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1 otein inhibitors and was further verified by alanine scanning.
2 n interactions using computational interface alanine scanning.
3 am positive pathogens was identified through alanine scanning.
4 mplex, we targeted 20 positions in Prp28 for alanine scanning.
5 GHRP-6 leads, which were further examined by alanine scanning.
6      The binding epitope was determined with alanine scanning.
7                       In previous work using alanine scanning (1), we identified residues Leu-53, Asp
8                                     Using an alanine-scanning 3A mutant, we show that Golgi targeting
9 n trafficking motifs in the channel and with alanine scanning across aa 610-620.
10                              We performed an alanine scanning analyses of 35 hGHv residues and determ
11           Using 167 sequences from a shotgun alanine scanning analysis of Site1, we have determined t
12 fected HEK-293 cells, we demonstrate through alanine scanning and amino acid substitutions that the p
13  and sequence, and it provides computational alanine scanning and change in solvent-accessible surfac
14                                              Alanine scanning and conservative substitutions identifi
15       Structure-function analysis of Ecm1 by alanine scanning and conservative substitutions identifi
16  and HhH domains of Escherichia coli LigA by alanine scanning and conservative substitutions, entaili
17                                              Alanine scanning and crystallographic structural analysi
18 a core determinant defined using a series of alanine scanning and deletion mutant variants.
19 inding, as well as computational analysis of alanine scanning and DMC data.
20 cin DNases through a combination of E9 DNase alanine scanning and double-mutant cycles (DMCs) coupled
21 bed via standard and nonstandard techniques (alanine scanning and hydrophile scanning, respectively),
22 ution of critical residues identified by the alanine scanning and NMR spectroscopy, along with the el
23 might contact RNA polymerase, we carried out alanine scanning and random mutagenesis of oxyR.
24 lix 3 of EntB-ArCP were generated by shotgun alanine scanning and selected for their ability to suppo
25                                 We generated alanine scanning and single and multiple amino acid subs
26 rved amino acids of Escherichia coli LigA by alanine scanning and thereby identified five new residue
27        We have now employed a combination of alanine-scanning and deletion mutagenesis to identify th
28                            Using the Rosetta alanine-scanning and design algorithms to predict destab
29      We have developed a novel computational alanine scanning approach that involves analysis of ense
30                                              Alanine-scanning assays suggested the absence of known h
31                                              Alanine scanning-based shotgun mutagenesis epitope mappi
32  Lyp substrate recognition using an "inverse alanine scanning" combinatorial library approach.
33 scence polarization displacement assays, and alanine scanning data all suggest that they bind to the
34                                              Alanine scanning defined seven individual amino acids as
35                                              Alanine scanning demonstrated that the epitope involves
36 idging may explain the previous finding from alanine scanning experiments that R333 contributes signi
37 s is also in line with systematic, in silico Alanine scanning free-energy simulations, which indicate
38 is using recombinant proteins and a panel of alanine-scanning GPC mutants revealed that F100G5 bindin
39                                              Alanine scanning helped resolve the boundaries of this c
40 OS-PDZ complex that correlated well with the alanine scanning identified regions of dystrophin.
41                                      Through alanine scanning, immunofluorescence cell staining and c
42                                Using shotgun alanine scanning in conjunction with this selection, we
43                                              Alanine scanning insertion mutagenesis was used to exami
44              Mutating residues into alanine (alanine scanning) is one of the fastest experimental mea
45                       Using this plasmid, an alanine scanning library has been constructed and the mu
46                   To do so, we created a tri-alanine scanning library that covers all of sigma(70) R4
47 activities are underscored by the results of alanine-scanning, limited proteolysis, and deletion anal
48 he hot spot residues known from experimental alanine scanning measurements.
