ライフサイエンスコーパス: allergic contact dermatitis

1 and characterize the role of MCs in chronic allergic contact dermatitis.

2 ty (CHS) of murine skin serves as a model of allergic contact dermatitis.

3 may notably influence the course of chronic allergic contact dermatitis.

4 onfirmed the generation of skin TRM cells in allergic contact dermatitis.

5 ffects the risk of contact sensitization and allergic contact dermatitis.

6 immunological processes like irritative and allergic contact dermatitis.

7 t and cosmetics, and they are known to cause allergic contact dermatitis.

8 e a potential therapeutic avenue in treating allergic contact dermatitis.

9 and in experimental animal studies to evoke allergic contact dermatitis.

10 ated with the development of T-cell-mediated allergic contact dermatitis.

11 lesional skin of patients with psoriasis or allergic contact dermatitis.

12 CD4+ and CD8+ T cells from individuals with allergic contact dermatitis.

13 contact dermatitis and oxazolone, a model of allergic contact dermatitis.

14 f psoriasis, fixed drug eruption, atopic and allergic contact dermatitis.

15 re examined utilizing models of irritant and allergic contact dermatitis.

16 including psoriasis, atopic dermatitis, and allergic contact dermatitis.

17 f occupational skin disease are irritant and allergic contact dermatitis.

18 NAAA inhibitor, ARN077, in a mouse model of allergic contact dermatitis.

19 contact hypersensitivity, an animal model of allergic contact dermatitis, a common pruritic disorder

20 We studied the role of cathelicidin in allergic contact dermatitis, a model requiring dendritic

21 Atopic dermatitis (AD) and allergic contact dermatitis (ACD) have a dynamic relatio

22 ce to haptens is an efficient way to prevent allergic contact dermatitis (ACD) in mice.

23 reviews the recent literature pertaining to allergic contact dermatitis (ACD) in the pediatric popul

24 Allergic contact dermatitis (ACD) is a common T-cell med

25 Allergic contact dermatitis (ACD) is the most common occ

26 Allergic contact dermatitis (ACD) is well recognized as

27 Further, allergic contact dermatitis (ACD) model was used to eval

28 o higher skin permeation was investigated in allergic contact dermatitis (ACD) mouse model.

29 (NKT cells) in cellular infiltrate of human allergic contact dermatitis (ACD) skin challenge sites.

30 xisting irritant contact dermatitis (ICD) or allergic contact dermatitis (ACD) that is sometimes clin

31 We evaluated the effect of OA-NO2 on allergic contact dermatitis (ACD) using an established m

32 y, a crucial role of Th2 responses in nickel allergic contact dermatitis (ACD) was demonstrated.

33 ell as irritant contact dermatitis (ICD) and allergic contact dermatitis (ACD), are common skin disea

34 nd inflammation are commonly associated with allergic contact dermatitis (ACD), it is not known if th

35 levance of SUCNR1/GPR91 expression mediating allergic contact dermatitis (ACD).

36 pothesize that IL-9 also has a role in human allergic contact dermatitis (ACD).

37 cal, and immunologic similarities to chronic allergic contact dermatitis (ACD).

38 le Kv1.3 blocker (EC50=2 nM), could suppress allergic contact dermatitis (ACD).

39 Allergic contact dermatitis affects 20% of children at s

40 Allergic contact dermatitis and ICD were characterized b

41 A close association was also found with allergic contact dermatitis and increased specific IgE t

42 Allergic contact dermatitis and its animal model, contac

43 ases in which NKT cells play a role, such as allergic contact dermatitis and psoriasis.

44 patients (n = 703) presenting with possible allergic contact dermatitis and subsequently undergoing

45 transiently compromised, such as psoriasis, allergic contact dermatitis, and blistering disorders.

