1 nzyme A thioesters of PUFAs antagonize HNF-4 alpha action.

2  were performed to identify the mechanism of alpha action.

3 AP4K4 expression is required for optimal TNF-alpha action.

4 oter likely involved in the mechanism of TNF-alpha action.

5 pa B) did not appear to be involved in SDF-1 alpha actions, alone or in combination with other cytoki

6      These data establish a mechanism of TNF alpha action and provide evidence that local expression

7 sm in vivo and the tissue specificity of TNF-alpha action are unclear.

8 ase reportedly initiates proinflammatory TNF alpha action by phosphorylating and activating Raf-1.

9                   Evidence suggests that TNF(alpha) action directly on vago-vagal reflex control circ

10    Inhibition of tumor necrosis factor (TNF)-alpha action has recently been shown to reverse insulin

11              Reports on the mechanism of TNF-alpha action have been somewhat contradictory.

12        Our data suggest that blockade of TNF-alpha action in adipose tissue may prevent TNF-alpha-ind

13                                To define TNF alpha action in epidermis, we compared the transcription

14 tive therapeutic antibody in suppressing TNF-alpha action in many autoimmune diseases, was able to re

15 Among the several effectors that mediate TNF-alpha action is AP-1, which consists of transcription fa

16 ed by Th1 cells may be only delayed when TNF-alpha action is blocked.

17 ulin sensitivity, and direct effects of PPAR-alpha action on the islet itself.

18            To determine the mechanism of TNF alpha action, splenic cells from control NOD and RIP-TNF

19 udy demonstrates that the activation of PPAR-alpha action via fenofibrate leads to neuroprotection by

20 Collectively these data demonstrate that TNF-alpha action within the brain is critical to the elabora