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1 early identify the active compound in AGE as allicin.
2 and NMR verified the active HPLC fraction as allicin.
3 sulphide (DAS), diallyl trisulphide (DATS), allicin, 3-vinyl-4H-1,3-dithiin (3-VD), 2-vinyl-4H-1,2-d
4 terminants of TRPV1 activation by capsaicin, allicin, acid, ammonia and voltage have been identified.
9 that produce organosulfur compounds, such as allicin and diallyl disulfide (DADS), which account for
12 of A. fistulosum contained 28.3-95.9 muL/mL allicin and exhibited significant antioxidant and antiba
13 ther experiments, three inhibitors (lithium, allicin and sodium) of acetyl CoA synthetase (EC 6.2.1.1
16 um bromide) cell viability assay to identify allicin as the active anticancer compound in aqueous gar
19 growth inhibition of Escherichia coli during allicin exposure coincides with a depletion of the gluta
20 hilic compounds such as acrolein (tear gas), allicin (garlic), and allyl isothiocyanate (AITC) (musta
21 d beta-carotene bleaching assays showed that allicin had an antiradical action mechanism, as well as
25 rgely inhibited by allicin treatment in vivo Allicin-induced protein modifications trigger protein ag
26 results indicate that the mode of action of allicin is a combination of a decrease of glutathione le
30 l isothiocyanate (AITC), cinnamaldehyde, and allicin, produce nociceptive sensation by directly activ
33 d candidate protein, is largely inhibited by allicin treatment in vivo Allicin-induced protein modifi
37 and reduced the number of binding sites for allicin, whereas the number of non-covalent binding site
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