ライフサイエンスコーパス: alcohol abuse

1 proteins that regulate HCV infection during alcohol abuse.

2 vity with potential for use as treatments of alcohol abuse.

3 effects in animal models relevant to drug or alcohol abuse.

4 onsumption that could underlie some forms of alcohol abuse.

5 and was negatively correlated with years of alcohol abuse.

6 offer unexplored targets in the treatment of alcohol abuse.

7 brain is one of the major targets of chronic alcohol abuse.

8 ic view of brain alterations associated with alcohol abuse.

9 to these cardiac diseases in the setting of alcohol abuse.

10 heart disease, hyperlipidemia, obesity, and alcohol abuse.

11 the aging process, therapeutics, and drug or alcohol abuse.

12 development of new therapeutic approaches to alcohol abuse.

13 l stress is an environmental risk factor for alcohol abuse.

14 and seeking in preclinical rodent models of alcohol abuse.

15 mong 14,727,591 patients, 268,084 (1.8%) had alcohol abuse.

16 tic stress disorder (PTSD), head injury, and alcohol abuse.

17 atitis, especially in those who are prone to alcohol abuse.

18 tion of additional miR-9 targets relevant to alcohol abuse.

19 naive subject can predict the likelihood of alcohol abuse.

20 ategies, can reduce the population burden of alcohol abuse.

21 ective drug development for the treatment of alcohol abuse.

22 al gambling, cigarette smoking, and drug and alcohol abuse.

23 bserved in those subjects with substance and alcohol abuse.

24 (BDNF) might be linked with vulnerability to alcohol abuse.

25 lumes than did patients without a history of alcohol abuse.

26 borative care (CC) intervention for PTSD and alcohol abuse.

27 tween subjects with and without a history of alcohol abuse.

28 oms only among those who screen positive for alcohol abuse.

29 nteraction of HIV infection and a history of alcohol abuse.

30 common polymorphism (MAOA-LPR) and risk for alcohol abuse.

31 itive men with and without a past history of alcohol abuse.

32 s, corticotropin-releasing factor (CRF), and alcohol abuse.

33 egative groups with and without a history of alcohol abuse.

34 eural mechanism for genetic predilection for alcohol abuse.

35 cute lung injury as a consequence of chronic alcohol abuse.

36 ver disease, often in the context of chronic alcohol abuse.

37 reported in humans with a history of chronic alcohol abuse.

38 ual-level risk factors for violence, such as alcohol abuse.

39 en dampened MAOA expression, elevated DA and alcohol abuse.

40 ons such as renal failure, diabetes, HIV and alcohol abuse.

41 ders, anxiety disorders, substance abuse, or alcohol abuse.

42 coholic individuals and patients with active alcohol abuse.

43 clinical liver disease, liver cirrhosis, or alcohol abuse.

44 duodenal biopsies from patients with chronic alcohol abuse.

45 isorders including depression, epilepsy, and alcohol abuse.

46 nt of future therapeutic strategies to treat alcohol abuse.

47 king behaviors and behaviors associated with alcohol abuse.

48 a new therapeutic direction for treatment of alcohol abuse.

49 ess also had a higher lifetime prevalence of alcohol abuse (10.1% vs 3.8%, P < .001) and drug abuse (

50 (20%), hypernatremia (12.6%), and extensive alcohol abuse (11.4%).

51 g management of suicide risk (3 indicators), alcohol abuse (2 indicators), and elderly patients; asse

52 One-year prevalence rates of alcohol abuse (2.3% vs. 3.9%; Z=-2.04; p<0.05) and depen

53 lipidemia (8%), metabolic syndrome (9%), and alcohol abuse (21%).

54 deaths [mean 1740] EU-wide) and deaths from alcohol abuse (28.0%, 12.30-43.70; 1550-5490 potential e

55 (40%), peripheral vascular disorders (39%), alcohol abuse (36%), valvular disease (32%), liver disea

56 t of psychiatric disorders; (3) substance or alcohol abuse; (4) compliance with medical regimens; (5)

57 oronary heart disease; 6) hyperlipidemia; 7) alcohol abuse; 8) tobacco use disorder; and 9) random ef

58 1 [0.82]; P = .02), substance use disorders (alcohol abuse, 96.5 [0.67]; P < .001; drug abuse, 97.6 [

59 Understanding the relationship between alcohol abuse, a common and theoretically modifiable con

60 risk factors, such as severe mental illness, alcohol abuse, a painful loss, exposure to violence, or

61 ed damage to cholangiocytes, but not ongoing alcohol abuse, affected liver stiffness.

