ライフサイエンスコーパス: allergy

1 ed therapy due to severity of their reported allergy.

2 s of age prevents hen's egg sensitization or allergy.

3 against helminths can be more susceptible to allergy.

4 nts with asthma, atopic dermatitis, and food allergy.

5 to 36 months with suspected or known peanut allergy.

6 nes for the diagnosis and management of food allergy.

7 hen compared with infants with transient egg allergy.

8 conventional disease model and/or human food allergy.

9 ith IgE-mediated delayed anaphylaxis in meat allergy.

10 -OIT as well as the role of iNKT cells in CM allergy.

11 can, cashew, pistachio, hazelnut, and almond allergy.

12 effects of EPIT for the treatment of peanut allergy.

13 ficantly decreased risk of developing peanut allergy.

14 ave a causal role in the development of food allergy.

15 rine mDCs and PBMCs from patients with birch allergy.

16 ion, but not with hay fever or biomarkers of allergy.

17 on from atopic dermatitis to asthma and food allergy.

18 n-specific IgE in patients with birch pollen allergy.

19 infancy to subsequent challenge-proven food allergy.

20 on and clinically relevant Hymenoptera venom allergy.

21 l transplant rejection, dry eye disease, and allergy.

22 iotics among inpatients reporting penicillin allergy.

23 B individuals might reduce the risk to this allergy.

24 n remained significant after conditioning by allergy.

25 25071 and challenge-proven IgE-mediated food allergy.

26 y be distinguish clinically from primary nut allergy.

27 onducted using sera from children with wheat allergy.

28 ic study and therapeutic targeting of peanut allergy.

29 ants associated with challenge-proven peanut allergy.

30 ons for the prevention and treatment of food allergy.

31 in TH2 cells from patients with grass pollen allergy.

32 ific IgE antibodies are a hallmark of type I allergy.

33 g individuals with multiple food (multifood) allergies.

34 and statistically coordinated with reported allergies.

35 atients with concurrent PN/TN and other food allergies.

36 revention and treatment of IgE-mediated food allergies.

37 0% of individuals suffering from respiratory allergies.

38 ndividuals who typically experience seasonal allergies.

39 lls is a central event in the development of allergies.

40 evention and management of asthma and airway allergies.

41 allergy, non-milk food allergies, and other allergies.

42 pet keeping did not alter associations with allergies.

43 ity of life is often reduced during seasonal allergies.

44 feeding modes for the future development of allergies.

45 asure quality of life in people experiencing allergies (0 = not troubled to 6 = extremely troubled).

46 e interval) (1.78; 1.28-2.48), non-milk food allergy (1.65; 1.27-2.14), and other allergies (3.07; 2.

47 nd Hodgkins'disease); - 3 patients with food allergy; - 1 patient with acute gastroenteritis; - 1 pat

48 lk food allergy (1.65; 1.27-2.14), and other allergies (3.07; 2.72-3.46) compared with children with

49 1-mediated birch pollen and associated food allergies, a single wild-type allergen does not provide

50 Short ragweed (Ambrosia artemisiifolia) allergies affect more than 36 million people annually.

51 ts with challenge-proven IgE-mediated peanut allergy against 148 non-allergic infants (all 1 year o

52 ontrolled study, 74 participants with peanut allergy (ages 4-25 years) were treated with placebo (n =

53 ort quality management of patients with drug allergies and better facilitate healthcare planning and

54 asic mechanisms underlying IgE-mediated food allergies and novel therapeutic approaches under investi

55 e in the setting of naturally resolving food allergy and accompany the acquisition of food allergen u

56 hylaxis associated with ovalbumin and peanut allergy and affects the epigenome of T cells, thereby pr

57 The new Global Allergy and Asthma European Network (GA(2)LEN) mobile ex

58 effective primary prevention strategies for allergy and asthma in the future.

