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1 in for adenylyl cyclase, in complex with the alpha2 adrenergic receptor.
2 h Galphaz coupled strongly to the endogenous alpha2 adrenergic receptor.
3 K1/2 is inhibited by epinephrine through the alpha2-adrenergic receptor.
4 restin is required for ERK activation by the alpha2-adrenergic receptor.
5 inhibition of presynaptic TRPV1 channels by alpha2 adrenergic receptors.
6 y yohimbine, consistent with contribution of alpha2 adrenergic receptors.
7 irming that it was mediated by activation of alpha2 adrenergic receptors.
8 ecline (PHFD) requires central activation of alpha2-adrenergic receptors.
9 n is specific for post-junctional alpha1- or alpha2-adrenergic receptors.
10 ined whether this was specific to alpha1- or alpha2-adrenergic receptors.
11 otein expression of NE transporter (NET) and alpha(2) adrenergic receptor.
12 that control song contain high densities of alpha(2)adrenergic receptors.
13 ted secretion, in the cell surface export of alpha(2)-adrenergic receptors.
14 morphisms of the adenosine A2A receptors and alpha(2)-adrenergic receptors.
15 a(1)- and beta-adrenergic receptors, but not alpha(2)-adrenergic receptors.
16 hat it is primarily mediated by postsynaptic alpha(2)-adrenergic receptors.
18 olin-stimulated cAMP accumulation induced by alpha(2)-adrenergic receptor activation is altered follo
20 ergic receptor antagonist, prazosin, and the alpha(2)-adrenergic receptor agonist, clonidine, reduced
21 ohimbine (1.0 microm), and attenuated by the alpha(2)-adrenergic receptor agonist, UK 14,304 (1.0 mic
23 te whether transdermal administration of the alpha2 adrenergic-receptor agonist, clonidine, can be us
25 ermal administration of the centrally acting alpha2-adrenergic receptor agonist, clonidine), and hypo
27 e is reduced responsiveness to alpha(1)- and alpha(2)-adrenergic receptor agonists in skeletal muscle
28 mpatholytic effects we observed suggest that alpha2 adrenergic-receptor agonists may offer a pharmaco
30 syndrome indicates that chronic exposure to alpha2-adrenergic receptor agonists produces strong depe
32 sly identified spinophilin as a regulator of alpha(2) adrenergic receptor (alpha(2)AR) trafficking an
33 receptor) and inhibitory (Galpha(i)-mediated alpha(2)-adrenergic receptor (alpha(2)-AR)) signals to a
35 d the role of the extracellular N termini of alpha(2)-adrenergic receptors (alpha(2)-ARs) in the ante
37 olateral localization and trafficking of the alpha(2)-adrenergic receptors (alpha(2)AR) are not; inst
39 everal studies have shown that activation of alpha(2)-adrenergic receptors (alpha(2)ARs) leads to mil
42 ers in cognition and emotion, by alpha1- and alpha2-adrenergic receptors (alpha1-ARs, alpha2-ARs) in
44 ole of arrestins in the trafficking of human alpha2-adrenergic receptors (alpha2-ARs) and the effect
45 the evolutionary precursor to the vertebrate alpha2-adrenergic receptors (alpha2-ARs) based upon sequ
46 the regulation of cell surface transport of alpha2-adrenergic receptors (alpha2-ARs) by GGA3 (Golgi-
47 strates subserving the inhibitory actions of alpha2-adrenergic receptors (alpha2-ARs) in the locus co
50 ve, and sympatholytic responses in vivo, and alpha2 adrenergic receptor (alpha2AR) agonists are used
55 ined the selectivity of interactions between alpha2-adrenergic receptors (alpha2R) and various combin
58 ng properties on I1 imidazoline receptors vs alpha2-adrenergic receptors and their blood pressure eff
59 sors are tested for the beta2-, alpha1-, and alpha2- adrenergic receptors, and adenosine type 1 recep
60 rolled by negative feedback from presynaptic alpha2-adrenergic receptors, and the targets of the rele
61 (iv) NA overflow for MA was enhanced by the alpha(2)-adrenergic receptor antagonist, yohimbine (1.0
63 factor 1 (CRF1) antagonist (antalarmin), and alpha2-adrenergic receptor antagonist (yohimbine; used a
64 d PLD activity was completely blocked by the alpha2-adrenergic receptor antagonist rauwolscine and by
65 mmonis 3 (CA3) epileptiform activity through alpha(2) adrenergic receptor (AR) activation on pyramida
66 regulating trafficking and signaling of the alpha(2)-adrenergic receptor (AR) both in vitro and in v
68 can be reversed by a potent centrally acting alpha2 adrenergic receptor (AR) agonist (dexmedetomidine
72 ediated via both post-junctional alpha1- and alpha2-adrenergic receptors are blunted in contracting h
76 was a result of activation of muscarinic and alpha2-adrenergic receptors because a specific alpha2-ad