62 on was associated with a period of decreased alcohol abuse, although recurrence of the alcohol-use di

63 2-fold increased risk of inpatient care for alcohol abuse among patients who had GBS compared with t

64 was no difference in inpatient treatment of alcohol abuse among patients who underwent gastric bypas

65 omic impact worldwide, pharmacotherapies for alcohol abuse and addiction remain limited.

66 ed sensitivity, is an important component of alcohol abuse and addiction.

67 Tolerance is a well described component of alcohol abuse and addiction.

68 l attractive targets for novel treatments of alcohol abuse and addiction.

69 US National Institute on Alcohol Abuse and Alcoholism (AA012388, AA017168, AA0059

70 ctions of alcohol, the National Institute on Alcohol Abuse and Alcoholism and the National Heart, Lun

71 Alcohol abuse and alcoholism are major health problems a

72 feeding mice alcohol (National Institute on Alcohol Abuse and Alcoholism binge model) or chow diets

73 nkers according to the National Institute on Alcohol Abuse and Alcoholism criteria.

74 erview Schedule of the National Institute on Alcohol Abuse and Alcoholism in a large representative s

75 h was organized by the National Institute on Alcohol Abuse and Alcoholism in collaboration with the O

76 mption to sex-specific National Institute on Alcohol Abuse and Alcoholism safe drinking levels.

77 eport published by the National Institute on Alcohol Abuse and Alcoholism showed that liver cirrhosis

78 ug Administration, the National Institute on Alcohol Abuse and Alcoholism, and the National Institute

79 nd could determine individual differences in alcohol abuse and alcoholism, as well as represent a the

80 National Institute on Alcohol Abuse and Alcoholism, US National Institutes of

81 bute to the development of new therapies for alcohol abuse and alcoholism.

82 velopment of numerous pathologies, including alcohol abuse and anxiety.

83 buse, making the association between chronic alcohol abuse and ARDS a common scenario in the intensiv

84 sought to investigate the associations among alcohol abuse and atrial fibrillation (AF), myocardial i

85 The main causes of pancreatitis are alcohol abuse and biliary disease.

86 or perpetuate existing patterns of excessive alcohol abuse and can enhance the probability of relapse

87 ote public education about the risks between alcohol abuse and certain types of cancer; * Support pol

88 Alcohol abuse and dependence (alcohol use disorders, AUD

89 Alcohol abuse and dependence disorders are common in the

90 ening tests to improve the identification of alcohol abuse and dependence disorders, the epidemiology

91 Early recognition of alcohol abuse and dependence is necessary and should pro

92 Alcohol abuse and dependence remain highly prevalent and

93 Alcohol abuse and dependence were the most common disord

94 ncident alcohol-related problems, and DSM-IV alcohol abuse and dependence).

95 he study of ethanol-related traits including alcohol abuse and dependence, and behavioral responses t

96 for DSM-III-R provided lifetime diagnoses of alcohol abuse and dependence, and onset and recency ques

97 Inventory of Consequences-Recent, and DSM-IV alcohol abuse and dependence.

98 ikelihood of meeting diagnostic criteria for alcohol abuse and dependence.

99 ol during adolescence is predictive of adult alcohol abuse and dependence.

100 hiatric comorbidity, and treatment of DSM-IV alcohol abuse and dependence.

101 Lifetime and 12-month DSM-IV alcohol abuse and dependence.

102 additive and interactive effects of previous alcohol abuse and HIV infection on cognition.

103 ts on cognitive function were found for past alcohol abuse and HIV infection, with significant intera

104 increased hepatic injury in a model of binge alcohol abuse and in response to TNF-alpha treatment.

105 osis is the most common consequence of acute alcohol abuse and may predispose to more severe hepatic

106 sk factors for progression of steatosis were alcohol abuse and overweight/obesity; cumulative exposur

107 hould be to change people's attitudes toward alcohol abuse and seeking professional help.

