ライフサイエンスコーパス: alanosine

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1 otency of geldanamycin, but did not affect L-alanosine.

2 and other MTAP-deficient malignancies with L-alanosine.

3 Thus, our results support the use of L-alanosine alone or in combination with a salvage agent a

4 is bound to carbamyl phosphate (CP) and to L-alanosine (an analogue of aspartate).

5 Alanosine, an inhibitor of AMP synthesis, inhibited the

6 ells are more sensitive to the toxicity of L-alanosine, an inhibitor of de novo AMP synthesis, than a

7 cells, allows for selective therapy using L-alanosine, an inhibitor of de novo AMP synthesis.

8 trexate, 5,10-dideazatetrahydrofolate, and L-alanosine and by methionine depletion.

9 rve as a predictor of cellular response to L-alanosine and glutathione-mediated resistance to geldana

10 rian SK-OV-3 cells, reduced the potency of L-alanosine and lowered intracellular glutathione levels.

11 e correlations, e.g., amino acid analogue, L-alanosine, and 296 with negative correlations, e.g., gel

12 e that SLC7A11 mediates cellular uptake of L-alanosine but confers resistance to geldanamycin by supp

13 The therapeutic index of L-alanosine can be increased by the use of a MTAP substrat

14 t strongly supports the kinetic results that alanosine did not inhibit the carbamylation of aspartate

15 Moreover, the alanosine position in this T-state is somewhat displaced

16 ctive agent for salvaging MTAP+ cells from L-alanosine toxicity and is superior to MTA due to lower c

17 ls obtained at relapse are as sensitive to L-alanosine toxicity as diagnosis samples.

18 electively rescued MTAP+ MOLT-4 cells from L-alanosine toxicity at 25 microM with negligible toxicity

19 TAP+ primary T-ALL cells were rescued from L-alanosine toxicity by the MTAP substrate 5'-deoxyadenosi

20 rinsically MTAP+, would be protected from L.-alanosine toxicity, whereas MTAP-tumor cells would be ki

21 P+ T-ALL cells and normal lymphocytes from L-alanosine toxicity.

22 MTAP(+) cells but not the MTAP(-) cells from alanosine toxicity.

23 MTAP-primary T-ALL cells was inhibited by L-alanosine with a mean IC50 of 4.8+/-5.3 ILM (range, 0.3-

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