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2 kocyte reactions were performed to assess DC allostimulatory ability and also the function of putativ
3 transduced ODN DCs exhibit markedly impaired allostimulatory ability and promote apoptosis of activat
4 gel shift assay results, and showed reduced allostimulatory ability of the CsA-treated cells compare
5 , adenosine-differentiated DCs have impaired allostimulatory activity and express high levels of angi
7 mined by flow cytometric analysis, and their allostimulatory activity assessed in primary mixed leuko
8 iated with marked impairment of their T-cell allostimulatory activity but with only modest prolongati
9 panded CD34+-selected cells showed increased allostimulatory activity compared to both cultured CD34+
11 s) from PD-L1(-/-) mice exhibited a stronger allostimulatory activity compared with that in wild-type
12 ing DC differentiation significantly reduced allostimulatory activity in a MLR using naive CD4(+) T c
15 nd CD86 expression and up-regulated the poor allostimulatory activity of the DCp assessed by MLR and
17 and costimulatory molecules and naive T cell allostimulatory activity than migratory wild-type (wt) C
21 /CD14- cells, a 42-fold increase (median) of allostimulatory activity was observed as compared with s
22 nistration and the resulting augmentation of allostimulatory activity within host lymphoid tissue, is
23 ndocytosis properties, cell surface markers, allostimulatory activity, and cytokine production follow
24 acity to produce IL-12, did not acquire full allostimulatory activity, and rapidly underwent apoptosi
25 rIL-4 were functionally immature in terms of allostimulatory activity, but this activity increased af
26 immature phenotype was linked to poor T cell allostimulatory activity, indicative of DC progenitors.
27 immature DCs partially restored the reduced allostimulatory activity, whereas alcohol given only dur
36 tolerogenic role of immature DC, the T cell allostimulatory and Th1-biasing function of CD14+ LC pre
38 crophages, but acquire, upon maturation, the allostimulatory capacity and surface phenotype of classi
39 from gammadelta-/- recipients exhibited less allostimulatory capacity compared to wt DCs after irradi
44 In summary, lack of CCR2 interferes with the allostimulatory capacity of DC and promotes the generati
47 lta T cells exacerbate GVHD by enhancing the allostimulatory capacity of host antigen-presenting cell
48 d NS3 (not E2) inhibited differentiation and allostimulatory capacity of immature DCs similar to defe
50 cal to that of interdigitating DC and potent allostimulatory capacity were released from FSDDC-A with
51 d costimulatory molecule expression and poor allostimulatory capacity when interacting with T cells,
52 loci in germline configuration and show low allostimulatory capacity, but produce type I IFN upon vi
53 ich are markedly impaired in Foxp3(-) T cell allostimulatory capacity, to favor the stimulation of mu
62 tial augmentation of the number of potential allostimulatory cells in donor organs before transplanta
63 and CD40 cross-linking increased the T-cell allostimulatory function of BM DCs, only LPS stimulation
69 PS was superior to CpG ODN in increasing the allostimulatory potential of lung DCs and their expressi
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