108 Alcohol abuse and/or dependence (AA/D) and mood disturba

109 The prevalence of lifetime alcohol abuse and/or dependence (alcoholism) in patients

110 ed problems or a current or prior history of alcohol abuse and/or dependence at the time of the 1981

111 ntabuse; a compound used in the treatment of alcohol abuse) and the antifungal agent chlordantoin.

112 e and 21 HIV positive) had a past history of alcohol abuse, and 47 (18 HIV negative and 29 HIV positi

113 eline, 40% of the patients had diabetes, 29% alcohol abuse, and 6% HIV or HBV coinfection.

114 research design issues, comorbid conditions, alcohol abuse, and being younger, unmarried, African Ame

115 is is associated with age, gender, diabetes, alcohol abuse, and coinfection with human immunodeficien

116 Younger age, black race, a history of alcohol abuse, and homelessness were predictors of clust

117 lude HCV infection, HBV infection, diabetes, alcohol abuse, and low CD4+ cell count.

118 it aggressiveness, impaired impulse control, alcohol abuse, and low CSF 5-hydroxyindoleacetic acid.

119 tiation, including non-injection drug abuse, alcohol abuse, and mental illness.

120 After adjusting for age, smoking, alcohol abuse, and nonimmunocompromising comorbidities,

121 umatic stress disorder [PTSD], substance and alcohol abuse, and others) and medical disorders (cardio

122 Unemployment, alcohol abuse, and smoking were associated with resistan

123 , diabetes (AOR, 1.79; 95% CI, 1.6-2.1), and alcohol abuse (AOR, 1.78; 95% CI, 1.5-2.1), whereas blac

124 other complex disease traits, alcoholism and alcohol abuse are influenced by the combined effects of

125 Use of alcohol abuse as a screening method for alcohol dependen

126 ns are significantly influenced by substance/alcohol abuse as well as postmortem interval; however, t

127 re identified as having a history of chronic alcohol abuse based on a positive response to an alcohol

128 preceded or was concurrent with the onset of alcohol abuse (Bipolar First), and 83 subjects with bipo

129 i in fecal samples from patients with active alcohol abuse but not in controls.

130 red in individuals with a history of chronic alcohol abuse but without ARDS (0.46 ng/mL [0.12-2.75],

131 g is a well-known risk factor for subsequent alcohol abuse, but the neural events underlying this ris

132 s is a well-known risk factor for subsequent alcohol abuse, but the neural mechanisms underlying inte

133 9 consecutive patients with ongoing or prior alcohol abuse, but without known liver disease.

134 (diabetes, insulin resistance, obesity) and alcohol abuse can be influenced before starting treatmen

135 Drug and alcohol abuse can produce a variety of ocular and neuro-

136 3 or more pressors simultaneously, extensive alcohol abuse, cancer history (nonskin), active meningit

137 tors, although an interaction with substance/alcohol abuse cannot be completely ruled out.

138 al abuse, neglect, caregiver's substance use/alcohol abuse, caregiver's depressive symptoms, caregive

139 Alcohol abuse causes half of all deaths from cirrhosis i

140 ABSTRACT: Alcohol abuse causes major social, economic and health-r

141 Alcohol abuse causes widespread changes in gene expressi

142 as attributable to CV risk factors, IHD, and alcohol abuse combined, whereas among RA subjects, only

143 these individuals with a history of chronic alcohol abuse compared with healthy controls, suggesting

144 Thus, alcohol abuse could be a co-factor in progression of HIV

145 measures (including duration and severity of alcohol abuse, craving, and anxiety or depressive sympto

146 ine dependence (4.5%; 95% CI, 3.6% to 5.4%), alcohol abuse/dependence (0.3%; 95% CI, 0.1% to 0.6%), a

147 UC patients with regard to PTSD (P =.01) and alcohol abuse/dependence (P =.048).

148 Bipolar II disorder predicted both alcohol abuse/dependence and benzodiazepine use and abus

149 showed on average a decrease in the rate of alcohol abuse/dependence of -24.2% (95% CI, -19.9% to -2

150 ar carcinoma and NHL and presence of HCV and alcohol abuse/dependence using International Classificat

151 Alcohol abuse/dependence was diagnosed by using the Alco

152 ement, (2) motivational interviews targeting alcohol abuse/dependence, and (3) evidence-based pharmac

153 of older age, those with obesity, those with alcohol abuse/dependence, and those who lacked insurance

154 ) of combat veterans in which PTSD, lifetime alcohol abuse/dependence, and Vietnam versus Gulf War se

155 ficantly greater risk for the later onset of alcohol abuse/dependence, cannabis use and abuse/depende

156 After adjusting for HCV infection and alcohol abuse/dependence, HIV status was not independent

157 atios of advanced fibrosis or cirrhosis were alcohol abuse/dependence, obesity, and being uninsured.

