure (NCPAP) or intubate-surfactant-extubate

182 The airway pressure needed to achieve comparable dependent l

183 6 kPa, and NHBD(2)NO 61+/-6 kPa; P=0.01) and airway pressures (NHBD(1) 30.8+/-3.5, NHBD(2) 32.5+/-5.6

184 fidence interval, 0.96-0.99; p = .017), peak airway pressure (odds ratio per 5-cm H2O increase: 1.11;

185 a plateau pressure of >30 cm H2O, and a peak airway pressure of >35 cm H2O.

186 t Fio2 1.0, 37 degrees C, 80% humidity, mean airway pressure of 20 cm H2O, and an inspiratory/expirat

187 io2 of 1.0, 37 degrees C, 80% humidity, mean airway pressure of 20 cm H2O, and an inspiratory/expirat

188 ontrol) lung was kept on continuous positive airway pressure of 20 cm H2O, and CO2 was partially remo

189 piratory pause with the lungs inflated to an airway pressure of 20 cm H2O.

190 ilator settings were an inspiratory positive airway pressure of 24 (IQR, 22-26) cm H2O, an expiratory

191 t recruitment maneuvers, targeted to maximal airway pressure of 30 cm H2O: 1) continuous positive air

192 (IQR, 22-26) cm H2O, an expiratory positive airway pressure of 4 (IQR, 4-5) cm H2O, and a backup rat

193 to occur up to and beyond a peak inspiratory airway pressure of 40 cm H(2)O, as demonstrated by both

194 airway occlusion and on continuous positive airway pressure of 5 and pressure support of 10 above po

195 per kg of predicted bodyweight and a plateau airway pressure of less than 30 cm H2O.

196 cording to the protocol of Webb and Tierney (airway pressures of 14/0, 30/0, 45/10, 45/0 cm H2O).

197 raphy (CT) during breath-holding sessions at airway pressures of 5, 15, and 45 cm H2O and Cine-CTs on

198 the beneficial effect of continuous positive airway pressure on quality of life, mood, and work absen

199 Bi-level positive airway pressure or adaptive servo-ventilation can be use

200 rst 72 hours (the use of continuous positive airway pressure or high-flow nasal cannula for at least

201 r obstacle to successful continuous positive airway pressure or noninvasive positive pressure ventila

202 uired a mask, continuous or bilevel positive airway pressure, or mechanical ventilation were classifi

203 respiratory system dynamic compliance, mean airway pressure, PaO2/FiO2 ratio, and oxygenation index

204 toms, adherence to using continuous positive airway pressure, patient satisfaction, and health care c

205 anging gas mixtures within each animal, mean airway pressure (Paw = 16.8 +/- 0.3 cm H(2)O) and freque

206 The difference between airway pressure (Paw) and Pes is a valid estimate of tra

207 th at least 8 cm H2O positive end-expiratory airway pressure (PEEP), and bilateral infiltrates consis

208 Central venous pressure, airway pressure, pericardial pressure, and pleural press

209 ntilation as a 35 cm H2O continuous positive airway pressure period lasting 3-4 seconds at different

210 ratio, tidal volume, respiratory rate, mean airway pressure, plateau pressure, and hemodynamic varia

211 0.3 +/- 0.1, and 0.3 +/- 0.1 mm Hg/mL/kg for airway pressure, pleural pressure, pericardial pressure,

212 uscle pressure, estimated in cm H2O as (peak airway pressure-positive end-expiratory pressure)x[(100-

213 culated from airway pressures alone (plateau airway pressure--positive end-expiratory pressure) did n

214 icted body weight with corresponding plateau airway pressures (PPlat) less than or equal to 30 cm H2O

215 spontaneous breathing or continuous positive airway pressure; pressure support ventilation 5-12 cm H2

216 Although regulated continuous positive airway pressure, pulse oximeters, and blenders are routi

217 show that important qualitative features of airway pressure-radius hysteresis loops are highly depen

218 ed by adding 5, 10, or 15 cm H2O to the mean airway pressure recorded during conventional mechanical

219 breathing exercises, and continuous positive airway pressure) reduce pulmonary risk.

220 Continuous positive airway pressure reduced extubation failure in comparison

221 ventional mechanical ventilation (n = 15) or airway pressure release ventilation (n = 12) for 48 hrs

222 Airway pressure release ventilation also significantly r

223 We compared the effects of airway pressure release ventilation and conventional mec

224 Mean airway pressures were higher in airway pressure release ventilation animals between 6 an

225 Respiratory rates were lower in airway pressure release ventilation at 24, 42, and 48 hr

226 Airway pressure release ventilation has been gaining a r

227 f high-frequency oscillatory ventilation and airway pressure release ventilation in ARDS is uncertain

228 The role of airway pressure release ventilation in the management of

229 release ventilation was conducted using the airway pressure release ventilation mode with an inspira

230 ween conventional mechanical ventilation and airway pressure release ventilation pigs.

231 PaO2/Fio2 ratio was lower in airway pressure release ventilation vs. conventional mec

232 Airway pressure release ventilation was set with a P(Hig

233 High-frequency oscillatory ventilation, airway pressure release ventilation, and partial liquid

234 mode to prevent intubation and then go on to airway pressure release ventilation, high-frequency osci

235 e open lung approach, recruitment maneuvers, airway pressure release ventilation, high-frequency osci

236 caused by severe smoke inhalation in swine, airway pressure release ventilation-treated animals deve

237 ween conventional mechanical ventilation and airway pressure release ventilation.

