ライフサイエンスコーパス: allozyme

1 her commonly used molecular markers, such as allozymes.

2 polygyne (multiple queen) social forms using allozymes.

3 fragment length polymorphism (RFLP) data and allozymes.

4 polymorphism with respect to the common A/B allozymes.

5 sly generated based on mitochondrial DNA and allozymes.

6 atellite loci (0.016) was lower than that of allozymes (0.036).

7 ampling sites in a manner similar to that of allozyme allele frequencies in Aequipecten opercularis,

8 in the absence of latitudinal clines for PGM allozyme alleles, the lack of association of PGM allozym

9 amino acid polymorphism segregating with the allozyme alleles.

10 lation showed significant differentiation in allozymes among neighbouring subpopulations.

11 Allozyme analysis revealed a high degree of fixed hetero

12 ing substantial gametic disequilibrium among allozyme and complementary DNA (cDNA) markers encompassi

13 and UV-irradiated experimental regimes using allozyme and DNA restriction fragment length polymorphis

14 ologically indistinguishable, and surveys of allozyme and DNA sequence variation have revealed signif

15 Overall, allozyme and ISSR techniques yielded similar results, in

16 The conclusion from the allozyme and life history data is that the flowering dog

17 divergence is consistent with evidence from allozyme and microsatellite data and is interpreted in t

18 Comparison with allozyme and mitochondrial DNA restriction fragment leng

19 Allozyme and PCR-based molecular markers have been widel

20 nt distribution of Fst estimates across both allozyme and RAPD loci.

21 We have now used allozyme and restriction fragment length polymorphism an

22 measure the extent of founder effects using allozymes and microsatellites, and ask whether this is c

23 nvicta using two classes of nuclear markers, allozymes and microsatellites, and markers of the mitoch

24 ilibrium that underlies associations between allozymes and susceptibility.

25 e relies on linkage between genetic markers (allozymes) and resistance loci.

26 tus) in 310 loci, including microsatellites, allozymes, and single nucleotide polymorphisms.

27 ed with constructs for wild type and variant allozymes; and recombinant proteins were measured by qua

28 dy of nucleotide variation observed that PGM allozymes are a heterogeneous mixture of amino acid poly

29 mon allozyme mobility alleles, we find these allozymes are the result of many different amino acid ch

30 he derived A allele, and differences between allozymes are twice as among B alleles.

31 Much of the scepticism that surrounds allozyme-associated heterosis comes from inconsistent an

32 allozyme-associated heterosis, and that the allozyme-associated heterosis detected in this study was

33 Allozyme-associated heterosis for growth rate was detect

34 in order to evaluate possible mechanisms of allozyme-associated heterosis for growth rate.

35 uating the genetic mechanisms that may cause allozyme-associated heterosis in natural populations has

36 ry for predictive power and repeatability of allozyme-associated heterosis, and that the allozyme-ass

37 e importance of heterosis, and in particular allozyme-associated heterosis, in natural populations re

38 to human genetic diseases.We looked for ACO2 allozymes by DNA sequencing and genotyping in a populati

39 The novel recombinant NAT2 allozymes catalyzed both N- and O-acetyltransferase acti

40 ever, strongly disagreed with those based on allozymes, chloroplast DNA, and morphological traits.

41 In addition, specific allozyme classes within species contain several amino ac

42 Alleles at all six of these allozymes correlate with the timing of adult eclosion, a

43 We demonstrate the method on allozyme data from two species of hybridizing trout, as

44 Previous allozyme data have suggested that there is a small but s

45 ess SA is observed for S locus data than for allozyme data in accordance with the hypothesis that SSI

46 e Me-2 locus and by F-statistics for all the allozyme data, is evidence of some local relatedness due

47 e replacement mutation that accounts for the allozyme difference.

48 The observed allozyme differences and surprisingly low estimates of a

49 No fixed allozyme differences distinguish the flowering dogwood f

50 on for transfection efficiency, the Trp(173) allozyme displayed 31%, Thr(287) 350%, Ile(306) 4.8%, an

51 The Arg146 allozyme displayed a significant decrease in protein qua

52 cells harboring these same cytosolic variant allozymes displayed significant increases in the EC(50)

53 The effect of inbreeding on allozyme diversity is consistently larger than has been

54 Allozyme diversity was surveyed in 19 putative loci over

55 Levels of allozyme diversity were studied in five species of the a

56 n aggregation in vitro for the three variant allozymes encoded by alleles that include the two TPMT*3

57 ely congruent to those produced by mtDNA and allozymes, except that the satellite DNA yields much lon

58 Patients with TPMT variant allozymes exhibit diminished levels of protein and/or en

59 The Ile(306) and Thr(287)/Ile(306) allozymes expressed too little activity for inclusion in

60 s virtually identical (0.036) to the average allozyme FST.

