ライフサイエンスコーパス: allergic reaction

1 influenza vaccination without an allergic reaction.

2 ases referred with a suspected perioperative allergic reaction.

3 ssess the contribution of pollensomes to the allergic reaction.

4 llergens, with potential implications in the allergic reaction.

5 s, which might have been due an IgE-mediated allergic reaction.

6 phils and CD4(+) T cells, all features of an allergic reaction.

7 they displayed decreased chemically induced allergic reaction.

8 ex vivo for the appearance of markers of an allergic reaction.

9 -4 is present and possibly contribute to the allergic reaction.

10 nd worse outcomes in the setting of an acute allergic reaction.

11 of the jelly product at home caused another allergic reaction.

12 could lead to absorption and consequently an allergic reaction.

13 No children developed allergic reaction.

14 s is a potentially life-threatening systemic allergic reaction.

15 olymerization of protein, which may alter an allergic reaction.

16 naphylaxis is a potentially life-threatening allergic reaction.

17 ssarily experience severe symptoms during an allergic reaction.

18 ly given during the provocation phase of the allergic reaction.

19 sensitization and elicitation phases of the allergic reaction.

20 ts received influenza vaccination without an allergic reaction.

21 -food allergies often associated with severe allergic reactions.

22 wan, and 60% of the exposed subjects develop allergic reactions.

23 weight compounds that modulate and stimulate allergic reactions.

24 There were no thrombotic events or severe allergic reactions.

25 s a frequent elicitor of severe IgE-mediated allergic reactions.

26 bsolute requirement for IgE glycosylation in allergic reactions.

27 vated in proportion to the severity of acute allergic reactions.

28 ease of mediators involved in immediate-type allergic reactions.

29 interest due to their known ability to cause allergic reactions.

30 capacity to induce house dust mite-mediated allergic reactions.

31 volunteers and from 41 patients during acute allergic reactions.

32 sk for developing significant peanut-induced allergic reactions.

33 nished density of faecal LAB associated with allergic reactions.

34 omponents potentially responsible for severe allergic reactions.

35 or tree nuts, which carries a risk of severe allergic reactions.

36 of inflammatory mediators that contribute to allergic reactions.

37 peanut, but not placebo, during acute peanut allergic reactions.

38 licy was associated with complete absence of allergic reactions.

39 Mast cells are key effector cells in allergic reactions.

40 RII mediates cell type-dependent function in allergic reactions.

41 There were no reports of severe systemic allergic reactions.

42 s histamine and leukotrienes, which initiate allergic reactions.

43 fy those most at risk of severe food-induced allergic reactions.

44 irculating factor responsible for triggering allergic reactions.

45 oglobulin G (IgG) plays a regulatory role in allergic reactions.

46 FcepsilonRI plays an important role in acute allergic reactions.

47 ter enrolment of 3232 patients due to severe allergic reactions.

48 se excipient of Inavir could cause immediate allergic reactions.

49 ergen in Mediterranean areas causing diverse allergic reactions.

50 rently formulated, is associated with severe allergic reactions.

51 asparaginase group) and asparaginase-related allergic reactions (14 [6%] vs 6 [3%]).

52 king the study powder because of a confirmed allergic reaction (25 of 407 [6.1%] compared with 6 of 4

53 -Cefaz) as well as the greatest frequency of allergic reactions (3.0% vs 2.4% for Hx-Cefaz and 1.7% f

54 6 of the 13 subjects experienced mild or no allergic reactions, 5 subjects had grade 2 reactions, an

55 nts investigated for suspected perioperative allergic reactions, 9.6% were diagnosed with allergy to

56 Allergic reactions affect millions of people worldwide.

57 led food challenge to cause life-threatening allergic reactions affects its clinical application.

58 Twenty-three patients with allergic reaction after Hymenoptera sting (11 wasp and 1

59 eters to visualize and quantify the regional allergic reaction after segmental endobronchial allergen

60 She also developed a systemic allergic reaction after sting from an unknown creature w

61 3-galactose (alpha-Gal) have reported severe allergic reactions after consumption of red meat.