238 Intraoperative peak airway pressures remained within normal limits for all p

239 ation index (p < .05); and 1/PaO2/Fio2, mean airway pressure, serum pH, and Paco2 were associated wit

240 We postulated that adding an airway pressure signal (Paw) to pressure tracings of cen

241 volume were simultaneously recorded at each airway pressure step.

242 eight, positive end-expiratory pressure, and airway pressure), subjects with an overweight fluid-bala

243 29 and 313 +/- 48 seconds, respectively) and airway pressure sustained during the last minute of load

244 Eadi index obtained during an occlusion from airway pressure swing was tightly correlated with that d

245 ic bifurcations also exhibit higher proximal airway pressures than symmetric ones, but the improvemen

246 the chest wall allows for calculation of an airway pressure that would place the lung at a desired v

247 However, under low airway pressure, the risk of pulmonary gas embolism incr

248 icant in those who were adherent to positive airway pressure therapy (-4.4 mm Hg vs. -1.6 mm Hg; P =

249 cant reduction in sleepiness in the positive airway pressure therapy group (P < 0.0001).

250 tal component summary scores in the positive airway pressure therapy group.

251 d at baseline and after 3 months of positive airway pressure therapy in a heterogeneous group of 52 c

252 Positive airway pressure therapy is frequently used to treat obst

253 This trial showed no effect of positive airway pressure therapy on glycemic control in patients

254 ex of 15 or more events per hour to positive airway pressure therapy or to usual care.

255 In OSA patients, continuous positive airway pressure therapy resulted in reduction of the pos

256 However, it is not known whether positive airway pressure therapy results in improvements in the n

257 Positive airway pressure therapy was associated with significant

258 We hypothesized that positive airway pressure therapy would be associated with improve

259 ction in children after 3 months of positive airway pressure therapy, even in developmentally delayed

260 After 4 weeks of continuous positive airway pressure therapy, flow-mediated dilation and expr

261 tient required noninvasive biphasic positive airway pressure throughout his course.

262 expenditure (effort), arterial blood gases, airway pressure, tidal volume and its coefficient of var

263 Analysis of the airway pressure-time curve (stress index) has been propo

264 and inflammation were assessed based on the airway pressure-time index, bronchoalveolar lavage (BAL)

265 nt of confirmed OSA with continuous positive airway pressure to reduce driving risk, rather than no t

266 t medications or empiric continuous positive airway pressure to reduce driving risk.

267 d by repeatedly lowering continuous positive airway pressure to subtherapeutic levels for 3 minutes d

268 ation and application of continuous positive airway pressure to the left lung for 48 hrs.

269 DATION 2: ACP recommends continuous positive airway pressure treatment as initial therapy for patient

270 n alternative therapy to continuous positive airway pressure treatment for patients diagnosed with OS

271 Continuous positive airway pressure treatment improves the functional outcom

272 Continuous positive airway pressure treatment was associated with reduction

273 Patients who received continuous positive airway pressure treatment were significantly less likely

274 idences of both OSA (AHI of >/=5 or positive airway pressure treatment) and OSA concomitant with habi

275 effects associated with continuous positive airway pressure treatment.

276 weeks of active or sham continuous positive airway pressure treatment.

277 ceived 8 weeks of active continuous positive airway pressure treatment.

278 morning after effective continuous positive airway pressure treatment.

279 sted of a 30-second breath hold at 40 cm H2O airway pressure under heavy sedation or paralysis.

280 spiratory distress syndrome, using high mean airway pressure under high-frequency oscillatory ventila

281 h-flow nasal cannula and continuous positive airway pressure use in a monitored setting to prevent re

282 mized clinical trials of continuous positive airway pressure versus mechanical ventilation.

283 Mean airway pressure was 18+/-3 cm H2O during conventional me

284 Plateau airway pressure was lower at Day 7 in the salbutamol gro

285 With A-CPR, high airway pressure was noted distal to the carina, which co

286 Peak airway pressure was similar at the three respiratory rat

287 injury occurred, tidal volume increased and airway pressure waveform changed.

288 The contour of the airway pressure waveform on a ventilator screen provides

289 s were observed: Tidal volume increased, and airway pressure waveform was transformed by extending th

290 of respiratory mechanics based on unmodified airway pressure were misleading regarding lung behavior

291 During stimulation at the T9 and L1 levels, airway pressures were 90 and 82 cm H2O, respectively.

292 Mean airway pressures were higher in airway pressure release

293 Dynamic airway pressures were in a safe range in which ventilato

294 Pre and Post plateau pressures and peak airway pressures were similar.

295 ar mechanics (derived from alveolar size and airway pressure) were determined in noninjured (n = 9) a

296 was measured 3 times at each of 9 levels of airway pressure, which was increased in increments of 5

297 others were examined over a range of static airway pressures, which varied the extents of regional P

298 y, chronic alcohol abuse, shock, higher peak airway pressure while being mechanically ventilated, cur

299 infants were ventilated, continuous positive airway pressure without ventilation increased from 7% (1

300 rategy improved the oxygenation index ([mean airway pressure x the ratio of the fraction of inspired