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1 mice reversed these exacerbated responses to allografting.
2 y disease (GCAD) and improves murine cardiac allografting.
3 derwent removal of the spleen before corneal allografting.
4 a significant advancement for clinical nerve allografting.
5 a significant advancement for clinical nerve allografting.
6 pand the clinical application of limb tissue allografting.
7 role in the elimination of malignancy after allografting.
8 estigated in the setting of nonmyeloablative allografting.
9 dramatically reduced the acute toxicities of allografting.
10 he role of nonmyeloablative conditioning for allografting.
11 terolemic recipients at 15 and 30 days after allografting.
12 matched-related or -unrelated T-cell-replete allografting.
13 uld one treat the patient who relapses after allografting?
14 l types reached donor tissue at 1 week after allografting and similarly after syngeneic grafting.
15 being developed to decrease the toxicity of allografting and to enhance allogeneic anti-multiple mye
16 marrow stimulation, osteochondral auto- and allografting, and autologous chondrocyte implantation.
17 questions of initial therapy choice, role of allografting, and changes in therapy remains a fluid dis
22 ng administered intravenously at the time of allografting (day 0) and on the following day (day 1).
32 cure are the excessive toxicity noted after allografting in multiple myeloma, contaminating tumor ce
36 lated (MUD) donor T-cell-replete bone marrow allografting, obviating the need for additional prophyla
38 d with 1 ml ALS on days -7 and 0 relative to allografting resulted in 100% permanent islet graft surv
39 th ALL in CR, reduced intensity conditioning allografting results in modest TRM, limited risk of rela
40 s by direct DNA injection, followed by liver allografting, results in donor-specific unresponsiveness
41 heir demonstrated enhancement of bone marrow allografting, suggests that the use of Tcm therapy in co
42 ular remission in patients who relapse after allografting; the mechanism for this graft-versus-leukem
43 eceived uniform conditioning, T-cell-replete allografting, then PTCy, mycophenolate mofetil, and tacr
44 regimens, designed to allow the benefits of allografting to be extended to patients in whom this pro
45 y allowing full extension of the benefits of allografting to the group of patients in highest need.
46 ated after nonmyeloablative conditioning and allografting using human leukocyte antigen (HLA)-mismatc
48 loped EBV-positive PTLD 8 to 94 months after allografting were uniformly treated with acyclovir and I
49 rrow transplantation (BMT) would allow organ allografting without chronic immunosuppressive therapy.
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