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1 recurrence and will not be offered a repeat allotransplant.
2 ow and its environment in a composite tissue allotransplant.
3 major histocompatibility complex mismatched allotransplants.
4 rejection of minor antigen disparate cardiac allotransplants.
5 and tapered corticosteroids, comparable with allotransplants.
6 n the elimination of malignant disease after allotransplants.
7 used myeloablative conditioning regimens for allotransplants.
8 (-) mice that spontaneously accepted a heart allotransplant and displayed donor-specific tolerance al
14 resulted in 100% survival of mice receiving allotransplants challenged with an otherwise invariably
20 CNA staining in lung cultures from rejecting allotransplanted dogs was significantly greater than tha
21 g the potential for better outcomes in islet allotransplant for type 1 diabetes mellitus with refinem
22 al rates of insulin independence after islet allotransplant for type 1 diabetes, long-term islet func
24 isolate human pancreatic islets intended for allotransplants generates a product that is hampered by
26 n cold University of Wisconsin solution, and allotransplanted into recipient dogs for either 4 hr (gr
27 (2) proliferation is increased in rejecting allotransplanted lungs, (3) endothelin-A receptors media
29 rade of rejection was 2.74+/-0.17 (n= 19) in allotransplanted lungs; evidence of infection was presen
30 We've established a nonhuman primate islet allotransplant model to address questions such as whethe
31 In a preclinical, nonhuman primate islet allotransplant model, the authors evaluated a novel immu
33 aftment/tolerance in exceptionally stringent allotransplant models by (1) limiting the early expansio
34 receptor blockade in mouse cardiac and islet allotransplant models has led to long-term engraftment a
39 f transplanted organs from the same species (allotransplant) or different species (xenotransplant).
40 One hundred thirty-two consecutive renal allotransplant patients, who underwent transplantation o
41 particles at postoperative day 6, a group of allotransplanted rats was treated with cyclosporin A (3
42 by using data from 1827 HLA-matched sibling allotransplant recipients reported to the International
43 ecretion was observed in either the auto- or allotransplant recipients, whereas healthy control subje
47 valuable in many clinical situations such as allotransplants, some autoimmunities, as well as with so
50 Sixty-seven percent of remitters received an allotransplant that delivered superior survival compared
51 ional successful multiple vascular composite allotransplants, thoracic, and abdominal organ recoverie
53 his shows that failure of orthotopic corneal allotransplants to elicit a CD4+ T-cell direct allorespo
54 bone marrow compartment in composite tissue allotransplants to potentially induce immune tolerance.
56 lity and function of Treg cells for inducing allotransplant tolerance or treating autoimmune conditio
57 esus monkeys undergoing MHC-mismatched islet allotransplants treated with belatacept and the mTOR inh
62 p I received composite musculocutaneous flap allotransplants (WF-->Lew) with immunosuppression allowi
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