ライフサイエンスコーパス: alcohol

1 reat analysis (396 IPA and 392 chlorhexidine-alcohol).

2 l SSI prevention compared with chlorhexidine-alcohol.

3 gression in AHAs, but only in the absence of alcohol.

4 component, which represents the short-chain alcohol.

5 vels in the brain, but enhanced responses to alcohol.

6 pecies yielded different amounts of furfuryl alcohol.

7 he anterior back and ventrum after consuming alcohol.

8 gical factor required for appetite evoked by alcohol.

9 ve a regulatory role in stress, anxiety, and alcohol.

10 alcium-dependent PLCs are not upregulated by alcohol.

11 , and to a panel of halogenated methanes and alcohols.

12 ](+*) for chloroalkanes, and [M-H2O](+*) for alcohols.

13 bilayer environments under the influence of alcohols.

14 d the conversion of crude products to chiral alcohols.

15 c oxidative methyl esterification of primary alcohols.

16 produce flavour and a group producing mainly alcohols.

17 des, or under dehydrative conditions with C1 alcohols.

18 These trends were dominated by the higher alcohols (1-hexanol, cis-3-hexenol, trans-2-hexenol) and

19 mer was transformed into ortho-nitrosobenzyl alcohol 11.

20 caused by sodium hypochlorite, 70% isopropyl alcohol, 3% hydrogen peroxide, ethyl alcohol, water imme

21 EtG) alcohol biomarker resulted in increased alcohol abstinence in outpatients with co-occurring seri

22 Six months of alcohol abstinence is typically required before liver tr

23 We determined the risk of an alcohol abuse diagnosis on incident AF, MI, and CHF.

24 reports on epigenetic mechanisms involved in alcohol abuse have focus on hepatic and neuronal regions

25 The population-attributable risk of alcohol abuse on each outcome was of similar magnitude t

26 1 [0.82]; P = .02), substance use disorders (alcohol abuse, 96.5 [0.67]; P < .001; drug abuse, 97.6 [

27 measures (including duration and severity of alcohol abuse, craving, and anxiety or depressive sympto

28 re another key feature of the persistence of alcohol addiction since it leads to relapse and for whic

29 roposal of the aSI model as a valid model of alcohol addiction vulnerability.

30 (EFhd2; Swiprosin-1) plays in the control of alcohol addiction-associated behaviours.

31 conditions were performed under intravenous alcohol administration (6% vol/vol infusion to achieve a

32 Participants stayed overnight after the alcohol administration, and discharge occurred on day 7

33 presence of primary, secondary and tertiary alcohols affords the corresponding glycosides in excelle

34 ide array of functional groups, including an alcohol, aldehyde, epoxide, indole, nitroalkane, and sul

35 Higher alcohols, aldehydes, and ketones characterized the aroma

36 Alcohol-amine combinations are discussed to the extent t

37 n a carboxylic acid, an aldehyde, an allylic alcohol, an aryl olefin, an alpha substituent, or amino

38 rt on a positive regulatory feedback loop of alcohol and FGF2 in rodent models.

39 abolic control and lifestyle choices such as alcohol and nicotine use.

40 es (CNTs) followed by adsorption of furfuryl alcohol and pyrolysis.

41 ants in the National Epidemiologic Survey on Alcohol and Related Conditions and 36309 participants in

42 ants in the National Epidemiologic Survey on Alcohol and Related Conditions III.

43 association was found only for the intake of alcohol and whole grains in relation to colorectal cance

44 everal other compounds, including aldehydes, alcohols and 2-alkylfurans that contribute to overall ar

45 ysis, to produce 4-substituted homopropargyl alcohols and 5-en-3-yn-1-ols, respectively, in one-pot w

46 The reaction between propargylic alcohols and allylic carbonates, engaging vanadium and p

47 ere investigated as potential photocages for alcohols and carboxylic acids, respectively.

48 rogen borrowing reaction employing secondary alcohols and Ph* (Me5C6) ketones to give beta-branched c

49 c oxidation reactions with different primary alcohols and shown to be compatible with substrates bear

50 es toward the synthesis of cis vicinal amino alcohols and tetrasubstituted alpha-borylamido complexes

51 ibiting reagents (e.g., chaotropic salts and alcohol) and (ii) the washing and elution steps that are

52 unds (2-propanol, ethylene glycol, propargyl alcohol, and 2-butoxyethanol) were found to be both mobi

53 Liquids studied to date include water, alcohols, and solutions with relevance to biology and en

54 ids, volatiles, sesquiterpenoids, diterpenes alcohols, and sugar esters from trichomes of the plants,

55 lifestyle factors, most notably smoking and alcohol, are associated with SP risk.