49                                        Using alanine scanning, molecular dynamics simulations, and tr
50                                              Alanine scanning mutagenesis analysis shows that four co
51 kappaB pathway signaling, using experimental alanine scanning mutagenesis and also the FTMap method f
52                                        Using alanine scanning mutagenesis and crystallographic approa
53 cid residues of a prototypic PPPSP motif via alanine scanning mutagenesis and demonstrate that each o
54 LIAGCITSTDPVLSALI(152)) in activity by using alanine scanning mutagenesis and examining salt toleranc
55                                              Alanine scanning mutagenesis and kinetic analysis reveal
56                       We performed saturated alanine scanning mutagenesis and other amino acid substi
57 5) to envelope function has been examined by alanine scanning mutagenesis and subsequent characteriza
58                In this work, we have used an alanine scanning mutagenesis approach to identify whethe
59 the vinylogous urea binding site, horizontal alanine scanning mutagenesis between p51 residues Lys-27
60                                              Alanine scanning mutagenesis confirms that these residue
61 o and TgRON2L1-bound forms complemented with alanine scanning mutagenesis data reveal an unexpected a
62                                              Alanine scanning mutagenesis demonstrated that positivel
63                                              Alanine scanning mutagenesis determined that the key res
64                                              Alanine scanning mutagenesis in sGC indicates that the H
65                                        Using alanine scanning mutagenesis in the activity subdomain o
66                                              Alanine scanning mutagenesis maps a novel epitope to a s
67                                              Alanine scanning mutagenesis of 12 Arg/Lys residues of e
68                  We thus performed extensive alanine scanning mutagenesis of A1AR-ECL2 to explore the
69 rformed the first complete charge-cluster-to-alanine scanning mutagenesis of an ARP and compared the
70                                              Alanine scanning mutagenesis of areas flanking Leu(172),
71                   In this work, we performed alanine scanning mutagenesis of aromatic residues locate
72                                 We performed alanine scanning mutagenesis of AuIB and alpha3beta4 nAC
73        To test this possibility, we employed alanine scanning mutagenesis of CB1 EC2 and identified t
74                                              Alanine scanning mutagenesis of conserved amino acid res
75                                   Charged-to-alanine scanning mutagenesis of CSM4 yielded one allele,
76                                              Alanine scanning mutagenesis of hVbeta2.1 wild-type and
77 ctions and receptor activation, we performed alanine scanning mutagenesis of loop residues and assess
78                                        Using alanine scanning mutagenesis of MBM1, we found that the
79                                              Alanine scanning mutagenesis of MPER indicates that it h
80 nd cofactor activity were assessed following alanine scanning mutagenesis of residues 555-571 that bo
81                                              Alanine scanning mutagenesis of residues 64-70, within f
82                                              Alanine scanning mutagenesis of the alpha-region reveals
83                                              Alanine scanning mutagenesis of the C-terminal region of
84                                              Alanine scanning mutagenesis of the conserved residues o
85                  In this study, we performed alanine scanning mutagenesis of the ExsA alpha-helix (re
86  of chimeras between these OCLN proteins and alanine scanning mutagenesis of the extracellular domain
87                                              Alanine scanning mutagenesis of the LIM interaction doma
88    Here, we report the results of saturating alanine scanning mutagenesis of the N-terminal domain of
89 f recombinant mutant CSFVs was created using alanine scanning mutagenesis of the p7 gene harboring se
90                                  We describe alanine scanning mutagenesis of the PhoP alpha loop and
91  random mutagenesis results, we performed an alanine scanning mutagenesis of the TLR2 DD loop and par
92                                              Alanine scanning mutagenesis of this region revealed tha
93                                 We performed alanine scanning mutagenesis of this region, and we quan
94                                              Alanine scanning mutagenesis of three to five consecutiv
95               Towards this end, we performed alanine scanning mutagenesis on selected residues in the
96 ole of NS4A in these processes, we conducted alanine scanning mutagenesis on the C-terminal acidic do
97                                              Alanine scanning mutagenesis revealed a requirement for
98                                              Alanine scanning mutagenesis revealed four classes of mu
99                                              Alanine scanning mutagenesis revealed that amino acids K
100                                              Alanine scanning mutagenesis revealed that the K/R-R/x-x
101                                              Alanine scanning mutagenesis studies revealed that the T
102 e, we performed a comprehensive deletion and alanine scanning mutagenesis study of this protein in th
103 (ATR) and curcumin (CCM), here we perform an alanine scanning mutagenesis study.