59 rrent allergic rhinitis (AR) related to mite allergy and asthma were based on yearly interviews at th

60 Allergy and atopic asthma have continued to become more

61 increasingly important as a way of advancing allergy and atopic asthma therapy, filling a need in tre

62 alities in genetic loci and pathways between allergy and autoimmune diseases to elucidate shared dise

63 ci, 48% had the same direction of effect for allergy and autoimmune diseases.

64 The relationship between allergy and autoimmune disorders is complex and poorly u

65 asing recognition that the alarming surge in allergy and autoimmunity in the industrialised and devel

66 tribution as modulators and effectors during allergy and autoimmunity should be considered when desig

67 e recommendations of the British Society for Allergy and Clinical Immunology (BSACI), the Association

68 r treatment followed the European Academy of Allergy and Clinical Immunology (EAACI) guidelines.

69 The European Academy of Allergy and Clinical Immunology (EAACI) has produced Gui

70 force initiative of the European Academy of Allergy and Clinical Immunology (EAACI), experts from ac

71 icles published since 2015 in the Journal of Allergy and Clinical Immunology and other high-impact jo

72 Task Force status by the European Academy of Allergy and Clinical Immunology in 2014.

73 d by a task force of the European Academy of Allergy and Clinical Immunology, aimed to assess the cur

74 of Care Committee of the British Society of Allergy and Clinical Immunology, using accredited method

75 maintains tissue homeostasis but can lead to allergy and fibrosis if not adequately regulated.

76 s with suspected histamine intolerance, food allergy and healthy controls.

77 cent advances relevant to the broad field of allergy and immunology.

78 ant diagnoses of asthma, hay fever, and food allergy and increased disease severity.

79 that emerged from the National Institute of Allergy and Infectious Disease (NIAID) workshop 'Complex

80 ction against multiple National Institute of Allergy and Infectious Disease-assigned category A prior

81 as the director of the National Institute of Allergy and Infectious Diseases at the National Institut

82 National Institute of Allergy and Infectious Diseases supported this analysis.

83 Beginning in 2008, the National Institute of Allergy and Infectious Diseases, working with other orga

84 articipants in a 2016 National Institutes of Allergy and Infectious Diseases-funded Technical Consult

85 -coupled receptors (Mrgprs) to contribute to allergy and inflammation.

86 me-wide association studies on self-reported allergy and sensitization comprising a total of 62,330 s

87 n also be used to monitor resolution of food allergy and the clinical response to immunomodulatory tr

88 c reviews on allergic rhinitis, asthma, food allergy and venom allergy, respectively.

89 ending on the country - namely 'hay fever', 'allergy' and 'pollen' - showing cultural differences.

90 gic diseases, such as asthma, rhinitis, food allergies, and atopic dermatitis, are generally classifi

91 undamental insights into the pathogenesis of allergies, and on the mechanisms of antibody production

92 tibiotic use and milk allergy, non-milk food allergies, and other allergies.

93 for precision medicine in food allergy, drug allergy, and anaphylaxis under the auspices of the PRACT

94 Asthma, allergy, and eczema were diagnosed prospectively, and lu

95 psia were analyzed regarding risk of asthma, allergy, and eczema.

96 I, 1.1-2.6]) and those with more than 2 food allergies (aOR, 1.9 [95% CI, 1.1-3.1]) were at greatest

97 Food allergies are a growing health problem, and the developm

98 Clinical manifestations of food allergy are largely mediated by IgE.

99 w nut allergies have a possibility of pectin allergies as well, and that pectin in the albedo of Citr

100 nt of evidence-based action plans to address allergy as a public health issue ought to be on national

101 gher prevalence of uncontrolled CRS, whereas allergy, asthma and smoking status did not alter the per

102 eyond the march to allergic conditions (food allergy, asthma, allergic rhinitis, allergic conjunctivi

103 evelopment of inflammatory diseases, such as allergy, asthma, and inflammatory bowel disease.

104 llergic sensitization or physician-diagnosed allergy at age 10 years.