158 After adjusting for HCV and alcohol abuse/dependence, the IRR for NHL comparing HIV-

159 xplained by the higher prevalence of HCV and alcohol abuse/dependence.

160 cidal ideation/attempt; nicotine dependence; alcohol abuse/dependence; and illicit drug abuse/depende

161 The alcohol-abuse deterrent disulfiram (DSF) is shown to hav

162 oints in a combined temporal sepsis network: Alcohol abuse, Diabetes and Cardio-vascular diagnoses.

163 In DSM-IV, an alcohol abuse diagnosis is preempted by dependence, alth

164 We determined the risk of an alcohol abuse diagnosis on incident AF, MI, and CHF.

165 Alcohol abuse disorders are associated with dysfunction

166 Childhood conduct and major depressive and alcohol abuse disorders were the most prevalent.

167 naling could be developed as medications for alcohol abuse disorders.

168 drug may be developed as a new treatment for alcohol abuse disorders.

169 this polymorphism increases the severity of alcohol abuse disorders.

170 lled consumption of alcohol is a hallmark of alcohol abuse disorders; however, the central molecular

171 have an important role in the development of alcohol-abuse disorders and studies from this laboratory

172 opathology; e.g., binge eating and opiate or alcohol abuse, disorders in which muORs and aberrant cor

173 Alcohol abuse does not always accompany alcohol dependen

174 Our studies indicate that alcohol abuse, even for a short duration, results in the

175 of dysthymia, major depressive disorder, and alcohol abuse for sexually abused white participants.

176 ugh posttraumatic stress disorder (PTSD) and alcohol abuse frequently occur among acutely injured tra

177 Acute pancreatitis is associated with alcohol abuse, gallstones and bacterial infection.

178 t severe disorder (P < .001), and those with alcohol abuse had a mean (SE) of 5.6 (1.2) points lower

179 In addition, patients who resumed alcohol abuse had decreased medial frontal gray matter v

180 nt score, patients with a history of chronic alcohol abuse had more severe nonpulmonary organ dysfunc

181 Alcohol abuse has long been known to adversely affect in

182 plexes (mRCC) that are adversely affected by alcohol abuse has not been studied.

183 Individuals with a history of chronic alcohol abuse have decreased concentrations of glutathio

184 reports on epigenetic mechanisms involved in alcohol abuse have focus on hepatic and neuronal regions

185 nstrates that HIV infection and a history of alcohol abuse have independent effects on some aspects o

186 cal comorbidity index, dementia, depression, alcohol abuse, head injury, smoking, body mass index, an

187 mans is induced by various insults including alcohol abuse, hepatitis B/C virus infection, autoimmune

188 Donor age, obesity, alcohol abuse, hepatitis serology, liver only donor, ima

189 ally severe in men and women with comparable alcohol abuse histories.

190 s remained significant after controlling for alcohol abuse history.

191 ently predicted by higher comorbidity index, alcohol abuse, history of trauma, and acquired abnormal

192 -Stage Liver Disease (MELD) score, diabetes, alcohol abuse, HIV, or HBV coinfection were collected at

193 r, strains and younger age, urban residency, alcohol abuse, homelessness, noninjection drug use, and

194 mechanism for developing myelosuppression in alcohol-abusing hosts with severe bacterial infection.

195 Although it is well recognized that alcohol abuse impairs alveolar macrophage immune functio

196 Alcohol abuse impairs immune defense.

197 the third theme in the paper - screening for alcohol abuse in college settings.

198 mechanisms for the synergism between HCV and alcohol abuse in liver disease progression.