61 conitase, we wished to find common ACO2 gene allozymes, functional polymorphisms that might be associ

62 The allozyme genetic structure of five populations (from Jap

63 effect of ontogeny on relationships between allozyme genotypes and fresh weight was measured weekly

64 Four variant allozymes had K(m) values that differed significantly fr

65 An early experiment with allozymes had shown linkage disequilibrium between these

66 showing low levels of allelic diversity (eg allozymes) has been well characterised and evaluated.

67 Most work to date, based on allozymes, has involved pattern analysis for individual

68 umulative effect was an increase in apparent allozyme heterosis later in ontogeny coinciding with a s

69 he glaciated history of the region, very low allozyme heterozygosity was found in these taxa (Ho = 0.

70 The rs5063 variant allozyme in transfected HEK293 cells was decreased to 55

71 racterize spatial genetic differentiation of allozymes in adult Delphinium nuttallianum plants within

72 Earlier studies of the common PGM allozymes in Drosophila melanogaster reported no in vitr

73 he failure to detect differences between PGM allozymes in functional studies.

74 lity of maternally (mtDNA) and biparentally (allozymes) inherited genes of 443 chamois (Rupicapra r.

75 Here, fixation, segregation, and linkage of allozymes is investigated in the progeny of self-fertili

76 The previously reported Est-6 allozyme latitudinal clines may be accounted for by the

77 ter, the investigators report the results of allozyme-level analyses of 2 lines derived from hybridiz

78 ulation that was heterozygous for two of the allozyme loci (Sharp Spring).

79 .e., all eight microsatellite loci, 12 of 16 allozyme loci and 44% of the 285 SNPs).

80 ytonuclear disequilibrium between 11 nuclear allozyme loci and both mitochondrial and chloroplast DNA

81 a population that was monomorphic for all 25 allozyme loci examined (Monkey Spring) than for fish fro

82 We used allozyme loci for a paternity analysis of 518 seeds prod

83 s k statistic to describe the variance among allozyme loci in 102 published data sets from fishes.

84 Electrophoretic analysis of 17 allozyme loci in 21 populations reveals significant freq

85 design was used to survey eight polymorphic allozyme loci in the parent and offspring generations of

86 Allelic variation for five allozyme loci is also consistent with the Spain-rest of

87 ferences from the hawthorn host race for six allozyme loci mapping to three different chromosomes.

88 the genetic structure of the nuclear-encoded allozyme loci should reflect the movement of both seeds

89 Two of six allozyme loci showed a significant increase in apparent

90 Three allozyme loci showed significant associations with mtDNA

91 Results from eight allozyme loci showed that there was considerable genetic

92 sure greater interpopulation divergence than allozyme loci suggested constraints on microsatellite ev

93 Allelic frequency differentiation at seven allozyme loci was relatively homogeneous across loci and

94 was evident in two of four populations, and allozyme loci were characterized by temporal variation i

95 Spatial autocorrelation analyses of 12 allozyme loci were used to compare genetic structure wit

96 same populations based on seven polymorphic allozyme loci yielded an estimate of Fst of 0.13.

97 well beyond the previously characterized six allozyme loci, covering substantial portions of chromoso

98 as not paralleled by allelic variation at 33 allozyme loci, which were used as nuclear DNA markers.

99 pulations at chloroplast markers but not for allozyme loci.

100 rkers and local heterozygote deficiencies at allozyme loci.

101 imates calculated from published data on six allozyme loci.

102 hort tandem repeat (microsatellite) loci and allozyme loci.

103 e loci directly or on loci tightly linked to allozyme loci.

104 al Pennsylvania, based on variation at eight allozyme loci.

105 ulation diversity is substantial for nuclear allozyme markers (mean P=0.610, A(e)=1.580, H(e)=0.277)

106 was derived from 251 microsatellite and four allozyme markers and covers 3350 cM (approximately 90% o

107 ts recombination fractions with ES1 and MDH2 allozyme markers consistent with previous localization o

108 Thus, fixation and segregation of allozyme markers in inbred oyster families deviates from

109 oral population structure of chloroplast and allozyme markers in the annual plant Phacelia dubia.