105 ng the first 2 years of life and the risk of allergies/atopies including hay fever, eczema, food alle

106 ar homeostasis, wound healing, inflammation, allergy, autoimmunity, and oncogenesis.

107 uals who do not have celiac disease or wheat allergy but who have intestinal symptoms, extraintestina

108 the microbiota of children with and without allergies, but there have been few studies on antibiotic

109 from respiratory and skin manifestations of allergy, but the routes and mechanisms of sensitization

110 ntial for new interventions to treat dietary allergies by modulation of the microbiota.

111 The prevalence of asthma and food allergy by 6 years of age was strongly increased among c

112 The clinical diagnosis of primary nut allergy can be made by the combination of a typical clin

113 fied all pointed out gaps or deficiencies in allergy care provision in primary care.

114 t differ between food sensitization and food allergy cases and controls.

115 IgE-mediated food allergy caused by jellyfish is rare worldwide.

116 ASA challenges and desensitizations from 10 allergy centers, as well as consensus among the related

117 and Care Excellence (NICE) Guidance on Drug Allergy-CG183.

118 specific IgE Ab cause a special form of meat allergy characterized by severe systemic reactions 3-7 h

119 siderable proportion of patients assessed in allergy clinics.

120 Cow milk (CM) allergy (CMA) affects up to 3% of the paediatric populat

121 examined the association between cow's milk allergy (CMA) and juvenile idiopathic arthritis (JIA).

122 Children with cow's milk allergy (CMA) have an increased risk of other allergic m

123 piratory allergies (P < .002) or respiratory allergies coexistent with atopic eczema (P < .001).

124 eding may carry a higher risk of eczema/skin allergy compared to direct feeding at the breast.

125 essful desensitization in patients with food allergy completing clinical trials and, in some studies,

126 In vespid venom allergy, cross-reactivity between venoms of different sp

127 Finally, we compared the allergy data with those of all known diseases.

128 Although the prevalence of food allergy decreased between age 1 year and age 4 years in

129 The primary outcome was IgE-mediated egg allergy defined by a positive pasteurized raw egg challe

130 ood-allergic), mean age 2.5 years, with food allergy defined by either clinical history of reactivity

131 CD36 and PAFR are important mediators of HDM allergy development and that inhibiting HDM engagement w

132 icrobiota during infancy precedes asthma and allergy development, possibly indicating an impaired muc

133 the microbiota and how it can be related to allergy development.

134 g the role of microbiome alterations in food allergy development.

135 not, in infant stool samples in relation to allergy development.

136 ted from mice and analyzed for mechanisms of allergy development.

137 We report a case of jellyfish allergy diagnosed via an oral food challenge.

138 foods and in peanut flour extracts used for allergy diagnosis and OIT.

139 ogy, aimed to assess the current practice of allergy diagnosis in Europe.

140 ecific IgE on sensitization to allergens and allergy diagnosis is described.

141 d a major effect on analytic specificity and allergy diagnosis.

142 tion of natural and recombinant oleosins for allergy diagnostic purposes.

143 dividuals (HC), were analyzed via singleplex allergy diagnostics and a newly established immunoblot d

144 une 2016, 3260 users had registered with the Allergy Diary and 2710 had completed the baseline questi

145 The Allergy Diary is available in 21 countries.

146 on Asthma) score ranging from 0 to 4 of the Allergy Diary was compared with EQ-5D (EuroQuol) and WPA

147 Patients with delayed meat allergy display IgE and IgG Ab that selectively recogniz

148 the potential for precision medicine in food allergy, drug allergy, and anaphylaxis under the auspice

149 The patient was diagnosed with a jellyfish allergy due to IgE mediated anaphylaxis after ingestion.

150 A number of food allergies (eg, fish, shellfish, and nuts) are lifelong,

151 database of published systematic reviews in allergy epidemiology.

152 that enable multivalent display of potential allergy epitopes for determining the immunogenicity of e

153 llergy highlights the critical need for more allergy evaluations.

154 We show that infants with persistent egg allergy exhibit a unique innate immune signature, charac

155 ines for investigation of perioperative drug allergy exist, but the quality of services is unknown.