104 ides that bound Gbetagamma were subjected to alanine scanning mutagenesis to determine their relevanc
105                                      We used alanine scanning mutagenesis to examine the contribution
106              We have used structure-directed alanine scanning mutagenesis to identify determinants in
107 ther analyzed by combining triple and single alanine scanning mutagenesis to identify individual resi
108                  We used NMR experiments and alanine scanning mutagenesis to identify residues in the
109                       In this study, we used alanine scanning mutagenesis to identify the key residue
110                                 We then used alanine scanning mutagenesis to locate the epitopes.
111                                      We used alanine scanning mutagenesis to replace selected amino a
112                                      We used alanine scanning mutagenesis to study the contributions
113                       The current study used alanine scanning mutagenesis to understand the selectivi
114  motif centered around the sequence "SQELD." Alanine scanning mutagenesis was carried out on the T(35
115                                              Alanine scanning mutagenesis was performed for the resid
116                                   Systematic alanine scanning mutagenesis was performed on the substr
117                                              Alanine scanning mutagenesis was performed to assess the
118 s parainfluenza virus 5 F protein TM domain, alanine scanning mutagenesis was performed.
119                               In this study, alanine scanning mutagenesis was used in combination wit
120                                              Alanine scanning mutagenesis was used to determine the r
121                                              Alanine scanning mutagenesis was used to map the specifi
122                                              Alanine scanning mutagenesis within the region of amino
123                     Using in vitro assembly, alanine scanning mutagenesis, and biophysical analyses,
124            Using an in vitro assembly assay, alanine scanning mutagenesis, and biophysical techniques
125      Using computational docking algorithms, alanine scanning mutagenesis, and surface plasmon resona
126 side chains at this interface, identified by alanine scanning mutagenesis, are conserved among DSX ho
127  probed the in vivo role of the linker using alanine scanning mutagenesis, assaying stressosome outpu
128 ude random peptide phage display mapping and alanine scanning mutagenesis, to identify residues in th
129                                        Using alanine scanning mutagenesis, we demonstrate that the bi
130                                        Using alanine scanning mutagenesis, we show that a pore helix
131                                        Using alanine scanning mutagenesis, we show that two distinct
132 gp41 HR1 has been systematically examined by alanine scanning mutagenesis, with subsequent characteri
133 esults from loss-of-binding studies using an alanine scanning mutagenesis-based epitope mapping appro
134 bution of the arm to folding was obtained by alanine scanning mutagenesis.
135 2 cytoplasmic tail were identified by single-alanine scanning mutagenesis.
136 helix 11, Thr978, as an essential residue by alanine scanning mutagenesis.
137  GABPbeta interface residues were chosen for alanine scanning mutagenesis.
138 nal mutagenesis, carbohydrate shielding, and alanine scanning mutagenesis.
139            We subjected HD5 to comprehensive alanine scanning mutagenesis.
140 ng of the epitopes was accomplished by using alanine scanning mutagenesis.
141                                  Deletion or alanine- scanning mutagenesis through this domain signif
142 nteracts with GP, here we used comprehensive alanine-scanning mutagenesis (shotgun mutagenesis), neut
143                             Here, we perform alanine-scanning mutagenesis across CDR H3 and make addi
144  explore this molecular environment, we used alanine-scanning mutagenesis across residues 660 to 680
145                                              Alanine-scanning mutagenesis along the entire length of
146 L binding site of AFABP/aP2 a combination of alanine-scanning mutagenesis and fluorescence resonance
147 ity binding at different DNA sequences using alanine-scanning mutagenesis and identified several key
148                                              Alanine-scanning mutagenesis and kinetic analysis identi
149                                      Peptide alanine-scanning mutagenesis and modeling of receptor-bo
150                                      We used alanine-scanning mutagenesis and patch clamp photometry
151 pes on VEGF for these three antibodies using alanine-scanning mutagenesis and structural analyses.