62 desensitization transiently prevents severe allergic reactions, allowing administration of life-savi

63 Allergic inflammation and severe allergic reactions (anaphylaxis) are important in allerg

64 cephalitis/myelitis, nonanaphylactic serious allergic reaction, anaphylaxis, Guillain-Barre syndrome,

65 ents' age, ethnicity, nature of the putative allergic reaction and coexisting clinical diseases such

66 The severity of the allergic reaction and the threshold dose during OFCs to

67 ht on clinical and mechanistic links between allergic reactions and abnormal desquamation, substantia

68 nisms by studying mast cells, key players in allergic reactions and anaphylaxis.

69 Mast cells (MCs) are well-known effectors of allergic reactions and are considered sentinels in the s

70 ion may be a potential modality for treating allergic reactions and bronchial asthma.

71 Reported allergic reactions and consumption of tree nuts and sesa

72 se and the cause of severe, life-threatening allergic reactions and death.

73 ute amount of contamination may cause strong allergic reactions and even death.

74 aucity of new therapeutic targets to control allergic reactions and forestall the rising trend of all

75 Ig isotype, yet it plays a critical role in allergic reactions and host protection from helminth inf

76 ts are available, but these carry a risk for allergic reactions and infection with blood-borne pathog

77 is an important immunomodulator involved in allergic reactions and inflammatory responses.

78 There were no infusional or allergic reactions and no difference in treatment-emerge

79 Allergic reactions and other skin disorders occurred sig

80 the commonest cause of life-threatening food-allergic reactions and significantly affect quality of l

81 ssues provides a novel strategy to alleviate allergic reactions and surpassed attenuation of tissue m

82 to be used to provide information regarding allergic reactions and therefore potentially aid in more

83 s, shrimps are the most predominant cause of allergic reactions and thus more extensively studied.

84 llergic swelling (mouth or throat), systemic allergic reactions, and asthma-related treatment-related

85 Mast cells are key effectors in allergic reactions, and decades of research have suggest

86 ch were grade 3 or 4 (five infections, three allergic reactions, and four cases of neutropenia), all

87 Mast cells are primary effectors in allergic reactions, and may have important roles in dise

88 e-resident immune cells that are involved in allergic reactions, and their numbers are increased in t

89 orrhage (anticoagulants), moderate to severe allergic reactions (antibiotics), and hypoglycemia with

90 oped in 4 patients, none of whom experienced allergic reactions, any bleeds requiring treatment, or c

91 We argue that appropriately targeted allergic reactions are beneficial, although they can bec

92 Late allergic reactions are common in the course of allergen-

93 is consistent with the observation that most allergic reactions are in connection with roasted peanut

94 Sensitization to food allergens and food allergic reactions are mostly caused by ingesting the al

95 nce and prompt recognition and management of allergic reactions are of the utmost importance.

96 peanuts are frequently consumed, but severe allergic reactions are rare.

97 ccurrence, elicitors and treatment of severe allergic reactions are recognized and reported different

98 Since allergic reactions are triggered by mast cell-mediated h

99 yet almost none will experience a fatal food-allergic reaction; asthma is not, in itself, a strong pr

100 tionnaire was used to collect data on severe allergic reactions based on the medical history and diag

101 stressful for the patient and avoids severe allergic reactions, BAT ought to precede challenge testi

102 chanistic studies reveal that the ability of allergic reaction blockade by the low-affinity anti-IgE

103 e frequently mentioned as amplifiers of food allergic reactions but only individual cases or small se

104 t mediator of protective immunity as well as allergic reaction, but how high affinity IgE antibodies

105 s occur because mediators released during an allergic reaction can interact with sensory nerves, chan

106 An acute MC-dependent allergic reaction can lead to systemic shock, but the ea

107 ed as causing allergies, but the majority of allergic reactions can be ascribed to a limited number o