156 Food allergy (FA) is an increasing problem that has no approv

157 posures protect against childhood asthma and allergy; few data exist on asthma and allergy in adults.

158 The rate of co-allergy for most tree-nuts was <30%.

159 consecutive inpatients' files documented as allergies from the University Hospital of Montpellier el

160 Few food allergy genetic risk variants have yet been identified.

161 The prevalence of food allergies has been increasing at an alarming rate over t

162 crease in the incidence and severity of food allergies has coincided with lifestyle changes in Wester

163 Furthermore, fungal allergy has been shown to play an important role in seve

164 king, there is a strong impression that food allergy has increased, and rates as high as approximatel

165 hould consider that patients with cashew nut allergies have a possibility of pectin allergies as well

166 Mothers of children with food allergy have increased anxiety, which may be influenced

167 The spectrum of severity observed with food allergy highlights the critical need for more allergy ev

168 er symptomatic therapy can be intensified or allergy immunotherapy should be administered.

169 Allergy immunotherapy targets the immunological cause of

170 validated International Study of Asthma and Allergies in Childhood questionnaires.

171 iota has been associated with development of allergies in infants.

172 breastfeeding can prevent the onset of food allergies in offspring by instructing T reg formation vi

173 Knowledge of coincident allergies in these pairs along with the combination of S

174 Consecutive eligible children with peanut allergy in 3 centers were prospectively invited to parti

175 factor (HRF) interactions with IgE and food allergy in a murine model.

176 studies have examined the prevalence of food allergy in adolescence using objective measures such as

177 ma and allergy; few data exist on asthma and allergy in adults.

178 allergen molecules causative for type I food allergy in animals, which, like in human patients, could

179 erning the persistence or resolution of food allergy in childhood are not understood.

180 prenatal risk factor for asthma, eczema, and allergy in childhood pointing toward in utero immune pro

181 While the prevalence of food allergy in humans has been well studied for some allerge

182 tential impact of climate change upon pollen allergy in humans, focusing upon common ragweed (Ambrosi

183 months reduces the risk of IgE-mediated egg allergy in infants with hereditary risk, but without ecz

184 revent the progression of Ova induced airway allergy in mice.

185 However, the role of allergy in severe asthma is still under debate.

186 n infancy and doctor's diagnosed eczema/skin allergy in the first 6 years of life (1387 infants), bas

187 cally significant higher risk of eczema/skin allergy in the first 6 years of life (PR = 1.46), adjust

188 patterns for doctor's diagnosed eczema/skin allergy in the first 6 years of life, adjusting for conf

189 on at age 4 to 6 months to prevent hen's egg allergy in the general population.

190 ants (52%) reported to have suffered from an allergy in the past, with allergic symptoms being presen

191 curs in CAD but is less studied than contact allergy in this exquisitely photosensitive condition.

192 Work Productivity and Activity Impairment in allergy) in 1288 users in 18 countries.

193 a human/mouse chimeric model of respiratory allergy independently of AIT-induced cellular changes.

194 Although specific immunotherapy for food allergies is becoming more effective, it is still labori

195 Nut allergy is a global disease.

196 Hymenoptera venom allergy is a potentially life-threatening allergic react

197 Allergy is a public health issue of high socioeconomic r

198 Food allergy is an important public health problem because it

199 Food allergy is an increasingly common health problem in West

200 A documented penicillin allergy is associated with increased morbidity including

201 Peanut allergy is common, life-threatening, and without therape

202 childhood asthma, and thorough assessment of allergy is crucial for optimal care of these children.

203 We suspect that this allergy is underdiagnosed because of the lack of diagnos

204 flammatory conditions, its potential role in allergy is unknown.