152 1) to envelope function has been examined by alanine-scanning mutagenesis and subsequent characteriza
153 lex as a guide, we undertook a comprehensive alanine-scanning mutagenesis approach at the TCR-pMHC-I
154 tersubunit interactions of K33, we performed alanine-scanning mutagenesis at charged residues in the
155  of flat beta-sheets, we performed extensive alanine-scanning mutagenesis experiments on the single-l
156                 Isolated A2 was subjected to alanine-scanning mutagenesis followed by expression of a
157                                              Alanine-scanning mutagenesis identified a functional TIL
158                                              Alanine-scanning mutagenesis identified an acidic-residu
159          We have performed surface acidic-to-alanine-scanning mutagenesis of 3C to identify the surfa
160                                              Alanine-scanning mutagenesis of 48 amino acids combined
161                  In this report we have used alanine-scanning mutagenesis of a putative coiled coil a
162                                              Alanine-scanning mutagenesis of B. subtilis alphaCTD unc
163                                              Alanine-scanning mutagenesis of DC-SIGN revealed that hi
164                                              Alanine-scanning mutagenesis of FNR amino acid residues
165                                              Alanine-scanning mutagenesis of HVEM was used to further
166                                 Results from alanine-scanning mutagenesis of hydrophobic residues in
167                                              Alanine-scanning mutagenesis of predicted catalytic resi
168                                              Alanine-scanning mutagenesis of residues predicted to li
169                                              Alanine-scanning mutagenesis of rho-TIA showed F18A had
170 he same TCR, complemented with computational alanine-scanning mutagenesis of SEA, SEB, SEC3, SEE, and
171                                        Using alanine-scanning mutagenesis of sigma32 and in vivo and
172 ermore, we report the solution structure and alanine-scanning mutagenesis of TACI_d2 along with co-cr
173 pe residues to CNTO607 binding, we performed alanine-scanning mutagenesis of the A-D region of IL-13.
174                                  We combined alanine-scanning mutagenesis of the A1AR second extracel
175                                              Alanine-scanning mutagenesis of the alternatively splice
176                                              Alanine-scanning mutagenesis of the ankyrin-B ANK (ankyr
177 ding determinants were identified by shotgun alanine-scanning mutagenesis of the BR3 ECD expressed on
178                                              Alanine-scanning mutagenesis of the GLUT4 amino terminus
179                                              Alanine-scanning mutagenesis of the intracellular C term
180                                              Alanine-scanning mutagenesis of the Na(v)1.6 N terminus
181 rlying biochemical mechanism by carrying out alanine-scanning mutagenesis of the PKR activation domai
182                       In this study, we used alanine-scanning mutagenesis of the residues between C13
183                                              Alanine-scanning mutagenesis of the TMH identified sever
184                                              Alanine-scanning mutagenesis of these nine side chains s
185                                 We performed alanine-scanning mutagenesis of this motif ((14)TFPLF(18
186                                              Alanine-scanning mutagenesis of this region suggested th
187             Here, we performed comprehensive alanine-scanning mutagenesis on csrA of E. coli and test
188                   In this report, we conduct alanine-scanning mutagenesis on the 14 other conserved s
189 s) on gp120, and its footprint as defined by alanine-scanning mutagenesis overlaps that of b12.
190                                              Alanine-scanning mutagenesis produced one rCPE variant,
191                                              Alanine-scanning mutagenesis revealed five additional re
192                                              Alanine-scanning mutagenesis revealed that an F protein
193                                              Alanine-scanning mutagenesis revealed that residues 1773
194                                              Alanine-scanning mutagenesis revealed that residues Phe4
195                                              Alanine-scanning mutagenesis revealed that the affinitie
196                                              Alanine-scanning mutagenesis revealed the molecular basi
197  containing mutations was generated using an alanine-scanning mutagenesis strategy.