108 Allergic reactions can be considered as maladaptive IgE

109 Allergic reactions can range from mild local symptoms to

110 cute, potentially fatal, multi-organ system, allergic reaction caused by the release of chemical medi

111 scribed the frequencies and risk factors for allergic reactions caused by accidental allergen ingesti

112 Immunoglobulin (Ig)E antibodies mediate the allergic reaction characterized by immediate hypersensit

113 ria and Switzerland who experienced a severe allergic reaction defined by the onset of severe pulmona

114 25% of the children experienced an allergic reaction due to a non-pre-packed food from bake

115 Only three subjects (0.6%) had a presumed allergic reaction during the test; another three reactio

116 Patients with allergic reactions during anesthesia were prospectively

117 ed to estimate the severity and threshold of allergic reactions during OFCs.

118 However, not all allergic reactions end up in the most severe form of ana

119 environmental chamber to stimulate an ocular allergic reaction followed by application of artificial

120 om allergy is a potentially life-threatening allergic reaction following a honeybee, vespid, or ant s

121 ent Reporting System has received reports of allergic reactions following immunization with egg-free

122 These results suggest that allergic reactions following influenza vaccination are n

123 lactose-containing dry powder inhalers cause allergic reactions for patients with cow's milk allergy

124 ll differences in sensitization and reported allergic reactions for select tree nuts, with levels bei

125 ied to automate the process of measuring the allergic reactions found by developing systems and algor

126 n of activating receptors might help prevent allergic reactions from developing, although most of the

127 number of physiological processes including allergic reactions, gastric acid secretion, neurotransmi

128 diator involved in the hypersensitivity-type allergic reaction has gained greater recognition.

129 Food-mediated allergic reactions have emerged as a major health proble

130 Mechanisms driving acute food allergic reactions have not been fully characterized.

131 nctional role as well as the implications on allergic reactions, however, are not clear yet.

132 s are significantly involved in IgE-mediated allergic reactions; however, their roles in health and d

133 from all studies, the eliciting dose for an allergic reaction in 1% of the population estimated for

134 for exposure to an allergen to result in an allergic reaction in specific IgE-positive patients.

135 d in milk are IgE reactive and can induce an allergic reaction in vitro.

136 frequent adverse reactions, including severe allergic reactions in 20%.

137 ypoallergenic Cyp c 1 mutant protect against allergic reactions in a murine model of fish allergy.

138 suitable for studying mechanisms involved in allergic reactions in a novel manner, with activity addr

139 sponses in lungs and to exacerbate pulmonary allergic reactions in animal models.

140 (CM) formulas are available for avoidance of allergic reactions in CM-allergic children and for preve

141 Because BSA can cause allergic reactions in humans the World Health Organizati

142 induction of defense responses in plants and allergic reactions in humans.

143 s on rates of epinephrine administration for allergic reactions in Massachusetts public schools.

144 al irritants but that also trigger exuberant allergic reactions in patients with allergies.

145 to the difference in the frequency of severe allergic reactions in patients with autosomal dominant h

146 ped us to estimate the severity and types of allergic reactions in patients.

147 ion of time in school but characteristics of allergic reactions in schools are not well studied.

148 can represent a potential risk for eliciting allergic reactions in sensitised individuals.

149 We sought to characterize severe allergic reactions in terms of elicitors, symptoms, emer

150 In contrast, it might result in frequent allergic reactions in the community considering that man

151 nts investigated for suspected perioperative allergic reactions in the Danish Anaesthesia Allergy Cen

152 od products are frequent elicitors of severe allergic reactions in the general population including c

153 Ps) represent a major cause of systemic food allergic reactions in the Mediterranean area.

154 mographic data, atopic status and details of allergic reactions in the previous year and reasons for

155 ability to severe outcomes from food-induced allergic reactions in the second and third decades.

156 It has been associated with allergic reactions in the skin and lungs but also homeos

157 ing transplantation may help prevent serious allergic reactions in transplant recipients.