205 ted antibiotic allergy (so-called antibiotic allergy labels [AALs]) and their impact on antibiotic pr

206 In the Learning Early About Peanut Allergy (LEAP) study, early peanut introduction in high-

207 hree serum groups, alpha-Gal-associated meat allergy (MA) patients, idiopathic anaphylaxis (IA) patie

208 ective Swedish birth cohort BAMSE (Children, Allergy, Milieu, Stockholm, Epidemiology).

209 prevented allergic sensitization in a mouse allergy model.

210 ying food allergy, suggesting that the EHR's allergy module has the potential to be used for clinical

211 and safe way of generating reliable data in allergy multicenter clinical trials.

212 Food allergy naturally resolves in a proportion of food-aller

213 es exist regarding the diagnosis of tree-nut allergy, necessitating either their elimination or the p

214 association between antibiotic use and milk allergy, non-milk food allergies, and other allergies.

215 These findings can explain lifelong food allergies observed in human subjects as the consequence

216 ients with CAD, with coexistent photocontact allergy occurring in a substantial proportion.

217 Photocontact allergy occurs in CAD but is less studied than contact a

218 antibiotic orders had a greater odds of milk allergy (Odds Ratio; 95% Confidence interval) (1.78; 1.2

219 ns (LTPs) are important causes of plant-food allergies often associated with severe allergic reaction

220 he total tested population) without reported allergy or allergic symptoms showed a positive SPT witho

221 tially IgE-mediated (affecting 50.8% of food allergy or intolerance patients) and 15.9% were anaphyla

222 croarray was performed in patients with food allergy or WAS.

223 allergic sensitization in patients with food allergy or Wiskott-Aldrich syndrome (WAS) and defined wh

224 develop normal humoral responses in a peanut allergy oral sensitization model.

225 Anaphylaxis was defined as per World Allergy Organisation (WAO) criteria.

226 Patients were recruited at random from allergy outpatient clinics in 101 health centers through

227 tations, and was associated with respiratory allergies (P < .002) or respiratory allergies coexistent

228 (P </= .002) and control subjects with venom allergy (P < .0001).

229 scores were worse in patients with comorbid allergy (P = 0.045) and chronic obstructive pulmonary di

230 crobiota of 138 infants at increased risk of allergy, participating in a clinical trial investigating

231 0-99% of patients with a label of penicillin allergy (PenA) are not allergic when comprehensively inv

232 During the symptomatic allergy period, patients showed (1) poorer performance i

233 nd memory test during and out of symptomatic allergy periods (pollen vs. non-pollen season).

234 Food allergies pose a considerable world-wide public health b

235 es/atopies including hay fever, eczema, food allergy, positive skin prick testing (SPT), or elevated

236 Primary nut allergy presents most commonly in the first five years o

237 Food allergy prevalence is reported to be increasing, but epi

238 portant for the development of strategies in allergy prevention.

239 role in secondary prevention of respiratory allergy progression.

240 Although there is some evidence for allergy-promoting effects in children because of exposur

241 Bacterial cowshed isolates are allergy protective in mice; however, the underlying mech

242 Defining the role of beneficial allergy-protective members of the microbiota in the regu

243 lly sensitized mice with ovalbumin or peanut allergy reduced anaphylactic responses to oral allergen

244 Food allergy-related quality of life improved from baseline t

245 but exact microbiome dysfunctions underlying allergies remain unclear.

246 he IgE response on a molecular level and put allergy research on the map of precision medicine.

247 ernative to the commercial ImmunoCAP ISAC in allergy research.

248 nts outgrowing their hen's egg or cow's milk allergy respectively.

249 gic rhinitis, asthma, food allergy and venom allergy, respectively.

