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1 le effect on primary T-cell reactivity after alloimmunization.
2 oimmunization and for abolishing established alloimmunization.
3 proved oxygenation, with a 1 percent rate of alloimmunization.
4 at risk for anemia due to maternal red-cell alloimmunization.
5 at risk for anemia due to maternal red-cell alloimmunization.
6 major complication of transfusion therapy is alloimmunization.
7 ocytes provides a potential stimulus for HbA alloimmunization.
8 antigen features that may also influence RBC alloimmunization.
9 tic counseling and prenatal assessment of Rh alloimmunization.
10 nts should be considered to prevent platelet alloimmunization.
11 as a means of preventing the development of alloimmunization.
12 a good serologic screen for the diagnosis of alloimmunization.
13 of an adjuvant, is sufficient to induce RBC alloimmunization.
14 eceptor is required for inflammation-induced alloimmunization.
15 significantly decreased in Akita mice after alloimmunization.
16 4 expression, thereby confirming its role in alloimmunization.
17 c acid-induced IFN-alpha/beta production and alloimmunization.
18 Prophylactic protocols prevented alloimmunization.
19 inflammatory state that is less conducive to alloimmunization.
20 ti-CD20 Ab has recently been used to prevent alloimmunization.
21 unknown to what extent this occurs in human alloimmunization.
22 ens, whereas nonresponders were resistant to alloimmunization.
23 nicity is a crucial factor in red blood cell alloimmunization.
24 binding motif for HLA-DR52a that can lead to alloimmunization.
25 mental factors may play a role in regulating alloimmunization.
26 mmation has a complex regulatory effect upon alloimmunization.
27 mit the further amplification of established alloimmunization.
28 mmation in mice has the capacity to regulate alloimmunization.
29 d the enhancing effects of poly (I:C) on RBC alloimmunization.
30 l leukocytes in blood transfusions can cause alloimmunization.
31 mmatory conditions associated with increased alloimmunization.
32 acid [poly (I:C)] significantly enhances RBC alloimmunization.
33 ion and, (5) the treatment and prevention of alloimmunization.
36 with such a microbe could predispose to RBC alloimmunization, a mouse model was developed using muri
40 We assessed protocols for preventing primary alloimmunization and for abolishing established alloimmu
41 m)IL-12 was given to C57BL/6 mice undergoing alloimmunization and found to transiently but profoundly
44 We used BALB/c donors (H-2(d)) to assess alloimmunization and islet transplantation outcomes in A
45 quiring transfusion with a very high risk of alloimmunization and life-threatening complications.
46 nfections, may have an increased risk of RBC alloimmunization and may benefit from personalized trans
47 le blood group loci, potentially eliminating alloimmunization and significantly improving transfusion
48 used leukocytes is critical to prevention of alloimmunization and transfusion-induced graft-versus-ho
51 transfused blood has been shown to decrease alloimmunization, and genotyping for antigen matching ma
53 ction of platelet products, prevention of Rh alloimmunization, and management of refractoriness to pl
54 tients to insufficient correction of anemia, alloimmunization, and organ iron overload (for which the
63 k of three complications of transfusion: HLA alloimmunization, cytomegalovirus transmission, and recu
70 eral concerns, including the consequences of alloimmunization in chronically transfused patients and
71 rphism have over a 3-fold lower risk for RBC alloimmunization in comparison with patients without thi
72 Advances have been made in understanding alloimmunization in granulocyte transfusion recipients a
74 the rate and the frequency of red blood cell alloimmunization in mouse models, may dictate responder/
76 These data describe the prevalence of Rh alloimmunization in patients with SCD transfused with ph
77 port findings from an observational study of alloimmunization in patients with sickle cell disease (S
84 olyclonal anti-KEL sera completely prevented alloimmunization in wild-type and single-knockout (KO) m
85 laxis with polyclonal anti-KEL sera prevents alloimmunization in wild-type recipients transfused with
88 ntly occurring phenomenon, prevention of HLA alloimmunization is an important management strategy.
89 bulin to prevent pregnancy associated anti-D alloimmunization, its mechanism of action remains elusiv
90 unologic consequences of transfusion such as alloimmunization may also be severe, resulting in acute
93 hat are at risk because of maternal red-cell alloimmunization, moderate and severe anemia can be dete
97 fractory to platelet transfusions because of alloimmunization require HLA-matched platelets, which is
98 RF) was strongly associated with a decreased alloimmunization risk (odds ratio [OR] 0.26, 95% confide
99 t donor RBC antigens, and valid estimates of alloimmunization risk are clinically important, but litt
100 s or patients with a known predisposition to alloimmunization, such as those with sickle cell disease
101 childhood are associated with lower rates of alloimmunization than are seen in SCD, suggesting immune
102 s who had a splenectomy had a higher rate of alloimmunization than patients who did not have a splene
103 ts that has a dramatically increased risk of alloimmunization that appears to be genetically determin
104 ing mixed lymphocyte reactions as a model of alloimmunization, the authors assessed the effect of inf
105 platelet concentrates for prevention of HLA alloimmunization, the findings of this study support tha
106 To discuss how inflammation affects humoral alloimmunization to antigens on transfused red blood cel
108 MNL) are responsible for transfusion-induced alloimmunization to donor major histocompatability compl
110 cessary to avert the adverse consequences of alloimmunization to human leukocyte antigens, immunohema
111 of the fetus and newborn (HDFN) is caused by alloimmunization to paternally derived RBC antigens.
114 MT, we wished to determine the prevalence of alloimmunization to platelets in transfused SCD patients
120 ently, three unique murine models of humoral alloimmunization to transfused RBCs have been described.
123 velopment of refractoriness to transfusions, alloimmunization, transfusion reactions, the transmissio
124 variety of immunologic responses, including alloimmunization, transfusion-associated graft-versus-ho
125 onor for male recipient, donor parity, donor alloimmunization, viral serology, nucleated cell dose, C
128 minority of untransfused patients at risk of alloimmunization who would benefit from more extensively
129 we discuss the risk factors associated with alloimmunization with emphasis on possible mechanisms th
130 7BL/6 splenocytes taken 7 days after in vivo alloimmunization with irradiated BALB/c spleen cells.
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