158 AHG2 inhibited peanut-specific IgE-mediated allergic reactions in vitro and in vivo.

159 , which translates into a reduction of acute allergic reactions in vivo, thus identifying CD63 as an

160 ystemic anaphylaxis, the most severe form of allergic reaction, in which an acute vascular response p

161 ansgenic mice and models of inflammatory and allergic reactions, in particular those of human activat

162 with strong desaturation after an immediate allergic reaction including erythema, abdominal pain, vo

163 ed 12-week MSSA cure, recurrence, and death; allergic reactions including major, minor, and potential

164 hrombotic or thromboembolic events, systemic allergic reactions (including anaphylaxis), or neutralis

165 e the secular trends of hospitalizations for allergic reactions, including anaphylaxis, among childre

166 lar trend of hospitalizations for paediatric allergic reactions, including anaphylaxis, are scarce.

167 individuals to develop severe IgE-associated allergic reactions, including fatal anaphylaxis, on subs

168 individuals to develop severe IgE-associated allergic reactions, including fatal anaphylaxis, upon su

169 -transgenic mice, in which various models of allergic reactions induced by IgG could be studied.

170 ronic inflammatory skin disease triggered by allergic reactions involving IgE antibodies directed tow

171 pe interact with allergen at the onset of an allergic reaction is of great importance for deciphering

172 gnosis to prevent potential life-threatening allergic reactions is crucial.

173 the time course of the early (EAR) and late allergic reaction (LAR).

174 The incidence of allergic reactions leading to hospitalizations between 1

175 vities to avoid the annoyingly intense itchy allergic reactions, leading to significant worsening of

176 Blue dye has, however, been associated with allergic reactions, long-term staining of skin, and incr

177 fortilin) has been implicated in late-phase allergic reactions (LPRs) and chronic allergic inflammat

178 in anaphylaxis and other acute IgE-mediated allergic reactions, mast cells (MCs) have been implicate

179 sis, or death, and few cases of grade 2 to 3 allergic reaction (n = 6), grade 1 to 3 hyperglycemia (n

180 ted; 1 nonserious treatment-related systemic allergic reaction occurred (assessed as moderate intensi

181 local allergic swellings or serious systemic allergic reactions occurred in subjects with asthma trea

182 to 4500 mg of peanut protein until objective allergic reactions occurred.

183 Treatment was well tolerated; no systemic allergic reactions occurred.

184 ed symptoms depend on the organ in which the allergic reaction occurs but can include red itchy eyes,

185 s associated with 47% reduced odds of peanut allergic reaction (odds ratio [OR], 0.53; 95% CI, 0.30-0

186 Patients given a diagnosis of an allergic reaction or anaphylaxis and a subset of patient

187 No allergic reactions or inhibitors were observed.

188 There were no acute allergic reactions or nonsignificant excesses of 6-month

189 No investigator-assessed anaphylactic allergic reactions or reactions requiring epinephrine we

190 s because of neuropathy, drug-induced lupus, allergic reaction, or fungal infection.

191 was no evidence of increased TEAEs, systemic allergic reactions, or severe local allergic swellings i

192 1999 and 2011 with the primary diagnosis of allergic reaction per International Classification of Di

193 is is the most severe and frightening of the allergic reactions, placing patients at high risk and de

194 Anaphylaxis is a systemic allergic reaction, potentially life-threatening that can

195 phase of a pan-European registry for severe allergic reactions provides for the first time data on a

196 such as confirming the etiology of systemic allergic reactions, providing written instructions for a

197 re were no serious adverse events, including allergic reaction, reported in either the antibiotic gro

198 press the recruitment of immune cells during allergic reactions, representing potential new drugs for

199 100% of the patients with grades 1, 2, and 3 allergic reactions, respectively.