198                                              Alanine-scanning mutagenesis studies targeting residues
199                            Here we report an alanine-scanning mutagenesis study of the Runt domain th
200  have contact residues within aa 412 to 423, alanine-scanning mutagenesis suggested that one subset,
201 te in CA for Ubc9 was mapped by deletion and alanine-scanning mutagenesis to a consensus motif for SU
202 orescence correlation spectroscopy (FCS) and alanine-scanning mutagenesis to characterize the interac
203                        In this work, we used alanine-scanning mutagenesis to elucidate the structural
204                          In this work we use alanine-scanning mutagenesis to explore the contribution
205                                      We used alanine-scanning mutagenesis to identify regions of huma
206                           This study applied alanine-scanning mutagenesis to investigate the role of
207                                    First, 32 alanine-scanning mutagenesis variants of dystrophin R16-
208                                              Alanine-scanning mutagenesis was applied to complete the
209                                              Alanine-scanning mutagenesis was applied to the correspo
210                                              Alanine-scanning mutagenesis was previously performed to
211                                              Alanine-scanning mutagenesis was previously performed to
212                                              Alanine-scanning mutagenesis was used to confirm that th
213                    In this study, charged to alanine-scanning mutagenesis was used to generate condit
214                                              Alanine-scanning mutagenesis was used to identify YscF m
215                                              Alanine-scanning mutagenesis was used to investigate the
216                                        Using alanine-scanning mutagenesis we found four residues, all
217               Through extensive deletion and alanine-scanning mutagenesis we have mapped key residues
218                                By performing alanine-scanning mutagenesis we identified a dilysine se
219                 For this study, by employing alanine-scanning mutagenesis, (125)I-SDF-1alpha competit
220 ccompanying article, we demonstrate, through alanine-scanning mutagenesis, a key role for extracellul
221 l residues were determined in two designs by alanine-scanning mutagenesis, and are consistent with th
222  by a combination of cysteine cross-linking, alanine-scanning mutagenesis, and computational simulati
223 d its membrane binding properties, performed alanine-scanning mutagenesis, and identified residues im
224                                        Using alanine-scanning mutagenesis, in cellulo bioluminescence
225                                        Using alanine-scanning mutagenesis, loss-of-function recombina
226                                        Using alanine-scanning mutagenesis, we have made mutations in
227                                        Using alanine-scanning mutagenesis, we identified two clusters
228                                        Using alanine-scanning mutagenesis, we probed the roles of two
229  G1 and G2, we performed point mutations and alanine-scanning mutagenesis.
230 im101 and ESCRT pathways, which we tested by alanine-scanning mutagenesis.
231  the residues within this loop, we performed alanine-scanning mutagenesis.
232 tide and peptide agonists were studied using alanine-scanning mutagenesis.
233 e-chains to GrpE-DnaK binding were probed by alanine-scanning mutagenesis.
234 the control of specific traits, we performed alanine-scanning mutagenesis.
235 f conserved switch II residues by performing alanine-scanning mutagenesis.
236 s explored by cross-competition analysis and alanine-scanning mutagenesis.
237  we generated the entire histone H2A and H2B alanine-scanning mutant strains in another background, w
238 pare the kinetic rates and affinities for 18 alanine scanning mutants comprising epitope residues 50-
239                             Binding of X5 to alanine scanning mutants of gp120JR-CSF complexed with C
240  species variants, (2) species chimeras, (3) alanine scanning mutants, and (4) site-specific mutants.
