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1 I, 0.42 to 0.74 per 1 g/dl increase in serum albumin concentration).
2 greater prognostic accuracy than total serum albumin concentration.
3 od urea nitrogen concentration and the serum albumin concentration.
4 ther factors that may affect muscle mass and albumin concentration.
5 and manage conditions that reduce the serum albumin concentration.
6 erum total cholesterol, LDL cholesterol, and albumin concentrations.
7 ation than those with normal or higher serum albumin concentrations.
8 ischarge weight and had repleted their serum albumin concentrations.
9 ated with lower BMI but not with lower serum albumin concentrations.
10 which Hpn knockout mice manifest low plasma albumin concentrations.
11 nd baseline information about eGFR and urine albumin concentrations.
12 mostly normal levels of serum bilirubin and albumin concentrations.
13 r 3 and blood urea nitrogen but higher serum albumin concentrations.
14 95% CI: 0.84, 0.99) for a 1-g/L higher serum albumin concentration].
15 (5.09 +/- 0.24) x 10(7) liters/mol at lower albumin concentrations (15 microm) to (0.54 +/- 0.05) x
16 8.1 yr; Child-Pugh score, 8.5 +/- 1.0; serum albumin concentration, 3.0 +/- 0.6 g/dl) were studied in
20 and then we used it to assess the effect of albumin concentration and buffer composition on binding.
23 Preoperative laboratory variables, including albumin concentration and donor-related information, wer
28 ought to stem from the prevailing low plasma albumin concentration and the decreased transcapillary o
29 exhibited elevated PGE2, reduced circulating albumin concentrations and EP2-mediated immunosuppressio
30 ckout and control rats showed higher urinary albumin concentrations and reduced amounts of megalin an
31 clude altered fluid status, changes in serum albumin concentrations and renal and hepatic function, a
32 ng a 12 min walk, haemoglobin concentration, albumin concentration, and age-corrected resting heart r
33 osis on initial biopsy as well as age, serum albumin concentration, and CKD stage at onset affected E
34 ion with the IL28B CC genotype, had a higher albumin concentration, and had a lower HCV viral load at
35 itrogen level, impaired sensorium, low serum albumin concentration, and partial thromboplastin time <
36 itrogen level, impaired sensorium, low serum albumin concentration, and partial thromboplastin time <
37 te were associated with drug dose and plasma albumin concentration, and were lower among men and amon
39 ning test solutes and artificial plasma with albumin concentration approximately 4 g/dl was dialyzed
40 dy suggests that decreases with age in serum albumin concentrations are associated with muscle loss (
41 ([normal albumin] [observed albumin]), where albumin concentrations are in g/L; if given in g/dL, the
42 be adjusted for the effect of abnormal serum albumin concentrations as follows: adjusted anion gap =
43 normalization of ALT at 6 months, low serum albumin concentration at diagnosis, and age at presentat
45 ysis, the serum monoclonal protein and serum albumin concentrations at diagnosis were the only risk f
49 er unit length, skinfold thickness and serum albumin concentration, but only in a sea lion colony exp
50 , C-reactive protein (CRP), haptoglobin, and albumin concentrations by radial immunodiffusion assays.
51 ntal studies have shown that a reduced serum albumin concentration can increase the volume of distrib
54 ation, which represents the fact that plasma albumin concentration does not reflect its function.
55 amount of weight loss and no improvement in albumin concentrations during the first month after hosp
57 pHo changes, and the presence of physiologic albumin concentrations failed to attenuate the pHo-media
58 d)/days, where albumin 1 and 2 are the serum albumin concentrations (g/L) at the beginning and end of
60 ssessed whether individuals with a low serum albumin concentration have delayed progression to AIDS,
62 d plasmatic parameters except an increase in albumin concentration in septic rats compared with the b
63 ors of poor outcome were adjusted for, serum albumin concentration in the hospital was a strong and i
64 re were no significant differences in plasma albumin concentrations in nonedematous and edematous chi
67 (as assessed by bronchoalveolar lavage fluid albumin concentration) in both NADPH oxidase-deficient m
71 l) porphine were determined as a function of albumin concentration, ionic strength in medium, pH, and
73 effects of the elevated circulating glycated albumin concentration is associated with reduction in pr
76 te phase protein (alpha2 macroglobulin), and albumin concentration is inversely proportional to that
78 1.9 vs. 2.2 +/- 0.6; P =.03) and lower serum albumin concentrations (low: 2.8 +/- 0.1 vs. normal: 3.3
79 = 75 y, use of > or = 3 medications, and an albumin concentration < 35.0 g/L were significant predic
80 Ninety-six percent of the patients had serum albumin concentration < or = 3 SD below the mean of the
83 f these compounds and/or inappropriately low albumin concentrations may blur the interpretation of th
86 was considered the upper limit of normal for albumin concentration, Micral-Test, sulfosalicylic acid
87 increased risk of disability with low serum albumin concentrations observed in the elderly may actua
88 blood urea nitrogen of > or =24 mg/dL; serum albumin concentration of < or =4.0 g/dL (< or =40.0 g/ L
90 als with hypoalbuminemia (defined as a serum albumin concentration of <35 g/L) at ART initiation had
91 in FcRn-deficient mice, and that the plasma albumin concentration of FcRn-deficient mice is less tha
92 07; P < .001) that of individuals with serum albumin concentrations of >/= 35 g/L, after multivariate
94 nvestigate the impact of pretransplant serum albumin concentration on post-transplant outcome in hear
95 en at relatively low hydraulic pressures the albumin concentration on the tissue side of the glycocal
96 at is only minimally sensitive to glycation, albumin concentration, or redox potential, unlike other
97 (S6K) pathway, but pathophysiologically high albumin concentrations overactivated mTORC1 and inhibite
99 P < .001) and was related to a greater serum albumin concentration (P < .001) and to a lower exposure
100 lations between the hematocrit and the serum albumin concentration (P = 0.009) and between the hemato
101 Albumin administration increased plasma albumin concentrations (p <.05 compared with placebo) an
103 the nephrotic syndrome (as assessed by serum albumin concentration), preexisting thrombophilic states
104 tes, serum creatinine concentration, urinary albumin concentration, previous cardiovascular event, an
105 g affinity remained heavily dependent on the albumin concentration (range (5.37 +/- 0.26) x 10(7) lit
106 y ratios (W/D) and tissue-to-plasma 125I-rat albumin concentration ratios (T/P) 8 h after the endotox
107 ting only studies in which, depending on the albumin concentration, real or extrapolated free concent
108 ntly during the study period, but only serum albumin concentrations showed a significant association
112 s implied that for every 1 mg/dL increase in albumin concentration, the post-transplant 1-year mortal
113 herein that the time required for the tissue albumin concentration to increase to values for a new st
114 e albumin:creatinine ratio (ACR) and urinary albumin concentration (UAC) obtained from a single sampl
121 , lymph node metastasis) and decreased serum albumin concentration were unfavorable for long-term sur
129 s (NHB) and Mexican Americans, whereas urine albumin concentrations were significantly higher in NHB
130 oxygen partial pressures, and intra-alveolar albumin concentrations) were the same in knockout mice a
131 observation led to the concept of effective albumin concentration, which represents the fact that pl
132 wer free fetuin-A, plasma pyrophosphate, and albumin concentrations, which accounted for 49% of the v
133 mimicked all the effects of pathophysiologic albumin concentrations, which disrupt normal signal tran
134 ndings support the concept of the "effective albumin concentration," which implies that the global HS
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