56 d on liquids, in particular, formic acid and alcohols, are highly attractive hydrogen carriers as the

57 of businesses where individuals can purchase alcohol as measured by rates per capita of liquor or con

58 ynthons with 1-aryl/1-heteroaryl propargylic alcohols as C2 synthons.

59 ssociation between the hydronium ion and the alcohol, as well as a higher intrinsic rate constant in

60 The relation between alcohol-attributable harm and socioeconomic status was i

61 The dehydrogenation of higher alcohols available from renewable feedstocks is also des

62 Using an alcohol-based extraction technique, we removed the lipid

63 To compare 2 alcohol-based, dual-action skin preparations for surgica

64 nding of how the BLA-->NAc pathway regulates alcohol behavior, and introduce a new methodology for th

65 ario in which the relative partitioning of n-alcohols between liquid-ordered and liquid-disordered ph

66 tervention using the ethyl glucuronide (EtG) alcohol biomarker resulted in increased alcohol abstinen

67 y driven by those with primary dependence on alcohol but not on opiates.

68 ation of meta- and para-substituted benzylic alcohols by either 1 or 5 displays a strong linear corre

69 within Flory-Huggins theory, the addition of alcohol causes an increase or decrease of the miscibilit

70 The impact of alcohol cessation initiatives on periodontal health shou

71 the monetary incentive delay task following alcohol challenge.

72 electivity and yields in the desaturation of alcohols compared to those obtained by the state-of-the-

73 imethyl sulfide, ethanethiol), and ester and alcohol compounds associated with distinct 'sulfur-cabba

74 flavour profiles, including 36 esters and 6 alcohols compounds, were statistically evaluated by clus

75 Mean amount of alcohol consumed was 82.59 grams over the evening.

76 and sCD163 were higher among ED with recent alcohol consumption (last drink <10 days before enrollme

77 Among females, no association between alcohol consumption and clinical AL progression was obse

78 asal amygdala to CeA projections showed that alcohol consumption and withdrawal were associated with

79 LDH2 and ADH1B, are strongly associated with alcohol consumption but have limited impact in European

80 independent phenotypes reflecting subjects' alcohol consumption during the past year, based on self-

81 Voluntary prolonged and excessive alcohol consumption in a 2-bottle choice procedure incre

82 ed a genome-wide association study (GWAS) of alcohol consumption in the large Genetic Epidemiology Re

83 BACKGROUND & AIMS: Chronic, excessive alcohol consumption leads to alcoholic liver disease (AL

84 Further, the results suggest that alcohol consumption may activate endothelial EVs towards

85 Low dosages (</=1.37 g of alcohol/day) of alcohol consumption may be beneficial to prevent periodo

86 cant SNP-based heritability of self-reported alcohol consumption of 13% (se=0.01).

87 sly reported SNP rs1229984 in ADH1B and both alcohol consumption phenotypes (OR=0.79, P=2.47 x 10(-20

88 ogical mechanism underpinning its effects on alcohol consumption remains to be determined.

89 Alcohol consumption was not associated with breast cance

90 late MSN activity, and their consequences on alcohol consumption were measured.

91 ctivity, healthy diet, and light-to-moderate alcohol consumption were positively associated with cogn

92 ate hypothetical effects of habits (smoking, alcohol consumption, and fat and carbohydrates consumpti

93 enarche, age at first birth, family history, alcohol consumption, and smoking status, which suggests

94 eta signaling dramatically reduced excessive alcohol consumption, as did selective inhibition of D1-M

95 biologically proximal environmental factors alcohol consumption, body mass index, smoking and matern

96 unacceptable harmful consequences of excess alcohol consumption, obesity, and viral hepatitis.

97 ss index, healthy diet, sedentary lifestyle, alcohol consumption, smoking, and urinary sodium excreti

98 seeking/low anxiety associated with enhanced alcohol consumption, which may be related to cortex func

99 ed negative binomial regression analysis for alcohol consumption.

100 ty, affording versatile tertiary propargylic alcohols containing two stereocenters.

101 nge significantly in the wines with modified alcohol content even at higher juice substitution rates.