199 Alcohol abuse increased the risk of AF, MI, and CHF to a

200 The results suggest that persistent alcohol abuse increases periodontitis development by hei

201 Alcohol abuse increases the incidence of acute respirato

202 Previous alcohol abuse increases the risk of developing mood diso

203 Chronic alcohol abuse induces liver injury and increases the sev

204 Alcohol abuse is a comorbid factor in many human immunod

205 Chronic alcohol abuse is a comorbid variable of acute respirator

206 Alcohol abuse is a leading cause of liver disease charac

207 Alcohol abuse is a leading cause of pancreatitis, accoun

208 Alcohol abuse is a leading cause of preventable death an

209 Alcohol abuse is a major cause of liver injury.

210 Alcohol abuse is a major cause of pancreatitis in people

211 Alcohol abuse is a major risk factor for pancreatitis an

212 Alcohol abuse is a significant medical and social proble

213 Alcohol abuse is a widespread and serious problem.

214 Relapse to alcohol abuse is an important clinical issue that is fre

215 We conclude that chronic alcohol abuse is an independent risk factor for acute re

216 A history of chronic alcohol abuse is associated with an increased incidence

217 Chronic alcohol abuse is associated with an increased incidence

218 Chronic alcohol abuse is associated with deficits in cognitive c

219 Alcohol abuse is associated with immunosuppressive and i

220 ovel clinical findings provide evidence that alcohol abuse is associated with significant zinc defici

221 de and other biomarkers for the detection of alcohol abuse is being better established.

222 Ill-health due to alcohol abuse is improving in some nations but deteriora

223 iver disease that develops in the absence of alcohol abuse is recognized increasingly as a major heal

224 ly increased incidence included substance or alcohol abuse, latent tuberculosis, diabetes mellitus, a

225 ultivariate predictors of mortality included alcohol abuse, low body mass index, and poor social adju

226 atients had a significant history of chronic alcohol abuse, making the association between chronic al

227 equent to comorbidity with chronic diseases, alcohol abuse, malignancy, immune deficient/suppression

228 aggravates the severity of ALD; conversely, alcohol abuse may be a cryptic co-factor in some cases o

229 Individuals with a history of past alcohol abuse may be at greater risk for cognitive dysfu

230 ALD patients is hefty and the prevalence of alcohol abuse may be increasing in both the developed an

231 for age, sex, socioeconomic status, smoking, alcohol abuse, medication, and comorbidity were estimate

232 Other causes include alcohol abuse, medications, and hepatitis B and C.

233 violations, aggressive driving, sleepiness, alcohol abuse, metabolic disorders, and multiple medicat

234 Thus, efforts to mitigate alcohol abuse might result in meaningful reductions of c

235 Offspring of an alcohol-abusing monozygotic twin whose co-twin was AD we

236 creatitis with pancreas divisum (n = 3), and alcohol abuse (n = 2).

237 d trauma, surgery, drug therapy, smoking, or alcohol abuse, nor was there any relevant family history

238 However, the effects of chronic alcohol abuse on acute lung injury and nonpulmonary orga

239 The population-attributable risk of alcohol abuse on each outcome was of similar magnitude t

240 odel of HIVE, we investigated the effects of alcohol abuse on the clearance of virus-infected macroph

241 tudy was to determine the effects of chronic alcohol abuse on the incidence and severity of the acute

242 , and smoking status, the effects of chronic alcohol abuse on the incidence of acute respiratory dist

243 , and smoking status, the effects of chronic alcohol abuse on the incidence of nonpulmonary organ dys

244 organ disease develops in a minority, due to alcohol abuse or concurrent genetic modifiers that are n

245 (OR, 0.29; 95% CI, 0.90 to 0.92) or lifetime alcohol abuse or dependence (OR, 0.30; 95% CI, 0.15 to 0

246 e disorder (ie, drug abuse or dependence and alcohol abuse or dependence), and any mental disorder (i

247 43 (1.27-1.64) for GAD, 1.20 (1.08-1.35) for alcohol abuse or dependence, 1.39 (1.18-1.67) for PTSD,

248 25.3% for social phobia, 9.1% vs. 25.9% for alcohol abuse or dependence, and 6.7% vs. 17.6% for drug

249 stment for a prior major depressive episode, alcohol abuse or dependence, and drug abuse or dependenc

250 d with an increased risk of depression, GAD, alcohol abuse or dependence, and PTSD and with some patt

251 ving while impaired sample reported 12-month alcohol abuse or dependence, compared with 1.0% and 1.8%

252 ving while impaired sample reported 12-month alcohol abuse or dependence, compared with to 2.0% and 1

253 history of depression did not predict later alcohol abuse or dependence.

254 sence or the absence of PTSD did not predict alcohol abuse or dependence.

