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1 or and normal single nucleotide polymorphism allelotypes.
2 ic recipient tissues all exhibited different allelotypes.
3 vents as a mechanism for generation of these allelotypes.
4 fficient and reliable method of high-density allelotyping.
6 utative tumor suppressor gene was defined by allelotyping 65 cases of sporadic ductal carcinoma in si
8 lly homogeneous cell subtype populations for allelotype analysis at chromosomes 1p36, 11q23, 14q32, a
9 onic myelocytic leukemia (CML), we performed allelotype analysis in 30 patients with CML as the disea
11 validated by comparison with microsatellite allelotype analysis of 118 markers in the same tumors.
12 o acute myeloid leukemia (AML), we performed allelotype analysis of 24 individuals using matched MDS
15 owerful combination of genome-wide molecular allelotyping and CGH has identified recurrent clonal DNA
16 pressor gene (TSG) was previously defined by allelotyping and recently refined by overlapping homozyg
17 was analyzed by Southern and microsatellite allelotyping at 53 different loci on 20 different chromo
19 x resistant and two susceptible strains were allelotyped for 10 genes and 49 random DNA markers to id
20 toid and lung cancer lines was amplified and allelotyped for 33 loci predicted by POMPOUS to be varia
23 llite markers was used to initially evaluate allelotypes in TK(+) revertants for patterns associated
25 r heterogeneity of NSCLC into microsatellite allelotyping in a cohort of 48 node-positive stage II pa
27 a new alternative approach to comprehensive allelotyping in which samples are genotyped for nearly 1
30 Additionally, we demonstrate that PCR-based allelotyping is a reliable method for the detection of c
32 d out padlock capture, a high-resolution RNA allelotyping method, to study X chromosome inactivation
33 gosity (LOH), we generated a high-resolution allelotype of 35 ductal carcinoma in situ cases and comp
35 These data represent the first comprehensive allelotype of esophageal adenocarcinoma and show the fea
37 mors, we performed a detailed microsatellite allelotype of lesions thought to arise from the renal co
41 the model tumor suppressor RB1, we performed allelotyping of single-nucleotide polymorphic sites and
42 In this report, we describe the sequence allelotyping of the Ha-ras variable number tandem repeat
43 tional profile for each patient based on the allelotyping of the primary tumor and lymph node deposit
45 Global patterns of LOH can be discerned by allelotyping of tumors with polymorphic genetic markers.
46 al patterns of LOH can be understood through allelotyping of tumors with polymorphic genetic markers.
48 velopment, we performed microsatellite-based allelotypes on primary mammary adenocarcinomas and lung
49 more stringent approach in computing robust allelotypes resulting in 94.4% of the 1095 informative r
54 t reported in Wilms' tumourigenesis and that allelotyping studies may fail to identify regions contai
60 up of 41 subjects whose primary tumours were allelotyped, the fractional allelic loss (FAL) at 39 aut
62 the four subclasses were microdissected and allelotyped using genome-wide single nucleotide polymorp
67 ybridization (CGH) and genome-wide molecular allelotyping were performed with a large group of uremia
70 d with aggressive growth by performing Chr 1 allelotyping with microsatellite markers in microdissect
71 possibility of individual tumor genome-wide allelotyping with potential prognostic and diagnostic ap
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