102 nt groups on any other measures of drinking, alcohol craving, or alcohol-related consequences.

103 lacebo for 16 weeks and administered an fMRI alcohol cue reactivity task at baseline and after 2 week

104 or placebo for 8 days, and underwent an fMRI alcohol cue-reactivity task (day 7; n=81) and a bar lab

105 Low dosages (</=1.37 g of alcohol/day) of alcohol consumption may be beneficial to

106 Cinnamyl alcohol dehydrogenase (CAD) catalyzes the final step in

107 oise responses and enables identification of alcohol dehydrogenase 5 (ADH5) as an enzyme that regulat

108 ly involved in natural product metabolism-an alcohol dehydrogenase and a cytochrome P450-produces une

109 D(+) and N(tz)ADH to be substrates for yeast alcohol dehydrogenase and lactate dehydrogenase, respect

110 ol oxidases (AAO) and the intracellular aryl-alcohol dehydrogenases (AAD) were also produced after ex

111 nd product yields of eight other short-chain alcohol dehydrogenases/reductases.

112 The different types of acceptorless alcohol dehydrogenation (AAD) reactions are discussed, f

113 Alcohol dependence (AD) and major depression (MD) are le

114 comprehensive miRNA expression data set for alcohol dependence and identify the causal miRNAs for al

115 nking risk levels between Waves 1 and 2, and alcohol dependence at Wave 2.

116 Yale-Penn study of the genetics of drug and alcohol dependence from February 14, 1999, to January 13

117 Here, we found that a history of alcohol dependence persistently decreased the expression

118 ve been implicated in the pathophysiology of alcohol dependence, but the specific molecular mechanism

119 fifty-two treatment-seeking individuals with alcohol dependence, half preselected to carry at least o

120 ALDH2 on lifetime measures, such as risk of alcohol dependence, our study adds further evidence of t

121 ntribute to various deficits associated with alcohol dependence.

122 g the neuroimmune system as therapeutics for alcohol dependence.

123 ependence and identify the causal miRNAs for alcohol-dependency in patients which were validated by t

124 We demonstrate that alcohol-dependent increases in Prosapip1 levels promote

125 orrelates as well as alcohol expectancies in alcohol-dependent patients and healthy controls and asse

126 ndent animals, but its potency was higher in alcohol-dependent rats.

127 Alcohol-dependent subjects were imaged 1-4 days (n=14) o

128 g reagent gave a very functionalized allylic alcohol derivative in 86% yield.

129 research for developing small-scale portable alcohol detection system has been accelerated with the a

130 tted for alcohol withdrawal before and after alcohol detoxification.

131 Age and alcohol diagnosis were significant predictors of alcohol

132 ific NOX4 KO mice and these were pair-fed on alcohol diet.

133 In addition, the role of alcohol drinking on outcomes in patients with cancer is

134 ing in the rural area, cigarette smoking and alcohol drinking were associated with insomnia.

135 ort for a neuroimmune mechanism of excessive alcohol drinking.

136 rol and decision-making in adults who abused alcohol during adolescence.

137 Exposing rats to alcohol during early-mid adolescence (PD28-42) increased

138 culations on FeO/Pt(111) reveals that benzyl alcohol enriches preferentially at the oxygen-terminated

139 lycerides, fatty amides, aldehydes, ketones, alcohols, epoxides, furans, pyrans and terpenic oxygenat

140 making and its neural correlates as well as alcohol expectancies in alcohol-dependent patients and h

141 eutic interventions should target subjective alcohol expectancies.

142 Longer alcohol exposure (7 d x 2.5 g/kg, i.p.) restricted these

143 Low, moderate to high, or binge-level alcohol exposure in the first trimester or throughout pr

144 Results showed that chronic alcohol exposure induced hepatocyte apoptosis in associa

145 translational mouse model of AUD showed that alcohol exposure leads to PCSK9 downregulation.

146 tion between craniofacial shape and prenatal alcohol exposure was observed at almost any level regard

147 neutrophils isolated from chronic plus binge alcohol fed mice or from human blood, and decreased the

148 hesis reduced PLIN2 and hepatic steatosis in alcohol-fed mice, but only de novo synthesis inhibition,

149 Chronic alcohol feeding causes lipid accumulation and apoptosis

150 We demonstrated that ten-day alcohol feeding primed the liver to LPS-induced lipid ac

151 We show that chronic alcohol feeding sensitizes rat hepatocytes to Ca(2+) -mo

152 ehicle was administered daily throughout the alcohol feeding study.

