ライフサイエンスコーパス: allyl isothiocyanate

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1 tivity (the latter against the TRPA1 agonist allyl isothiocyanate).

2 neurons do not respond to the TrpA1b agonist allyl isothiocyanate.

3 ed antimicrobial packaging incorporated with allyl isothiocyanate (AIT) and carbon nanotube (CNT), an

4 s acrolein (tear gas), allicin (garlic), and allyl isothiocyanate (AITC) (mustard oil).

5 alenol (alpha-ZOL) on a solution model using allyl isothiocyanate (AITC) and also determines the bioa

6 dPGJ(2) evoked currents similar to equimolar allyl isothiocyanate (AITC) in the nominal absence of ca

7 e of cabbage that accounted for the enhanced allyl isothiocyanate (AITC) in the volatile oils of the

8 psulation and controlled release of volatile allyl isothiocyanate (AITC) molecules.

9 Allyl isothiocyanate (AITC) occurs in many commonly cons

10 (1)H and (13)C NMR spectra of allyl isothiocyanate (AITC) were measured, and the excha

11 TRPA1 activators, including cinnamaldehyde, allyl isothiocyanate (AITC), and 4-hydroxynonenal, incre

12 Pungent chemicals such as allyl isothiocyanate (AITC), cinnamaldehyde, and allicin

13 The effect of allyl isothiocyanate (AITC), in combination with low tem

14 Allyl isothiocyanate (AITC; aka, mustard oil) is a power

15 h increasing doses of the chemical irritants allyl isothiocyanate (AITC; also known as mustard oil) o

16 The pungent component of mustard, allyl-isothiocyanate (AITC), activates the extreme cold

17 e neurons while capsaicin (TRPV1 agonist) or allyl-isothiocyanate (AITC, TRPA1 agonist) elicited resp

18 The TRPA1 agonists allyl isothiocyanate and cinnamaldehyde and the TRPV4 ag

19 hey responded to the electrophilic compounds allyl isothiocyanate and cinnamaldehyde as well as heat.

20 , and some pungent plant ingredients such as allyl isothiocyanate and cinnamaldehyde.

21 Both allyl isothiocyanate and THC mediate their excitatory ef

22 s, which also responded to the TRPA1 agonist allyl isothiocyanate and the TRPV1 agonist capsaicin.

23 oducing a significant potentiating effect on allyl isothiocyanate- and diclofenac-induced currents of

24 Treatment with allyl isothiocyanate, cinnamaldehyde, or GSK1016790A cau

25 (EC(50) = 34 microM) than the TRPA1 agonist allyl isothiocyanate (EC(50) = 400 microM) or the TRPV3

26 gredients of pungent natural products (e.g., allyl isothiocyanate), environmental irritants (e.g., ac

27 humans find pungent and irritating, such as allyl isothiocyanate (in wasabi) and acrolein (in cigare

28 neurons that also responded to mustard oil (allyl isothiocyanate), indicating a presynaptic action.

29 gredients of pungent natural products [e.g., allyl isothiocyanate (ITC), cinnamaldehyde, allicin, and

30 ed responses triggered by the TRPA1 agonists allyl-isothiocyanate (mustard oil), carvacrol, and polyu

31 Here we show that AITC (allyl isothiocyanate; mustard oil) and menthol represent

32 lencing TRPV1(+) or TRPA1(+) neurons allowed allyl isothiocyanate or capsaicin, respectively, to evok

33 nd corresponding quenching when activated by allyl isothiocyanate or heat suggest lipid bilayer-indep

34 Here we show that allicin and DADS excite an allyl isothiocyanate-sensitive subpopulation of sensory

35 the nociceptive and inflammatory response to allyl isothiocyanate (the agonist of TRPA1) and reversed

36 and DADS share structural similarities with allyl isothiocyanate, the pungent ingredient in wasabi a

37 Allyl isothiocyanate, the pungent principle of wasabi an

38 Topical application of mustard oil (allyl isothiocyanate) to the skin activates underlying s

39 n of KS(13)CN with allyl bromide to give the allyl isothiocyanate, which upon Trofimov pyrrole synthe

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