200 ironmental nickel exposure is known to cause allergic reactions, respiratory illness, and may be resp

201 Peanut-induced allergic reactions resulted in a significant decrease in

202 acute, severe, life-threatening multisystem allergic reaction resulting from the sudden systemic rel

203 nt of cashew nut allergen may cause a severe allergic reaction, suggesting high potency comparable wi

204 dhood immunity culminating in sensitivity to allergic reactions, susceptibility to microbial infectio

205 Pepsinized cashew proteins elicited weaker allergic reactions than native cashew proteins but impor

206 emia, pancreatitis, pancreatic neoplasms, or allergic reactions than was placebo.

207 Anaphylaxis is a severe allergic reaction that can be rapidly progressing and fa

208 Anaphylaxis is a serious allergic reaction that can cause death; however, the act

209 g, life-threatening, generalized or systemic allergic reaction that is classically elicited by antige

210 Anaphylaxis is a severe allergic reaction that is rapid in onset and might cause

211 thophysiology of anaphylaxis and other acute allergic reactions that it can be difficult to think of

212 Several authors have reported on allergic reactions that resulted in presentation to the

213 s can contribute to the immunologic basis of allergic reactions, the molecular underpinnings associat

214 1R and H2R blockers in the therapy for acute allergic reactions, there is little mechanistic evidence

215 l injections received PI prophylaxis without allergic reactions, thus demonstrating a lack of true PI

216 develops as the result of a non-IgE-mediated allergic reaction to a variety of organic materials or l

217 nine of 27 patients, and one patient had an allergic reaction to carboplatin.

218 acute infusional toxicity (attributed to an allergic reaction to dimethyl sulfoxide) in the 29 patie

219 her (1) a convincing history of an immediate allergic reaction to egg, milk, or both with a positive

220 nimising exposure and reducing incidences of allergic reaction to fish in contaminated products.

221 Many patients developing an allergic reaction to fruit are diagnosed as having oral

222 istory of accidental ingestion leading to an allergic reaction to hazelnut had a predictive value of

223 with a self-reported egg allergy or previous allergic reaction to inactivated influenza vaccine.

224 zation to airborne allergens causes a Type 2 allergic reaction to ingested foods.

225 of infants randomized to receive egg had an allergic reaction to the egg powder and did not continue

226 if a particular patient has had an apparent allergic reaction to the medication, potential allergy t

227 mune tolerant (IT) if they had no detectable allergic reactions to a peanut oral food challenge after

228 nificantly between the two groups, but fewer allergic reactions to asparaginase were observed in the

229 h penicillin in the diagnostic evaluation of allergic reactions to beta-lactam antibiotics, mimicking

230 We studied 387 patients with immediate allergic reactions to beta-lactams and 1124 paired contr

231 Allergic reactions to beta-lactams are among the most fr

232 ncluded in the guideline are epidemiology of allergic reactions to beta-lactams, molecular structure,

233 We conducted a systematic review of (1) allergic reactions to botulinum antitoxin and (2) the pr

234 osures causing food allergy, such as delayed allergic reactions to carbohydrate moieties in mammalian

235 IgE-mediated allergic reactions to cashews and other nuts can trigger

236 However, the nature of allergic reactions to chickpea has not been characterize

237 Perioperative allergic reactions to chlorhexidine are often severe and

238 Allergic reactions to drugs are a serious public health

239 Eighteen patients (28%) had a history of allergic reactions to egg, legumes, and nuts and strictl

240 the body, are the same as the mechanisms of allergic reactions to external allergens.

241 ding perceptions of severe, life-threatening allergic reactions to food by different stakeholders, wi

242 Allergic reactions to food can have serious consequences

243 venom-related anaphylaxis also occur during allergic reactions to food in 22 patients with peanut al

244 sed to identify those at most risk of severe allergic reactions to food, and the consequences of misi

245 l immune components of the effector phase of allergic reactions to food.

246 de guidance to reduce the risk of accidental allergic reactions to foods in the community.

247 Allergic reactions to gelatin are comparatively rare, bu

248 lp to identify patients at risk of objective allergic reactions to hazelnut.

