ライフサイエンスコーパス: allergic disorder

1 20% of the global population suffers from an allergic disorder.

2 PIES) is a non-IgE-mediated gastrointestinal allergic disorder.

3 inking their possible roles with exacerbated allergic disorders.

4 ternal-fetal interactions may occur in other allergic disorders.

5 osed as one mechanism to explain the rise in allergic disorders.

6 ical target for the regulation of autoimmune/allergic disorders.

7 of cytokines and chemokines associated with allergic disorders.

8 fundamentally new concept for the control of allergic disorders.

9 cologically targeting arginine metabolism in allergic disorders.

10 eosinophils in a variety of inflammatory or allergic disorders.

11 develop safe immunotherapeutic strategy for allergic disorders.

12 to the eosinophilia seen in asthma and other allergic disorders.

13 d implicated as a genetic predisposition for allergic disorders.

14 also be elevated in individuals with severe allergic disorders.

15 hn's disease and rheumatoid arthritis and in allergic disorders.

16 rovide a novel strategy for the treatment of allergic disorders.

17 tality associated with anaphylaxis and other allergic disorders.

18 extracellular mediators of inflammation and allergic disorders.

19 rease the susceptibility to inflammatory and allergic disorders.

20 of the IL-21/IL-21R system in the context of allergic disorders.

21 classes of fatigue, sleep disturbances, and allergic disorders.

22 al integrity, such as acute inflammatory and allergic disorders.

23 iated with risk of childhood asthma or other allergic disorders.

24 ls are frequently increased in patients with allergic disorders.

25 tecting pollen allergens as measures against allergic disorders.

26 as well as in autoimmune, inflammatory, and allergic disorders.

27 ential adjuvants for FcepsilonRI-MC-mediated allergic disorders.

28 Biologicals have also been tested in allergic disorders.

29 ould be exploited for the treatment of human allergic disorders.

30 ir detrimental contributions to IgE-mediated allergic disorders.

31 to an even broader list of hypersensitivity/allergic disorders.

32 may affect the initiation and maintenance of allergic disorders.

33 e associated with having single and multiple allergic disorders.

34 ts on the development of single and multiple allergic disorders.

35 lturally applicable classification system of allergic disorders.

36 heir joint importance in the pathogenesis of allergic disorders.

37 for the design of novel immunotherapies for allergic disorders.

38 allergic asthma and other mast cell-mediated allergic disorders.

39 ratory disorders, such as asthma and related allergic disorders.

40 the only causative treatment of IgE-mediated allergic disorders.

41 preventative and therapeutic strategies for allergic disorders.

42 that manifest clinically as asthma and other allergic disorders.

43 n be used to analyze complex respiratory and allergic disorders.

44 nd their association with the development of allergic disorders.

45 harbors helminth infections or suffers from allergic disorders.

46 ent of novel approaches for the treatment of allergic disorders.

47 pathogenesis of infectious, autoimmune, and allergic disorders.

48 rtant in the pathogenesis of anaphylaxis and allergic disorders.

49 ssue resident cells with a principal role in allergic disorders.

50 e adaptive immune response that causes these allergic disorders.

51 particles (DEP), promotes the development of allergic disorders.

52 onic inflammation associated with asthma and allergic disorders.

53 y responses associated with asthma and other allergic disorders.

54 innate immunity and regulation of autoimmune/allergic disorders.

55 significantly higher odds of developing any allergic disorders (adjusted OR, 3.04; 95% CI, 1.08-8.60

56 apacity of pDCs in patients with one type of allergic disorder, allergic asthma, and controls; 2) to

57 IgE) has a major role in the pathogenesis of allergic disorders and asthma.

58 ciated with pathologic Th2 responses such as allergic disorders and asthma.

59 Mast cells play critical roles in allergic disorders and asthma.

60 olvement of cytokines and other mediators in allergic disorders and described novel approaches for un

61 ely recognized as critical effector cells in allergic disorders and other immunoglobulin E-associated

62 lated goals of preventing the development of allergic disorders and providing the most effective diag

63 elopmental contribution to the risk of later allergic disorders and suggest that involvement of epige

64 uction and treatment of autoimmune diseases, allergic disorders, and graft rejection by depleting und

65 The manifestations of allergic disorders are closely tied to the biologic effe

66 nical trials of anti-IgE in the treatment of allergic disorders are discussed.

67 Because many chronic inflammatory and allergic disorders are intimately linked to excessive ma

68 ulatory cells in immunoglobulin E-associated allergic disorders, as well as in certain innate and ada

69 Important allergic disorder associations will be missed without su

70 min D may be involved in the pathogenesis of allergic disorders, asthma and decreased lung function.

71 on in early childhood and atopy and reported allergic disorders at the age of 6.5 years in an Ethiopi

72 j 2, leading to preventive measures against allergic disorders caused by Japanese cedar pollen.