250 ntal Factors and Their Influence on Newborns Allergy Risk (LINA) and correlated with asthma developme

251 ergic sensitization in their effects on food allergy risk (relative excess risk due to interaction, 1

252 robiotics may improve quality of life during allergy season by increasing the percentage of regulator

253 eneficial effects on MRQLQ scores throughout allergy season in individuals who typically experience s

254 t be used across all European countries, but allergy seasonality can be compared across Europe provid

255 beta-lactam allergy skin testing (BLAST) is recommended by antimicro

256 atory sites by calculating the enrichment of allergy SNPs falling in gene regulatory regions in vario

257 Additionally, we observed an enrichment of allergy SNPs falling within immune pathways and regions

258 gh prevalence of patient-reported antibiotic allergy (so-called antibiotic allergy labels [AALs]) and

259 IgE-mediated food allergy status at 1 year was determined by formal challe

260 adolescents who had only self-reported food allergy status available, the prevalence of self-reporte

261 cant results were reanalyzed conditioning on allergy status.

262 plication cohorts (the Manchester Asthma and Allergy Study [n = 30] and the Childhood Origins of Asth

263 f 722 participants in the German Multicenter Allergy Study, a birth cohort started in 1990.

264 eviously validated methods for studying food allergy, suggesting that the EHR's allergy module has th

265 rial colonization and food sensitization and allergy suggests that the microbiome may have a causal r

266 = 0.04), sensitization without any clinical allergy symptoms (aOR = 1.40; 1.06-1.85; P = 0.02), and

267 Since wheat flour, a cause of food allergy, tends to disperse rapidly in air, it can uninte

268 From the 118 antibiotic allergy-tested patients, 226 AALs were reported (mean, 1

269 tic prescribing, incorporation of antibiotic allergy testing (AAT) into antimicrobial stewardship (AM

270 was to identify whether inpatient penicillin allergy testing affected clinical outcomes during hospit

271 ammatory dermatoses, 127 controls undergoing allergy testing for IgE levels, and 30 controls with pem

272 Inpatient penicillin allergy testing led to a change in antibiotic selection

273 story and examination, supported by specific allergy tests.

274 presenting the association between PUFA and allergy; therefore, estimates could not be pooled.

275 mong students with current IgE-mediated food allergy, those with resolved or current asthma (adjusted

276 last 10 years, we have collected 28 cases of allergy to Argas reflexus from several European countrie

277 uture diagnostics and immunotherapeutics for allergy to HDM.

278 ined as disease progression, relapse, death, allergy to rituximab, or severe infection) after transpl

279 aker report of healthcare provider-diagnosed allergy to the above foods prior to age 3 with evidence

280 py (EPIT) by using Viaskin Peanut for peanut allergy treatment.

281 butes to human diseases such as asthma, food allergies, type 1 and type 2 diabetes, hepatic steatosis

282 reactions to food in 22 patients with peanut allergy undergoing double-blind, placebo-controlled food

283 Allergy vaccines should be easily applicable, safe, and

284 important implications for the design of new allergy vaccines.

285 h pollen allergen Bet v 1 for suitability as allergy vaccines.

286 The clinical presentation of a C. sativa allergy varies from mild to life-threatening reactions a

287 ), and 2.1% were confirmed to have hen's egg allergy versus 0.6% in the placebo group (relative risk,

288 Wheat allergy (WA) and celiac disease (CD) are well-defined en

289 the prevalence of self-reported current food allergy was 5.5% (95% CI, 4.9-6.2), with peanut, 2.8% (9

290 healthy donors and 120 patients with peanut allergy was collected into EDTA or heparin tubes, and sa

291 Food allergy was defined as caretaker report of healthcare pr

292 ion affects offspring susceptibility to food allergy, we epicutaneously sensitized female mice with o

293 Those with asthma and more than 2 food allergies were at the greatest risk for adverse food rea

294 G1-4 Ab in individuals with and without meat allergy were assessed by ELISA.

295 ny adverse food reaction, and those with nut allergy were most at risk of severe reactions (aOR, 2.9

296 ed IgA recognition patterns in children with allergy were observed already at 1 month of age, when th

297 participants with birch pollen-related apple allergy were randomized to daily sublingual application

298 option to be considered for adults with food allergy who were not able to acquire immune tolerance du

299 ciency led to exacerbated HDM-induced airway allergy, with increased airway and tissue eosinophilia,

300 ical pollutant and the chief cause of pollen allergy worldwide.