46 in diseases, including psoriasis, atopic and allergic contact dermatitis, and cutaneous T-cell lympho

47 sensitizing chemicals in the development of allergic contact dermatitis, and suggest that the irrita

48 lergic background such as atopic dermatitis, allergic contact dermatitis, and urticaria are very comm

49 i-inflammatory activity in both irritant and allergic contact dermatitis animal models.

50 Additional cases of allergic contact dermatitis are being reported with temp

51 Atopic dermatitis and allergic contact dermatitis are both common skin disease

52 ich could be applicable for the treatment of allergic contact dermatitis, are still largely unknown.

53 s diagnosed with either atopic dermatitis or allergic contact dermatitis as well as in an inducible m

54 by prominent barrier abnormalities (subacute allergic contact dermatitis, atopic dermatitis), topical

55 anaphylatoxin C5a is a critical mediator of allergic contact dermatitis, bridging essential aspects

56 to Ni(++) is one of the most common forms of allergic contact dermatitis, but how the T-cell receptor

57 rin barrier defects might also predispose to allergic contact dermatitis by allowing greater penetrat

58 patients suffering from psoriasis, atopic or allergic-contact dermatitis express CCR10.

59 The role of MCs in chronic allergic contact dermatitis has not been investigated, i

60 In the past decade, mechanisms underlying allergic contact dermatitis have been intensively invest

61 In addition, AFC inhibits in vivo allergic contact dermatitis in a mouse model utilizing s

62 The documented rates of allergic contact dermatitis in children are on the rise.

63 epidemiology and clinical manifestations of allergic contact dermatitis in children.

64 reviews the recent literature pertaining to allergic contact dermatitis in the pediatric population.

65 ct of topically applied THC on DNFB-mediated allergic contact dermatitis in wild-type and CB1/2 recep

66 s included dyskeratosis follicularis Darier, allergic contact dermatitis, infectious folliculitis, va

67 Allergic contact dermatitis is a chronic T cell-driven i

68 Allergic contact dermatitis is a common disorder that ha

69 Allergic contact dermatitis is a common skin disease ass

70 Allergic contact dermatitis is commonly associated with

71 ults provide support for the hypothesis that allergic contact dermatitis is not a classic delayed typ

72 Allergic contact dermatitis is one of the most common oc

73 Allergic contact dermatitis is the most frequent occupat

74 ective mechanism in those who do not develop allergic contact dermatitis is tolerance induction by re

75 Allergic contact dermatitis may affect as many as 20% of

76 reports have suggested that chemical-induced allergic contact dermatitis may not be a traditional typ

77 Allergic contact dermatitis occurs less frequently in th

78 , females have significantly higher rates of allergic contact dermatitis on the face.

79 ne (acute irritant contact dermatitis, acute allergic contact dermatitis) or by prominent barrier abn

80 ere observed in sensory neurons of mice with allergic contact dermatitis- or dry skin-elicited itch;

81 tor-alpha agonists in models of irritant and allergic contact dermatitis produced in mouse ears by to

82 h AD, despite a trend toward weaker clinical allergic contact dermatitis reactions.

83 tcomes of CD39 deficiency in irritant versus allergic contact dermatitis, reflecting its diverse role

84 Allergic contact dermatitis, such as in response to pois

85 In a well-established model of allergic contact dermatitis, the absence of Mincle leads

86 inflammatory responses in a mouse model for allergic contact dermatitis, the contact hypersensitivit

87 The prevalence of allergic contact dermatitis to allergens (eg, fragrance)

88 this study we use an in vivo mouse model of allergic contact dermatitis to investigate how nanoparti

89 ver, the frequency of DNCB sensitization and allergic contact dermatitis to topically applied mechlor

90 Irritant or allergic contact dermatitis usually presents as an eczem

91 n the group using white petrolatum developed allergic contact dermatitis vs 4 patients (0.9%) in the

92 molecular and cellular mechanism underlying allergic contact dermatitis, we evaluated oxazolone-indu

93 IV hypersensitivity reactions, resulting in allergic contact dermatitis, which clinically resembles