110 Spatial analysis of mtDNA and allozyme markers revealed strong geographical structure

111 ysis of 288 microsatellite polymorphisms, 13 allozyme markers, and phenotypic sex in four backcross f

112 The method is appropriate for use with allozymes, microsatellites, or restriction fragment leng

113 D. melanogaster and D. simulans share common allozyme mobility alleles, we find these allozymes are t

114 tected polymorphisms that would lead to ACO2 allozymes, nor have we observed differences in ACO2 isof

115 (m) values for sodium arsenite with the same allozymes of 11.8, 8.9, and 4.5mum.

116 Of the recombinant allozymes, only the double mutant (Arg39Cys264) displaye

117 However, estimates of U based on changes in allozymes or DNA sequences and fitness traits are discor

118 collected from natural populations, and the allozyme phenotypes of infected hosts were compared to t

119 parallels the paradox described earlier for allozyme polymorphism and challenges simple equilibrium

120 an amino acid variant, leading to the common allozyme polymorphism at Gld, remains in low to intermed

121 utocorrelation (SA) treatment of S locus and allozyme polymorphism data for four loci indicates that

122 sential to understanding the significance of allozyme polymorphism in adaptation.

123 the Pro/Leu mutation responsible for the A/B allozyme polymorphism in each variant.

124 t polymorphism, which corresponds to the S-F allozyme polymorphism.

125 morphisms, one of which accounts for the S-F allozyme polymorphism.

126 lected site is responsible for the EST-6 F/S allozyme polymorphism; the same site is responsible for

127 s significant clinal differentiation for the allozyme polymorphisms as well as for many of the crypti

128 id mutations responsible for the common G6PD allozyme polymorphisms in Drosophila melanogaster have b

129 uency-dependent selection protecting natural allozyme polymorphisms through differential resource uti

130 e phosphoglucomutase (PGM) possesses several allozyme polymorphisms, it is unique in that it had been

131 ults are consistent with the hypothesis that allozyme population structure is primarily the result of

132 e loci was significantly higher than that of allozymes, probably because the former experience consid

133 The allozyme results only partially supported the patterns r

134 s264, Thr364, and double variant Arg39Cys264 allozymes showed significant decreases in levels of acti

135 hila willistoni was the subject of intensive allozyme studies and the locus coding for alcohol dehydr

136 ogen metabolism and is highly polymorphic in allozyme studies of many species.

137 Allozyme studies revealed complete reproductive isolatio

138 A combination of field experiments and allozyme studies was used to test whether two spring-eph

139 ore than twice the level detected in earlier allozyme surveys.

140 Using allozyme techniques, we uncovered genetic differentiatio

141 ADH allozyme type has no detectable effect on development.

142 s of QTL suggests that coding variants (e.g. allozymes) underlie a fraction of quantitative variation

143 ues, but there was nevertheless considerable allozyme variability in two of the four sets of inbred p

144 multiple continents for several phenotypes, allozyme variants, sequence variants, and chromosome inv

145 Canary Islands was investigated by studying allozyme variation at nine putative loci in five populat

146 We studied allozyme variation in metapopulations of two species of

147 We studied allozyme variation in rock pool metapopulations of two s

148 al outcrossing distances and the patterns of allozyme variation in T. erectum and T. grandiflorum pop

149 Here, allozyme variation is analysed in over 1100 individuals

150 bit a positive correlation between mtDNA and allozyme variation, suggesting that mtDNA diversity may

151 t contrasts sharply with previous studies of allozyme variation, which have showed significantly less

152 ll (South America) associated with the Est-6 allozyme variation.

153 hich are not perfectly associated with Est-6 allozyme variation.

154 s linked to Thr(287), so this double variant allozyme was also studied functionally.

155 The intracellular stability of the *1 and *2 allozymes was examined in insect as well as mammalian ce

156 Using four polymorphic allozymes, we compared the outcrossing-rate estimates of

157 The enzyme activities of the SULT1A1 allozymes were studied with a variety of substrates, inc

158 GSTP1 variant allozymes were then expressed in COS-1 cells, and five d

159 rotein level was also observed for the Arg39 allozyme, whereas Met201 displayed no significant change

160 similar to the wild-type cytosolic (WT Cyt) allozyme, whereas the R424C and S457F cytosolic variants

161 zyme alleles, the lack of association of PGM allozymes with the cosmopolitan In(3L)P inversion, and t

162 protein was observed for the Arg146 variant allozyme, with 20% of wild-type protein, primarily becau

163 ) 6.2% of the activity of the wild type (WT) allozyme, with 20, 190, 4.4, and 7.9% of the level of WT

164 1, and 11 mum for WT, Trp(173), and Thr(287) allozymes, with K(m) values for sodium arsenite with the