255 nia or schizoaffective disorder and lifetime alcohol abuse or dependence; 64 men with DSM-III-R schiz

256 Alcohol dependence and alcohol abuse or harmful use cause substantial morbidity

257 ology and for all subjects with a history of alcohol abuse or major depression (but no PTSD), the Cz

258 liver dysfunction, or reported a history of alcohol abuse or recent anemia therapy left 4,940 NHANES

259 usion (OR 2.75, 95% CI 1.22-6.37, p = .016), alcohol abuse (OR 2.09, 95% CI .88-5.10, p = 0.098), rec

260 ), peripheral vascular disorders (OR = 1.8), alcohol abuse (OR = 1.8), neurological disorders (OR = 1

261 S, coagulopathy, deficiency anemia, obesity, alcohol abuse, or drug abuse) were associated with highe

262 liver illnesses, including viral hepatitis, alcohol abuse, or metabolic disease.

263 in individuals without a history of chronic alcohol abuse (p < .001).

264 In multivariable analysis, living alone, alcohol abuse, perception of medical care as being a sub

265 V-positive subjects, those with a history of alcohol abuse performed more poorly on tests of verbal I

266 e than double the risk of inpatient care for alcohol abuse postoperatively compared with patients und

267 e (PR = 1.41; 95% CI: 1.21-1.63; P = 0.001), alcohol abuse (PR = 2.57; 95% CI: 2.33-2.84; P = 0.001),

268 that homelessness, intravenous drug use, and alcohol abuse predisposed patients to emm32.2 iGAS disea

269 Alcohol abuse predisposes the host to bacterial infectio

270 Differences between men and women in alcohol abuse prevalence have long been attributed to so

271 21) subjects had higher risks of drug abuse, alcohol abuse, rapid cycling, and suicide attempts.

272 disturbance, in turn, increases the risk of alcohol abuse relapse.

273 quency of mood disorders among patients with alcohol abuse relapse.

274 e higher IL-8 levels, liver surgery, chronic alcohol abuse, shock, higher peak airway pressure while

275 ated with a period of recovery from comorbid alcohol abuse, suggesting this posthospital time may pro

276 correlation of reduced binding with years of alcohol abuse suggests an involvement of CB1 receptors i

277 y influence the development or expression of alcohol abuse syndromes in animal models or humans.

278 ere lower in feces from patients with active alcohol abuse than controls.

279 more likely to receive a diagnosis of DSM-IV alcohol abuse than their peers not attending college; de

280 promising lead for the development of novel alcohol abuse therapies.

281 drug targets would be clinically relevant in alcohol abuse treatment and may serve to provide a bette

282 Individuals with a history of chronic alcohol abuse underwent bronchoalveolar lavage within 7

283 9H-SAT, men with a history of incarceration, alcohol abuse, use ever of intravenous drugs, younger ag

284 Prevalence of lifetime and 12-month alcohol abuse was 17.8% and 4.7%; prevalence of lifetime

285 patients with a positive history of chronic alcohol abuse was 70% (46 of 66), compared with 31% (47

286 volving 571 intensive care patients, chronic alcohol abuse was a significant comorbid variable that i

287 After multivariable adjustment, alcohol abuse was associated with an increased risk of i

288 A history of chronic alcohol abuse was determined by a Short Michigan Alcohol

289 Overall, prevalence of smoking and alcohol abuse was higher among patients with AD than the

290 Current alcohol abuse was more prevalent among men, whites, and

291 The relative risk of inpatient care for alcohol abuse was studied before and after surgery.

292 Chronic alcohol abuse weakens alveolar tight junctions, priming

293 ectin levels from the patients with ARDS and alcohol abuse were also significantly elevated compared

294 Symptoms of dysthymia, major depression, and alcohol abuse were assessed using the National Institute

295 ronic renal failure, diabetes mellitus, HIV, alcohol abuse) were more common in nonwhite sepsis patie

296 in relation to depressive symptomatology and alcohol abuse with conflicting findings.

297 y (2003-2014) to estimate the association of alcohol abuse with liver fibrosis.

298 been reported in animal models of epilepsy, alcohol abuse, withdrawal, and stress.

299 omfort-care), intraabdominal conditions, and alcohol abuse/withdrawal.

300 m of the OPN gene, compared to patients with alcohol abuse without liver disease.