ライフサイエンスコーパス: alkalization

1 he Rim101 transcription factor upon external alkalization.

2 creasing the voltage sensitivity of internal alkalization.

3 g during acidification and decreasing during alkalization.

4 complex results in defective phagolysosomal alkalization.

5 dependent NHE1-activation and subsequent pHi alkalization.

6 This was potentiated with intracellular alkalization.

7 tion in the ER, were defective for endosomal alkalization.

8 080 led to marked enhancement of the rate of alkalization.

9 prospectively abandoned hyperventilation and alkalization.

10 the negative effects of hyperventilation and alkalization.

11 r disruption byd-ceramide-C6 causes Golgi pH alkalization.

12 s patients treated with hyperventilation and alkalization, 13 before and 16 after the availability of

13 Ammonia and intracellular alkalization activate TRPV1 through a mechanism that inv

14 n murine sperm, SLO3 mediates a voltage- and alkalization-activated K(+) current essential to male fe

15 Paradoxically, AE3 enhances intracellular re-alkalization after MAc removal in neurons and astrocytes

16 AE3 speeds re-alkalization after removal of MAc in neurons and astrocy

17 of this kinase paralleled or preceded medium alkalization and activation of the defense gene phenylal

18 ression of cell wall elicitor-induced medium alkalization and PAL gene activation, suggesting a regul

19 ubstrate permeability, measurements of media alkalization, and proton decoupling that AQP9 acts as a

20 Moderate alkalizations ( approximately 0.5 to 1.0 pH unit alkaliz

21 Intracellular acidification inhibited while alkalization augmented the inward current.

22 Alkalization by ammonium pre-pulse to pHi 7.78 +/- 0.02

23 vitro and in vivo studies support intestinal alkalization by sodium bicarbonate supplementation as a

24 slower recovery kinetics from intracellular alkalization, consistent with the Cl(-)/HCO3(-) exchange

25 containing BME were added to chocolates with alkalization degrees (pH 4.5, 6, 7.5) at conching temper

26 liary subunit that helps define the specific alkalization dependence of KSper activation.

27 increase is not attributable to cytoplasmic alkalization; depolarization induces a slight acidificat

28 With physiological ionic gradients, alkalization depolarizes Slo3(-/-) spermatozoa, presumab

29 ow internal pH and low external K, such that alkalization does not reopen the channels unless externa

30 olved in HC feedback but that synaptic cleft alkalization during light-evoked hyperpolarization of HC

31 Alkalization hyperpolarizes sperm and produces Ca(2+) en

32 The resulting endolysosomal alkalization impedes macrophage antigen processing and p

33 extracellular Cl(-) removal led to a larger alkalization in slc4a10-transfected cells than in contro

34 t is critical for fertility, is activated by alkalization in the range of pH 6.4-7.2 at membrane pote

35 ial K(+)/H(+) exchanger, as well as external alkalization inhibited a recovery phase of the reversibl

36 ments in penumbral cortex and post-ischaemic alkalization of brain.

37 atidylcholine-phospholipase C inhibition and alkalization of endolysosomal compartments blocked its a

38 by a mechanism that probably depends on the alkalization of endolysosomes.

39 Alkalization of pH(i) from 7.2 to 7.8 increased gap junc

40 ma membrane Na(+)/Ca(2+) exchanger (NCX) via alkalization of the bath solution, by adding lanthanum,

41 H in yeast, CHX20 elicited acidification and alkalization of the cytosol and vacuole, respectively.

42 The results reveal the alkalization of the electrolyte close to the oxygen redu

43 The ensuing alkalization of the endosomal lumen increased translocat

44 that actin-depolymerizing agents promote the alkalization of the Golgi stack and thetrans-Golgi netwo

45 mentation (5 mg/ml in drinking water) led to alkalization of the intestinal contents.

46 d killing of yeast by primary macrophages by alkalization of the phagosome.

47 sitive dye pyranine resulted in an immediate alkalization of the vesicle lumen by an average pH chang

48 ogether with an NH4Cl-mediated intracellular alkalization (or lesser acidification) of AQP4-expressin

49 Hyperventilation and alkalization remain common therapies.

50 during ischemia with glial acidification or alkalization, respectively.

51 In addition, this alkalization resulted in the reduction of the histopatho

52 controls chemotactic steering; a concomitant alkalization serves as a highly cooperative mechanism th

53 Soil alkalization severely affects crop growth and agricultur

54 Thus, intracellular alkalization stimulates, while intracellular acidificati

55 rents that are less effectively activated by alkalization than are native KSper currents.

56 and pH, and light-induced acidification and alkalization that are asso-ciated with inorganic carbon

57 that high external pH induces intracellular alkalization that directly or indirectly activates plasm

58 ype II approximately 4 Hz) and the degree of alkalization that silenced the cells (type I 7.4-7.6, ty

59 cidification (up to pH 5.0), but also during alkalization (up to pH 8.0), an original and inducible p

60 lizations ( approximately 0.5 to 1.0 pH unit alkalization) were observed during exposure to inhibitor

61 Na/H exchange (NHE2) results in cell alkalization, whereas intracellular [Na] is regulated by

62 ing cascade involving TGFalpha>PI3K>NHE1>pHi alkalization, which leads to a permissible environment f