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1 ission blockade with the chitinase inhibitor allosamidin.
2 in total syntheses of (+)-trehazolin and (+)-allosamidin.
3 erential sensitivity of PfCHT1 and PgCHT1 to allosamidin.
4 ctive AMCase was abrogated by treatment with allosamidin.
5 to invade the mosquito, and the presence of allosamidin, a chitinase inhibitor, in a blood meal prev
7 inase inhibitor allosamidin, suggesting that allosamidins act as transition state analogs of an oxazo
8 structures of the active enzyme complexed to allosamidin (an analogue of a proposed reaction intermed
9 ation that raises questions about the use of allosamidin and components like it as potential malaria
10 for 6 weeks with constant administration of allosamidin and fed an atherogenic diet showed aggravate
11 ensitive to the specific chitinase inhibitor allosamidin and has the ability to bind to chitin partic
13 including the potent inhibitors argadin and allosamidin, and the structures of these in complex with
14 profile of PfCHT1 and its IC(50) (40 nM) to allosamidin are distinct from endochitinase activities s
17 ut: the presence of the chitinase inhibitor, allosamidin, in an infectious blood meal prevents oocyst
19 ver, PbCHT1 activity appeared insensitive to allosamidin inhibition, an observation that raises quest
22 igated the effects of a chitinase inhibitor, allosamidin, on macrophage function in vitro and on athe
23 ion is mediated via a PI3K/Akt-dependent and allosamidin-sensitive pathway that is independent of the
24 are similar to the known chitinase inhibitor allosamidin, suggesting that allosamidins act as transit
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