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1 or output induced by the neuropeptide C-type allatostatin.
2 silenced by application of an insect peptide allatostatin.
3 endocrine cells) expressing the neuropeptide allatostatin A (AstA) inhibits or limits several starvat
6 signed to products of 10 neuropeptide genes (allatostatins A, B, C, SIFamide, allatotropin, FMRFamide
14 ere we show that myoinhibitory peptide (MIP)/allatostatin-B, a pleiotropic neuropeptide widespread am
15 e raised an antibody against Cancer borealis allatostatin-B1 (CbAST-B1; VPNDWAHFRGSWa) and used it to
18 licing variants of three neuropeptide genes (allatostatin CC, CCHamide 1, and short neuropeptide F) a
21 ng with antisera against Diploptera punctata allatostatin (Dip-AST), Manduca sexta allatotropin (Mas-
22 ated transfected neurons with the Drosophila allatostatin G protein-coupled receptor (AlstR)/ligand s
25 e learned in order to understand the role of allatostatins in the modulation of hormone production.
26 afferents of this receptor demonstrate that allatostatin increases the conductance of the neurons, c
27 d H. gammarus, all of the serotonin-like and allatostatin-like immunoreactivity colocalizes in neurop
29 examined the distribution of serotonin-like, allatostatin-like, and FLRF(NH2)-like immunoreactivities
30 neurons of the adult contain serotonin-like, allatostatin-like, and Phe-Leu-Arg-Phe-amide (FLRF(NH2))
31 ; the remaining five families are related to allatostatin, myomodulin, buccalin/drosulfakinin, orcoki
32 us system of various insects and include the allatostatins of cockroaches and crickets, the schistost
34 rent populations of neighboring neurons with allatostatin or HM4D Gi/o-coupled receptors, we analyzed
35 2012) showed that bath application of C-type allatostatin produced either increases or decreases in t
37 ity; Channelrhodopsin-2 for photoactivation; allatostatin receptor for inactivation by ligand applica
38 ociated virus 2, we expressed the Drosophila allatostatin receptor in somatostatin (Sst)-expressing n
39 aterally to express an inhibitory Drosophila allatostatin receptor were silenced by application of an
40 G-protein-coupled receptor AlstR (Drosophila allatostatin receptor) [corrected] after application of
42 rgic C1 neurons, using inhibitory Drosophila allatostatin receptors, for the enhanced expiratory-rela
43 e peptides, tyrosine hydroxylase, proctolin, allatostatin, serotonin, Cancer borealis tachykinin-rela
44 g the viral CREB-expressing neurons with the allatostatin system occludes the spatial memory enhancem
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