73 Eosinophilic esophagitis (EoE) is an allergic disorder characterized by infiltration of the o

74 Eosinophilic esophagitis (EoE) is an allergic disorder characterized by the accumulation of e

75 ed of health professionals' attitudes toward allergic disorders classification supports the need to u

76 mplification process of the hypersensitivity/allergic disorders classification was carried out to bet

77 f healthcare professionals' attitudes toward allergic disorders classification was proposed to the me

78 These pathological features of allergic disorder, common in developed countries, appear

79 Allergic disorders comprise a major component of pediatr

80 Many patients with allergic disorders continue to have uncontrolled symptom

81 Atopic (allergic) disorders develop out of a close interaction b

82 and possibly other Ab-mediated autoimmune or allergic disorders, especially when given in short cours

83 esentatives to update the classifications of allergic disorders for the ICD-11 revision.

84 urticaria, venom allergy and other probable allergic disorders) from the Scottish Primary Care Clini

85 iologic mechanisms underlying IgE-associated allergic disorders has led to the identification of nove

86 pothesis that IgE, which also contributes to allergic disorders, has an important function in protect

87 Allergic disorders have increased substantially in recen

88 Allergic disorders have now become a major worldwide pub

89 FLG) variants are important risk factors for allergic disorders; however, knowledge on their individu

90 correlates with a reduced risk of atopy and allergic disorders; however, limited data are available

91 the pathophysiology of bronchial asthma and allergic disorders; however, this concept was challenged

92 roteins in IgE-based serologic assessment of allergic disorders in a helminth-infected population, we

93 sk factors and may be responsible for severe allergic disorders in atopic individuals.

94 associated with the development of multiple allergic disorders in humans, indicating that TSLP is a

95 se dust mite-derived proteases contribute to allergic disorders in part by disrupting epithelial barr

96 or autoimmune, infectious, inflammatory, and allergic disorders in the etiology of multiple myeloma (

97 ve useful in the treatment of autoimmune and allergic disorders in which iNKT cell activation is dest

98 genes that contribute to the development of allergic disorders include leukocyte histocompatibility

99 mplicated in the pathogenesis of a number of allergic disorders including asthma.

100 ells contribute to immunoglobulin-E-mediated allergic disorders including atopic dermatitis.

101 been implicated by their presence in diverse allergic disorders including bronchial asthma, allergic

102 Allergic disorders, including asthma, have increased dra

103 aureus, a major human pathogen, exacerbates allergic disorders, including atopic dermatitis, nasal p

104 e so inextricably linked to the pathology of allergic disorders, including fatal anaphylaxis, that it

105 The coexistence of allergic disorders is common, with approximately 2% of t

106 The coexistence of allergic disorders is frequent, and allergic sensitizati

107 presented whereby the evolution of specific allergic disorders is predicated on the confluence of pr

108 The role of T(H) in autoimmune-disorders and allergic-disorders is now re-evaluated, with current dat

109 ly in the context of lung- and skin-specific allergic disorders, it is becoming increasingly clear th

110 le of mast cells, critical effector cells in allergic disorders, known to interact with ILC2s in vivo

111 e of the IgE response in the pathogenesis of allergic disorders, little is known about the role of fo

112 the presence of degranulated MC in various (allergic) disorders, MC-derived EV should be considered

113 evalence and incidence of atopy and reported allergic disorders (measured at age of 6.5 years) were d

114 Information on respiratory and allergic disorders, medical visits, and medications was

115 C) are well known for their effector role in allergic disorders; moreover, they are associated with d

116 n-IgE-mediated gastrointestinal food-induced allergic disorders (non-IgE-GI-FAs) account for an unkno

117 n the brain, heart, synovium and intestines, allergic disorders of the lung and skin, and microbial i

118 Indeed, allergic disorders often have their roots in early child

119 oduction leads to epidermal and consequently allergic disorders, our findings emphasize the need for

120 of adult-onset respiratory and dermatologic allergic disorders remains unclear.

121 Gastrointestinal allergic disorders represent a diverse spectrum of infla

122 ole in type I hypersensitivity reactions and allergic disorders such as anaphylaxis and asthma.

123 We did not find associations with allergic disorders such as asthma or with blood pressure

124 new therapeutic target for the treatment of allergic disorders such as atopic dermatitis (AD).

125 However, patients with allergic disorders such as atopic dermatitis show a pauc

126 sing pollen allergens is required to prevent allergic disorders such as pollinosis.

127 nes, which promote distinctive aspects of an allergic disorder, such as tissue localization.

128 hought to be critical to the pathogenesis of allergic disorders, such as anaphylaxis and asthma, and

129 tical role in the pathology of IgE-dependent allergic disorders, such as anaphylaxis and asthma.

130 Allergic disorders, such as anaphylaxis, hay fever, ecze

131 proinflammatory mediators that contribute to allergic disorders, such as asthma and anaphylaxis.

132 to the proinflammatory role of mast cells in allergic disorders, the results obtained in this study e

133 of the GSTP1 gene may influence the risk for allergic disorders through an impaired defence against o

134 athway has been previously linked to various allergic disorders, we provide unexpected evidence for i

135 on and FLG variants with single and multiple allergic disorders were tested in log-binomial regressio

136 worm burden tended to have increased risk of allergic disorder, whereas low IgE/high worm burden tend

137 pted to be an antigen-driven, TH2-associated allergic disorder, whereas the cause of PPI-REE remains

138 lic esophagitis (EoE), a recently recognized allergic disorder with poorly understood pathogenesis.