77 ms using drug treatments targeting beta- and alpha2-adrenergic receptors, but little is known about t
78 that PHFD does not require the activation of alpha2-adrenergic receptors, but that alpha2-adrenergic
79 e activation of either supraspinal or spinal alpha(2) adrenergic receptors can induce antinociception
80 ion of alpha2-adrenergic receptors, but that alpha2-adrenergic receptors can modulate resting and pos
81 ctivity, which was increased by 5-HT(1A) and alpha2-adrenergic receptor co-activation in an activator
82 teract with a photoaffinity derivative of an alpha2-adrenergic receptor-derived peptide from the thir
83 voltage-dependent Ca2+ channels mediated by alpha2-adrenergic receptors desensitizes slowly with pro
84 Chinese hamster ovary-K1 cells impaired the alpha(2)-adrenergic receptor G(s)-mediated stimulation o
85 ed: Galpha(oA) = Galpha(oB) > Galpha(i2) for alpha(2)-adrenergic receptor; Galpha(i2) > Galpha(oA) =
88 vious studies in several animal species, the alpha2 adrenergic receptors in the human ciliary body, r
89 Stimulated GTPgammaS binding indicated that alpha2-adrenergic receptors in HET hippocampus were more
90 ast to that which occurs in the hippocampus, alpha(2)-adrenergic receptor inhibition of NE release is
92 gulates the Golgi to cell surface traffic of alpha(2)-adrenergic receptors, likely through a physical
93 on a natively expressed signalling pathway, alpha(2)-adrenergic receptor-mediated I(Ca) modulation,
95 hile rolipram and PDE4D deficiency shortened alpha2 adrenergic receptor-mediated anesthesia, a surrog
96 eleased glutamate in the mNTS and results in alpha2 adrenergic receptor-mediated inhibition of DMV ne
98 ctions by approximately 50% and enhanced the alpha2-adrenergic receptor-mediated inhibition of forsko
99 btype influence signaling efficiency for the alpha2-adrenergic receptor-mediated signaling pathway.
100 e from studies in animals demonstrating that alpha2-adrenergic receptor-mediated vasoconstrictor resp
101 r nerve injury, the cascade of activation of alpha(2) adrenergic receptors-muscarinic receptors-NO in
103 he level of arousal by selectively targeting alpha2 adrenergic receptors on LC neurons, resulting in
104 (3)R) was increased but not signaling of the alpha(2) adrenergic receptor or bradykinin BK(2) recepto
106 ts suggest that spinal (but not supraspinal) alpha(2) adrenergic receptors play a significant role in
108 aused an approximate 50% decrease in NET and alpha(2) adrenergic receptor protein expression in sever
109 ic actions of norepinephrine are exerted via alpha2 adrenergic receptors residing on targets of norad
113 cular properties of each Gialpha, Gbeta, and alpha2-adrenergic receptor subtype influence signaling e
114 -PCR was performed to detect the mRNA of the alpha(2)-adrenergic receptor subtypes alpha(2)A, alpha(2
115 ecluded understanding the role of individual alpha2 adrenergic receptor subtypes in the antiepileptog
118 ugh regulation of TNF expression, transforms alpha(2)-adrenergic receptors such that they function to
119 at low dose SNRIs, by acting on 5-HT(1A) and alpha2-adrenergic receptors, synergistically reduced AMP
120 ation/down-regulation of the chromaffin cell alpha(2)-adrenergic receptors that normally inhibit CA s
122 Here, we show that noradrenaline activates alpha2-adrenergic receptors to control short-term and lo
123 lymorphisms (GPIIb, FcgammaRIIa, P-selectin, alpha2 adrenergic receptor, transforming growth factor [
124 BRET signals were observed for AGS4-RLuc and alpha(2)-adrenergic receptor-Venus, which were Galpha(i)
125 le in vivo interactions between dopamine and alpha(2)-adrenergic receptors was investigated in quail,
126 a high binding affinity of M1 muscarinic and alpha2 adrenergic receptors was associated with a greate
129 ution and concentration of beta1, beta2, and alpha2 adrenergic receptors were examined in the frontal
130 mice to sedation elicited by stimulation of alpha2 adrenergic receptors, whereas arrestin 3 knockout
131 ne binding sites in plasma membrane and lack alpha2-adrenergic receptors, which recognize many I1-imi
132 avian brain, indicating that the density of alpha(2) adrenergic receptors within the song system mig
133 volumes of song nuclei, and the densities of alpha(2) adrenergic receptors within the song system of
134 e largest, T was highest, and the density of alpha(2) adrenergic receptors (within HVc and RA) was lo
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