249 The inflammatory mechanisms of allergic reactions to hormone allergens, which are intri

250 Included were 121 patients with systemic allergic reactions to Hymenoptera stings, 76 with double

251 Allergic reactions to mosquito bites are an increasing c

252 The aim of this study was to characterize allergic reactions to non-pre-packed foods and to invest

253 rved in school or brought from home reported allergic reactions to nuts.

254 Biomarkers of severity and threshold of allergic reactions to peanut could significantly improve

255 associated with the threshold (P = .020) of allergic reactions to peanut during OFCs.

256 Human allergic reactions to peanut proteins and the associated

257 sitivity is associated with the threshold of allergic reactions to peanut.

258 he management of immediate and non-immediate allergic reactions to penicillins and other beta-lactams

259 Central European patients displaying strong allergic reactions to plant-derived food.

260 d an examination of the relationship between allergic reactions to red meat and sensitization to gela

261 Allergic reactions to stinging insects may be unexpected

262 The cause might be allergic reactions to the gram-positive pathogen Staphyl

263 ood allergens and aeroallergens, or reported allergic reactions to tree nuts and sesame.

264 t allergen can result in diverse patterns of allergic reactions to various foods.

265 Fifty-nine individuals with allergic reactions to vespid stings and positive ImmunoC

266 Allergic reactions to walnut can be life-threatening.

267 Hospitalizations for allergic reactions totalled 1987 in Finland and 5433 in

268 this mechanism opens new avenues to inhibit allergic reactions triggered by IgE.

269 lergens that has been identified to cause an allergic reaction, typically the symptoms are localized

270 sensitization is not a definite indicator of allergic reaction upon ingestion.

271 the basis of absence or presence of systemic allergic reactions upon sting challenge.

272 There were no systemic allergic reactions (upper 95% CI for population, 1.3%).

273 le the dynamic transcriptome of acute peanut allergic reactions using serial peripheral blood samples

274 lostridium difficile infection and potential allergic reactions (using epinephrine administration as

275 severe local allergic swelling, or systemic allergic reactions versus subjects without asthma.

276 t FcepsilonRII may play a role in regulating allergic reactions via modulating IFN-gamma production i

277 covery ended the belief that an anaphylactic/allergic reaction was caused by poisons, but to the cont

278 lerated, and only 1 patient with a suspected allergic reaction was noted.

279 Allergic reaction was triggered by a single intraperiton

280 Surgeons' concern for allergic reactions was associated with decreased rates o

281 As mast cell mediators trigger allergic reactions, we here determine the function of TR

282 drawal (IT, n = 7), whereas those who had an allergic reaction were categorized as nontolerant (NT; n

283 ugh to August 2013, 1334 (0.51%) episodes of allergic reactions were detected, and 532 (0.20%) episod

284 Allergic reactions were induced in mice nasally pretreat

285 Allergic reactions were mainly caused by food (children

286 Severe allergic reactions were reported in ten (1%) of 1605 pat

287 lenges (DBPCFCs) with a positive outcome for allergic reactions were selected from the clinical datab

288 Hormone allergy is an allergic reaction where the offending allergens are one'

289 vel form of food allergy with severe delayed allergic reactions where IgE antibodies are directed aga

290 lls has been well documented in IgE-mediated allergic reactions, whereas other mast cell functions ar

291 food allergic people, the incidence of food allergic reactions which are coded as anaphylaxis by hea

292 This is particularly evident in allergic reactions, which represent ideal systems to stu

293 gainst food proteins and a history of severe allergic reactions) who underwent specific food oral imm

294 Anaphylaxis is a serious systemic allergic reaction with rapid onset and potentially life-

295 consciousness are known to occur with severe allergic reactions with IgE-mediated food allergy, repor

296 This study investigates the risk of an allergic reaction within the milk-, wheat-, hazelnut- an

297 FA diagnosis was based on recognized allergic reactions within 4 hours after exposure to know

298 atory state, and is associated with dampened allergic reactions within the host.

299 follow-up in consideration of development of allergic reactions without exercise.

300 Shrimp is a frequent cause of severe allergic reactions world-wide.