153 ses in control livers, but not after chronic alcohol-feeding, suggesting desensitization to the inhib

154 to perform the electro-oxidation of primary alcohols, followed by the separation and detection of th

155 icipants performed a task to gain points for alcohol, food or no reward.

156 e povacrylex-alcohol (IPA) and chlorhexidine-alcohol for elective, clean-contaminated colorectal surg

157 spectrometer, native cholesterol in its free alcohol form is readily detected from a few hundred nano

158 e effect of the moisture content on furfuryl alcohol formation was found to be of little impact.

159 ontrol group and none of the patients in the alcohol-free group.

160 sion mediated by vitamins C, E, and aromatic alcohols has been recently shown to be feasible from a c

161 ous biochemical compounds, from simple diene-alcohols have been carried out in a cascade fashion.

162 n of its predominant product, 4-hydroxycumyl alcohol (HCA), in tandem with transformation of a synthe

163 The inhibition by trans addition of alcohols implies that they alter the planar membrane str

164 on, correlated with increased motivation for alcohol in alcoholics.

165 Results show that coffee produces furfuryl alcohol in larger quantities (418microg/g) compared to o

166 (P) rats intermittent access to water or 20% alcohol in their homecage for 7 weeks (three 24-h sessio

167 d deoxy derivatives, disaccharides and sugar alcohols in (0.05, 0.15, 0.25 and 0.35)molkg(-1) thiamin

168 e the association between hydronium ions and alcohols in a steric environment resembling the constrai

169 and p-coumaraldehyde to their corresponding alcohols in an NADPH-dependent manner.

170 nd to rapidly react with nucleophiles (e.g., alcohols) in reactions that were first-order in cation r

171 We show that alcohol increases Fgf2 expression in the dorsal striatum

172 phases evolves as the chain length of the n-alcohol increases.

173 dy assessed both retrograde facilitation and alcohol induced memory impairment using two independent

174 lar disorder, Tourette's syndrome, dementia, alcohol-induced delusions and obsessive-compulsive disor

175 ext found that S1P effectively could reverse alcohol-induced endothelial barrier dysfunction using bo

176 al reactive oxygen species and contribute to alcohol-induced hepatocyte injury.

177 mice or from human blood, and decreased the alcohol-induced increased liver triglyceride and fat dro

178 phenotype, which could have implications for alcohol-induced tumor angiogenesis.

179 pylene oxide, using a functionalized primary alcohol initiator and magnesium 2,6-di-tert-butyl phenox

180 ear, based on self-reported information: any alcohol intake (drinker/non-drinker status) and the regu

181 Myoclonus improved significantly after alcohol intake (p = 0.016).

182 and Signature E4 is unique in ESCC linked to alcohol intake and genetic variants in alcohol-metaboliz

183 Clinically, low and moderate alcohol intake improves human health with protection aga

184 Repeated alcohol intake leads to mesostriatal neuroadaptations, r

185 cally or intra-PLmPFC, interacted with prior alcohol intake to escalate aggression in ANAs.

186 g years-of-life-lost were 0.5 years for high alcohol intake, 0.7 years for obesity, 3.9 years for dia

187 We directly investigated how alcohol intoxication impacts hemorrhagic shock and resus

188 nferiority trial comparing iodine povacrylex-alcohol (IPA) and chlorhexidine-alcohol for elective, cl

189 While alcohol is a risk factor for breast cancer, less is know

190 l carcinoma with risk factors of tobacco and alcohol is more frequent among blacks.

191 men who are or may become pregnant, avoiding alcohol is the safest option.

192 tive cross-coupling of primary and secondary alcohols is demonstrated.

193 The dehydration of alcohols is involved in many organic conversions but has

194 e also produced after exposure to other aryl-alcohols, known substrates and inducers of AAOs, and dur

195 88c produced approximately double the higher alcohol levels and around twenty times more esters compa

196 ation of this device in wearable transdermal alcohol measurements.

197 Well-known genetic variants in alcohol metabolizing genes, for example, ALDH2 and ADH1B

198 ed to alcohol intake and genetic variants in alcohol-metabolizing enzymes.

199 instruments, we derived separate dimensional alcohol misuse scales and applied a proxy-phenotype stud

200 iderable promise for continuous non-invasive alcohol monitoring in real-life situations.

