ライフサイエンスコーパス: allostatic

1 triction (SR) sleep may be regulated by both allostatic and homeostatic mechanisms.

2 he hypothesis that chronic stress induces an allostatic attenuation of the mesolimbic dopaminergic sy

3 ay be explained by a maladaptive feedforward allostatic cascade model and how understanding migraine

4 l forebrain and cingulate cortex may mediate allostatic changes in sleep time and intensity observed

5 rlying these adaptive, or more specifically, allostatic, changes in sleep homeostasis are unknown.

6 ng behaviors by reversing an alcohol-induced allostatic DA deficiency in the mesolimbic system.

7 also review how homeostatic, circadian, and allostatic drives help regulate sleep state switching an

8 neighborhood risk of psychosocial stress on allostatic load (AL) among adolescents as a mechanism th

9 Allostatic load (AL) has been proposed as a new conceptu

10 Allostatic load (AL) is a measure of overall physiologic

11 With increasing severity and over time, the allostatic load (increase in glycemia) may have patholog

12 king speed, lower fluid intelligence, higher allostatic load and increased mortality risk.

13 imulated leukocytes would show signs of high allostatic load and low levels of protective psychosocia

14 the brain and body that can lead to disease (allostatic load and overload).

15 GR gene expression can lead to an increased allostatic load and result in an "aging-like" phenotype

16 Consequently, allostasis and allostatic load can jointly affect vulnerability to brai

17 In this Personal View, we propose an allostatic load framework to account for biomarkers of p

18 Allostatic load may alter brain networks both functional

19 understanding migraine within the context of allostatic load model suggests alternative treatments fo

20 ts of CRS or an example of adaptation to the allostatic load produced by CRS.

21 reflects an allostatic process, and that the allostatic load produced by SD has direct effects on the

22 Chronic allostatic load provides a unifying perspective with imp

23 gins by noting two new terms, allostasis and allostatic load that are intended to supplement and clar

24 ipheral physiology and can contribute to the allostatic load throughout the body.

25 es can become dysregulated and maladaptive ("allostatic load").

26 tics and experiences of societal inequality (allostatic load) increase the complexity of studying bre

27 n, disability and markers of chronic stress (allostatic load).

28 llostasis) and maladaptive in the long term (allostatic load).

29 Chronic stress is measured by allostatic load, a biological marker of cumulative wear

30 dults from low-income families manifest more allostatic load, an index of chronic physiological stres

31 ople with type 2 diabetes experience chronic allostatic load, manifest as dynamic disturbances in rea

32 We consider chronic stress/allostatic load, mental distress, coping skills and reso

33 eoretical constructs, such as weathering and allostatic load, regarding the power of chronic environm

34 specific physiological norms, allostasis and allostatic load, stochasticity, and decline in stress re

35 increases incentive salience of HP foods and allostatic load.

36 in general or mental health or estimates of allostatic load.

37 hat can, in a vicious cycle, lead to further allostatic load.

38 depressive-like behavior, indicating that an allostatic mechanism exists in this difference.

39 The allostatic model of maintaining "stability through chang

40 tization, aberrant learning, and anti-reward allostatic neuroadaptations.

41 ntervention for the prevention of subsequent allostatic overload that auto-perpetuates obesity.

42 Two types of "allostatic overload" are described: type 1 is an essenti

43 xt of repeated sleep restriction reflects an allostatic process, and that the allostatic load produce

44 n as a motivational incentive because due to allostatic reference resetting, more stimuli are require

45 ts in withdrawal mediated through persistent allostatic reference resetting.

46 ticipatory processing which is essential for allostatic regulation.

47 unrelated to physiological needs induces an allostatic regulatory state that defends a supranormal a

48 ogical stressors are frequent and/or severe, allostatic responses can become dysregulated and maladap

49 ctors, injury (or stressors), and the body's allostatic responses.

50 or is lost in either neuronal population, an allostatic shift will occur leading to a long-term dysre

51 mical dysregulations that ultimately form an allostatic state (decreased dopamine and opioid peptide

52 This allostatic state is hypothesized to be reflected in a ch

53 and are hypothesized to repeatedly drive the allostatic state.