241 we measured their binding to a panel of 11 G alanine-scanning mutants and identified two mutants, P18
242  We constructed a t-toxin library of ProTx-I alanine-scanning mutants and screened this library again
243 In addition, it was found that when the BLIP alanine-scanning mutants were tested in the strain, the
244 -sulfated analogues, truncated peptides, and alanine-scanning mutants, suggested that each of the 12-
245                                              Alanine scanning mutation of Epep revealed residues crit
246                          We describe here an alanine scanning mutational analysis of the Abeta(1-40)
247                                              Alanine-scanning mutational analysis of the first 62 ami
248 ain by single amino acid substitutions and 3-alanine scanning mutations identified important but not
249                                  A series of alanine scanning mutations of hot-spot residues at the i
250                                  Most of the alanine-scanning mutations in the CT had little effect o
251 rotein (CP) rescued RNA synthesis by several alanine-scanning mutations in the N-terminal alpha helix
252 of glycoprotein incorporation, we introduced alanine-scanning mutations into this region of the trans
253 function has been systematically examined by alanine scanning of all gp41 loop residues and the subse
254                                              Alanine scanning of AP-Cav-B revealed that Thr-90 and -9
255                              Second, shotgun alanine scanning of BCMA was used to map critical residu
256                                     Finally, alanine scanning of CDR1 and CDR2 sequences of TRBV4-1 r
257                                              Alanine scanning of GpTx-1 revealed that residues Trp(29
258                                  Comparative alanine scanning of Im2 and Im9 residues involved in bin
259                                              Alanine scanning of MPER residues 664 to 680 revealed th
260                                              Alanine scanning of the active-site residues shows that
261 ucture-activity relations of the Spt5 CTD by alanine scanning of the consensus nonapeptide.
262                                              Alanine scanning of the E6AP-UbcH7 binding interface ide
263 ar determinants of interaction, we performed alanine scanning of the Kv4.3 S3b region.
264                                              Alanine scanning of the last five residues revealed the
265                                              Alanine scanning of the loops surrounding the protease a
266                                              Alanine scanning of the peptide sequence, combined with
267                                 Furthermore, alanine scanning of the RBD has identified several resid
268                                              Alanine scanning of their complementarity-determining re
269                                              Alanine scanning of this interface has identified the ho
270 n to a previously described "DXL" motif, the alanine scanning results predicted four amino acid posit
271                                              Alanine scanning revealed that hydrophobic amino acids i
272 of the TCR-peptide-HLA ternary complexes and alanine scanning revealed that the autologously derived
273  of UBL-containing proteins and IKKbeta, and alanine scanning revealed that the leucine at position 3
274                                 Furthermore, alanine scanning reveals modest differences in the ssDNA
275                                              Alanine scanning reveals residues crucial for GEF activi
276                                  An unbiased alanine-scanning screen covering the entire region combi
277                               We identify by alanine scanning seven functionally important amino acid
278 ium exchange mass spectrometry (H/DXMS), and alanine scanning site-directed mutagenesis.
279                                              Alanine scanning studies of the secreted recombinant rec
280                     We found from exhaustive alanine scanning studies that fibrillation of this WW do
281 analyzing the binding of Brd4 to a series of alanine-scanning substitution mutants of the human papil
282                                              Alanine-scanning substitution of this 14 amino acid regi
283 characterized using overlapping peptides and alanine scanning substitutions and were localized to two
284 itope for gp120, as previously identified by alanine scanning substitutions on the gp120 surface.
285                                Here, we used alanine scanning substitutions spanning residues 1023 to
286 stigate the bioactive surface of ProTx-II by alanine-scanning the toxin and analyzing the interaction
287  box C/D small RNP complex, we have employed alanine scanning to evaluate the interaction between the
288              We have performed computational alanine scanning to gain insight into thermodynamic aspe
289                             Finally, we used alanine scanning to identify determinants in the C-termi
290 ting affinities of protein variants (shotgun alanine scanning) to analysis of GB1 stability.
291                                Computational alanine scanning using the molecular mechanics Poisson-B
292 ssays for receptor binding and calcium flux, alanine-scanning variants of nociceptin indicated that f
293                                Computational alanine scanning was also conducted to identify putative
294    Each of the 23 BLIP positions examined by alanine scanning was randomized to create libraries cont
295                                              Alanine scanning was used to construct mutants of hOAT1,
296                                Computational alanine scanning was used to identify key residues in th
297                        Combinatorial shotgun alanine-scanning was used to assess intramolecular coope
298                                        Using alanine scanning, we demonstrated that Leu-127 and Leu-1
299                                           By alanine scanning, we identified a single mutation in the
300 p these determinants, we performed global E2 alanine scanning with a panel of 16 human monoclonal ant

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