201 e biosensor performance towards subcutaneous alcohol monitoring was substantiated by the ex vivo mice

202 Alcohol, nicotine, and cannabis are among the most commo

203 rmine the impact of complete abstinence from alcohol on long-term survival and (2) identify prognosti

204 erved a similar influence of the length of n-alcohols on the direction of the shift in Tmix.

205 opioids, cocaine, cannabis, methamphetamine, alcohol, or tobacco.

206 n peroxide (H2O2) enzymatically generated by alcohol oxidase (AOx).

207 One of the best-studied is vanillyl-alcohol oxidase (VAO) from the fungus Penicillium simpli

208 SSPs, aryl-alcohol oxidases (AAO) and the intracellular aryl-alcoho

209 transition temperature the largest, when the alcohol partitions equally between the two phases.

210 e neutral condition allows acidic protons of alcohols, phenols, and malonates to be present, and frag

211 We gave Wistar or alcohol-preferring (P) rats intermittent access to water

212 temperature irrespective of which phase the alcohol prefers.

213 Counselling for Alcohol Problems (CAP), a brief intervention delivered b

214 Alcohol produced a pathological-like pattern of aggressi

215 SF bread, characterized by highest level of alcohols, received the highest score for aroma and sweet

216 Alcohol relapse (>/=30 g/day) was not associated with mo

217 Cumulative incidence rate of first alcohol relapse was 25.2%, 33.7%, and 35.2% at 1, 3, and

218 Alcohol relapse was similar in Group 1 versus Group 2: 2

219 rapeutic improvement, long-term influence of alcohol relapse, and their interaction.

220 injury should be extended to drug-related or alcohol-related and violent injury in adolescence.

221 ation, mortality, and HCC in HBV-, HCV-, and alcohol-related cirrhosis.

222 er measures of drinking, alcohol craving, or alcohol-related consequences.

223 Risks of drug-related or alcohol-related death increased by a similar magnitude.

224 ective and cost-effective policies to reduce alcohol-related harm.

225 aging data showed that ghrelin increased the alcohol-related signal in the amygdala (P=0.01) and modu

226 five causal groups (suicide, drug-related or alcohol-related, homicide, accidental, and other causes

227 injury (ie, self-inflicted, drug-related or alcohol-related, or violent injury) affects 4% of 10-19-

228 ity-related (self-inflicted, drug-related or alcohol-related, or violent injury) or accident-related

229 As a result, much preclinical alcohol research aims to identify relevant CeA neuroadap

230 immune signaling and has been implicated in alcohol responses in animal models and human alcoholics.

231 The alcohol responsiveness of clinical symptoms was evaluate

232 Clinical Institute Withdrawal Assessment for Alcohol, Revised (CIWA-Ar) scores.

233 Alcohol's effect on BAT was assessed by histology, qPCR,

234 king, for which treatments exist, compulsive alcohol seeking is therefore another key feature of the

235 Furthermore, the impaired alcohol seeking was associated with structural alteratio

236 s necessary for cue-induced reinstatement of alcohol seeking, expand our understanding of how the BLA

237 rol extinction and relapse in a rat model of alcohol seeking.

238 disorder that is associated with compulsive alcohol-seeking behavior.

239 earning during extinction of cue-conditioned alcohol-seeking behavior.

240 get to facilitate long-lasting extinction of alcohol-seeking behavior.SIGNIFICANCE STATEMENT Alcohol

241 udies showed that context-induced relapse to alcohol-seeking was attenuated by bilateral microinfusio

242 Alcohol self-administration and cue-induced reinstatemen

243 Both drugs attenuated alcohol self-administration in Wt rats but not in NOP (-

244 ADX71441 dose-dependently decreased alcohol self-administration of both dependent and non-de

245 Profiling of the OFC synaptome identified alcohol-sensitive proteins that control glutamate releas

246 abstraction from the carbinol carbon of the alcohol substrate.

247 The racemic allylic alcohol substrates can be converted to the enantioenrich

248 alkynyl carbonyl compounds employing allylic alcohol substrates in contrast to more traditional conju

249 strain-release "cyclopentylation" of amines, alcohols, thiols, carboxylic acids, and other heteroatom

250 and 53 patients relapsed as indicated by the Alcohol Timeline Followback method.

251 ng of combining the oil with a metal base in alcohol to form a solution, followed by addition of reac

252 xcluded volume interactions (which cause the alcohol to prefer one phase) just balances the effect of

253 eta-amino analogs (via in situ conversion of alcohols to imidates, directed C-H amination, and hydrol

254 amlined protocol enables rapid conversion of alcohols to their beta-amino analogs (via in situ conver

255 Acute alcohol treatment (25 mm) completely inhibited hormone-i

256 from alcohol users and after in vitro acute alcohol treatment of human MDDCs.

257 The synthesis of homopropargylic alcohols under metal-free and mild condition is describe

258 cardiovascular events, diabetes, obesity, or alcohol use (all p<0.01).

259 Heavy alcohol use can lead to progressive liver damage, especi

260 Precision medicine for alcohol use disorder (AUD) allows optimal treatment of t

261 ent directions in medication development for alcohol use disorder (AUD) emphasize the need to identif

262 omplex diseases, but has not been applied to alcohol use disorder (AUD) or other psychiatric diseases

263 Alcohol use disorder has been linked to dysregulation of

264 ire-9, Generalised Anxiety Disorder-7 scale, Alcohol Use Disorder Identification Test (AUDIT), and se

265 result supported by the dFNC outcome and the alcohol use disorder identification test.

266 ohol-seeking behavior.SIGNIFICANCE STATEMENT Alcohol use disorder is a chronic relapsing disorder tha

267 everal risk factors have been identified for alcohol use disorder, many individuals with these factor

268 ovides evidence for a role of aldosterone in alcohol use disorders (AUDs).

269 key strategy to reduce the treatment gap for alcohol use disorders, one of the leading causes of the

270 eases in many chronic comorbidities in which alcohol use has a substantial role.

271 Moderate alcohol use is associated with improved insulin sensitiv

272 current well-being and past-year problematic alcohol use of past-year ayahuasca users and comparison

273 a critical review of the effect of moderate alcohol use on cardiovascular and liver disease in patie

274 gnificantly higher prevalence of tobacco and alcohol use than did high socioeconomic groups.

275 Light/moderate alcohol use was not substantially associated with accele

276 Differences in self-reported alcohol use were maintained at the 3-month follow-up.

277 bstance use disorder (excluding cannabis and alcohol use).

278 jection drug use, 45% a history of hazardous alcohol use, and 57% a comorbid psychiatric diagnosis.

279 factors included vigorous physical activity, alcohol use, fruits, vegetables and foods rich in dietar

280 non-alcoholic fatty liver disease, hazardous alcohol use, or type 2 diabetes, reported higher prevale

281 estyle factors were included: smoking, heavy alcohol use, unhealthy diet and physical inactivity.

282 ied Charlson comorbidity index, smoking, and alcohol use.

283 can be used as the biomarkers for excessive alcohol use.

284 ulated the prevalence of the five major HRBs-alcohol use; cigarette smoking, physical inactivity, unh

285 etylases are modulated in cells derived from alcohol users and after in vitro acute alcohol treatment

286 alpha-olefination of nitriles using primary alcohols, via dehydrogenative coupling of alcohols with

287 opropyl alcohol, 3% hydrogen peroxide, ethyl alcohol, water immersion, ultraviolet light, and heat ex

288 e meta-derivatives are better photocages for alcohols, whereas ortho-derivatives are better protectiv

289 starts from l-lactic acid-derived propargyl alcohol, which is submitted to sequential Sonogashira/Su

290 Alcohols with chain lengths of 10-14 carbons decrease Tm

291 the coupling of primary and secondary benzyl alcohols with indoles to form 3-benzylated indoles and H

292 ry alcohols, via dehydrogenative coupling of alcohols with nitriles, is presented.

293 06) in heavy drinkers primarily admitted for alcohol withdrawal before and after alcohol detoxificati

294 Measures included occurrence of alcohol withdrawal syndrome and delirium tremens, injury

295 Trauma patients with alcohol withdrawal syndrome experience a high occurrence

296 Alcohol withdrawal syndrome severity was defined by CIWA

297 inical outcomes, pharmacologic treatment for alcohol withdrawal syndrome, and Clinical Institute With

298 ns, injury characteristics, risk factors for alcohol withdrawal syndrome, clinical outcomes, pharmaco

299 , as determined by M30 levels, occurs during alcohol withdrawal, and survival data point toward a nov

300 This resulted in an alcohol yield of approximately 7%.